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Artigo de periodico
Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells (2020)
Librais, Gabriela Nunes Marsiglio ; Vilas-Boas, Eloisa Aparecida ; Carlein, Christopher; Hoffmann, Markus Daniel Alexander; Roma, Leticia Prates ; Carpinelli, Angelo Rafael
Fonte: Redox Report. Unidade: ICB
Assuntos: FISIOLOGIA, INSULINA, ILHOTAS DE LANGERHANS, ÁCIDOS GRAXOS, GLICOSE, MEMBRANA PLASMÁTICA, PÂNCREAS, ÁCIDO LINOLEICO
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LIBRAIS, Gabriela Nunes Marsiglio et al. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, v. 25, n. 1, p. 41–50, 2020Tradução . . Disponível em: https://doi.org/10.1080/13510002.2020.1757877. Acesso em: 11 nov. 2024.
APA
Librais, G. N. M., Vilas-Boas, E. A., Carlein, C., Hoffmann, M. D. A., Roma, L. P., & Carpinelli, A. R. (2020). Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells. Redox Report, 25( 1), 41–50. doi:10.1080/13510002.2020.1757877
NLM
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Vancouver
Librais GNM, Vilas-Boas EA, Carlein C, Hoffmann MDA, Roma LP, Carpinelli AR. Evidence for NADPH oxidase activation by GPR40 in pancreatic β-cells [Internet]. Redox Report. 2020 ; 25( 1): 41–50.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1080/13510002.2020.1757877
Artigo de periodico
Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice (2020)
Estrela, Gabriel Rufino ; Wasinski, Frederick ; Gregnani, Marcos Fernandes ; Lima, Leandro Ceotto Freitas ; Arruda, Adriano C.; Morais, Rafael Leite; Malheiros, Denise Maria Avancini Costa; Câmara, Niels Olsen Saraiva ; Pesquero, João Bosco ; Bader, Michael ; Barros, Carlos Castilho ; Araújo, Ronaldo Carvalho
Fonte: Frontiers in Molecular Biosciences. Unidades: FM, ICB
Assuntos: IMUNOLOGIA, ANGIOTENSINAS, INIBIDORES DE ENZIMAS, INFLAMAÇÃO, CAMUNDONGOS, QUIMIOTERÁPICOS, CININAS, RATOS, CREATINA, TÚBULOS RENAIS
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ESTRELA, Gabriel Rufino et al. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice. Frontiers in Molecular Biosciences, v. 7, p. 10 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fmolb.2020.00096. Acesso em: 11 nov. 2024.
APA
Estrela, G. R., Wasinski, F., Gregnani, M. F., Lima, L. C. F., Arruda, A. C., Morais, R. L., et al. (2020). Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice. Frontiers in Molecular Biosciences, 7, 10 . doi:10.3389/fmolb.2020.00096
NLM
Estrela GR, Wasinski F, Gregnani MF, Lima LCF, Arruda AC, Morais RL, Malheiros DMAC, Câmara NOS, Pesquero JB, Bader M, Barros CC, Araújo RC. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice [Internet]. Frontiers in Molecular Biosciences. 2020 ; 7 10 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fmolb.2020.00096
Vancouver
Estrela GR, Wasinski F, Gregnani MF, Lima LCF, Arruda AC, Morais RL, Malheiros DMAC, Câmara NOS, Pesquero JB, Bader M, Barros CC, Araújo RC. Angiotensin-converting enzyme inhibitor protects against cisplatin nephrotoxicity by modulating kinin B1 receptor expression and aminopeptidase P activity in mice [Internet]. Frontiers in Molecular Biosciences. 2020 ; 7 10 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fmolb.2020.00096
Artigo de periodico-carta/editorial
Editorial: Harnessing the participation of dendritic cells in immunity and tolerance (2020)
Boscardin, Silvia Beatriz ; Dudziak, Diana ; Münz, Christian ; Rosa, Daniela Santoro
Fonte: Frontiers in Immunology. Unidade: ICB
Assuntos: PARASITOLOGIA, CÉLULAS DENDRÍTICAS, IMUNIDADE, INTERFERON TIPO I, ONTOGENIA, IMUNOTERAPIA, SEPSE, INFECÇÕES BACTERIANAS E MICOSES, CÉLULAS CULTIVADAS DE TUMOR, LINFÓCITOS T
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BOSCARDIN, Silvia Beatriz et al. Editorial: Harnessing the participation of dendritic cells in immunity and tolerance. Frontiers in Immunology. Lausanne: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.3389/fimmu.2020.595841. Acesso em: 11 nov. 2024. , 2020
APA
Boscardin, S. B., Dudziak, D., Münz, C., & Rosa, D. S. (2020). Editorial: Harnessing the participation of dendritic cells in immunity and tolerance. Frontiers in Immunology. Lausanne: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.3389/fimmu.2020.595841
NLM
Boscardin SB, Dudziak D, Münz C, Rosa DS. Editorial: Harnessing the participation of dendritic cells in immunity and tolerance [Internet]. Frontiers in Immunology. 2020 ; 11 4 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fimmu.2020.595841
Vancouver
Boscardin SB, Dudziak D, Münz C, Rosa DS. Editorial: Harnessing the participation of dendritic cells in immunity and tolerance [Internet]. Frontiers in Immunology. 2020 ; 11 4 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.3389/fimmu.2020.595841
Artigo de periodico
Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function (2020)
Vilas-Boas, Eloisa Aparecida ; Karabacz, Noémie; Librais, Gabriela Nunes Marsiglio ; Valle, Maíra Mello Rezende ; Nalbach, Lisa; Ampofo, Emmanuel; Morgan, Bruce ; Carpinelli, Angelo Rafael ; Roma, Leticia Prates
Fonte: Pharmacological Reports. Unidade: ICB
Assuntos: FISIOLOGIA, ÁCIDOS GRAXOS, INSULINA, ESTRESSE OXIDATIVO, PÂNCREAS, APOPTOSE
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ABNT
VILAS-BOAS, Eloisa Aparecida et al. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function. Pharmacological Reports, v. 72, n. 6, p. 1725-1737, 2020Tradução . . Disponível em: https://doi.org/10.1007/s43440-020-00101-6. Acesso em: 11 nov. 2024.
