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Artigo de periodico
Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology (2025)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Mori, Yasuo; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: IQ, ICB
Subjects: DELEÇÃO DE GENES, DOENÇA DE PARKINSON, RATOS
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, v. 62, p. 5333-5346, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s12035-024-04611-9. Acesso em: 21 out. 2025.
APA
Ferreira, A. F. F., Ulrich, H., Mori, Y., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology. Molecular Neurobiology, 62, 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 out. 21 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Ulrich H, Mori Y, Feng Z-P, Sun H-S, Britto LRG de. Deletion of the transient receptor potential Melastatin 2 gene mitigates the 6-hydroxydopamine-induced parkinson’s disease–like pathology [Internet]. Molecular Neurobiology. 2025 ; 62 5333-5346.[citado 2025 out. 21 ] Available from: https://dx.doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology (2025)
Ferreira, Ana Flávia Fernandes ; Mori, Yasuo ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Molecular Neurobiology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, APOPTOSE, ATIVIDADE MOTORA, RECEPTORES DE DOPAMINA, ESTRESSE OXIDATIVO, CAMUNDONGOS
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, v. 62, n. 4, p. 5333-5346, 2025Tradução . . Disponível em: https://doi.org/10.1007/s12035-024-04611-9. Acesso em: 21 out. 2025.
APA
Ferreira, A. F. F., Mori, Y., Feng, Z. -P., Sun, H. -S., Ulrich, H., & Britto, L. R. G. de. (2025). Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology. Molecular Neurobiology, 62( 4), 5333-5346. doi:10.1007/s12035-024-04611-9
NLM
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 out. 21 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Vancouver
Ferreira AFF, Mori Y, Feng Z-P, Sun H-S, Ulrich H, Britto LRG de. Deletion of the transient receptor potential melastatin 2 gene mitigates the 6-hyydroxydopamine-induced Parkinson’s Disease–Like Pathology [Internet]. Molecular Neurobiology. 2025 ; 62( 4): 5333-5346.[citado 2025 out. 21 ] Available from: https://doi.org/10.1007/s12035-024-04611-9
Artigo de periodico
Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease (2025)
Juvenal, Guilherme Araújo ; Higa, Guilherme Shigueto Vilar ; Marques, Lucas Bonfim; Zampieri, Thais Tessari; Viana, Felipe José Costa ; Britto, Luiz Roberto Giorgetti de ; Tang, Yong; Illes, Peter ; Virgilio, Francesco Di ; Ulrich, Henning ; Pasquale, Roberto de
Source: Purinergic Signalling. Unidades: ICB, IQ
Subjects: NEUROTRANSMISSORES, RECEPTORES
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ABNT
JUVENAL, Guilherme Araújo et al. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling, v. 21, p. 149-177, 2025Tradução . . Disponível em: https://dx.doi.org/10.1007/s11302-024-10034-x. Acesso em: 21 out. 2025.
APA
Juvenal, G. A., Higa, G. S. V., Marques, L. B., Zampieri, T. T., Viana, F. J. C., Britto, L. R. G. de, et al. (2025). Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease. Purinergic Signalling, 21, 149-177. doi:10.1007/s11302-024-10034-x
NLM
Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2025 ;21 149-177.[citado 2025 out. 21 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x
Vancouver
Juvenal GA, Higa GSV, Marques LB, Zampieri TT, Viana FJC, Britto LRG de, Tang Y, Illes P, Virgilio FD, Ulrich H, Pasquale R de. Regulation of GABAergic neurotransmission by purinergic receptors in brain physiology and disease [Internet]. Purinergic Signalling. 2025 ;21 149-177.[citado 2025 out. 21 ] Available from: https://dx.doi.org/10.1007/s11302-024-10034-x
