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Artigo de periodico
miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation (2021)
Lima, Vanessa Morais; Liu, Jianming; Brandão, Bruna B. ; Lino, Caroline Antunes ; Silva, Camila Stephanie Balbino ; Ribeiro, Márcio Augusto Campos; Silva, Tiago Eugênio Oliveira da; Gregório, Caroline Cristiano Real ; Faria, Daniele de Paula ; Cederquist, Carly; Huang, Zhan-Peng; Hu, Xiaoyun ; Barreto-Chaves, Maria Luiza Morais ; Ferreira, Julio Cesar Batista ; Festuccia, William Tadeu Lara ; Mori, Marcelo A. ; Kahn, C. Ronald ; Wang, Da-Zhi ; Diniz, Gabriela Placoná
Fonte: Metabolism. Unidades: FM, ICB
Assuntos: ANATOMIA, FISIOLOGIA, TECIDO ADIPOSO, DIETA, OBESIDADE, TERMOGÊNESE, METABOLISMO DE GORDURA, MICRORNAS, CAMUNDONGOS, ADIPOSIDADE
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LIMA, Vanessa Morais et al. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, v. 117, p. 13 , 2021Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2021.154723. Acesso em: 03 nov. 2025.
APA
Lima, V. M., Liu, J., Brandão, B. B., Lino, C. A., Silva, C. S. B., Ribeiro, M. A. C., et al. (2021). miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation. Metabolism, 117, 13 . doi:10.1016/j.metabol.2021.154723
NLM
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Vancouver
Lima VM, Liu J, Brandão BB, Lino CA, Silva CSB, Ribeiro MAC, Silva TEO da, Gregório CCR, Faria D de P, Cederquist C, Huang Z-P, Hu X, Barreto-Chaves MLM, Ferreira JCB, Festuccia WTL, Mori MA, Kahn CR, Wang D-Z, Diniz GP. miRNA-22 deletion limits white adipose expansion and activates brown fat to attenuate high-fat diet-induced fat mass accumulation [Internet]. Metabolism. 2021 ; 117 13 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.metabol.2021.154723
Artigo de periodico
Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure (2020)
Fontes, Milene Tavares ; Paula, Suliana Mesquita ; Lino, Caroline Antunes ; Senger, Nathalia ; Couto, Gisele Kruger; Barreto-Chaves, Maria Luiza Morais ; Mill, José Geraldo ; Rossoni, Luciana Venturini
Fonte: Clinical Science. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, TECIDO ADIPOSO, INSUFICIÊNCIA CARDÍACA ANIMAL, DOENÇAS CARDIOVASCULARES EM ANIMAL, ENDOTÉLIO VASCULAR, INFARTO DO MIOCÁRDIO, RATOS, ANGIOTENSINAS, AORTA DE ANIMAL, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, AORTA TORÁCICA
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FONTES, Milene Tavares et al. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201099. Acesso em: 03 nov. 2025.
APA
Fontes, M. T., Paula, S. M., Lino, C. A., Senger, N., Couto, G. K., Barreto-Chaves, M. L. M., et al. (2020). Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science. doi:10.1042/CS20201099
NLM
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20201099
Vancouver
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20201099
Artigo de periodico
Impact of hyperthyroidism on cardiac hypertrophy (2020)
Barreto-Chaves, Maria Luiza Morais ; Senger, Nathalia; Fevereiro, Marina Reingruber; Cesaro, Alice Cristina Parletta ; Takano, Ana Paula Cremasco
Fonte: Endocrine Connections. Unidade: ICB
Assuntos: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, CÉLULAS MUSCULARES, INSUFICIÊNCIA CARDÍACA, SISTEMA NERVOSO SIMPÁTICO
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BARRETO-CHAVES, Maria Luiza Morais et al. Impact of hyperthyroidism on cardiac hypertrophy. Endocrine Connections, v. 9, n. 3, p. R59–R69, 2020Tradução . . Disponível em: https://doi.org/10.1530/EC-19-0543. Acesso em: 03 nov. 2025.
