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Artigo de periodico
Diminished cholesterol efflux mediated by HDL and coronary artery disease in young male anabolic androgenic steroid users (2019)
Souza, Francis Ribeiro de; Santos, Marcelo Rodrigues dos; Porello, Rafael Armani; Fonseca, Guilherme Wesley Peixoto da; Sayegh, Ana Luiza Carrari; Lima, Thaís Pinheiro; Ferreira, Fabiana Dias; Oliveira, Tiago Franco de; Yonamine, Maurício ; Takayama, Liliam; Pereira, Rosa Maria Rodrigues; Negrão, Carlos Eduardo; Passarelli, Marisa; Rochitte, Carlos Eduardo; Alves, Maria Janieire de Nazaré Nunes
Source: Atherosclerosis. Unidades: FCF, FM, EEFE
Subjects: COLESTEROL, DOENÇAS CARDIOVASCULARES, ESTEROIDES
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ABNT
SOUZA, Francis Ribeiro de et al. Diminished cholesterol efflux mediated by HDL and coronary artery disease in young male anabolic androgenic steroid users. Atherosclerosis, v. 283, p. 100-105, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.atherosclerosis.2019.02.006. Acesso em: 05 nov. 2024.
APA
Souza, F. R. de, Santos, M. R. dos, Porello, R. A., Fonseca, G. W. P. da, Sayegh, A. L. C., Lima, T. P., et al. (2019). Diminished cholesterol efflux mediated by HDL and coronary artery disease in young male anabolic androgenic steroid users. Atherosclerosis, 283, 100-105. doi:10.1016/j.atherosclerosis.2019.02.006
NLM
Souza FR de, Santos MR dos, Porello RA, Fonseca GWP da, Sayegh ALC, Lima TP, Ferreira FD, Oliveira TF de, Yonamine M, Takayama L, Pereira RMR, Negrão CE, Passarelli M, Rochitte CE, Alves MJ de NN. Diminished cholesterol efflux mediated by HDL and coronary artery disease in young male anabolic androgenic steroid users [Internet]. Atherosclerosis. 2019 ; 283 100-105.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2019.02.006
Vancouver
Souza FR de, Santos MR dos, Porello RA, Fonseca GWP da, Sayegh ALC, Lima TP, Ferreira FD, Oliveira TF de, Yonamine M, Takayama L, Pereira RMR, Negrão CE, Passarelli M, Rochitte CE, Alves MJ de NN. Diminished cholesterol efflux mediated by HDL and coronary artery disease in young male anabolic androgenic steroid users [Internet]. Atherosclerosis. 2019 ; 283 100-105.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.atherosclerosis.2019.02.006
Artigo de periodico
Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 (2018)
Bozi, Luiz Henrique Marchesi; Takano, Ana Paula Cremasco ; Campos, Juliane Cruz; Rolim, Natale Pinheiro Lage; Dourado, Paulo Magno Martins; Voltarelli, Vanessa Azevedo; Wisløff, Ulrik; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Brum, Patricia Chakur
Source: International journal of cardiology. Unidades: ICB, EEFE
Subjects: RETÍCULO ENDOPLASMÁTICO, INFARTO DO MIOCÁRDIO, DOENÇAS CARDIOVASCULARES, PROTEÍNAS, RATOS WISTAR
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ABNT
BOZI, Luiz Henrique Marchesi et al. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, v. 272, p. 194-201, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2018.08.070. Acesso em: 05 nov. 2024.
APA
Bozi, L. H. M., Takano, A. P. C., Campos, J. C., Rolim, N. P. L., Dourado, P. M. M., Voltarelli, V. A., et al. (2018). Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, 272, 194-201. doi:10.1016/j.ijcard.2018.08.070
NLM
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Vancouver
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Artigo de periodico
Alpha2A-adrenergic receptor and eNOS genetic polymorphisms are associated with exercise muscle vasodilatation in apparently healthy individuals (2016)
Nunes, Rafael Amorim Belo; Barroso, Lucia Pereira; Pereira, Alexandre da Costa; Rondon, M. U.; Negrão, C. E.; Krieger, José Eduardo; Mansur, Alfredo José
Source: International Journal of Cardiology. Heart & Vasculature. Unidades: IME, EEFE, FM
Subjects: VASODILATAÇÃO, EXERCÍCIO FÍSICO, POLIMORFISMO, ADRENORECEPTORES, ÓXIDO NÍTRICO, SISTEMA CARDIOVASCULAR, VARIAÇÃO GENÉTICA
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ABNT
NUNES, Rafael Amorim Belo et al. Alpha2A-adrenergic receptor and eNOS genetic polymorphisms are associated with exercise muscle vasodilatation in apparently healthy individuals. International Journal of Cardiology. Heart & Vasculature, v. 13, p. 14-18, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijcha.2016.10.001. Acesso em: 05 nov. 2024.
