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Artigo de periodico
Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms (2024)
Glaser, Talita ; Martins, Patrícia; Beco, Renata; Bento, Carolina Adriane ; Cappellari, Angelica R; Oliveira, Sophia La Banca de; Merkel, Christian A; Sampaio, Vanessa Fernandes Arnaud ; Lameu, Claudiana ; Battastini, Ana Maria ; Ulrich, Henning
Fonte: Frontiers in Pharmacology. Unidades: FM, IQ
Assuntos: ADENOSINA, CÉLULAS-TRONCO, CAFEÍNA
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GLASER, Talita et al. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms. Frontiers in Pharmacology, v. 14, p. 1-16 art. 1328398, 2024Tradução . . Disponível em: https://dx.doi.org/10.3389/fphar.2023.1328398. Acesso em: 18 jun. 2024.
APA
Glaser, T., Martins, P., Beco, R., Bento, C. A., Cappellari, A. R., Oliveira, S. L. B. de, et al. (2024). Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms. Frontiers in Pharmacology, 14, 1-16 art. 1328398. doi:10.3389/fphar.2023.1328398
NLM
Glaser T, Martins P, Beco R, Bento CA, Cappellari AR, Oliveira SLB de, Merkel CA, Sampaio VFA, Lameu C, Battastini AM, Ulrich H. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms [Internet]. Frontiers in Pharmacology. 2024 ; 14 1-16 art. 1328398.[citado 2024 jun. 18 ] Available from: https://dx.doi.org/10.3389/fphar.2023.1328398
Vancouver
Glaser T, Martins P, Beco R, Bento CA, Cappellari AR, Oliveira SLB de, Merkel CA, Sampaio VFA, Lameu C, Battastini AM, Ulrich H. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms [Internet]. Frontiers in Pharmacology. 2024 ; 14 1-16 art. 1328398.[citado 2024 jun. 18 ] Available from: https://dx.doi.org/10.3389/fphar.2023.1328398
Artigo de periodico-resenha
Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats (2024)
Ferreira, Julio Cesar Batista ; Campos, Juliane Cruz ; Qvit, Nir ; Qi, Xin; Bozi, Luiz Henrique Marchesi ; Bechara, Luiz Roberto Grassmann ; Lima, Vanessa Morais ; Queliconi, Bruno Barros ; Disatnik, Marie-Helene; Dourado, Paulo Magno Martins ; Kowaltowski, Alicia Juliana ; Rosen, Daria Mochly
Fonte: Nature Communications. Unidades: ICB, FM, IQ
Assuntos: ANATOMIA, INSUFICIÊNCIA CARDÍACA ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, ORGANELAS CELULARES, MITOCÔNDRIAS
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FERREIRA, Julio Cesar Batista et al. Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats. Nature Communications. London: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1038/s41467-018-08276-6,. Acesso em: 18 jun. 2024. , 2024
APA
Ferreira, J. C. B., Campos, J. C., Qvit, N., Qi, X., Bozi, L. H. M., Bechara, L. R. G., et al. (2024). Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats. Nature Communications. London: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:https://doi.org/10.1038/s41467-024-47288-3
NLM
Ferreira JCB, Campos JC, Qvit N, Qi X, Bozi LHM, Bechara LRG, Lima VM, Queliconi BB, Disatnik M-H, Dourado PMM, Kowaltowski AJ, Rosen DM. Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats [Internet]. Nature Communications. 2024 ; 15 12 .[citado 2024 jun. 18 ] Available from: https://doi.org/10.1038/s41467-018-08276-6,
Vancouver
Ferreira JCB, Campos JC, Qvit N, Qi X, Bozi LHM, Bechara LRG, Lima VM, Queliconi BB, Disatnik M-H, Dourado PMM, Kowaltowski AJ, Rosen DM. Author Correction: A selective inhibitor of mitofusin 1-βIIPKC association improves heart failure outcome in rats [Internet]. Nature Communications. 2024 ; 15 12 .[citado 2024 jun. 18 ] Available from: https://doi.org/10.1038/s41467-018-08276-6,
Artigo de periodico
DNA methylation of the promoter region at the CREB1 binding site is a mechanism for the epigenetic regulation of brain-specific PKM zeta (2023)
Pramio, Dimitrius Tansini ; Vieceli, Felipe Monteleone ; Branco, Elisa Varella ; Goes, Carolina Purcell ; Kobayashi, Gerson Shigeru ; Pelegrina, Diogo Vieira da Silva; Moraes, Beatriz Caroline de ; Allam, Aicha El ; Kumar, Bony De ; Jara, Gabriel ; Farfel, José Marcelo ; Reis, Eduardo Moraes ; Passos-Bueno, Maria Rita ; Schechtman, Deborah
Fonte: Biochimica et biophysica acta-gene regulatory mechanisms. Unidades: IQ, IB, FM, ESALQ
Assuntos: EPIGÊNESE GENÉTICA, PROTEÍNAS QUINASES, DOENÇA DE ALZHEIMER, METILAÇÃO DE DNA
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PRAMIO, Dimitrius Tansini et al. DNA methylation of the promoter region at the CREB1 binding site is a mechanism for the epigenetic regulation of brain-specific PKM zeta. Biochimica et biophysica acta-gene regulatory mechanisms, v. 1866, n. 1, 2023Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/54748. Acesso em: 18 jun. 2024.