Artigo de periodico-carta/editorial
Editorial: 15 years of frontiers in neuroanatomy: the origin of Parkinson's disease (2025)
Falquetto, Bárbara ; Nombela, Cristina ; Britto, Luiz Roberto Giorgetti de
Source: Frontiers in Neuroanatomy. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇAS NEURODEGENERATIVAS, NEUROANATOMIA, DOENÇA DE PARKINSON
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ABNT
FALQUETTO, Bárbara e NOMBELA, Cristina e BRITTO, Luiz Roberto Giorgetti de. Editorial: 15 years of frontiers in neuroanatomy: the origin of Parkinson's disease. Frontiers in Neuroanatomy. Lausanne: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.3389/fnana.2025.1649700. Acesso em: 21 out. 2025. , 2025
APA
Falquetto, B., Nombela, C., & Britto, L. R. G. de. (2025). Editorial: 15 years of frontiers in neuroanatomy: the origin of Parkinson's disease. Frontiers in Neuroanatomy. Lausanne: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.3389/fnana.2025.1649700
NLM
Falquetto B, Nombela C, Britto LRG de. Editorial: 15 years of frontiers in neuroanatomy: the origin of Parkinson's disease [Internet]. Frontiers in Neuroanatomy. 2025 ; 19 2 .[citado 2025 out. 21 ] Available from: https://doi.org/10.3389/fnana.2025.1649700
Vancouver
Falquetto B, Nombela C, Britto LRG de. Editorial: 15 years of frontiers in neuroanatomy: the origin of Parkinson's disease [Internet]. Frontiers in Neuroanatomy. 2025 ; 19 2 .[citado 2025 out. 21 ] Available from: https://doi.org/10.3389/fnana.2025.1649700
Artigo de periodico
The role of interleukin-17 in neurological disorders (2025)
Parka, Seung Eun ; Ferreira, Ana Flávia Fernandes ; Kwon, Hyeokbin ; Yu, Jeongwoo ; Diniz, Leila; Kim, Eunha ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Animal Cells and Systems. Unidades: IQ, ICB
Subjects: FISIOLOGIA, INTERLEUCINAS, TRANSTORNOS MENTAIS, DOENÇAS NEURODEGENERATIVAS, CITOCINAS, DOENÇAS DO SISTEMA NERVOSO CENTRAL
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ABNT
PARKA, Seung Eun et al. The role of interleukin-17 in neurological disorders. Animal Cells and Systems, v. 29, n. 1, p. 372-386, 2025Tradução . . Disponível em: https://doi.org/10.1080/19768354.2025.2510994. Acesso em: 21 out. 2025.
APA
Parka, S. E., Ferreira, A. F. F., Kwon, H., Yu, J., Diniz, L., Kim, E., et al. (2025). The role of interleukin-17 in neurological disorders. Animal Cells and Systems, 29( 1), 372-386. doi:10.1080/19768354.2025.2510994
NLM
Parka SE, Ferreira AFF, Kwon H, Yu J, Diniz L, Kim E, Ulrich H, Britto LRG de. The role of interleukin-17 in neurological disorders [Internet]. Animal Cells and Systems. 2025 ; 29( 1): 372-386.[citado 2025 out. 21 ] Available from: https://doi.org/10.1080/19768354.2025.2510994
Vancouver
Parka SE, Ferreira AFF, Kwon H, Yu J, Diniz L, Kim E, Ulrich H, Britto LRG de. The role of interleukin-17 in neurological disorders [Internet]. Animal Cells and Systems. 2025 ; 29( 1): 372-386.[citado 2025 out. 21 ] Available from: https://doi.org/10.1080/19768354.2025.2510994
Parte de monografia/livro
Criação do Curso de Ciências Biomédicas (2024)
Britto, Luiz Roberto Giorgetti de
Source: Instituto de Ciencias Biomédicas da Universidade de São Paulo: 55 anos de criação: sua história, seu progresso e suas realizações. Unidade: ICB
Subjects: BIOMEDICINA, BIOCIÊNCIAS
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ABNT
BRITTO, Luiz Roberto Giorgetti de. Criação do Curso de Ciências Biomédicas. Instituto de Ciencias Biomédicas da Universidade de São Paulo: 55 anos de criação: sua história, seu progresso e suas realizações. Tradução . São Paulo: ICB/USP, 2024. p. 314 . Disponível em: https://doi.org/10.11606/9788593820021. Acesso em: 21 out. 2025.