APA
Barreto-Chaves, M. L. M., Senger, N., Fevereiro, M. R., Cesaro, A. C. P., & Takano, A. P. C. (2020). Impact of hyperthyroidism on cardiac hypertrophy. Endocrine Connections, 9( 3), R59–R69. doi:10.1530/EC-19-0543
NLM
Barreto-Chaves MLM, Senger N, Fevereiro MR, Cesaro ACP, Takano APC. Impact of hyperthyroidism on cardiac hypertrophy [Internet]. Endocrine Connections. 2020 ; 9( 3): R59–R69.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1530/EC-19-0543
Vancouver
Barreto-Chaves MLM, Senger N, Fevereiro MR, Cesaro ACP, Takano APC. Impact of hyperthyroidism on cardiac hypertrophy [Internet]. Endocrine Connections. 2020 ; 9( 3): R59–R69.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1530/EC-19-0543
Artigo de periodico
Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R (2020)
Lino, Caroline Antunes ; Teixeira, Larissa de Bortoli ; Simões, Sarah Capelupe; Silva, Tábatha de Oliveira ; Diniz, Gabriela Placoná ; Costa Neto, Claudio Miguel da ; Barreto-Chaves, Maria Luiza Morais
Fonte: Journal of Cellular Physiology. Unidades: FMRP, ICB
Assuntos: ANATOMIA, HIPERTROFIA VENTRICULAR ESQUERDA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, SISTEMA CARDIOVASCULAR, IMUNOHISTOQUÍMICA, PROTEÍNAS QUINASES
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LINO, Caroline Antunes et al. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, v. 236, n. 6, p. 4640-4654, 2020Tradução . . Disponível em: https://doi.org/10.1002/jcp.30187. Acesso em: 03 nov. 2025.
APA
Lino, C. A., Teixeira, L. de B., Simões, S. C., Silva, T. de O., Diniz, G. P., Costa Neto, C. M. da, & Barreto-Chaves, M. L. M. (2020). Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R. Journal of Cellular Physiology, 236( 6), 4640-4654. doi:10.1002/jcp.30187
NLM
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.30187
Vancouver
Lino CA, Teixeira L de B, Simões SC, Silva T de O, Diniz GP, Costa Neto CM da, Barreto-Chaves MLM. Beta‐arrestin 2 mediates cardiac hypertrophy induced by thyroid hormones via AT1R [Internet]. Journal of Cellular Physiology. 2020 ; 236( 6): 4640-4654.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.30187
Artigo de periodico
The endocrinological component and signaling pathways associated to cardiac hypertrophy (2020)
Takano, Ana Paula Cremasco ; Senger, Nathalia ; Barreto-Chaves, Maria Luiza Morais
Fonte: Molecular and Cellular Endocrinology. Unidade: ICB
Assuntos: ANATOMIA, DOENÇAS CARDIOVASCULARES, HORMÔNIOS TIREOIDIANOS, INSULINA, SISTEMA RENINA-ANGIOTENSINA, CATECOLAMINAS, INSUFICIÊNCIA CARDÍACA
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TAKANO, Ana Paula Cremasco e SENGER, Nathalia e BARRETO-CHAVES, Maria Luiza Morais. The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, v. 30, p. 13 , 2020Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2020.110972. Acesso em: 03 nov. 2025.