APA
Pramio, D. T., Vieceli, F. M., Branco, E. V., Goes, C. P., Kobayashi, G. S., Pelegrina, D. V. da S., et al. (2023). DNA methylation of the promoter region at the CREB1 binding site is a mechanism for the epigenetic regulation of brain-specific PKM zeta. Biochimica et biophysica acta-gene regulatory mechanisms, 1866( 1). doi:10.1016/j.bbagrm.2023.194909
NLM
Pramio DT, Vieceli FM, Branco EV, Goes CP, Kobayashi GS, Pelegrina DV da S, Moraes BC de, Allam AE, Kumar BD, Jara G, Farfel JM, Reis EM, Passos-Bueno MR, Schechtman D. DNA methylation of the promoter region at the CREB1 binding site is a mechanism for the epigenetic regulation of brain-specific PKM zeta [Internet]. Biochimica et biophysica acta-gene regulatory mechanisms. 2023 ; 1866( 1):[citado 2024 jun. 18 ] Available from: https://observatorio.fm.usp.br/handle/OPI/54748
Vancouver
Pramio DT, Vieceli FM, Branco EV, Goes CP, Kobayashi GS, Pelegrina DV da S, Moraes BC de, Allam AE, Kumar BD, Jara G, Farfel JM, Reis EM, Passos-Bueno MR, Schechtman D. DNA methylation of the promoter region at the CREB1 binding site is a mechanism for the epigenetic regulation of brain-specific PKM zeta [Internet]. Biochimica et biophysica acta-gene regulatory mechanisms. 2023 ; 1866( 1):[citado 2024 jun. 18 ] Available from: https://observatorio.fm.usp.br/handle/OPI/54748
Trabalho de evento-resumo periodico
Protein Disulfide Isomerase overexpression induces mitochondrial reorganization and a differentiated VSMC phenotype: role of mitofusin-2 (2023)
Wosniak Junior, João; Rodrigues, Rafael Felipe Gonçalves ; Kakimoto, Pâmela Aiako Hypólito Brito ; Silva, Camille Cristine Caldeira da ; Antunes, Fernanda ; Strauss, Bryan Eric ; Kowaltowski, Alicia Juliana ; Laurindo, Francisco Rafael Martins
Fonte: Free Radical Biology & Medicine. Nome do evento: Biennial Congress of the Society for Free Radical Research International/SFRRI. Unidades: FM, IQ
Assuntos: PROTEÍNAS, MITOCÔNDRIAS
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WOSNIAK JUNIOR, João et al. Protein Disulfide Isomerase overexpression induces mitochondrial reorganization and a differentiated VSMC phenotype: role of mitofusin-2. Free Radical Biology & Medicine. New York: Faculdade de Medicina, Universidade de São Paulo. Disponível em: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1. Acesso em: 18 jun. 2024. , 2023
APA
Wosniak Junior, J., Rodrigues, R. F. G., Kakimoto, P. A. H. B., Silva, C. C. C. da, Antunes, F., Strauss, B. E., et al. (2023). Protein Disulfide Isomerase overexpression induces mitochondrial reorganization and a differentiated VSMC phenotype: role of mitofusin-2. Free Radical Biology & Medicine. New York: Faculdade de Medicina, Universidade de São Paulo. Recuperado de https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
NLM
Wosniak Junior J, Rodrigues RFG, Kakimoto PAHB, Silva CCC da, Antunes F, Strauss BE, Kowaltowski AJ, Laurindo FRM. Protein Disulfide Isomerase overexpression induces mitochondrial reorganization and a differentiated VSMC phenotype: role of mitofusin-2 [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S98 res. 207.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Vancouver
Wosniak Junior J, Rodrigues RFG, Kakimoto PAHB, Silva CCC da, Antunes F, Strauss BE, Kowaltowski AJ, Laurindo FRM. Protein Disulfide Isomerase overexpression induces mitochondrial reorganization and a differentiated VSMC phenotype: role of mitofusin-2 [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S98 res. 207.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Artigo de periodico
Immunodominant antibody responses directed to SARS-CoV-2 hotspot mutation sites and risk of immune escape (2023)
Oliveira, Jamille Ramos ; Ruiz, Cesar Manuel Remuzgo ; Machado, Rafael Rahal Guaragna ; Urbanski, Alysson Henrique ; Magawa, Jhosiene Yukari; Daher, Isabela Pazotti; Urbanski, Alysson Henrique ; Schmitz, Gabriela Justamante Händel ; Arcuri, Helen Andrade; Ferreira, Marcelo Alves ; Sasahara, Greyce Luri ; Medeiros, Giuliana Xavier de ; Silva Júnior, Roberto Carlos Vieira da ; Rosa, Daniela Santoro ; Nakaya, Helder Takashi Imoto ; Gadermaier, Gabriele; Coelho, Verônica ; Santos, Keity Souza ; Durigon, Edison Luiz ; Boscardin, Silvia Beatriz ; Schechtman, Deborah ; Cunha Neto, Edecio; Kalil Filho, Jorge Elias ; Coelho, Veronica Porto Carreiro de Vasconcellos
Fonte: Frontiers in Immunology. Unidades: ICB, IQ, FM, FCF
Assuntos: MICROBIOLOGIA, PARASITOLOGIA, ANTICORPOS, COVID-19, VACINAS VIRAIS, EPITOPOS, IMUNOGLOBULINAS, VARIAÇÃO GENÉTICA, MUTAÇÃO GENÉTICA, PEPTÍDEOS
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OLIVEIRA, Jamille Ramos et al. Immunodominant antibody responses directed to SARS-CoV-2 hotspot mutation sites and risk of immune escape. Frontiers in Immunology, v. 13, p. 1-15, 2023Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.1010105. Acesso em: 18 jun. 2024.