APA
Britto, L. R. G. de. (2024). Criação do Curso de Ciências Biomédicas. In Instituto de Ciencias Biomédicas da Universidade de São Paulo: 55 anos de criação: sua história, seu progresso e suas realizações (p. 314 ). São Paulo: ICB/USP. doi:10.11606/9788593820021
NLM
Britto LRG de. Criação do Curso de Ciências Biomédicas [Internet]. In: Instituto de Ciencias Biomédicas da Universidade de São Paulo: 55 anos de criação: sua história, seu progresso e suas realizações. São Paulo: ICB/USP; 2024. p. 314 .[citado 2025 out. 21 ] Available from: https://doi.org/10.11606/9788593820021
Vancouver
Britto LRG de. Criação do Curso de Ciências Biomédicas [Internet]. In: Instituto de Ciencias Biomédicas da Universidade de São Paulo: 55 anos de criação: sua história, seu progresso e suas realizações. São Paulo: ICB/USP; 2024. p. 314 .[citado 2025 out. 21 ] Available from: https://doi.org/10.11606/9788593820021
Artigo de periodico
Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model (2024)
Bianchetti, Maria Eduada Goulart ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Frontiers in Neuroanatomy. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE PARKINSON, ATROFIA DE MÚLTIPLOS SISTEMAS, CITOCINAS, DOENÇAS NEURODEGENERATIVAS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, IMUNOHISTOQUÍMICA, ELISA EM ANIMAL
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ABNT
BIANCHETTI, Maria Eduada Goulart e FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, v. 18, p. 12 , 2024Tradução . . Disponível em: https://doi.org/10.3389/fnana.2024.1511951. Acesso em: 21 out. 2025.
APA
Bianchetti, M. E. G., Ferreira, A. F. F., & Britto, L. R. G. de. (2024). Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model. Frontiers in Neuroanatomy, 18, 12 . doi:10.3389/fnana.2024.1511951
NLM
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 out. 21 ] Available from: https://doi.org/10.3389/fnana.2024.1511951
Vancouver
Bianchetti MEG, Ferreira AFF, Britto LRG de. Inhibition of neuroinflammation by GIBH-130 (AD-16) reduces neurodegeneration, motor deficits, and proinflammatory cytokines in a hemiparkinsonian model [Internet]. Frontiers in Neuroanatomy. 2024 ; 18 12 .[citado 2025 out. 21 ] Available from: https://doi.org/10.3389/fnana.2024.1511951
Artigo de periodico
Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years (2024)
Ferreira, Ana Flávia Fernandes ; Meira Neta, Marina da Silva ; Lemuchi, Livia M. ; Bianchetti, Maria E. ; Kamidai, Nicole M. ; Kilinsky, Livia Marques ; Britto, Luiz Roberto Giorgetti de
Source: Advanced in Neurology. Unidade: ICB
Subjects: FISIOLOGIA, MODELOS ANIMAIS DE DOENÇAS, ROEDORES, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years. Advanced in Neurology, v. 3, n. 3, p. 19 , 2024Tradução . . Disponível em: https://doi.org/10.36922/an.2903. Acesso em: 21 out. 2025.
APA
Ferreira, A. F. F., Meira Neta, M. da S., Lemuchi, L. M., Bianchetti, M. E., Kamidai, N. M., Kilinsky, L. M., & Britto, L. R. G. de. (2024). Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years. Advanced in Neurology, 3( 3), 19 . doi:10.36922/an.2903
NLM
Ferreira AFF, Meira Neta M da S, Lemuchi LM, Bianchetti ME, Kamidai NM, Kilinsky LM, Britto LRG de. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years [Internet]. Advanced in Neurology. 2024 ; 3( 3): 19 .[citado 2025 out. 21 ] Available from: https://doi.org/10.36922/an.2903
Vancouver
Ferreira AFF, Meira Neta M da S, Lemuchi LM, Bianchetti ME, Kamidai NM, Kilinsky LM, Britto LRG de. Most utilized rodent models for Alzheimer’s and Parkinson’s disease: a critical review of the past 5 years [Internet]. Advanced in Neurology. 2024 ; 3( 3): 19 .[citado 2025 out. 21 ] Available from: https://doi.org/10.36922/an.2903
Artigo de periodico
Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model (2024)
Ferreira, Ana Flávia Fernandes ; Ulrich, Henning ; Feng, Zhong-Ping ; Sun, Hong-Shuo ; Britto, Luiz Roberto Giorgetti de
Source: Experimental Neurology. Unidades: ICB, IQ
Subjects: FISIOLOGIA, DOENÇAS NEURODEGENERATIVAS, DOENÇA DE PARKINSON, MODELOS ANIMAIS DE DOENÇAS, CÉLULAS MORTAS, ASTRÓCITOS, NEURÓGLIA, CAMUNDONGOS, RATOS, TÉCNICAS ESTEREOTÁXICAS, IMUNOHISTOQUÍMICA, REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
FERREIRA, Ana Flávia Fernandes et al. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, v. 377, p. 15 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.expneurol.2024.114780. Acesso em: 21 out. 2025.