APA
Vilas-Boas, E. A., Karabacz, N., Librais, G. N. M., Valle, M. M. R., Nalbach, L., Ampofo, E., et al. (2020). Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function. Pharmacological Reports, 72( 6), 1725-1737. doi:10.1007/s43440-020-00101-6
NLM
Vilas-Boas EA, Karabacz N, Librais GNM, Valle MMR, Nalbach L, Ampofo E, Morgan B, Carpinelli AR, Roma LP. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function [Internet]. Pharmacological Reports. 2020 ; 72( 6): 1725-1737.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s43440-020-00101-6
Vancouver
Vilas-Boas EA, Karabacz N, Librais GNM, Valle MMR, Nalbach L, Ampofo E, Morgan B, Carpinelli AR, Roma LP. Chronic activation of GPR40 does not negatively impact upon BRIN-BD11 pancreatic β-cell physiology and function [Internet]. Pharmacological Reports. 2020 ; 72( 6): 1725-1737.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1007/s43440-020-00101-6
Artigo de periodico
Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein (2019)
Cavalcante, Paula Andréa Malveira ; Alenina, Natalia ; Budu, Alexandre ; Lima, Leandro Ceotto Freitas ; Silva, Thaís Alves da ; Agudelo, Juan Sebastian Henao ; Qadri, Fatimunnisa ; Câmara, Niels Olsen Saraiva ; Bader, Michael ; Araújo, Ronaldo de Carvalho
Fonte: Mediators of Inflammation. Unidade: ICB
Assuntos: IMUNOLOGIA, NEFROPATIAS EM ANIMAL, MACRÓFAGOS, PROTEÍNAS, MEDULA ÓSSEA DE ANIMAL, TÍBIA, PRESSÃO SANGUÍNEA, RATOS, REAÇÃO EM CADEIA POR POLIMERASE, ELISA EM ANIMAL, WESTERN BLOTTING
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CAVALCANTE, Paula Andréa Malveira et al. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein. Mediators of Inflammation, v. 2019, p. 14 , 2019Tradução . . Disponível em: https://doi.org/10.1155/2019/9086758. Acesso em: 11 nov. 2024.
APA
Cavalcante, P. A. M., Alenina, N., Budu, A., Lima, L. C. F., Silva, T. A. da, Agudelo, J. S. H., et al. (2019). Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein. Mediators of Inflammation, 2019, 14 . doi:10.1155/2019/9086758
NLM
Cavalcante PAM, Alenina N, Budu A, Lima LCF, Silva TA da, Agudelo JSH, Qadri F, Câmara NOS, Bader M, Araújo R de C. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein [Internet]. Mediators of Inflammation. 2019 ; 2019 14 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1155/2019/9086758
Vancouver
Cavalcante PAM, Alenina N, Budu A, Lima LCF, Silva TA da, Agudelo JSH, Qadri F, Câmara NOS, Bader M, Araújo R de C. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein [Internet]. Mediators of Inflammation. 2019 ; 2019 14 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1155/2019/9086758
Artigo de periodico
Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells (2019)
Gimenez, Marcela; Verissimo Filho, Sidney; Wittig, Ilka ; Schickling, Brandon M; Hahner, Fabian ; Schürmann, Christoph ; Netto, Luis Eduardo Soares ; Rosa, José César ; Brandes, Ralf P ; Sartoretto, Simone Marcieli; Camargo, Livia de Lucca ; Abdulkader, Fernando Rodrigues de Moraes ; Miller Junior, Francis J ; Lopes, Lucia Rossetti
Fonte: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidades: FMRP, ICB, IB
Assuntos: FISIOLOGIA, FARMACOLOGIA, ESPECTROMETRIA DE MASSAS, PROTEÍNAS RECOMBINANTES, ATIVAÇÃO ENZIMÁTICA, MÚSCULO LISO VASCULAR, CATALISADORES, FATORES DE CRESCIMENTO, CÉLULAS MUSCULARES
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GIMENEZ, Marcela et al. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 39, n. 2, p. 224-236, 2019Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.118.311038. Acesso em: 11 nov. 2024.
APA
Gimenez, M., Verissimo Filho, S., Wittig, I., Schickling, B. M., Hahner, F., Schürmann, C., et al. (2019). Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells. Arteriosclerosis, Thrombosis, and Vascular Biology, 39( 2), 224-236. doi:10.1161/ATVBAHA.118.311038
NLM
Gimenez M, Verissimo Filho S, Wittig I, Schickling BM, Hahner F, Schürmann C, Netto LES, Rosa JC, Brandes RP, Sartoretto SM, Camargo L de L, Abdulkader FR de M, Miller Junior FJ, Lopes LR. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2019 ; 39( 2): 224-236.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1161/ATVBAHA.118.311038
Vancouver
Gimenez M, Verissimo Filho S, Wittig I, Schickling BM, Hahner F, Schürmann C, Netto LES, Rosa JC, Brandes RP, Sartoretto SM, Camargo L de L, Abdulkader FR de M, Miller Junior FJ, Lopes LR. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2019 ; 39( 2): 224-236.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1161/ATVBAHA.118.311038

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