APA
Oliveira, J. R., Ruiz, C. M. R., Machado, R. R. G., Urbanski, A. H., Magawa, J. Y., Daher, I. P., et al. (2023). Immunodominant antibody responses directed to SARS-CoV-2 hotspot mutation sites and risk of immune escape. Frontiers in Immunology, 13, 1-15. doi:10.3389/fimmu.2022.1010105
NLM
Oliveira JR, Ruiz CMR, Machado RRG, Urbanski AH, Magawa JY, Daher IP, Urbanski AH, Schmitz GJH, Arcuri HA, Ferreira MA, Sasahara GL, Medeiros GX de, Silva Júnior RCV da, Rosa DS, Nakaya HTI, Gadermaier G, Coelho V, Santos KS, Durigon EL, Boscardin SB, Schechtman D, Cunha Neto E, Kalil Filho JE, Coelho VPC de V. Immunodominant antibody responses directed to SARS-CoV-2 hotspot mutation sites and risk of immune escape [Internet]. Frontiers in Immunology. 2023 ; 13 1-15.[citado 2024 jun. 18 ] Available from: https://doi.org/10.3389/fimmu.2022.1010105
Vancouver
Oliveira JR, Ruiz CMR, Machado RRG, Urbanski AH, Magawa JY, Daher IP, Urbanski AH, Schmitz GJH, Arcuri HA, Ferreira MA, Sasahara GL, Medeiros GX de, Silva Júnior RCV da, Rosa DS, Nakaya HTI, Gadermaier G, Coelho V, Santos KS, Durigon EL, Boscardin SB, Schechtman D, Cunha Neto E, Kalil Filho JE, Coelho VPC de V. Immunodominant antibody responses directed to SARS-CoV-2 hotspot mutation sites and risk of immune escape [Internet]. Frontiers in Immunology. 2023 ; 13 1-15.[citado 2024 jun. 18 ] Available from: https://doi.org/10.3389/fimmu.2022.1010105
Artigo de periodico
Novel Gadolinium-Free Ultrasmall nanostructured positive contrast for magnetic resonance angiography and imaging (2023)
Kawassaki, Rodrigo Ken ; Romano, Mariana ; Uchiyama, Mayara Klimuk ; Cardoso, Roberta Mansini ; Baptista, Maurício da Silva ; Farsky, Sandra Helena Poliselli ; Chaim, Khallil Taverna ; Guimarães, Robson Raphael; Araki, Koiti
Fonte: Nano Letters. Unidades: IQ, FCF, FM
Assuntos: RESSONÂNCIA MAGNÉTICA, NANOPARTÍCULAS
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KAWASSAKI, Rodrigo Ken et al. Novel Gadolinium-Free Ultrasmall nanostructured positive contrast for magnetic resonance angiography and imaging. Nano Letters, v. 23, p. 5497-5505, 2023Tradução . . Disponível em: https://doi.org/10.1021/acs.nanolett.3c00665. Acesso em: 18 jun. 2024.
APA
Kawassaki, R. K., Romano, M., Uchiyama, M. K., Cardoso, R. M., Baptista, M. da S., Farsky, S. H. P., et al. (2023). Novel Gadolinium-Free Ultrasmall nanostructured positive contrast for magnetic resonance angiography and imaging. Nano Letters, 23, 5497-5505. doi:10.1021/acs.nanolett.3c00665
NLM
Kawassaki RK, Romano M, Uchiyama MK, Cardoso RM, Baptista M da S, Farsky SHP, Chaim KT, Guimarães RR, Araki K. Novel Gadolinium-Free Ultrasmall nanostructured positive contrast for magnetic resonance angiography and imaging [Internet]. Nano Letters. 2023 ; 23 5497-5505.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1021/acs.nanolett.3c00665
Vancouver
Kawassaki RK, Romano M, Uchiyama MK, Cardoso RM, Baptista M da S, Farsky SHP, Chaim KT, Guimarães RR, Araki K. Novel Gadolinium-Free Ultrasmall nanostructured positive contrast for magnetic resonance angiography and imaging [Internet]. Nano Letters. 2023 ; 23 5497-5505.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1021/acs.nanolett.3c00665
Artigo de periodico
Mitochondrial dysfunction mediates neuronal cell response to DMMB photodynamic therapy (2023)
Gomes, Raphael de Ávila Narciso; Garza, Alejandro Marmolejo; Haan, Floris-Jan; García, Teresa Mitchell ; Chen, Tingting; Mauthe, Mario; Franco, Yollanda Edwirges Moreira ; Murakami Junior, Mario Minor; Marie, Suely Kazue Nagahashi ; Baptista, Maurício da Silva ; Dolga, Amalia M; Lima, Marina Trombetta
Fonte: BBA - Molecular Cell Research. Unidades: FM, IQ
Assuntos: TERAPIA FOTODINÂMICA, MITOCÔNDRIAS, SISTEMA NERVOSO CENTRAL
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GOMES, Raphael de Ávila Narciso et al. Mitochondrial dysfunction mediates neuronal cell response to DMMB photodynamic therapy. BBA - Molecular Cell Research, v. 1870, p. 1-10 art. 119429, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.bbamcr.2022.119429. Acesso em: 18 jun. 2024.