APA
Takano, A. P. C., Senger, N., & Barreto-Chaves, M. L. M. (2020). The endocrinological component and signaling pathways associated to cardiac hypertrophy. Molecular and Cellular Endocrinology, 30, 13 . doi:10.1016/j.mce.2020.110972
NLM
Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
Vancouver
Takano APC, Senger N, Barreto-Chaves MLM. The endocrinological component and signaling pathways associated to cardiac hypertrophy [Internet]. Molecular and Cellular Endocrinology. 2020 ; 30 13 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2020.110972
Artigo de periodico
The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity (2020)
Gewehr, Mayara Calegaro Ferrari ; Teixeira, Alexandre Abilio de Souza ; Santos, Bruna de Araújo Cardoso dos; Biondo, Luana Amorim ; Gozzo, Fábio Cesar ; Marques, Amanda Miquilini Cordibello ; Eichler, Rosangela Aparecida dos Santos ; Reckziegel, Patrícia ; Silva, Renée de Nazaré Oliveira da ; Santos, Nilton Barretos dos ; Câmara, Niels Olsen Saraiva ; Castoldi, Angela ; Barreto-Chaves, Maria Luiza Morais ; Dale, Camila Squarzoni ; Senger, Nathalia; Lima, joanna Darck Carola Correia ; Seelaender, Marilia Cerqueira Leite ; Inada, Aline Carla ; Akamine, Eliana Hiromi ; Castro, Leandro Mantovani de ; Rodrigues, Alice Cristina ; Rosa Neto, José Cesar ; Ferro, Emer Suavinho
Fonte: Biomolecules. Unidade: ICB
Assuntos: IMUNOLOGIA, ANATOMIA, BIOLOGIA CELULAR, FARMACOLOGIA, METABOLISMO ENERGÉTICO, OBESIDADE, RESISTÊNCIA À INSULINA, DIETA ANIMAL, EXPRESSÃO GÊNICA, ENZIMAS HIDROLÍTICAS, ENZIMAS PROTEOLÍTICAS, RATOS, DIETA ANIMAL
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GEWEHR, Mayara Calegaro Ferrari et al. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity. Biomolecules, v. 10, n. 2, p. 40 , 2020Tradução . . Disponível em: https://doi.org/10.3390/biom10020321. Acesso em: 03 nov. 2025.
APA
Gewehr, M. C. F., Teixeira, A. A. de S., Santos, B. de A. C. dos, Biondo, L. A., Gozzo, F. C., Marques, A. M. C., et al. (2020). The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity. Biomolecules, 10( 2), 40 . doi:10.3390/biom10020321
NLM
Gewehr MCF, Teixeira AA de S, Santos B de AC dos, Biondo LA, Gozzo FC, Marques AMC, Eichler RA dos S, Reckziegel P, Silva R de NO da, Santos NB dos, Câmara NOS, Castoldi A, Barreto-Chaves MLM, Dale CS, Senger N, Lima joanna DCC, Seelaender MCL, Inada AC, Akamine EH, Castro LM de, Rodrigues AC, Rosa Neto JC, Ferro ES. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity [Internet]. Biomolecules. 2020 ; 10( 2): 40 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.3390/biom10020321
Vancouver
Gewehr MCF, Teixeira AA de S, Santos B de AC dos, Biondo LA, Gozzo FC, Marques AMC, Eichler RA dos S, Reckziegel P, Silva R de NO da, Santos NB dos, Câmara NOS, Castoldi A, Barreto-Chaves MLM, Dale CS, Senger N, Lima joanna DCC, Seelaender MCL, Inada AC, Akamine EH, Castro LM de, Rodrigues AC, Rosa Neto JC, Ferro ES. The relevance of thimet oligopeptidase in the regulation of energy metabolism and diet-induced obesity [Internet]. Biomolecules. 2020 ; 10( 2): 40 .[citado 2025 nov. 03 ] Available from: https://doi.org/10.3390/biom10020321
Artigo de periodico
Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders (2020)
Silva, Tábatha de Oliveira ; Lino, Caroline Antunes ; Buzatto, Vanessa C.; Asprino, Paula Fontes ; Luc, Yao Wei; Lima, Vanessa Morais ; Fonseca, Renata Inzinna Bernardo; Jensen, Leonardo; Murata, Gilson Masahiro ; Verissimo Filho, Sidney; Ribeiro, Márcio Augusto Campos; Irigoyen, Maria Claudia Costa ; Huangg, Zhan-Peng; Galante, Pedro Alexandre Favoretto ; Wang, Da‐Zhi ; Diniz, Gabriela Placoná ; Donato Junior, José ; Ferreira, Julio Cesar Batista ; Rodrigues, Alice Cristina ; Barreto-Chaves, Maria Luiza Morais
Fonte: Cellular Physiology and Biochemistry. Unidades: FM, ICB
Assuntos: ANATOMIA, FARMACOLOGIA, FISIOLOGIA, OBESIDADE, MICRORNAS, HIPERTROFIA VENTRICULAR ESQUERDA, DOENÇAS METABÓLICAS, ADIPOSIDADE, CAMUNDONGOS, TECIDO ADIPOSO, ECOCARDIOGRAFIA, HORMÔNIOS TIREOIDIANOS
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SILVA, Tábatha de Oliveira et al. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders. Cellular Physiology and Biochemistry, v. 54, n. 6, p. 1199-1217, 2020Tradução . . Disponível em: https://doi.org/10.33594/000000309. Acesso em: 03 nov. 2025.