APA
Ferreira, A. F. F., Ulrich, H., Feng, Z. -P., Sun, H. -S., & Britto, L. R. G. de. (2024). Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model. Experimental Neurology, 377, 15 . doi:10.1016/j.expneurol.2024.114780
NLM
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Vancouver
Ferreira AFF, Ulrich H, Feng Z-P, Sun H-S, Britto LRG de. Neurodegeneration and glial morphological changes are both prevented by TRPM2 inhibition during the progression of a Parkinson's disease mouse model [Internet]. Experimental Neurology. 2024 ; 377 15 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.expneurol.2024.114780
Artigo de periodico
Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study (2024)
Singulani, Monique Patricio ; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de ; Camarini, Rosana
Source: Alzheimer's & Dementia. Unidades: FM, ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇA DE ALZHEIMER, LÍTIO, TRANSTORNO BIPOLAR, CAMUNDONGOS, IMUNOHISTOQUÍMICA
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ABNT
SINGULANI, Monique Patricio et al. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study. Alzheimer's & Dementia, v. 20, n. S1, p. 2 , 2024Tradução . . Disponível em: https://doi.org/10.1002/alz.090340. Acesso em: 21 out. 2025.
APA
Singulani, M. P., Talib, L. L., Forlenza, O. V., Britto, L. R. G. de, & Camarini, R. (2024). Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study. Alzheimer's & Dementia, 20( S1), 2 . doi:10.1002/alz.090340
NLM
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study [Internet]. Alzheimer's & Dementia. 2024 ; 20( S1): 2 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1002/alz.090340
Vancouver
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. Lithium as a pharmacological approach to treat behavioral complications in Alzheimer’s disease: a preclinical study [Internet]. Alzheimer's & Dementia. 2024 ; 20( S1): 2 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1002/alz.090340
Artigo de periodico
‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study (2024)
Singulani, Monique Patricio ; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de ; Camarini, Rosana
Source: Alzheimer's & Dementia. Unidades: FM, ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, DOENÇA DE ALZHEIMER, CAMUNDONGOS, IMUNOHISTOQUÍMICA, MODELOS ANIMAIS DE DOENÇAS, COMPORTAMENTO ANIMAL, ANSIEDADE
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ABNT
SINGULANI, Monique Patricio et al. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study. Alzheimer's & Dementia, v. 21, n. S1, p. 5 , 2024Tradução . . Disponível em: https://doi.org/10.1002/alz.087517. Acesso em: 21 out. 2025.
APA
Singulani, M. P., Talib, L. L., Forlenza, O. V., Britto, L. R. G. de, & Camarini, R. (2024). ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study. Alzheimer's & Dementia, 21( S1), 5 . doi:10.1002/alz.087517
NLM
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study [Internet]. Alzheimer's & Dementia. 2024 ; 21( S1): 5 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1002/alz.087517
Vancouver
Singulani MP, Talib LL, Forlenza OV, Britto LRG de, Camarini R. ‘Latent’ mild behavioral impairment as a potential diagnostic strategy for preclinical Alzheimer’s disease: an animal model study [Internet]. Alzheimer's & Dementia. 2024 ; 21( S1): 5 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1002/alz.087517
Artigo de periodico
Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease (2024)
Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Figueroa, Paulina Belén Sepulveda ; Cuyul-Vásquez, Iván; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de
Source: Ageing Research Reviews. Unidades: ICB, FM
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇA DE PARKINSON, LÍTIO, DOENÇAS NEURODEGENERATIVAS, REVISÃO SISTEMÁTICA
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ABNT
SINGULANI, Monique Patricio et al. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, v. 95, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2024.102231. Acesso em: 21 out. 2025.
