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Artigo de periodico
Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice (2018)
Staurengo-Ferrari, Larissa; Ruiz-Miyazawa, Kenji W.; Pinho-Ribeiro, Felipe A.; Fattori, Victor; Zaninelli, Tiago H.; Badaro-Garcia, Stephanie; Borghi, Sergio M.; Carvalho, Thacyana T.; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Verri Júnior, Waldiceu Aparecido
Source: Frontiers in Pharmacology. Unidade: FMRP
Subjects: FLAVONOIDES, ARTRITE, INFLAMAÇÃO, ARTICULAÇÕES, DOENÇAS REUMÁTICAS, PRODUTOS NATURAIS
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ABNT
STAURENGO-FERRARI, Larissa et al. Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice. Frontiers in Pharmacology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphar.2018.01123. Acesso em: 04 nov. 2024.
APA
Staurengo-Ferrari, L., Ruiz-Miyazawa, K. W., Pinho-Ribeiro, F. A., Fattori, V., Zaninelli, T. H., Badaro-Garcia, S., et al. (2018). Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice. Frontiers in Pharmacology, 9. doi:10.3389/fphar.2018.01123
NLM
Staurengo-Ferrari L, Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Fattori V, Zaninelli TH, Badaro-Garcia S, Borghi SM, Carvalho TT, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice [Internet]. Frontiers in Pharmacology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphar.2018.01123
Vancouver
Staurengo-Ferrari L, Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Fattori V, Zaninelli TH, Badaro-Garcia S, Borghi SM, Carvalho TT, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Trans-chalcone attenuates pain and inflammation in experimental acute gout arthritis in mice [Internet]. Frontiers in Pharmacology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphar.2018.01123
Artigo de periodico
The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β (2018)
Ruiz-Miyazawa, Kenji W.; Borghi, Sergio M.; Ribeiro, Felipe A. Pinho; Ferrari, Larissa Staurengo; Fattori, Victor; Fernandes, Glaura S. A.; Casella, Antonio M.; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Cunha, Fernando de Queiroz ; Casagrande, Rubia; Verri, Waldiceu A.
Source: Journal of Functional Foods. Unidade: FMRP
Subjects: ARTRITE, DOR, INFLAMAÇÃO, RATOS, MACRÓFAGOS
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ABNT
RUIZ-MIYAZAWA, Kenji W. et al. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, v. 48, p. 106-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jff.2018.06.025. Acesso em: 04 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Borghi, S. M., Ribeiro, F. A. P., Ferrari, L. S., Fattori, V., Fernandes, G. S. A., et al. (2018). The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β. Journal of Functional Foods, 48, 106-116. doi:10.1016/j.jff.2018.06.025
NLM
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Vancouver
Ruiz-Miyazawa KW, Borghi SM, Ribeiro FAP, Ferrari LS, Fattori V, Fernandes GSA, Casella AM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri WA. The citrus flavanone naringenin reduces gout-induced joint pain and inflammation in mice by inhibiting the activation of NFκB and macrophage release of IL-1β [Internet]. Journal of Functional Foods. 2018 ; 48 106-116.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jff.2018.06.025
Artigo de periodico
Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation (2018)
Ruiz-Miyazawa, Kenji W.; Pinho-Ribeiro, Felipe A; Borghi, Sérgio M.; Staurengo-Ferrari, Larissa; Fattori, Victor; Amaral, Flavio A.; Teixeira, Mauro M.; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu Aparecido
Source: Journal of Agricultural and Food Chemistry. Unidade: FMRP
Subjects: ARTRITE, CITOCINAS, ESTRESSE OXIDATIVO
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ABNT
RUIZ-MIYAZAWA, Kenji W. et al. Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation. Journal of Agricultural and Food Chemistry, v. 66, n. 25, p. 6269-6280, 2018Tradução . . Disponível em: https://doi.org/10.1021/acs.jafc.8b00959. Acesso em: 04 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Pinho-Ribeiro, F. A., Borghi, S. M., Staurengo-Ferrari, L., Fattori, V., Amaral, F. A., et al. (2018). Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation. Journal of Agricultural and Food Chemistry, 66( 25), 6269-6280. doi:10.1021/acs.jafc.8b00959
NLM
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Borghi SM, Staurengo-Ferrari L, Fattori V, Amaral FA, Teixeira MM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation [Internet]. Journal of Agricultural and Food Chemistry. 2018 ; 66( 25): 6269-6280.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1021/acs.jafc.8b00959
Vancouver
Ruiz-Miyazawa KW, Pinho-Ribeiro FA, Borghi SM, Staurengo-Ferrari L, Fattori V, Amaral FA, Teixeira MM, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Verri Júnior WA. Hesperidin methylchalcone suppresses experimental gout arthritis in mice by inhibiting NF-KB activation [Internet]. Journal of Agricultural and Food Chemistry. 2018 ; 66( 25): 6269-6280.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1021/acs.jafc.8b00959
Artigo de periodico
Budlein A, a sesquiterpene lactone from viguiera robusta, alleviates pain and inflammation in a model of acute gout arthritis in mice (2018)
Fattori, Victor; Zarpelon, Ana C.; Staurengo-Ferrari, Larissa; Borghi, Sergio M.; Zaninelli, Tiago H.; Costa, Fernando Batista da; Alves Filho, José Carlos Farias; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Casagrande, Rúbia; Arakawa, Nilton Syogo; Verri Júnior, Waldiceu Aparecido
Source: Frontiers in Pharmacology. Unidades: FCFRP, FMRP
Subjects: ANALGESIA, ARTRITE, ARTICULAÇÃO DO JOELHO, DOENÇAS REUMÁTICAS, SESQUITERPENOS
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ABNT
FATTORI, Victor et al. Budlein A, a sesquiterpene lactone from viguiera robusta, alleviates pain and inflammation in a model of acute gout arthritis in mice. Frontiers in Pharmacology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fphar.2018.01076. Acesso em: 04 nov. 2024.