APA
Nunes, R. A. B., Barroso, L. P., Pereira, A. da C., Rondon, M. U., Negrão, C. E., Krieger, J. E., & Mansur, A. J. (2016). Alpha2A-adrenergic receptor and eNOS genetic polymorphisms are associated with exercise muscle vasodilatation in apparently healthy individuals. International Journal of Cardiology. Heart & Vasculature, 13, 14-18. doi:10.1016/j.ijcha.2016.10.001
NLM
Nunes RAB, Barroso LP, Pereira A da C, Rondon MU, Negrão CE, Krieger JE, Mansur AJ. Alpha2A-adrenergic receptor and eNOS genetic polymorphisms are associated with exercise muscle vasodilatation in apparently healthy individuals [Internet]. International Journal of Cardiology. Heart & Vasculature. 2016 ; 13 14-18.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcha.2016.10.001
Vancouver
Nunes RAB, Barroso LP, Pereira A da C, Rondon MU, Negrão CE, Krieger JE, Mansur AJ. Alpha2A-adrenergic receptor and eNOS genetic polymorphisms are associated with exercise muscle vasodilatation in apparently healthy individuals [Internet]. International Journal of Cardiology. Heart & Vasculature. 2016 ; 13 14-18.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcha.2016.10.001
Artigo de periodico
Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice (2016)
Bacurau, Aline Villa Nova; Jannig, Paulo Roberto; Moraes, Wilson Max Almeida Monteiro de; Cunha, Telma Fátima da; Medeiros, Alessandra; Barberi, Laura; Coelho, Marcele A.; Bacurau, Reury Frank Pereira; Ugrinowitsch, Carlos; Musarò, Antonio; Brum, Patricia Chakur
Source: International Journal of Cardiology. Unidades: EACH, EEFE
Subjects: INSUFICIÊNCIA CARDÍACA, MÚSCULO ESQUELÉTICO, TREINAMENTO AERÓBIO, ATROFIA MUSCULAR
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ABNT
BACURAU, Aline Villa Nova et al. Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice. International Journal of Cardiology, v. 214, p. 137-147, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2016.03.071. Acesso em: 05 nov. 2024.
APA
Bacurau, A. V. N., Jannig, P. R., Moraes, W. M. A. M. de, Cunha, T. F. da, Medeiros, A., Barberi, L., et al. (2016). Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice. International Journal of Cardiology, 214, 137-147. doi:10.1016/j.ijcard.2016.03.071
NLM
Bacurau AVN, Jannig PR, Moraes WMAM de, Cunha TF da, Medeiros A, Barberi L, Coelho MA, Bacurau RFP, Ugrinowitsch C, Musarò A, Brum PC. Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice [Internet]. International Journal of Cardiology. 2016 ; 214 137-147.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2016.03.071
Vancouver
Bacurau AVN, Jannig PR, Moraes WMAM de, Cunha TF da, Medeiros A, Barberi L, Coelho MA, Bacurau RFP, Ugrinowitsch C, Musarò A, Brum PC. Akt/mTOR pathway contributes to skeletal muscle anti-atrophic effect of aerobic exercise training in heart failure mice [Internet]. International Journal of Cardiology. 2016 ; 214 137-147.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2016.03.071
Trabalho de evento-resumo periodico
Functional characteristics of plasma lipoproteins from patients with metabolic syndrome are modulated by changes in their size and composition after short-term exercise (2009)
Casella Filho, Antonio; Cesena, Fernando Henpin Yue; Trombetta, Ivani Credidio; Denardi, Celise A. S; Dourado, Paulo Magno Martins; Silva, Vanessa Mendes da; Negrão, C. E.; Maranhão, Raul Cavalcante
Source: Atherosclerosis Supplements. Conference titles: Brazilian Congress of Atherosclerosis. Unidades: EEFE, FCF
Subjects: LIPOPROTEÍNAS, ARTERIOSCLEROSE
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ABNT
CASELLA FILHO, Antonio et al. Functional characteristics of plasma lipoproteins from patients with metabolic syndrome are modulated by changes in their size and composition after short-term exercise. Atherosclerosis Supplements. Clare: Escola de Educação Física e Esporte, Universidade de São Paulo. . Acesso em: 05 nov. 2024. , 2009
APA
Casella Filho, A., Cesena, F. H. Y., Trombetta, I. C., Denardi, C. A. S., Dourado, P. M. M., Silva, V. M. da, et al. (2009). Functional characteristics of plasma lipoproteins from patients with metabolic syndrome are modulated by changes in their size and composition after short-term exercise. Atherosclerosis Supplements. Clare: Escola de Educação Física e Esporte, Universidade de São Paulo.
NLM
Casella Filho A, Cesena FHY, Trombetta IC, Denardi CAS, Dourado PMM, Silva VM da, Negrão CE, Maranhão RC. Functional characteristics of plasma lipoproteins from patients with metabolic syndrome are modulated by changes in their size and composition after short-term exercise. Atherosclerosis Supplements. 2009 ; 10( 3): 32-33 res. L008.[citado 2024 nov. 05 ]
Vancouver
Casella Filho A, Cesena FHY, Trombetta IC, Denardi CAS, Dourado PMM, Silva VM da, Negrão CE, Maranhão RC. Functional characteristics of plasma lipoproteins from patients with metabolic syndrome are modulated by changes in their size and composition after short-term exercise. Atherosclerosis Supplements. 2009 ; 10( 3): 32-33 res. L008.[citado 2024 nov. 05 ]

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