APA
Silva, T. de O., Lino, C. A., Buzatto, V. C., Asprino, P. F., Luc, Y. W., Lima, V. M., et al. (2020). Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders. Cellular Physiology and Biochemistry, 54( 6), 1199-1217. doi:10.33594/000000309
NLM
Silva T de O, Lino CA, Buzatto VC, Asprino PF, Luc YW, Lima VM, Fonseca RIB, Jensen L, Murata GM, Verissimo Filho S, Ribeiro MAC, Irigoyen MCC, Huangg Z-P, Galante PAF, Wang D‐Z, Diniz GP, Donato Junior J, Ferreira JCB, Rodrigues AC, Barreto-Chaves MLM. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders [Internet]. Cellular Physiology and Biochemistry. 2020 ; 54( 6): 1199-1217.[citado 2025 nov. 03 ] Available from: https://doi.org/10.33594/000000309
Vancouver
Silva T de O, Lino CA, Buzatto VC, Asprino PF, Luc YW, Lima VM, Fonseca RIB, Jensen L, Murata GM, Verissimo Filho S, Ribeiro MAC, Irigoyen MCC, Huangg Z-P, Galante PAF, Wang D‐Z, Diniz GP, Donato Junior J, Ferreira JCB, Rodrigues AC, Barreto-Chaves MLM. Deletion of miRNA-22 induces cardiac hypertrophy in females but attenuates obesogenic diet-mediated metabolic disorders [Internet]. Cellular Physiology and Biochemistry. 2020 ; 54( 6): 1199-1217.[citado 2025 nov. 03 ] Available from: https://doi.org/10.33594/000000309
Artigo de periodico
Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism (2019)
Lino, Caroline Antunes; Demasi, Marilene; Barreto-Chaves, Maria Luiza Morais
Fonte: Molecular and Cellular Endocrinology. Unidade: ICB
Assuntos: ANATOMIA, METABOLISMO DE PROTEÍNA, HORMÔNIOS TIREOIDIANOS, CORAÇÃO
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LINO, Caroline Antunes e DEMASI, Marilene e BARRETO-CHAVES, Maria Luiza Morais. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, v. 493, p. 1-9, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110451. Acesso em: 03 nov. 2025.
APA
Lino, C. A., Demasi, M., & Barreto-Chaves, M. L. M. (2019). Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism. Molecular and Cellular Endocrinology, 493, 1-9. doi:10.1016/j.mce.2019.110451
NLM
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Vancouver
Lino CA, Demasi M, Barreto-Chaves MLM. Ubiquitin proteasome system (UPS) activation in the cardiac hypertrophy ofhyperthyroidism [Internet]. Molecular and Cellular Endocrinology. 2019 ; 493 1-9.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2019.110451
Trabalho de evento-resumo periodico
In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites (2019)
Alvarez, José Maria ; Santana D. Y.; Silva do Nascimento, R.; Fevereiro, M.; Borges da Silva, H.; Barreto-Chaves, Maria Luiza Morais ; Marinho, Claudio Romero Farias ; Epiphanio, Sabrina ; Câmara, Niels Olsen Saraiva ; Lima, Maria Regina D'Império
Fonte: European Journal of Immunology. Unidades: ICB, FCF
Assuntos: DOENÇA DE CHAGAS EM ANIMAL, TRYPANOSOMA CRUZI, CARDIOPATIAS, CAMUNDONGOS, ANIMAIS PARASITOS, IMUNOLOGIA, IMUNOPARASITOLOGIA, ANTIPARASITÁRIOS
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ALVAREZ, José Maria et al. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. Disponível em: https://doi.org/10.1002/eji.201970400. Acesso em: 03 nov. 2025. , 2019
APA
Alvarez, J. M., Santana D. Y.,, Silva do Nascimento, R., Fevereiro, M., Borges da Silva, H., Barreto-Chaves, M. L. M., et al. (2019). In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. doi:10.1002/eji.201970400
NLM
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/eji.201970400
Vancouver
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/eji.201970400
Artigo de periodico
Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan (2019)
Leme, Janaina M. M.; Ohar, Erina; Santiago, Veronica Feijoli ; Barros, Mário Henrique de ; Netto, Luis Eduardo Soares ; Pimenta, Daniel Carvalho ; Mariano, Douglas Oscar Ceolin ; Oliveira, Cristiano Luis Pinto de ; Bicev, Renata Naporano ; Barreto-Chaves, Maria Luiza Morais ; Lino, Caroline Antunes ; Demasi, Marilene
Fonte: Archives of Biochemistry and Biophysics. Unidades: ICB, IF
Assuntos: MUTAÇÃO, ANATOMIA, MICROBIOLOGIA, CRONOLOGIA
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ABNT
LEME, Janaina M. M. et al. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, v. 666, p. 63-72, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.abb.2019.03.012. Acesso em: 03 nov. 2025.