APA
Gomes, R. de Á. N., Garza, A. M., Haan, F. -J., García, T. M., Chen, T., Mauthe, M., et al. (2023). Mitochondrial dysfunction mediates neuronal cell response to DMMB photodynamic therapy. BBA - Molecular Cell Research, 1870, 1-10 art. 119429. doi:10.1016/j.bbamcr.2022.119429
NLM
Gomes R de ÁN, Garza AM, Haan F-J, García TM, Chen T, Mauthe M, Franco YEM, Murakami Junior MM, Marie SKN, Baptista M da S, Dolga AM, Lima MT. Mitochondrial dysfunction mediates neuronal cell response to DMMB photodynamic therapy [Internet]. BBA - Molecular Cell Research. 2023 ; 1870 1-10 art. 119429.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1016/j.bbamcr.2022.119429
Vancouver
Gomes R de ÁN, Garza AM, Haan F-J, García TM, Chen T, Mauthe M, Franco YEM, Murakami Junior MM, Marie SKN, Baptista M da S, Dolga AM, Lima MT. Mitochondrial dysfunction mediates neuronal cell response to DMMB photodynamic therapy [Internet]. BBA - Molecular Cell Research. 2023 ; 1870 1-10 art. 119429.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1016/j.bbamcr.2022.119429
Artigo de periodico
Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time (2023)
Leite, Giuseppe Gianini Figueiredo ; Brunialti, Milena Karina Colo ; Pietrobom, Paula M. Peçanha ; Ferreira, Paulo Roberto Abrão ; Arakaki, Jaquelina Sonoe Ota ; Cunha Neto, Edecio ; Ferreira, Bianca Lima ; Ronsein, Graziella Eliza ; Tashima, Alexandre Keiji ; Salomão, Reinaldo
Fonte: iScience. Unidades: FM, IQ
Assuntos: COVID-19, PROTEÔMICA
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LEITE, Giuseppe Gianini Figueiredo et al. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time. iScience, v. 26, n. 10, p. 1-16, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.107824. Acesso em: 18 jun. 2024.
APA
Leite, G. G. F., Brunialti, M. K. C., Pietrobom, P. M. P., Ferreira, P. R. A., Arakaki, J. S. O., Cunha Neto, E., et al. (2023). Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time. iScience, 26( 10), 1-16. doi:10.1016/j.isci.2023.107824
NLM
Leite GGF, Brunialti MKC, Pietrobom PMP, Ferreira PRA, Arakaki JSO, Cunha Neto E, Ferreira BL, Ronsein GE, Tashima AK, Salomão R. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time [Internet]. iScience. 2023 ; 26( 10): 1-16.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.107824
Vancouver
Leite GGF, Brunialti MKC, Pietrobom PMP, Ferreira PRA, Arakaki JSO, Cunha Neto E, Ferreira BL, Ronsein GE, Tashima AK, Salomão R. Understanding COVID-19 progression with longitudinal peripheral blood mononuclear cell proteomics: changes in the cellular proteome over time [Internet]. iScience. 2023 ; 26( 10): 1-16.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.107824
Trabalho de evento-resumo periodico
Mitochondrial calcium dynamics and bioenergetics in Autosomal Dominant Polycystic Kidney Disease: more pieces to the puzzle (2023)
Amaral, Andressa Godoy ; Serna, Julian David Cualcialpud ; Silva, Camille Cristine Caldeira da ; Costa, Eliene; Onuchic, Luiz Fernando ; Kowaltowski, Alicia Juliana
Fonte: Free Radical Biology & Medicine. Nome do evento: Biennial Congress of the Society for Free Radical Research International/SFRRI. Unidades: ICB, FM, IQ
Assuntos: MITOCÔNDRIAS, NEFROPATIAS
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ABNT
AMARAL, Andressa Godoy et al. Mitochondrial calcium dynamics and bioenergetics in Autosomal Dominant Polycystic Kidney Disease: more pieces to the puzzle. Free Radical Biology & Medicine. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1. Acesso em: 18 jun. 2024. , 2023
APA
Amaral, A. G., Serna, J. D. C., Silva, C. C. C. da, Costa, E., Onuchic, L. F., & Kowaltowski, A. J. (2023). Mitochondrial calcium dynamics and bioenergetics in Autosomal Dominant Polycystic Kidney Disease: more pieces to the puzzle. Free Radical Biology & Medicine. New York: Instituto de Ciências Biomédicas, Universidade de São Paulo. Recuperado de https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
NLM
Amaral AG, Serna JDC, Silva CCC da, Costa E, Onuchic LF, Kowaltowski AJ. Mitochondrial calcium dynamics and bioenergetics in Autosomal Dominant Polycystic Kidney Disease: more pieces to the puzzle [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S62 res. 125.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Vancouver
Amaral AG, Serna JDC, Silva CCC da, Costa E, Onuchic LF, Kowaltowski AJ. Mitochondrial calcium dynamics and bioenergetics in Autosomal Dominant Polycystic Kidney Disease: more pieces to the puzzle [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S62 res. 125.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Trabalho de evento-resumo
Metabolismo oxidativo do ácido úrico e a relação com a progressão da sepse (2023)
Alves, Priscilla D'Avila Mazzuco ; Silva, Railmara Pereira da ; Meotti, Flavia Carla
Fonte: Resumos. Nome do evento: Simpósio Internacional de Iniciação Científica e Tecnológica da USP/SIICUSP. Unidades: IQ, FM
Assuntos: ÁCIDO ÚRICO, SEPSE
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ALVES, Priscilla D'Avila Mazzuco e SILVA, Railmara Pereira da e MEOTTI, Flavia Carla. Metabolismo oxidativo do ácido úrico e a relação com a progressão da sepse. 2023, Anais.. São Paulo: Pró-Reitoria de Pesquisa da USP, 2023. Disponível em: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210. Acesso em: 18 jun. 2024.
