Source: Inflammation Research. Unidades: CENA, FMRP, FCFRP, FORP
Subjects: GENGIVA, INTERLEUCINAS, PERIODONTITE, FARMACOTERAPIA, ANIMAIS DE LABORATÓRIO
ABNT
FRANCHIN, Marcelo et al. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model. Inflammation Research, v. 72, p. 107-114, 2022Tradução . . Disponível em: https://doi.org/10.1007/s00011-022-01662-3. Acesso em: 29 abr. 2026.APA
Franchin, M., Taira, T. M., Prado, D. da S., Hernandez, C. A. S., Andrade, F. B. de, Abdalla, H. B., et al. (2022). PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model. Inflammation Research, 72, 107-114. doi:10.1007/s00011-022-01662-3NLM
Franchin M, Taira TM, Prado D da S, Hernandez CAS, Andrade FB de, Abdalla HB, Napimoga MH, Cunha TM, Alves SYF, Rosalen PL. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model [Internet]. Inflammation Research. 2022 ; 72 107-114.[citado 2026 abr. 29 ] Available from: https://doi.org/10.1007/s00011-022-01662-3Vancouver
Franchin M, Taira TM, Prado D da S, Hernandez CAS, Andrade FB de, Abdalla HB, Napimoga MH, Cunha TM, Alves SYF, Rosalen PL. PI3Kγ controls IL-17A expression and attenuates alveolar bone loss in an experimental periodontitis model [Internet]. Inflammation Research. 2022 ; 72 107-114.[citado 2026 abr. 29 ] Available from: https://doi.org/10.1007/s00011-022-01662-3