APA
Fattori, V., Zarpelon, A. C., Staurengo-Ferrari, L., Borghi, S. M., Zaninelli, T. H., Costa, F. B. da, et al. (2018). Budlein A, a sesquiterpene lactone from viguiera robusta, alleviates pain and inflammation in a model of acute gout arthritis in mice. Frontiers in Pharmacology, 9. doi:10.3389/fphar.2018.01076
NLM
Fattori V, Zarpelon AC, Staurengo-Ferrari L, Borghi SM, Zaninelli TH, Costa FB da, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Arakawa NS, Verri Júnior WA. Budlein A, a sesquiterpene lactone from viguiera robusta, alleviates pain and inflammation in a model of acute gout arthritis in mice [Internet]. Frontiers in Pharmacology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphar.2018.01076
Vancouver
Fattori V, Zarpelon AC, Staurengo-Ferrari L, Borghi SM, Zaninelli TH, Costa FB da, Alves Filho JCF, Cunha TM, Cunha F de Q, Casagrande R, Arakawa NS, Verri Júnior WA. Budlein A, a sesquiterpene lactone from viguiera robusta, alleviates pain and inflammation in a model of acute gout arthritis in mice [Internet]. Frontiers in Pharmacology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fphar.2018.01076
Artigo de periodico
Neurological and inflammatory manifestations in Sjögren’s syndrome: the role of the kynurenine metabolic pathway (2018)
Oliveira, Fabíola Reis de; Fantucci, Marina Zílio; Adriano, Leidiane; Valim, Valéria; Cunha, Thiago Mattar; Louzada Júnior, Paulo; Rocha, Eduardo Melani
Source: International Journal of Molecular Sciences. Unidade: FMRP
Subjects: SÍNDROME DE SJOGREN, DOR, INFLAMAÇÃO
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ABNT
OLIVEIRA, Fabíola Reis de et al. Neurological and inflammatory manifestations in Sjögren’s syndrome: the role of the kynurenine metabolic pathway. International Journal of Molecular Sciences, v. 19, n. 12, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19123953. Acesso em: 04 nov. 2024.
APA
Oliveira, F. R. de, Fantucci, M. Z., Adriano, L., Valim, V., Cunha, T. M., Louzada Júnior, P., & Rocha, E. M. (2018). Neurological and inflammatory manifestations in Sjögren’s syndrome: the role of the kynurenine metabolic pathway. International Journal of Molecular Sciences, 19( 12). doi:10.3390/ijms19123953
NLM
Oliveira FR de, Fantucci MZ, Adriano L, Valim V, Cunha TM, Louzada Júnior P, Rocha EM. Neurological and inflammatory manifestations in Sjögren’s syndrome: the role of the kynurenine metabolic pathway [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 12):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19123953
Vancouver
Oliveira FR de, Fantucci MZ, Adriano L, Valim V, Cunha TM, Louzada Júnior P, Rocha EM. Neurological and inflammatory manifestations in Sjögren’s syndrome: the role of the kynurenine metabolic pathway [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 12):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/ijms19123953
Artigo de periodico
15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice (2018)
Ruiz-Miyazawa, Kenji W; Staurengo-Ferrari, Larissa; Pinho-Ribeiro, Felipe A; Fattori, Victor ; Zaninelli, Tiago H ; Badaro-Garcia, Stephanie; Borghi, Sergio M; Andrade, Ketlem C; Clemente-Napimoga, Juliana T; Alves Filho, José Carlos Farias ; Cunha, Thiago Mattar ; Fraceto, Leonardo F; Cunha, Fernando de Queiroz ; Napimoga, Marcelo H ; Casagrande, Rúbia ; Verri Júnior, Waldiceu Aparecido
Source: Scientific Reports. Unidade: FMRP
Subjects: ARTRITE, INFLAMAÇÃO, DOR, RATOS
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ABNT
RUIZ-MIYAZAWA, Kenji W et al. 15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice. Scientific Reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-32334-0. Acesso em: 04 nov. 2024.