APA
Alves, P. D. 'A. M., Silva, R. P. da, & Meotti, F. C. (2023). Metabolismo oxidativo do ácido úrico e a relação com a progressão da sepse. In Resumos. São Paulo: Pró-Reitoria de Pesquisa da USP. Recuperado de https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
NLM
Alves PD'AM, Silva RP da, Meotti FC. Metabolismo oxidativo do ácido úrico e a relação com a progressão da sepse [Internet]. Resumos. 2023 ;[citado 2024 jun. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Vancouver
Alves PD'AM, Silva RP da, Meotti FC. Metabolismo oxidativo do ácido úrico e a relação com a progressão da sepse [Internet]. Resumos. 2023 ;[citado 2024 jun. 18 ] Available from: https://uspdigital.usp.br/siicusp/siicPublicacao.jsp?codmnu=7210
Artigo de periodico
Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display (2023)
Carnero, Luis Antonio Rodriguez ; Kuramoto, Andréia; Oliveira, Lea Campos de; Monteiro, Jhonatas Sirino ; Setubal, João Carlos ; Cunha Neto, Edecio ; Sabino, Ester Cerdeira ; Giordano, Ricardo José
Fonte: Plos Neglected Tropical Diseases. Unidades: IQ, FM
Assuntos: DOENÇA DE CHAGAS, GENOMAS, ANTICORPOS, TRYPANOSOMA CRUZI
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ABNT
CARNERO, Luis Antonio Rodriguez et al. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display. Plos Neglected Tropical Diseases, v. 17, n. 1, p. 1-20 art. e0011019, 2023Tradução . . Disponível em: https://doi.org/10.1371/journal.pntd.0011019. Acesso em: 18 jun. 2024.
APA
Carnero, L. A. R., Kuramoto, A., Oliveira, L. C. de, Monteiro, J. S., Setubal, J. C., Cunha Neto, E., et al. (2023). Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display. Plos Neglected Tropical Diseases, 17( 1), 1-20 art. e0011019. doi:10.1371/journal.pntd.0011019
NLM
Carnero LAR, Kuramoto A, Oliveira LC de, Monteiro JS, Setubal JC, Cunha Neto E, Sabino EC, Giordano RJ. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display [Internet]. Plos Neglected Tropical Diseases. 2023 ; 17( 1): 1-20 art. e0011019.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1371/journal.pntd.0011019
Vancouver
Carnero LAR, Kuramoto A, Oliveira LC de, Monteiro JS, Setubal JC, Cunha Neto E, Sabino EC, Giordano RJ. Anti-Trypanosoma cruzi antibody profiling in patients with Chagas disease treated with benznidazole assessed by genome phage display [Internet]. Plos Neglected Tropical Diseases. 2023 ; 17( 1): 1-20 art. e0011019.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1371/journal.pntd.0011019
Artigo de periodico
GBM cells Exhibit susceptibility to metformin treatment according to TLR4 pathway activation and metabolic and antioxidant status (2023)
Moretti, Isabele Fattori ; Lerario, Antonio Marcondes ; Sola, Paula Rodrigues ; Silva, Janaína Macedo da; Baptista, Mauricio da Silva ; Palmisano, Giuseppe ; Shinjo, Sueli Mieko Oba ; Marie, Suely Kazue Nagahashi
Fonte: Cancers. Unidades: FM, IQ, ICB
Assuntos: PARASITOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS CEREBRAIS, GLIOMA, LIPOPOLISSACARÍDEOS, ANTIOXIDANTES, ANTIDIABÉTICOS (HIPOGLICEMIANTES), ESTRESSE OXIDATIVO, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORETTI, Isabele Fattori et al. GBM cells Exhibit susceptibility to metformin treatment according to TLR4 pathway activation and metabolic and antioxidant status. Cancers, v. 15, n. 3, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.3390/cancers15030587. Acesso em: 18 jun. 2024.
APA
Moretti, I. F., Lerario, A. M., Sola, P. R., Silva, J. M. da, Baptista, M. da S., Palmisano, G., et al. (2023). GBM cells Exhibit susceptibility to metformin treatment according to TLR4 pathway activation and metabolic and antioxidant status. Cancers, 15( 3), 1-18. doi:10.3390/cancers15030587
NLM
Moretti IF, Lerario AM, Sola PR, Silva JM da, Baptista M da S, Palmisano G, Shinjo SMO, Marie SKN. GBM cells Exhibit susceptibility to metformin treatment according to TLR4 pathway activation and metabolic and antioxidant status [Internet]. Cancers. 2023 ; 15( 3): 1-18.[citado 2024 jun. 18 ] Available from: https://doi.org/10.3390/cancers15030587
Vancouver
Moretti IF, Lerario AM, Sola PR, Silva JM da, Baptista M da S, Palmisano G, Shinjo SMO, Marie SKN. GBM cells Exhibit susceptibility to metformin treatment according to TLR4 pathway activation and metabolic and antioxidant status [Internet]. Cancers. 2023 ; 15( 3): 1-18.[citado 2024 jun. 18 ] Available from: https://doi.org/10.3390/cancers15030587
Artigo de periodico
COVID-19 induces more pronounced extracellular matrix deposition than other causes of ARDS (2023)
Costa, Natália de Souza Xavier ; Ribeiro Júnior, Gabriel ; Nascimento, Ellen Caroline Toledo do; Brito, Jôse Mára de; Antonangelo, Leila ; Faria, Caroline Silverio; Monteiro, Jhonatas Sirino ; Setubal, João Carlos ; Pinho, Joao Renato Rebello ; Pereira, Roberta Verciano; Seelaender, Marília Cerqueira ; Castro, Gabriela Salim Fereira de ; Lima, Joanna D. C. C; Monteiro, Renata Aparecida de Almeida; Duarte Neto, Amaro Nunes ; Saldiva, Paulo Hilário Nascimento ; Silva, Luiz Fernando Ferraz da ; Dolhnikoff, Marisa ; Mauad, Thais
Fonte: Respiratory Research. Unidades: FM, IQ, CEBIMAR
Assuntos: COVID-19, FIBROSE PULMONAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Natália de Souza Xavier et al. COVID-19 induces more pronounced extracellular matrix deposition than other causes of ARDS. Respiratory Research, v. 24, p. 1-11 art. 281, 2023Tradução . . Disponível em: https://dx.doi.org/10.1186/s12931-023-02555-7. Acesso em: 18 jun. 2024.