APA
Singulani, M. P., Ferreira, A. F. F., Figueroa, P. B. S., Cuyul-Vásquez, I., Talib, L. L., Forlenza, O. V., & Britto, L. R. G. de. (2024). Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, 95, 20 . doi:10.1016/j.arr.2024.102231
NLM
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Vancouver
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Artigo de periodico
Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease (2023)
Pereira, Carolina Parga Martins; Oliveira, José Francis ; Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Journal of Neuroimmunology. Unidades: ICB, FM
Subjects: FISIOLOGIA, CAMUNDONGOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, NEURÓGLIA, TOXINAS, COORDENAÇÃO MOTORA, RECEPTORES DOPAMINÉRGICOS, NEURÔNIOS MOTORES
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ABNT
PEREIRA, Carolina Parga Martins et al. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, v. 375, p. 1-11, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.jneuroim.2023.578019. Acesso em: 21 out. 2025.
APA
Pereira, C. P. M., Oliveira, J. F., Singulani, M. P., Ferreira, A. F. F., & Britto, L. R. G. de. (2023). Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease. Journal of Neuroimmunology, 375, 1-11. doi:10.1016/j.jneuroim.2023.578019
NLM
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Vancouver
Pereira CPM, Oliveira JF, Singulani MP, Ferreira AFF, Britto LRG de. Microglial depletion exacerbates motor impairment and dopaminergic neuron loss in a 6-OHDA model of Parkinson's disease [Internet]. Journal of Neuroimmunology. 2023 ; 375 1-11.[citado 2025 out. 21 ] Available from: https://doi.org/10.1016/j.jneuroim.2023.578019
Artigo de periodico
Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions (2023)
Hamasaki, Mike Yoshio ; Mendes, Caroline; Batagello, Daniella Sabino ; Hirata, Mario Hiroyuki ; Britto, Luiz Roberto Giorgetti de ; Nogueira, Maria Inês
Source: NeuroReport. Unidades: FCF, ICB
Subjects: ANÓXIA, CÓRTEX CEREBRAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HAMASAKI, Mike Yoshio et al. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, v. 34, n. 11, p. 575-582, 2023Tradução . . Disponível em: https://doi.org/10.1097/WNR.0000000000001927. Acesso em: 21 out. 2025.
APA
Hamasaki, M. Y., Mendes, C., Batagello, D. S., Hirata, M. H., Britto, L. R. G. de, & Nogueira, M. I. (2023). Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions. NeuroReport, 34( 11), 575-582. doi:10.1097/WNR.0000000000001927
NLM
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2025 out. 21 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Vancouver
Hamasaki MY, Mendes C, Batagello DS, Hirata MH, Britto LRG de, Nogueira MI. Pathophysiological aspects of neonatal anoxia andtemporal expression of S100β in different brain regions [Internet]. NeuroReport. 2023 ; 34( 11): 575-582.[citado 2025 out. 21 ] Available from: https://doi.org/10.1097/WNR.0000000000001927
Artigo de periodico
The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? (2023)
Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Neural Regeneration Research. Unidade: ICB
Subjects: FISIOLOGIA, CANAIS IÔNICOS, ESTRESSE OXIDATIVO, DOENÇA DE PARKINSON, PROTEÍNAS DA MEMBRANA, DOENÇAS NEURODEGENERATIVAS, DOENÇAS NEUROMUSCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Ana Flávia Fernandes e BRITTO, Luiz Roberto Giorgetti de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease?. Neural Regeneration Research, v. 18, n. 8, p. 1652-1656, 2023Tradução . . Disponível em: https://doi.org/10.4103/1673-5374.360343. Acesso em: 21 out. 2025.
APA
Ferreira, A. F. F., & Britto, L. R. G. de. (2023). The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? Neural Regeneration Research, 18( 8), 1652-1656. doi:10.4103/1673-5374.360343
NLM
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2025 out. 21 ] Available from: https://doi.org/10.4103/1673-5374.360343
Vancouver
Ferreira AFF, Britto LRG de. The transient receptor potential melastatin 2: a new therapeutical target for Parkinson’s disease? [Internet]. Neural Regeneration Research. 2023 ; 18( 8): 1652-1656.[citado 2025 out. 21 ] Available from: https://doi.org/10.4103/1673-5374.360343
Parte de monografia/livro
Purinergic signaling in Parkinson’s disease (2023)
Silva, Jean Bezerra ; Ferreira, Ana Flávia Fernandes ; Glaser, Talita ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Unidades: IQ, ICB
Assunto: DOENÇA DE PARKINSON
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ABNT
SILVA, Jean Bezerra et al. Purinergic signaling in Parkinson’s disease. Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Tradução . Cham: Springer, 2023. . . Acesso em: 21 out. 2025.