APA
Ruiz-Miyazawa, K. W., Staurengo-Ferrari, L., Pinho-Ribeiro, F. A., Fattori, V., Zaninelli, T. H., Badaro-Garcia, S., et al. (2018). 15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice. Scientific Reports, 8. doi:10.1038/s41598-018-32334-0
NLM
Ruiz-Miyazawa KW, Staurengo-Ferrari L, Pinho-Ribeiro FA, Fattori V, Zaninelli TH, Badaro-Garcia S, Borghi SM, Andrade KC, Clemente-Napimoga JT, Alves Filho JCF, Cunha TM, Fraceto LF, Cunha F de Q, Napimoga MH, Casagrande R, Verri Júnior WA. 15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 04 ] Available from: https://doi.org/10.1038/s41598-018-32334-0
Vancouver
Ruiz-Miyazawa KW, Staurengo-Ferrari L, Pinho-Ribeiro FA, Fattori V, Zaninelli TH, Badaro-Garcia S, Borghi SM, Andrade KC, Clemente-Napimoga JT, Alves Filho JCF, Cunha TM, Fraceto LF, Cunha F de Q, Napimoga MH, Casagrande R, Verri Júnior WA. 15d-PGJ2-loaded nanocapsules ameliorate experimental gout arthritis by reducing pain and inflammation in a PPAR-gamma-sensitive manner in mice [Internet]. Scientific Reports. 2018 ; 8[citado 2024 nov. 04 ] Available from: https://doi.org/10.1038/s41598-018-32334-0
Artigo de periodico
Participation of transient receptor potential vanilloid 1 in paclitaxel-induced acute visceral and peripheral nociception in rodents (2018)
Rossato, Mateus Fortes; Rigo, Flavia Karine; Oliveira, Sara Marchesan; Guerra, Gustavo Petri; Silva, Cássia Regina; Cunha, Thiago Mattar; Gomez, Marcus Vinicius; Ferreira, Juliano; Trevisan, Gabriela
Source: European Journal of Pharmacology. Unidade: FMRP
Subjects: FÁRMACOS, QUIMIOTERÁPICOS, DOR
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ABNT
ROSSATO, Mateus Fortes et al. Participation of transient receptor potential vanilloid 1 in paclitaxel-induced acute visceral and peripheral nociception in rodents. European Journal of Pharmacology, v. 828, p. 42-51, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2018.03.033. Acesso em: 04 nov. 2024.
APA
Rossato, M. F., Rigo, F. K., Oliveira, S. M., Guerra, G. P., Silva, C. R., Cunha, T. M., et al. (2018). Participation of transient receptor potential vanilloid 1 in paclitaxel-induced acute visceral and peripheral nociception in rodents. European Journal of Pharmacology, 828, 42-51. doi:10.1016/j.ejphar.2018.03.033
NLM
Rossato MF, Rigo FK, Oliveira SM, Guerra GP, Silva CR, Cunha TM, Gomez MV, Ferreira J, Trevisan G. Participation of transient receptor potential vanilloid 1 in paclitaxel-induced acute visceral and peripheral nociception in rodents [Internet]. European Journal of Pharmacology. 2018 ; 828 42-51.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.ejphar.2018.03.033
Vancouver
Rossato MF, Rigo FK, Oliveira SM, Guerra GP, Silva CR, Cunha TM, Gomez MV, Ferreira J, Trevisan G. Participation of transient receptor potential vanilloid 1 in paclitaxel-induced acute visceral and peripheral nociception in rodents [Internet]. European Journal of Pharmacology. 2018 ; 828 42-51.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.ejphar.2018.03.033
Artigo de periodico
Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis (2018)
Staurengo-Ferrari, Larissa; Trevelin, Silvia C.; Fattori, Victor; Nascimento, Daniele C.; Lima, Kalil A. de; Pelayo, Jacinta S.; Figueiredo, Florêncio; Casagrande, Rubia; Fukada, Sandra Yasuyo; Teixeira, Mauro M.; Cunha, Thiago Mattar; Liew, Foo Y.; Oliveira, Rene D.; Louzada Júnior, Paulo; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias; Verri, Waldiceu A.