APA
Costa, N. de S. X., Ribeiro Júnior, G., Nascimento, E. C. T. do, Brito, J. M. de, Antonangelo, L., Faria, C. S., et al. (2023). COVID-19 induces more pronounced extracellular matrix deposition than other causes of ARDS. Respiratory Research, 24, 1-11 art. 281. doi:10.1186/s12931-023-02555-7
NLM
Costa N de SX, Ribeiro Júnior G, Nascimento ECT do, Brito JM de, Antonangelo L, Faria CS, Monteiro JS, Setubal JC, Pinho JRR, Pereira RV, Seelaender MC, Castro GSF de, Lima JDCC, Monteiro RA de A, Duarte Neto AN, Saldiva PHN, Silva LFF da, Dolhnikoff M, Mauad T. COVID-19 induces more pronounced extracellular matrix deposition than other causes of ARDS [Internet]. Respiratory Research. 2023 ; 24 1-11 art. 281.[citado 2024 jun. 18 ] Available from: https://dx.doi.org/10.1186/s12931-023-02555-7
Vancouver
Costa N de SX, Ribeiro Júnior G, Nascimento ECT do, Brito JM de, Antonangelo L, Faria CS, Monteiro JS, Setubal JC, Pinho JRR, Pereira RV, Seelaender MC, Castro GSF de, Lima JDCC, Monteiro RA de A, Duarte Neto AN, Saldiva PHN, Silva LFF da, Dolhnikoff M, Mauad T. COVID-19 induces more pronounced extracellular matrix deposition than other causes of ARDS [Internet]. Respiratory Research. 2023 ; 24 1-11 art. 281.[citado 2024 jun. 18 ] Available from: https://dx.doi.org/10.1186/s12931-023-02555-7
Artigo de periodico
4-Hydroxynonenal impairs miRNA maturation in heart failure via Dicer post-translational modification (2023)
Kiyuna, Ligia Akemi ; Cândido, Darlan da Silva ; Bechara, Luiz Roberto Grassmann ; Jesus, Itamar Couto Guedes ; Ramalho, Lisley Santos ; Krum, Barbara Nunes ; Albuquerque, Rudá Prestes e ; Campos, Juliane Cruz ; Bozi, Luiz Henrique Marchesi ; Zambelli, Vanessa Olzon ; Alves, Ariane Ferreira Nunes ; Campolo, Nicolás; Mastrogiovanni, Mauricio ; Silvina Bartesaghi ; Leyva, Alejandro ; Durán, Rosario ; Rafael Radi ; Arantes, Guilherme Menegon ; Cunha Neto, Edecio ; Mori, Marcelo Alves da Silva ; Chen, Che-Hong ; Yang, Wenjin; Mochly-Rosen, Daria ; Ian J. MacRae, Ian J. ; Ferreira, Ludmila Rodrigues Pinto ; Ferreira, Julio Cesar Batista
Fonte: European Heart Journal. Unidades: IQ, FM, ICB
Assuntos: ANATOMIA, INSUFICIÊNCIA CARDÍACA, MICRORNAS, ALDEÍDOS, MITOCÔNDRIAS, RIBONUCLEOSÍDEOS, ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES
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ABNT
KIYUNA, Ligia Akemi et al. 4-Hydroxynonenal impairs miRNA maturation in heart failure via Dicer post-translational modification. European Heart Journal, v. 44, n. 44, p. 4696-4712, 2023Tradução . . Disponível em: https://doi.org/10.1093/eurheartj/ehad662. Acesso em: 18 jun. 2024.
APA
Kiyuna, L. A., Cândido, D. da S., Bechara, L. R. G., Jesus, I. C. G., Ramalho, L. S., Krum, B. N., et al. (2023). 4-Hydroxynonenal impairs miRNA maturation in heart failure via Dicer post-translational modification. European Heart Journal, 44( 44), 4696-4712. doi:10.1093/eurheartj/ehad662
NLM
Kiyuna LA, Cândido D da S, Bechara LRG, Jesus ICG, Ramalho LS, Krum BN, Albuquerque RP e, Campos JC, Bozi LHM, Zambelli VO, Alves AFN, Campolo N, Mastrogiovanni M, Silvina Bartesaghi, Leyva A, Durán R, Rafael Radi, Arantes GM, Cunha Neto E, Mori MA da S, Chen C-H, Yang W, Mochly-Rosen D, Ian J. MacRae IJ, Ferreira LRP, Ferreira JCB. 4-Hydroxynonenal impairs miRNA maturation in heart failure via Dicer post-translational modification [Internet]. European Heart Journal. 2023 ; 44( 44): 4696-4712.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1093/eurheartj/ehad662
Vancouver
Kiyuna LA, Cândido D da S, Bechara LRG, Jesus ICG, Ramalho LS, Krum BN, Albuquerque RP e, Campos JC, Bozi LHM, Zambelli VO, Alves AFN, Campolo N, Mastrogiovanni M, Silvina Bartesaghi, Leyva A, Durán R, Rafael Radi, Arantes GM, Cunha Neto E, Mori MA da S, Chen C-H, Yang W, Mochly-Rosen D, Ian J. MacRae IJ, Ferreira LRP, Ferreira JCB. 4-Hydroxynonenal impairs miRNA maturation in heart failure via Dicer post-translational modification [Internet]. European Heart Journal. 2023 ; 44( 44): 4696-4712.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1093/eurheartj/ehad662
Trabalho de evento-resumo periodico
Protein disulphide isomerase A1 (PDI) supports platelet-endothelium interaction in hyperglycaemia: impacts on endothelial membrane biophysics and secretion of adhesionrelated proteins (2023)
Gaspar, Renato Simões ; França, Álefe Roger Silva; Diniz Filho, Joel Félix Silva ; Santos, Clenilton Costa dos ; Teixeira, L; Massafera, Mariana Pereira ; Bustos, Silvina Odete ; Alencar, Luciana Magalhães Rebelo ; Ronsein, Graziella Eliza ; Laurindo, Francisco Rafael Martins
