Filtros : "2012" "Endocrinology" Removido: "HU-SCPACIN-62" Limpar

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  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      LELLIS-SANTOS, Camilo et al. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, v. 153, n. 8, p. 3668-3678, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1135. Acesso em: 17 out. 2024.
    • APA

      Lellis-Santos, C., Sakamoto, L. H., Bromati, C. R., Nogueira, T. C. de A., Leite, A. R., Yamanaka, T. S., et al. (2012). The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, 153( 8), 3668-3678. doi:10.1210/en.2012-1135
    • NLM

      Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1135
    • Vancouver

      Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1135
  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      OLIVEIRA, Ariclécio Cunha de et al. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment. Endocrinology, v. 153, p. 2178-2188, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2011-1675. Acesso em: 17 out. 2024.
    • APA

      Oliveira, A. C. de, Andreotti, S., Farias, T. da S. M., Torres-Leal, F. L., Proença, A. R. G. de, Campaña, A. B., et al. (2012). Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment. Endocrinology, 153, 2178-2188. doi:10.1210/en.2011-1675
    • NLM

      Oliveira AC de, Andreotti S, Farias T da SM, Torres-Leal FL, Proença ARG de, Campaña AB, Souza AH de, Sertié RAL, Carpinelli AR, Cipolla-Neto J, Lima FB. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment [Internet]. Endocrinology. 2012 ; 153 2178-2188.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2011-1675
    • Vancouver

      Oliveira AC de, Andreotti S, Farias T da SM, Torres-Leal FL, Proença ARG de, Campaña AB, Souza AH de, Sertié RAL, Carpinelli AR, Cipolla-Neto J, Lima FB. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment [Internet]. Endocrinology. 2012 ; 153 2178-2188.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2011-1675
  • Source: Endocrinology. Unidade: FMRP

    Subjects: ENDOCRINOLOGIA, RECEPTORES (CONTROLE), HORMÔNIOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      TRASLAVIÑA, Guilhermo A. Ariza e FRANCI, Celso Rodrigues. Divergent roles of the CRH receptors in the control of gonadotropin secretion induced by acute restraint stress at proestrus. Endocrinology, v. 153, n. 10, p. 4838-4848, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1333. Acesso em: 17 out. 2024.
    • APA

      Traslaviña, G. A. A., & Franci, C. R. (2012). Divergent roles of the CRH receptors in the control of gonadotropin secretion induced by acute restraint stress at proestrus. Endocrinology, 153( 10), 4838-4848. doi:10.1210/en.2012-1333
    • NLM

      Traslaviña GAA, Franci CR. Divergent roles of the CRH receptors in the control of gonadotropin secretion induced by acute restraint stress at proestrus [Internet]. Endocrinology. 2012 ; 153( 10): 4838-4848.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1333
    • Vancouver

      Traslaviña GAA, Franci CR. Divergent roles of the CRH receptors in the control of gonadotropin secretion induced by acute restraint stress at proestrus [Internet]. Endocrinology. 2012 ; 153( 10): 4838-4848.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1333
  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      CARVALHO FILHO, M. A. et al. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, v. 153, n. 11, p. 5261-5274, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1400. Acesso em: 17 out. 2024.
    • APA

      Carvalho Filho, M. A., Carvalho, B. M., Oliveira, A. G., Guadagnini, D., Ueno, M., Dias M. M.,, et al. (2012). Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, 153( 11), 5261-5274. doi:10.1210/en.2012-1400
    • NLM

      Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1400
    • Vancouver

      Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1400
  • Source: Endocrinology. Unidade: FMRP

    Subjects: FARMACOLOGIA, OCITOCINA, RECEPTORES

    Acesso à fonteDOIHow to cite
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    • ABNT

      BUSNARDO, Cristiane et al. Ionotropic glutamate receptors in hypothalamic paraventricular and supraoptic nuclei mediate vasopressin and oxytocin release in unanesthetized rats. Endocrinology, v. 153, n. 5, p. 2323-2331, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2011-2079. Acesso em: 17 out. 2024.
    • APA

      Busnardo, C., Crestani, C. C., Resstel, L. B. M., Tavares, R. F., Antunes-Rodrigues, J., & Corrêa, F. M. de A. (2012). Ionotropic glutamate receptors in hypothalamic paraventricular and supraoptic nuclei mediate vasopressin and oxytocin release in unanesthetized rats. Endocrinology, 153( 5), 2323-2331. doi:10.1210/en.2011-2079
    • NLM

      Busnardo C, Crestani CC, Resstel LBM, Tavares RF, Antunes-Rodrigues J, Corrêa FM de A. Ionotropic glutamate receptors in hypothalamic paraventricular and supraoptic nuclei mediate vasopressin and oxytocin release in unanesthetized rats [Internet]. Endocrinology. 2012 ; 153( 5): 2323-2331.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2011-2079
    • Vancouver

      Busnardo C, Crestani CC, Resstel LBM, Tavares RF, Antunes-Rodrigues J, Corrêa FM de A. Ionotropic glutamate receptors in hypothalamic paraventricular and supraoptic nuclei mediate vasopressin and oxytocin release in unanesthetized rats [Internet]. Endocrinology. 2012 ; 153( 5): 2323-2331.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2011-2079
  • Source: Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      KINOTE, Andrezza et al. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, v. 153, n. 8, p. 3633-3645, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1341. Acesso em: 17 out. 2024.
    • APA

      Kinote, A., Faria, J. A., Roman, E. A., Solon, C., Razolli, D. S., Ignacio-Souza, L. M., et al. (2012). Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, 153( 8), 3633-3645. doi:10.1210/en.2012-1341
    • NLM

      Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1341
    • Vancouver

      Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 out. 17 ] Available from: https://doi.org/10.1210/en.2012-1341

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