Source: Frontiers in Immunology. Unidades: FCFRP, FMRP
Subjects: MACRÓFAGOS, STAPHYLOCOCCUS, INTERLEUCINAS, ÓXIDO NÍTRICO, ARTRITE
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ABNT
STAURENGO-FERRARI, Larissa et al. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, v. 9, 2018Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2018.00962. Acesso em: 04 nov. 2024.
APA
Staurengo-Ferrari, L., Trevelin, S. C., Fattori, V., Nascimento, D. C., Lima, K. A. de, Pelayo, J. S., et al. (2018). Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis. Frontiers in Immunology, 9. doi:10.3389/fimmu.2018.00962
NLM
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Vancouver
Staurengo-Ferrari L, Trevelin SC, Fattori V, Nascimento DC, Lima KA de, Pelayo JS, Figueiredo F, Casagrande R, Fukada SY, Teixeira MM, Cunha TM, Liew FY, Oliveira RD, Louzada Júnior P, Cunha F de Q, Alves Filho JCF, Verri WA. Interleukin-33 receptor (ST2) deficiency improves the outcome of staphylococcus aureus-induced septic arthritis [Internet]. Frontiers in Immunology. 2018 ; 9[citado 2024 nov. 04 ] Available from: https://doi.org/10.3389/fimmu.2018.00962
Artigo de periodico
Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis (2018)
Silva, Maria C; Ferreira, Marcela Davoli; Medina, Tiago S; Costa, Renata Sesti; Silva, Grace K; Lopes, Carla D; Cardozo, Lucas Esteves; Gava, Fábio N; Lyroni, Konstantina; Dias, Fabricio C; Frade, Amanda F; Baron, Monique; Nakaya, Helder Takashi Imoto ; Figueiredo, Florêncio; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Tsatsanis, Christos; Chevillard, Christophe; Cunha-Neto, Edécio; Hirsch, Emilio; Silva, João Santana da; Cunha, Thiago Mattar
Source: Nature Communications. Unidades: FCF, FMRP, FM
Subjects: TRYPANOSOMA CRUZI, MIOCARDIOPATIAS
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ABNT
SILVA, Maria C et al. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, v. 9, p. 1-14 art. 1513, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41467-018-03986-3. Acesso em: 04 nov. 2024.
APA
Silva, M. C., Ferreira, M. D., Medina, T. S., Costa, R. S., Silva, G. K., Lopes, C. D., et al. (2018). Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis. Nature Communications, 9, 1-14 art. 1513. doi:10.1038/s41467-018-03986-3
NLM
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Vancouver
Silva MC, Ferreira MD, Medina TS, Costa RS, Silva GK, Lopes CD, Cardozo LE, Gava FN, Lyroni K, Dias FC, Frade AF, Baron M, Nakaya HTI, Figueiredo F, Alves Filho JCF, Cunha F de Q, Tsatsanis C, Chevillard C, Cunha-Neto E, Hirsch E, Silva JS da, Cunha TM. Canonical PI3Kγ signaling in myeloid cells restricts Trypanosoma cruzi infection and dampens chagasic myocarditis [Internet]. Nature Communications. 2018 ; 9 1-14 art. 1513.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1038/s41467-018-03986-3
Artigo de periodico
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis (2018)
Peres, Raphael Sanches; Donate, Paula Barbim; Talbot, Jhimmy; Cecílio, Nerry Tatiana; Lobo, Patricia Rolim Mendonça; Machado, Caio Cavalcante; Lima, Kalil William Alves de; Oliveira, Renê Donizete Ribeiro de; Carregaro, Vanessa; Nakaya, Helder Takashi Imoto ; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Liew, Foo Yew; Louzada Júnior, Paulo; Cunha, Fernando Queiróz
Source: Journal of Autoimmunity. Unidades: FMRP, FCF
Subjects: ARTRITE REUMATOIDE, METOTREXATO
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ABNT
PERES, Raphael Sanches et al. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, v. 90, p. 49-58, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jaut.2018.01.004. Acesso em: 04 nov. 2024.
APA
Peres, R. S., Donate, P. B., Talbot, J., Cecílio, N. T., Lobo, P. R. M., Machado, C. C., et al. (2018). TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis. Journal of Autoimmunity, 90, 49-58. doi:10.1016/j.jaut.2018.01.004
NLM
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Vancouver
Peres RS, Donate PB, Talbot J, Cecílio NT, Lobo PRM, Machado CC, Lima KWA de, Oliveira RDR de, Carregaro V, Nakaya HTI, Cunha TM, Alves Filho JCF, Liew FY, Louzada Júnior P, Cunha FQ. TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis [Internet]. Journal of Autoimmunity. 2018 ; 90 49-58.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jaut.2018.01.004
Artigo de periodico
Cortical stimulation in conscious rats controls joint inflammation (2018)
Bassi, Gabriel Shimizu; Ulloa, Luis; Santos, Victor Rodrigues; Del Vecchio, Flávio; Pereira, Polianna Delfino; Rodrigues, Gerson Jhonatan; Castania, Jaci Airton; Cunha, Fernando de Queiroz; Salgado, Hélio César; Cunha, Thiago Mattar; Garcia-Cairasco, Norberto; Kanashiro, Alexandre
Source: Progress in Neuro-Psychopharmacology & Biological Psychiatry. Unidade: FMRP
Subjects: NERVO VAGO, INFLAMAÇÃO, ARTRITE REUMATOIDE
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ABNT
BASSI, Gabriel Shimizu et al. Cortical stimulation in conscious rats controls joint inflammation. Progress in Neuro-Psychopharmacology & Biological Psychiatry, v. 84, p. 201–213, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.pnpbp.2018.02.013. Acesso em: 04 nov. 2024.