Fonte: Free Radical Biology & Medicine. Nome do evento: Biennial Congress of the Society for Free Radical Research International/SFRRI. Unidades: IQ, FM, ECA
Assuntos: HIPERGLICEMIA, PROTEÍNAS
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ABNT
GASPAR, Renato Simões et al. Protein disulphide isomerase A1 (PDI) supports platelet-endothelium interaction in hyperglycaemia: impacts on endothelial membrane biophysics and secretion of adhesionrelated proteins. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. Disponível em: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1. Acesso em: 18 jun. 2024. , 2023
APA
Gaspar, R. S., França, Á. R. S., Diniz Filho, J. F. S., Santos, C. C. dos, Teixeira, L., Massafera, M. P., et al. (2023). Protein disulphide isomerase A1 (PDI) supports platelet-endothelium interaction in hyperglycaemia: impacts on endothelial membrane biophysics and secretion of adhesionrelated proteins. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. Recuperado de https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
NLM
Gaspar RS, França ÁRS, Diniz Filho JFS, Santos CC dos, Teixeira L, Massafera MP, Bustos SO, Alencar LMR, Ronsein GE, Laurindo FRM. Protein disulphide isomerase A1 (PDI) supports platelet-endothelium interaction in hyperglycaemia: impacts on endothelial membrane biophysics and secretion of adhesionrelated proteins [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S87 res. 182.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Vancouver
Gaspar RS, França ÁRS, Diniz Filho JFS, Santos CC dos, Teixeira L, Massafera MP, Bustos SO, Alencar LMR, Ronsein GE, Laurindo FRM. Protein disulphide isomerase A1 (PDI) supports platelet-endothelium interaction in hyperglycaemia: impacts on endothelial membrane biophysics and secretion of adhesionrelated proteins [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S87 res. 182.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Artigo de periodico
Anserine is expressed in human cardiac and skeletal muscles (2023)
Gonçalves, Lívia de Souza ; Pereira, Wagner Ribeiro ; Silva, Rafael Pires da ; Yamaguchi, Guilherme Carvalho; Carvalho, Victor Henrique; Vargas, Bianca Scigliano ; Jensen, Leonardo ; Medeiros, Marisa Helena Gennari de ; Roschel, Hamilton ; Artioli, Guilherme Giannini
Fonte: Physiological Reports. Unidades: IQ, EEFE, FM
Assuntos: FISIOLOGIA CARDIOVASCULAR, CARDIOLOGIA, MÚSCULO ESQUELÉTICO
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ABNT
GONÇALVES, Lívia de Souza et al. Anserine is expressed in human cardiac and skeletal muscles. Physiological Reports, v. 11, n. 19, 2023Tradução . . Disponível em: https://doi.org/10.14814/phy2.15833. Acesso em: 18 jun. 2024.
APA
Gonçalves, L. de S., Pereira, W. R., Silva, R. P. da, Yamaguchi, G. C., Carvalho, V. H., Vargas, B. S., et al. (2023). Anserine is expressed in human cardiac and skeletal muscles. Physiological Reports, 11( 19). doi:10.14814/phy2.15833
NLM
Gonçalves L de S, Pereira WR, Silva RP da, Yamaguchi GC, Carvalho VH, Vargas BS, Jensen L, Medeiros MHG de, Roschel H, Artioli GG. Anserine is expressed in human cardiac and skeletal muscles [Internet]. Physiological Reports. 2023 ; 11( 19):[citado 2024 jun. 18 ] Available from: https://doi.org/10.14814/phy2.15833
Vancouver
Gonçalves L de S, Pereira WR, Silva RP da, Yamaguchi GC, Carvalho VH, Vargas BS, Jensen L, Medeiros MHG de, Roschel H, Artioli GG. Anserine is expressed in human cardiac and skeletal muscles [Internet]. Physiological Reports. 2023 ; 11( 19):[citado 2024 jun. 18 ] Available from: https://doi.org/10.14814/phy2.15833
Artigo de periodico
Transcriptome and secretome analyses of endophyte methylobacterium mesophilicum and pathogen Xylella fastidiosa interacting show nutrient competition (2023)
Dourado, Manuella Nóbrega ; Pierry, Paulo Marques ; Feitosa Junior, Oséias Rodrigues ; Uceda-Campos, Guillermo ; Barbosa, Deibs ; Zaini, Paulo Adriano ; Dandekar, Abhaya M. ; Da Silva, Aline Maria ; Araújo, Welington Luiz de
Fonte: Microorganisms. Unidades: ICB, IQ, BIOINFORMÁTICA, FM
Assuntos: MICROBIOLOGIA, BACTÉRIAS GRAM-NEGATIVAS, INFECÇÕES BACTERIANAS GRAM-NEGATIVAS, FITOPATÓGENOS, ANTIOXIDANTES, TRANSCRIÇÃO GÊNICA, PROTEÍNAS DE PLANTAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DOURADO, Manuella Nóbrega et al. Transcriptome and secretome analyses of endophyte methylobacterium mesophilicum and pathogen Xylella fastidiosa interacting show nutrient competition. Microorganisms, v. 11, n. 11, p. 28 , 2023Tradução . . Disponível em: https://doi.org/10.3390/ microorganisms11112755. Acesso em: 18 jun. 2024.