APA
Leme, J. M. M., Ohar, E., Santiago, V. F., Barros, M. H. de, Netto, L. E. S., Pimenta, D. C., et al. (2019). Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan. Archives of Biochemistry and Biophysics, 666, 63-72. doi:10.1016/j.abb.2019.03.012
NLM
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Vancouver
Leme JMM, Ohar E, Santiago VF, Barros MH de, Netto LES, Pimenta DC, Mariano DOC, Oliveira CLP de, Bicev RN, Barreto-Chaves MLM, Lino CA, Demasi M. Mutations of cys and Ser residues in the α5-subunit of the 20S proteasome from Saccharomyces cerevisiae affects gating and chronological lifespan [Internet]. Archives of Biochemistry and Biophysics. 2019 ; 666 63-72.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.abb.2019.03.012
Artigo de periodico
Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus (2019)
Palombit, Kelly ; Mendes, Cristina Eusébio ; Lima, Wothan Tavares de ; Barreto-Chaves, Maria Luiza Morais ; Castelucci, Patricia
Fonte: Digestive Diseases and Sciences. Unidade: ICB
Assuntos: ANATOMIA, FARMACOLOGIA, PLEXO MIENTÉRICO, ISQUEMIA, REPERFUSÃO, ÍLEO
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PALOMBIT, Kelly et al. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus. Digestive Diseases and Sciences, v. 64, p. 1815-1829, 2019Tradução . . Disponível em: https://doi.org/10.1007/s10620-019-05496-8. Acesso em: 03 nov. 2025.
APA
Palombit, K., Mendes, C. E., Lima, W. T. de, Barreto-Chaves, M. L. M., & Castelucci, P. (2019). Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus. Digestive Diseases and Sciences, 64, 1815-1829. doi:10.1007/s10620-019-05496-8
NLM
Palombit K, Mendes CE, Lima WT de, Barreto-Chaves MLM, Castelucci P. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus [Internet]. Digestive Diseases and Sciences. 2019 ; 64 1815-1829.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1007/s10620-019-05496-8
Vancouver
Palombit K, Mendes CE, Lima WT de, Barreto-Chaves MLM, Castelucci P. Blockage of the P2X7 receptor attenuates harmful changes produced by ischemia and reperfusion in the myenteric plexus [Internet]. Digestive Diseases and Sciences. 2019 ; 64 1815-1829.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1007/s10620-019-05496-8
Artigo de periodico
Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia (2019)
Lima, Vanessa Morais ; Lino, Caroline Antunes ; Senger, Nathalia ; Silva, Tábatha de Oliveira ; Fonseca, Renata Inzinna Bernardo; Bader, Michael ; Santos, Robson Augusto Sousa ; Donato Junior, José ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Fonte: Molecular and Cellular Endocrinology. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA, ANGIOTENSINA II, METABOLISMO, OBESIDADE, GLICOSE
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ABNT
LIMA, Vanessa Morais et al. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, v. 498, p. 1-10, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2019.110576. Acesso em: 03 nov. 2025.