APA
Bassi, G. S., Ulloa, L., Santos, V. R., Del Vecchio, F., Pereira, P. D., Rodrigues, G. J., et al. (2018). Cortical stimulation in conscious rats controls joint inflammation. Progress in Neuro-Psychopharmacology & Biological Psychiatry, 84, 201–213. doi:10.1016/j.pnpbp.2018.02.013
NLM
Bassi GS, Ulloa L, Santos VR, Del Vecchio F, Pereira PD, Rodrigues GJ, Castania JA, Cunha F de Q, Salgado HC, Cunha TM, Garcia-Cairasco N, Kanashiro A. Cortical stimulation in conscious rats controls joint inflammation [Internet]. Progress in Neuro-Psychopharmacology & Biological Psychiatry. 2018 ; 84 201–213.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.pnpbp.2018.02.013
Vancouver
Bassi GS, Ulloa L, Santos VR, Del Vecchio F, Pereira PD, Rodrigues GJ, Castania JA, Cunha F de Q, Salgado HC, Cunha TM, Garcia-Cairasco N, Kanashiro A. Cortical stimulation in conscious rats controls joint inflammation [Internet]. Progress in Neuro-Psychopharmacology & Biological Psychiatry. 2018 ; 84 201–213.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.pnpbp.2018.02.013
Artigo de periodico
Aqueous extracts from Uncaria tomentosa (Willd. ex Schult.) DC. reduce bronchial hyperresponsiveness and inflammation in a murine model of asthma (2018)
Azevedo, Bruna Cestari ; Morel, Lucas Junqueira Freitas; Carmona, Fabio ; Cunha, Thiago Mattar ; Contini, Silvia Helena Taleb; Delprete, Piero Giuseppe; Ramalho, Fernando Silva; Crevelin, Eduardo José ; Bertoni, Bianca Waléria; França, Suzelei Castro; Borges, Marcos de Carvalho ; Pereira, Ana Maria Soares
Source: Journal of Ethnopharmacology. Unidades: FMRP, FFCLRP
Subjects: ASMA, FÁRMACOS, PLANTAS MEDICINAIS, FITOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AZEVEDO, Bruna Cestari et al. Aqueous extracts from Uncaria tomentosa (Willd. ex Schult.) DC. reduce bronchial hyperresponsiveness and inflammation in a murine model of asthma. Journal of Ethnopharmacology, v. 218, p. 76-89, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jep.2018.02.013. Acesso em: 04 nov. 2024.
APA
Azevedo, B. C., Morel, L. J. F., Carmona, F., Cunha, T. M., Contini, S. H. T., Delprete, P. G., et al. (2018). Aqueous extracts from Uncaria tomentosa (Willd. ex Schult.) DC. reduce bronchial hyperresponsiveness and inflammation in a murine model of asthma. Journal of Ethnopharmacology, 218, 76-89. doi:10.1016/j.jep.2018.02.013
NLM
Azevedo BC, Morel LJF, Carmona F, Cunha TM, Contini SHT, Delprete PG, Ramalho FS, Crevelin EJ, Bertoni BW, França SC, Borges M de C, Pereira AMS. Aqueous extracts from Uncaria tomentosa (Willd. ex Schult.) DC. reduce bronchial hyperresponsiveness and inflammation in a murine model of asthma [Internet]. Journal of Ethnopharmacology. 2018 ; 218 76-89.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jep.2018.02.013
Vancouver
Azevedo BC, Morel LJF, Carmona F, Cunha TM, Contini SHT, Delprete PG, Ramalho FS, Crevelin EJ, Bertoni BW, França SC, Borges M de C, Pereira AMS. Aqueous extracts from Uncaria tomentosa (Willd. ex Schult.) DC. reduce bronchial hyperresponsiveness and inflammation in a murine model of asthma [Internet]. Journal of Ethnopharmacology. 2018 ; 218 76-89.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.jep.2018.02.013
Artigo de periodico
The atypical chemokine receptor ACKR2 is protective against sepsis (2018)
Castanheira, Fernanda Vargas e Silva; Borges, Vanessa de Fátima; Sônego, Fabiane; Kanashiro, Alexandre ; Donate, Paula Barbim; Melo, Paulo Henrique de; Pallas, Kenneth; Russo, Remo C.; Amaral, Flávio A.; Teixeira, Mauro M.; Ramalho, Fernando Silva ; Cunha, Thiago Mattar ; Liew, Foo Y.; Alves Filho, José Carlos Farias ; Graham, Gerard J.; Cunha, Fernando de Queiroz
Source: SHOCK. Unidade: FMRP
Subjects: SEPSE, QUIMIOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASTANHEIRA, Fernanda Vargas e Silva et al. The atypical chemokine receptor ACKR2 is protective against sepsis. SHOCK, v. 49, n. 6, p. 682-689, 2018Tradução . . Disponível em: https://doi.org/10.1097/shk.0000000000000969. Acesso em: 04 nov. 2024.
