Filtros : "BRENTANI, RICARDO RENZO" "Molecular Brain Research" Limpar

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  • Source: Molecular Brain Research. Unidade: FM

    Subjects: CAMUNDONGOS, PROTEÍNAS, HIPOCAMPU DE ANIMAL, ENCEFALITE (COMPLICAÇÕES)

    Acesso à fonteDOIHow to cite
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    • ABNT

      MAGLIO, Laura E. et al. Hippocampal synaptic plasticity in mice devoid of cellular prion protein. Molecular Brain Research, v. 131, p. 58-64, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.molbrainres.2004.08.004. Acesso em: 23 ago. 2024.
    • APA

      Maglio, L. E., Perez, M. F., Martins, V. R., Brentani, R. R., & Ramirez, O. A. (2004). Hippocampal synaptic plasticity in mice devoid of cellular prion protein. Molecular Brain Research, 131, 58-64. doi:10.1016/j.molbrainres.2004.08.004
    • NLM

      Maglio LE, Perez MF, Martins VR, Brentani RR, Ramirez OA. Hippocampal synaptic plasticity in mice devoid of cellular prion protein [Internet]. Molecular Brain Research. 2004 ; 131 58-64.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/j.molbrainres.2004.08.004
    • Vancouver

      Maglio LE, Perez MF, Martins VR, Brentani RR, Ramirez OA. Hippocampal synaptic plasticity in mice devoid of cellular prion protein [Internet]. Molecular Brain Research. 2004 ; 131 58-64.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/j.molbrainres.2004.08.004
  • Source: Molecular Brain Research. Unidade: FM

    Assunto: RADIOLOGIA

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    • ABNT

      COITINHO, Adriana S. et al. Decreased hyperlocomotion induced by MK-801, but not amphetamine and caffeine in mice lacking cellular prion protein (PrPc). Molecular Brain Research, v. 107, p. 190-194, 2002Tradução . . Disponível em: https://doi.org/10.1016/s0169-328x(02)00526-0. Acesso em: 23 ago. 2024.
    • APA

      Coitinho, A. S., Dietrich, M. O., Hoffmann, A., Dall'Igna, O., Souza, D. O., Martins, V. R., et al. (2002). Decreased hyperlocomotion induced by MK-801, but not amphetamine and caffeine in mice lacking cellular prion protein (PrPc). Molecular Brain Research, 107, 190-194. doi:10.1016/s0169-328x(02)00526-0
    • NLM

      Coitinho AS, Dietrich MO, Hoffmann A, Dall'Igna O, Souza DO, Martins VR, Brentani RR, Izquierdo I, Lara DR. Decreased hyperlocomotion induced by MK-801, but not amphetamine and caffeine in mice lacking cellular prion protein (PrPc) [Internet]. Molecular Brain Research. 2002 ; 107 190-194.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(02)00526-0
    • Vancouver

      Coitinho AS, Dietrich MO, Hoffmann A, Dall'Igna O, Souza DO, Martins VR, Brentani RR, Izquierdo I, Lara DR. Decreased hyperlocomotion induced by MK-801, but not amphetamine and caffeine in mice lacking cellular prion protein (PrPc) [Internet]. Molecular Brain Research. 2002 ; 107 190-194.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(02)00526-0
  • Source: Molecular Brain Research. Unidade: FM

    Assunto: RADIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      GRANER, Edgard et al. Cellular prion protein binds laminin and mediates neuritogenesish. Molecular Brain Research, v. 76, p. 85-92, 2000Tradução . . Disponível em: https://doi.org/10.1016/s0169-328x(99)00334-4. Acesso em: 23 ago. 2024.
    • APA

      Graner, E., Mercadante, A. F., Zanata, S. M., Forlenza, O. V., Cabral, A. L. B., Veiga, S. S., et al. (2000). Cellular prion protein binds laminin and mediates neuritogenesish. Molecular Brain Research, 76, 85-92. doi:10.1016/s0169-328x(99)00334-4
    • NLM

      Graner E, Mercadante AF, Zanata SM, Forlenza OV, Cabral ALB, Veiga SS, Juliano MA, Roesler R, Walz R, Minetti A, Izquierdo I, Martins VR, Brentani RR. Cellular prion protein binds laminin and mediates neuritogenesish [Internet]. Molecular Brain Research. 2000 ; 76 85-92.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(99)00334-4
    • Vancouver

      Graner E, Mercadante AF, Zanata SM, Forlenza OV, Cabral ALB, Veiga SS, Juliano MA, Roesler R, Walz R, Minetti A, Izquierdo I, Martins VR, Brentani RR. Cellular prion protein binds laminin and mediates neuritogenesish [Internet]. Molecular Brain Research. 2000 ; 76 85-92.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(99)00334-4
  • Source: Molecular Brain Research. Unidade: FM

    Assunto: RADIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      ROESLER, Rafael et al. Normal inhibitory avoidance learning and anxiety, but increased locomotor activity in mice devoid of PrP. Molecular Brain Research, v. 71, p. 349-53, 1999Tradução . . Disponível em: https://doi.org/10.1016/s0169-328x(99)00193-x. Acesso em: 23 ago. 2024.
    • APA

      Roesler, R., Walz, R., Quevedo, J., Paris, F. de, Zanata, S. M., Graner, E., et al. (1999). Normal inhibitory avoidance learning and anxiety, but increased locomotor activity in mice devoid of PrP. Molecular Brain Research, 71, 349-53. doi:10.1016/s0169-328x(99)00193-x
    • NLM

      Roesler R, Walz R, Quevedo J, Paris F de, Zanata SM, Graner E, Izquierdo I, Martins VR, Brentani RR. Normal inhibitory avoidance learning and anxiety, but increased locomotor activity in mice devoid of PrP [Internet]. Molecular Brain Research. 1999 ; 71 349-53.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(99)00193-x
    • Vancouver

      Roesler R, Walz R, Quevedo J, Paris F de, Zanata SM, Graner E, Izquierdo I, Martins VR, Brentani RR. Normal inhibitory avoidance learning and anxiety, but increased locomotor activity in mice devoid of PrP [Internet]. Molecular Brain Research. 1999 ; 71 349-53.[citado 2024 ago. 23 ] Available from: https://doi.org/10.1016/s0169-328x(99)00193-x

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