APA
Lima, V. M., Lino, C. A., Senger, N., Silva, T. de O., Fonseca, R. I. B., Bader, M., et al. (2019). Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia. Molecular and Cellular Endocrinology, 498, 1-10. doi:10.1016/j.mce.2019.110576
NLM
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Vancouver
Lima VM, Lino CA, Senger N, Silva T de O, Fonseca RIB, Bader M, Santos RAS, Donato Junior J, Barreto-Chaves MLM, Diniz GP. Angiotensin II type 2 receptor mediates high fat diet-induced cardiomyocyte hypertrophy and hypercholesterolemia [Internet]. Molecular and Cellular Endocrinology. 2019 ; 498 1-10.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.mce.2019.110576
Artigo de periodico
High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury (2019)
Guedes, Elaine Castilho; Silva, Ivson Bezerra da; Lima, Vanessa Morais; Miranda, Juliane Braga ; Albuquerque, Rudá Prestes e ; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: ANATOMIA, OBESIDADE, CORAÇÃO, ANÓXIA, MIOCÁRDIO, ISQUEMIA DO MIOCÁRDIO ANIMAL, CAMUNDONGOS, CARDIOLOGIA VETERINÁRIA, DIETA ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUEDES, Elaine Castilho et al. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury. Journal of Cellular Physiology, v. 234, n. 6, p. 9399-9407, 2019Tradução . . Disponível em: https://doi.org/10.1002/jcp.27624. Acesso em: 03 nov. 2025.
APA
Guedes, E. C., Silva, I. B. da, Lima, V. M., Miranda, J. B., Albuquerque, R. P. e, Ferreira, J. C. B., et al. (2019). High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury. Journal of Cellular Physiology, 234( 6), 9399-9407. doi:10.1002/jcp.27624
NLM
Guedes EC, Silva IB da, Lima VM, Miranda JB, Albuquerque RP e, Ferreira JCB, Barreto-Chaves MLM, Diniz GP. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury [Internet]. Journal of Cellular Physiology. 2019 ; 234( 6): 9399-9407.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.27624
Vancouver
Guedes EC, Silva IB da, Lima VM, Miranda JB, Albuquerque RP e, Ferreira JCB, Barreto-Chaves MLM, Diniz GP. High fat diet reduces the expression of miRNA‐29b in heart and increases susceptibility of myocardium to ischemia/reperfusion injury [Internet]. Journal of Cellular Physiology. 2019 ; 234( 6): 9399-9407.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.27624
Artigo de periodico
MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control (2018)
Santana, Danni Yohani; Salgado, Rafael Moysés; Fevereiro, Marina Reingruber; Nascimento, Rogério Silva do; Fonseca, Raissa; Câmara, Niels Olsen Saraiva ; Epiphanio, Sabrina ; Marinho, Claudio Romero Farias ; Barreto-Chaves, Maria Luiza Morais ; Lima, Maria Regina D'Império ; Alvarez, José Maria
Fonte: PloS Neglected Tropical Diseases. Unidades: ICB, FCF
Assuntos: TRYPANOSOMA CRUZI, CORAÇÃO, DOENÇA DE CHAGAS, PARASITISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTANA, Danni Yohani et al. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control. PloS Neglected Tropical Diseases, v. 12, n. 8, p. 1-19, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0006617. Acesso em: 03 nov. 2025.
APA
Santana, D. Y., Salgado, R. M., Fevereiro, M. R., Nascimento, R. S. do, Fonseca, R., Câmara, N. O. S., et al. (2018). MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control. PloS Neglected Tropical Diseases, 12( 8), 1-19. doi:10.1371/journal.pntd.0006617
NLM
Santana DY, Salgado RM, Fevereiro MR, Nascimento RS do, Fonseca R, Câmara NOS, Epiphanio S, Marinho CRF, Barreto-Chaves MLM, Lima MRD'I, Alvarez JM. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control [Internet]. PloS Neglected Tropical Diseases. 2018 ; 12( 8): 1-19.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1371/journal.pntd.0006617
Vancouver
Santana DY, Salgado RM, Fevereiro MR, Nascimento RS do, Fonseca R, Câmara NOS, Epiphanio S, Marinho CRF, Barreto-Chaves MLM, Lima MRD'I, Alvarez JM. MyD88 activation in cardiomyocytes contributes to the heart immune response to acute Trypanosoma cruzi infection with no effect on local parasite control [Internet]. PloS Neglected Tropical Diseases. 2018 ; 12( 8): 1-19.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1371/journal.pntd.0006617
Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia ; Melo, Marcos Barrouin; Diniz, Gabriela Placoná ; Santos, Maria José Campagnole dos ; Santos, Robson Augusto Souza dos; Barreto-Chaves, Maria Luiza Morais
Fonte: Clinical Science. Unidade: ICB
Assuntos: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 03 nov. 2025.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20171606
Artigo de periodico
Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 (2018)
Bozi, Luiz Henrique Marchesi; Takano, Ana Paula Cremasco ; Campos, Juliane Cruz; Rolim, Natale Pinheiro Lage; Dourado, Paulo Magno Martins; Voltarelli, Vanessa Azevedo; Wisløff, Ulrik; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Brum, Patricia Chakur
Fonte: International journal of cardiology. Unidades: ICB, EEFE
Assuntos: RETÍCULO ENDOPLASMÁTICO, INFARTO DO MIOCÁRDIO, DOENÇAS CARDIOVASCULARES, PROTEÍNAS, RATOS WISTAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOZI, Luiz Henrique Marchesi et al. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, v. 272, p. 194-201, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2018.08.070. Acesso em: 03 nov. 2025.