APA
Castanheira, F. V. e S., Borges, V. de F., Sônego, F., Kanashiro, A., Donate, P. B., Melo, P. H. de, et al. (2018). The atypical chemokine receptor ACKR2 is protective against sepsis. SHOCK, 49( 6), 682-689. doi:10.1097/shk.0000000000000969
NLM
Castanheira FV e S, Borges V de F, Sônego F, Kanashiro A, Donate PB, Melo PH de, Pallas K, Russo RC, Amaral FA, Teixeira MM, Ramalho FS, Cunha TM, Liew FY, Alves Filho JCF, Graham GJ, Cunha F de Q. The atypical chemokine receptor ACKR2 is protective against sepsis [Internet]. SHOCK. 2018 ; 49( 6): 682-689.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/shk.0000000000000969
Vancouver
Castanheira FV e S, Borges V de F, Sônego F, Kanashiro A, Donate PB, Melo PH de, Pallas K, Russo RC, Amaral FA, Teixeira MM, Ramalho FS, Cunha TM, Liew FY, Alves Filho JCF, Graham GJ, Cunha F de Q. The atypical chemokine receptor ACKR2 is protective against sepsis [Internet]. SHOCK. 2018 ; 49( 6): 682-689.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1097/shk.0000000000000969
Artigo de periodico
Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB (2018)
Manchope, Marília F.; Artero, Nayara A.; Fattori, Victor; Mizokami, Sandra S.; Pitol, Dimitrius Leonardo; Issa, João Paulo Mardegan; Fukada, Sandra Yasuyo; Cunha, Thiago Mattar; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Casagrande, Rubia; Verri Júnior, Waldiceu A.
Source: Inflammation Research. Unidades: FORP, FCFRP, FMRP
Subjects: ARTRITE, ARTROPLASTIA, FLAVONOIDES, DOR, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MANCHOPE, Marília F. et al. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB. Inflammation Research, v. 67, n. 11-12, p. 997-1012, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00011-018-1195-y. Acesso em: 04 nov. 2024.
APA
Manchope, M. F., Artero, N. A., Fattori, V., Mizokami, S. S., Pitol, D. L., Issa, J. P. M., et al. (2018). Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB. Inflammation Research, 67( 11-12), 997-1012. doi:10.1007/s00011-018-1195-y
NLM
Manchope MF, Artero NA, Fattori V, Mizokami SS, Pitol DL, Issa JPM, Fukada SY, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB [Internet]. Inflammation Research. 2018 ; 67( 11-12): 997-1012.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1007/s00011-018-1195-y
Vancouver
Manchope MF, Artero NA, Fattori V, Mizokami SS, Pitol DL, Issa JPM, Fukada SY, Cunha TM, Alves Filho JCF, Cunha F de Q, Casagrande R, Verri Júnior WA. Naringenin mitigates titanium dioxide (TiO2)-induced chronic arthritis in mice: role of oxidative stress, cytokines, and NFκB [Internet]. Inflammation Research. 2018 ; 67( 11-12): 997-1012.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1007/s00011-018-1195-y
Artigo de periodico
Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner (2018)
Wanderley, Carlos W.; Colon, David F.; Luiz, Joao Paulo Mesquita; Oliveira, Francisco F.; Viacava, Paula R.; Leite, Caio A.; Pereira, Janaina A.; Silva, Camila M.; Silva, Cassia R.; Silva, Rangel L.; Hernandez, Cesar A. Speck; Mota, Jose M.; Alves Filho, José Carlos Farias; Lima-Júnior, Roberto C.; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz
Source: Cancer Research. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, IMUNOLOGIA CELULAR, NEOPLASIAS, MACRÓFAGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WANDERLEY, Carlos W. et al. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner. Cancer Research, v. 78, n. 20, p. 5891-5900, 2018Tradução . . Disponível em: https://doi.org/10.1158/0008-5472.can-17-3480. Acesso em: 04 nov. 2024.