APA
Silva, J. B., Ferreira, A. F. F., Glaser, T., Ulrich, H., & Britto, L. R. G. de. (2023). Purinergic signaling in Parkinson’s disease. In Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer.
NLM
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2025 out. 21 ]
Vancouver
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2025 out. 21 ]
Artigo de periodico
Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation (2023)
Yonamine, Caio Yogi ; Passarelli, Marisa ; Suemoto, Claudia Kimie ; Pasqualucci, Carlos Augusto ; Jacob-Filho, Wilson ; Alves, Venâncio Avancini Ferreira ; Marie, Suely Kazue Nagahashi ; Giannella, Maria Lucia Correa ; Britto, Luiz Roberto ; Machado, Ubiratan Fabres
Source: Cells. Unidades: FM, ICB
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DIABETES MELLITUS, HIPOCAMPO, NEUROBLASTOMA, OBESIDADE, DOENÇAS NEURODEGENERATIVAS, ESTRESSE OXIDATIVO, GLICOSE, DNA POLIMERASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
YONAMINE, Caio Yogi et al. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, v. 12, n. 9, p. 1-21, 2023Tradução . . Disponível em: https://doi.org/10.3390/cells12091250. Acesso em: 21 out. 2025.
APA
Yonamine, C. Y., Passarelli, M., Suemoto, C. K., Pasqualucci, C. A., Jacob-Filho, W., Alves, V. A. F., et al. (2023). Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation. Cells, 12( 9), 1-21. doi:10.3390/cells12091250
NLM
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/cells12091250
Vancouver
Yonamine CY, Passarelli M, Suemoto CK, Pasqualucci CA, Jacob-Filho W, Alves VAF, Marie SKN, Giannella MLC, Britto LR, Machado UF. Postmortem brains from subjects with Diabetes Mellitus display reduced GLUT4 expression and soma area in hippocampal neurons: potential involvement of inflammation [Internet]. Cells. 2023 ; 12( 9): 1-21.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/cells12091250
Artigo de periodico
Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease (2022)
Maglione, Andréa V. ; Nascimento, Bruna P. P. do ; Ribeiro, Miriam O. ; Souza, Talytha J. L. de ; Silva, Renata E. C. da; Sato, Monica A. ; Penatti, Carlos A. A.; Britto, Luiz Roberto Giorgetti de ; Souza, Janaina Sena de ; Maciel, Rui M. B. ; Conceição, Rodrigo Rodrigues da ; Laureano-Melo, Roberto ; Giannocco, Gisele
Source: Metabolic Brain Disease. Unidade: ICB
Subjects: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇAS NEURODEGENERATIVAS, HIPOCAMPU DE ANIMAL, DOENÇAS DO SISTEMA NERVOSO CENTRAL EM ANIMAL, MODELOS ANIMAIS DE DOENÇAS, TRIIODOTIRONINA, DEPRESSÃO, CAMUNDONGOS, COMPORTAMENTO ANIMAL, SUPERÓXIDO DISMUTASE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGLIONE, Andréa V. et al. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease. Metabolic Brain Disease, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1007/s11011-022-01055-9. Acesso em: 21 out. 2025.