APA
Bozi, L. H. M., Takano, A. P. C., Campos, J. C., Rolim, N. P. L., Dourado, P. M. M., Voltarelli, V. A., et al. (2018). Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, 272, 194-201. doi:10.1016/j.ijcard.2018.08.070
NLM
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Vancouver
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Artigo de periodico
MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone (2017)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Moreno, Camila Rodrigues; Senger, Nathalia; Barreto-Chaves, Maria Luiza Morais
Fonte: Journal of Cellular Physiology. Unidade: ICB
Assuntos: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, RNA DE TRANSFERÊNCIA, SISTEMA RENINA-ANGIOTENSINA, RATOS WISTAR, BLOQUEIO CARDÍACO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, v. 232, n. 12, p. 3360-3368, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25781. Acesso em: 03 nov. 2025.
APA
Diniz, G. P., Lino, C. A., Moreno, C. R., Senger, N., & Barreto-Chaves, M. L. M. (2017). MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, 232( 12), 3360-3368. doi:10.1002/jcp.25781
NLM
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.25781
Vancouver
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.25781
Artigo de periodico
S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone (2017)
Takano, Ana Paula Cremasco ; Munhoz, Carolina Demarchi ; Moriscot, Anselmo Sigari ; Gupta, Sudhiranjan; Barreto-Chaves, Maria Luiza Morais
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 03 nov. 2025.
APA
Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y
NLM
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Vancouver
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Fonte: Clinical Science. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 03 nov. 2025.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets (2016)
Guedes, Elaine Castilho ; França, Gustavo Starvaggi ; Lino, Caroline Antunes ; Koyama, Fernanda Christtanini ; Moreira, Luana do Nascimento; Alexandre, Juliana Gomes ; Galante, Pedro Alexandre Favoretto ; Barreto-Chaves, Maria Luiza Morais ; Diniz, Gabriela Placoná
Fonte: Journal of Cellular Physiology. Unidades: ICB, IQ
Assuntos: ANATOMIA, MICRORNAS, EXPRESSÃO GÊNICA, HIPERTROFIA VENTRICULAR ESQUERDA, MIOCÁRDIO, DIETA ANIMAL, CAMUNDONGOS, GLICEMIA, JEJUNO DE ANIMAL, INSULINA, RNA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUEDES, Elaine Castilho et al. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, v. 231, n. 8, p. 1771-1783, 2016Tradução . . Disponível em: https://doi.org/10.1002/jcp.25280. Acesso em: 03 nov. 2025.
APA
Guedes, E. C., França, G. S., Lino, C. A., Koyama, F. C., Moreira, L. do N., Alexandre, J. G., et al. (2016). MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets. Journal of Cellular Physiology, 231( 8), 1771-1783. doi:10.1002/jcp.25280
NLM
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.25280
Vancouver
Guedes EC, França GS, Lino CA, Koyama FC, Moreira L do N, Alexandre JG, Galante PAF, Barreto-Chaves MLM, Diniz GP. MicroRNA expression signature is altered in the cardiac remodeling induced by high fat diets [Internet]. Journal of Cellular Physiology. 2016 ; 231( 8): 1771-1783.[citado 2025 nov. 03 ] Available from: https://doi.org/10.1002/jcp.25280

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