APA
Wanderley, C. W., Colon, D. F., Luiz, J. P. M., Oliveira, F. F., Viacava, P. R., Leite, C. A., et al. (2018). Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner. Cancer Research, 78( 20), 5891-5900. doi:10.1158/0008-5472.can-17-3480
NLM
Wanderley CW, Colon DF, Luiz JPM, Oliveira FF, Viacava PR, Leite CA, Pereira JA, Silva CM, Silva CR, Silva RL, Hernandez CAS, Mota JM, Alves Filho JCF, Lima-Júnior RC, Cunha TM, Cunha F de Q. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner [Internet]. Cancer Research. 2018 ; 78( 20): 5891-5900.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1158/0008-5472.can-17-3480
Vancouver
Wanderley CW, Colon DF, Luiz JPM, Oliveira FF, Viacava PR, Leite CA, Pereira JA, Silva CM, Silva CR, Silva RL, Hernandez CAS, Mota JM, Alves Filho JCF, Lima-Júnior RC, Cunha TM, Cunha F de Q. Paclitaxel reduces tumor growth by reprogramming tumor-associated macrophages to an M1- profile in a TLR4-dependent manner [Internet]. Cancer Research. 2018 ; 78( 20): 5891-5900.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1158/0008-5472.can-17-3480
Artigo de periodico
Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation (2018)
Kanashiro, Alexandre; Franchin, Marcelo; Bassi, Gabriel Shimizu; Santana, Dênis Augusto Reis; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ulloa, Luis; Rodrigues, Gerson Jonathan
Source: Fundamental and Clinical Pharmacology. Unidade: FMRP
Subjects: ARTRITE, RECEPTORES ANDROGÊNICOS, SISTEMA NERVOSO PARASSIMPÁTICO, NEUTRÓFILOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KANASHIRO, Alexandre et al. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, v. 32, n. 2, p. 155-162, 2018Tradução . . Disponível em: https://doi.org/10.1111/fcp.12338. Acesso em: 04 nov. 2024.
APA
Kanashiro, A., Franchin, M., Bassi, G. S., Santana, D. A. R., Cunha, T. M., Cunha, F. de Q., et al. (2018). Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, 32( 2), 155-162. doi:10.1111/fcp.12338
NLM
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/fcp.12338
Vancouver
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1111/fcp.12338
Artigo de periodico
Liver immune cells release type 1 interferon due to DNA sensing and amplify liver injury from acetaminophen overdose (2018)
Araujo, Alan Moreira de; Antunes, Maísa; Mattos, Matheus; Diniz, Ariane Barros; Alvarenga, Débora; Nakagaki, Brenda; Carvalho, Érika; Lacerda, Viviane; Carvalho-Gontijo, Raquel; Goulart, Jorge; Cunha, Thiago Mattar
Source: Cells. Unidade: FMRP
Subjects: HEPATOPATIAS, SISTEMA IMUNE, IMUNIDADE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, Alan Moreira de et al. Liver immune cells release type 1 interferon due to DNA sensing and amplify liver injury from acetaminophen overdose. Cells, v. 7, n. 8, 2018Tradução . . Disponível em: https://doi.org/10.3390/cells7080088. Acesso em: 04 nov. 2024.
APA
Araujo, A. M. de, Antunes, M., Mattos, M., Diniz, A. B., Alvarenga, D., Nakagaki, B., et al. (2018). Liver immune cells release type 1 interferon due to DNA sensing and amplify liver injury from acetaminophen overdose. Cells, 7( 8). doi:10.3390/cells7080088
NLM
Araujo AM de, Antunes M, Mattos M, Diniz AB, Alvarenga D, Nakagaki B, Carvalho É, Lacerda V, Carvalho-Gontijo R, Goulart J, Cunha TM. Liver immune cells release type 1 interferon due to DNA sensing and amplify liver injury from acetaminophen overdose [Internet]. Cells. 2018 ; 7( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/cells7080088
Vancouver
Araujo AM de, Antunes M, Mattos M, Diniz AB, Alvarenga D, Nakagaki B, Carvalho É, Lacerda V, Carvalho-Gontijo R, Goulart J, Cunha TM. Liver immune cells release type 1 interferon due to DNA sensing and amplify liver injury from acetaminophen overdose [Internet]. Cells. 2018 ; 7( 8):[citado 2024 nov. 04 ] Available from: https://doi.org/10.3390/cells7080088
Artigo de periodico
Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes (2018)
Saraiva, André L.; Veras, Flávio P.; Peres, Raphael S.; Talbot, Jhimmy; Lima, Kalil A. de; Luiz, João P.; Carballido, José M.; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ryffel, Bernhard; Alves Filho, José Carlos Farias
Source: The FASEB Journal. Unidade: FMRP
Subjects: DOENÇAS AUTOIMUNES, INFLAMAÇÃO, METABOLISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARAIVA, André L. et al. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes. The FASEB Journal, v. 32, n. 12, p. 6550-6558, 2018Tradução . . Disponível em: https://doi.org/10.1096/fj.201800285. Acesso em: 04 nov. 2024.