APA
Maglione, A. V., Nascimento, B. P. P. do, Ribeiro, M. O., Souza, T. J. L. de, Silva, R. E. C. da, Sato, M. A., et al. (2022). Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease. Metabolic Brain Disease, 1-16. doi:10.1007/s11011-022-01055-9
NLM
Maglione AV, Nascimento BPP do, Ribeiro MO, Souza TJL de, Silva REC da, Sato MA, Penatti CAA, Britto LRG de, Souza JS de, Maciel RMB, Conceição RR da, Laureano-Melo R, Giannocco G. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease [Internet]. Metabolic Brain Disease. 2022 ; 1-16.[citado 2025 out. 21 ] Available from: https://doi.org/10.1007/s11011-022-01055-9
Vancouver
Maglione AV, Nascimento BPP do, Ribeiro MO, Souza TJL de, Silva REC da, Sato MA, Penatti CAA, Britto LRG de, Souza JS de, Maciel RMB, Conceição RR da, Laureano-Melo R, Giannocco G. Triiodothyronine treatment reverses Depression-Like behavior in a triple-transgenic animal model of Alzheimer’s Disease [Internet]. Metabolic Brain Disease. 2022 ; 1-16.[citado 2025 out. 21 ] Available from: https://doi.org/10.1007/s11011-022-01055-9
Artigo de periodico
The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model (2022)
Santamarina, Aline Boveto ; Moraes, Ruan Carlos Macêdo ; Nehmi Filho, Victor; Murata, Gilson Masahiro ; Freitas, Jéssica Alves de ; Miranda, Danielle Araujo de ; Cerqueira, Anderson Romério Azevedo ; Costa, Soraia Katia Pereira ; Ferreira, Ana Flávia Fernandes ; Britto, Luiz Roberto Giorgetti de
Source: Antioxidants. Unidades: FM, ICB
Subjects: FARMACOLOGIA, FISIOLOGIA, PRODUTOS NATURAIS, INTERLEUCINA 6, OBESIDADE, SUPLEMENTAÇÃO ALIMENTAR, DIABETES MELLITUS, MINERAIS NA DIETA, ENZIMAS OXIRREDUTORAS, ANTIOXIDANTES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANTAMARINA, Aline Boveto et al. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model. Antioxidants, v. 11, p. 1-24, 2022Tradução . . Disponível em: https://doi.org/10.3390/antiox11030447. Acesso em: 21 out. 2025.
APA
Santamarina, A. B., Moraes, R. C. M., Nehmi Filho, V., Murata, G. M., Freitas, J. A. de, Miranda, D. A. de, et al. (2022). The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model. Antioxidants, 11, 1-24. doi:10.3390/antiox11030447
NLM
Santamarina AB, Moraes RCM, Nehmi Filho V, Murata GM, Freitas JA de, Miranda DA de, Cerqueira ARA, Costa SKP, Ferreira AFF, Britto LRG de. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model [Internet]. Antioxidants. 2022 ; 11 1-24.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/antiox11030447
Vancouver
Santamarina AB, Moraes RCM, Nehmi Filho V, Murata GM, Freitas JA de, Miranda DA de, Cerqueira ARA, Costa SKP, Ferreira AFF, Britto LRG de. The symbiotic effect of a new nutraceutical with Yeast β-Glucan, Prebiotics, Minerals, and Silybum marianum (Silymarin) for recovering metabolic homeostasis via Pgc-1α, Il-6, and Il-10 gene expression in a Type-2 Diabetes Obesity model [Internet]. Antioxidants. 2022 ; 11 1-24.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/antiox11030447
Artigo de periodico
Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study (2022)
Faria, Daniele de Paula ; Souza, Larissa Estessi de ; Duran, Fabio Luis de Souza ; Buchpiguel, Carlos Alberto ; Britto, Luiz Roberto Giorgetti de ; Crippa, José Alexandre de Souza ; Busatto Filho, Geraldo ; Gregório, Caroline Cristiano Real
Source: International Journal of Molecular Sciences. Unidades: FM, ICB, FMRP
Subjects: FISIOLOGIA, CANABINOIDES, DOENÇA DE ALZHEIMER, MODELOS ANIMAIS DE DOENÇAS, RATOS WISTAR, ANTINEOPLÁSICOS, MEMÓRIA ANIMAL, GLICOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FARIA, Daniele de Paula et al. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, v. 23. n. 3, p. 1-10, 2022Tradução . . Disponível em: https://doi.org/10.3390/ijms23031076. Acesso em: 21 out. 2025.
APA
Faria, D. de P., Souza, L. E. de, Duran, F. L. de S., Buchpiguel, C. A., Britto, L. R. G. de, Crippa, J. A. de S., et al. (2022). Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study. International Journal of Molecular Sciences, 23. n. 3, 1-10. doi:10.3390/ijms23031076
NLM
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/ijms23031076
Vancouver
Faria D de P, Souza LE de, Duran FL de S, Buchpiguel CA, Britto LRG de, Crippa JA de S, Busatto Filho G, Gregório CCR. Cannabidiol treatment improves glucose metabolism and memory in sreptozotocin-induced Alzheimer’s disease rat model: a proof-of-concept study [Internet]. International Journal of Molecular Sciences. 2022 ; 23. n. 3 1-10.[citado 2025 out. 21 ] Available from: https://doi.org/10.3390/ijms23031076

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