APA
Saraiva, A. L., Veras, F. P., Peres, R. S., Talbot, J., Lima, K. A. de, Luiz, J. P., et al. (2018). Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes. The FASEB Journal, 32( 12), 6550-6558. doi:10.1096/fj.201800285
NLM
Saraiva AL, Veras FP, Peres RS, Talbot J, Lima KA de, Luiz JP, Carballido JM, Cunha TM, Cunha F de Q, Ryffel B, Alves Filho JCF. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes [Internet]. The FASEB Journal. 2018 ; 32( 12): 6550-6558.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1096/fj.201800285
Vancouver
Saraiva AL, Veras FP, Peres RS, Talbot J, Lima KA de, Luiz JP, Carballido JM, Cunha TM, Cunha F de Q, Ryffel B, Alves Filho JCF. Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes [Internet]. The FASEB Journal. 2018 ; 32( 12): 6550-6558.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1096/fj.201800285
Artigo de periodico
Synthetic chalcones as potential tool for acute- and chronic-pain control (2018)
Rocha, Lilian W.; Sonza, Daniele R.; Broering, Milena Fronza; Nunes, Roberta; Buzzi, Fátima de Campos; Corrêa, Rogério; Silva, Rangel Leal; Cunha, Thiago Mattar; Santin, José Roberto; Quintão, Nara Lins Meira
Source: Biomedicine & Pharmacotherapy. Unidade: FMRP
Subjects: HIPERSENSIBILIDADE, CALCOGÊNIOS, PÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROCHA, Lilian W. et al. Synthetic chalcones as potential tool for acute- and chronic-pain control. Biomedicine & Pharmacotherapy, v. 104, p. 437-450, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.biopha.2018.05.075. Acesso em: 04 nov. 2024.
APA
Rocha, L. W., Sonza, D. R., Broering, M. F., Nunes, R., Buzzi, F. de C., Corrêa, R., et al. (2018). Synthetic chalcones as potential tool for acute- and chronic-pain control. Biomedicine & Pharmacotherapy, 104, 437-450. doi:10.1016/j.biopha.2018.05.075
NLM
Rocha LW, Sonza DR, Broering MF, Nunes R, Buzzi F de C, Corrêa R, Silva RL, Cunha TM, Santin JR, Quintão NLM. Synthetic chalcones as potential tool for acute- and chronic-pain control [Internet]. Biomedicine & Pharmacotherapy. 2018 ; 104 437-450.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.biopha.2018.05.075
Vancouver
Rocha LW, Sonza DR, Broering MF, Nunes R, Buzzi F de C, Corrêa R, Silva RL, Cunha TM, Santin JR, Quintão NLM. Synthetic chalcones as potential tool for acute- and chronic-pain control [Internet]. Biomedicine & Pharmacotherapy. 2018 ; 104 437-450.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.biopha.2018.05.075
Artigo de periodico
Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors (2018)
Sonego, Andreza B.; Prado, Douglas S.; Vale, Gabriel T.; Sepulveda-Diaz, Julia E.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato ; Del Bel, Elaine A.; Raisman-Vozari, Rita; Guimarães, Francisco Silveira
Source: Brain, Behavior, and Immunity. Unidades: FMRP, EERP, FORP
Subjects: CANABINOIDES, INFLAMAÇÃO, ESTRESSE OXIDATIVO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SONEGO, Andreza B. et al. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, v. 74, p. 241-251, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbi.2018.09.014. Acesso em: 04 nov. 2024.
APA
Sonego, A. B., Prado, D. S., Vale, G. T., Sepulveda-Diaz, J. E., Cunha, T. M., Tirapelli, C. R., et al. (2018). Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors. Brain, Behavior, and Immunity, 74, 241-251. doi:10.1016/j.bbi.2018.09.014
NLM
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014
Vancouver
Sonego AB, Prado DS, Vale GT, Sepulveda-Diaz JE, Cunha TM, Tirapelli CR, Del Bel EA, Raisman-Vozari R, Guimarães FS. Cannabidiol prevents haloperidol-induced vacuos chewing movements and inflammatory changes in mice via PPARγ receptors [Internet]. Brain, Behavior, and Immunity. 2018 ; 74 241-251.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bbi.2018.09.014

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