APA
Dourado, M. N., Pierry, P. M., Feitosa Junior, O. R., Uceda-Campos, G., Barbosa, D., Zaini, P. A., et al. (2023). Transcriptome and secretome analyses of endophyte methylobacterium mesophilicum and pathogen Xylella fastidiosa interacting show nutrient competition. Microorganisms, 11( 11), 28 . doi:https://doi.org/10.3390/ microorganisms11112755
NLM
Dourado MN, Pierry PM, Feitosa Junior OR, Uceda-Campos G, Barbosa D, Zaini PA, Dandekar AM, Da Silva AM, Araújo WL de. Transcriptome and secretome analyses of endophyte methylobacterium mesophilicum and pathogen Xylella fastidiosa interacting show nutrient competition [Internet]. Microorganisms. 2023 ; 11( 11): 28 .[citado 2024 jun. 18 ] Available from: https://doi.org/10.3390/ microorganisms11112755
Vancouver
Dourado MN, Pierry PM, Feitosa Junior OR, Uceda-Campos G, Barbosa D, Zaini PA, Dandekar AM, Da Silva AM, Araújo WL de. Transcriptome and secretome analyses of endophyte methylobacterium mesophilicum and pathogen Xylella fastidiosa interacting show nutrient competition [Internet]. Microorganisms. 2023 ; 11( 11): 28 .[citado 2024 jun. 18 ] Available from: https://doi.org/10.3390/ microorganisms11112755
Trabalho de evento-resumo periodico
Uric acid oxidation products correlate with sepsis and promote modification on serum albumin in intensive care unit patients (2023)
Silva, Railmara Pereira da ; Alves, Priscilla D'Avila Mazzuco ; Brandão, Luciana Severo ; Vileigas, Danielle Fernandes ; Souza Junior, Douglas Ricardo de ; Ronsein, Graziella Eliza ; Pinto, Lucia Andrade; Meotti, Flavia Carla
Fonte: Free Radical Biology & Medicine. Nome do evento: Biennial Congress of the Society for Free Radical Research International/SFRRI. Unidades: IQ, FM, EE
Assuntos: OXIDAÇÃO, ÁCIDO ÚRICO
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ABNT
SILVA, Railmara Pereira da et al. Uric acid oxidation products correlate with sepsis and promote modification on serum albumin in intensive care unit patients. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. Disponível em: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1. Acesso em: 18 jun. 2024. , 2023
APA
Silva, R. P. da, Alves, P. D. 'A. M., Brandão, L. S., Vileigas, D. F., Souza Junior, D. R. de, Ronsein, G. E., et al. (2023). Uric acid oxidation products correlate with sepsis and promote modification on serum albumin in intensive care unit patients. Free Radical Biology & Medicine. New York: Instituto de Química, Universidade de São Paulo. Recuperado de https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
NLM
Silva RP da, Alves PD'AM, Brandão LS, Vileigas DF, Souza Junior DR de, Ronsein GE, Pinto LA, Meotti FC. Uric acid oxidation products correlate with sepsis and promote modification on serum albumin in intensive care unit patients [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S48-S49 res. 97.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Vancouver
Silva RP da, Alves PD'AM, Brandão LS, Vileigas DF, Souza Junior DR de, Ronsein GE, Pinto LA, Meotti FC. Uric acid oxidation products correlate with sepsis and promote modification on serum albumin in intensive care unit patients [Internet]. Free Radical Biology & Medicine. 2023 ; 208 S48-S49 res. 97.[citado 2024 jun. 18 ] Available from: https://www.sciencedirect.com/journal/free-radical-biology-and-medicine/vol/208/suppl/S1
Artigo de periodico
Biallelic variants in DNA2 cause poikiloderma with congenital cataracts and severe growth failure reminiscent of Rothmund-Thomson syndrome (2023)
Lazzaro Filho, Ricardo Di; Yamamoto, Guilherme Lopes; Silva, Tiago J; Rocha, Leticia A; Linnenkamp, Bianca D. W; Castro, Matheus Augusto Araújo; Bartholdi, Deborah; Schaller, André; Leeb, Tosso; Kelmann, Samantha; Utagawa, Claudia Y; Steiner, Carlos E; Steinmetz, Leandra; Honjo, Rachel Sayuri; Kim, Chong Ae ; Wang, Lisa; Abourjaili-Bilodeau, Raphaël; Campeau, Philippe M; Warma, Matthew; Passos-Bueno, Maria Rita ; Hoch, Nicolas Carlos ; Bertola, Débora Romeo
Fonte: Journal of Medical Genetics. Unidades: BIOINFORMÁTICA, FM, IB, IQ
Assuntos: GENÉTICA MÉDICA, ANÁLISE DE SEQUÊNCIA DE DNA, DOENÇAS GENÉTICAS, MUTAÇÃO GENÉTICA
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ABNT
LAZZARO FILHO, Ricardo Di et al. Biallelic variants in DNA2 cause poikiloderma with congenital cataracts and severe growth failure reminiscent of Rothmund-Thomson syndrome. Journal of Medical Genetics, 2023Tradução . . Disponível em: https://doi.org/10.1136/jmg-2022-109119. Acesso em: 18 jun. 2024.
APA
Lazzaro Filho, R. D., Yamamoto, G. L., Silva, T. J., Rocha, L. A., Linnenkamp, B. D. W., Castro, M. A. A., et al. (2023). Biallelic variants in DNA2 cause poikiloderma with congenital cataracts and severe growth failure reminiscent of Rothmund-Thomson syndrome. Journal of Medical Genetics. doi:10.1136/jmg-2022-109119
NLM
Lazzaro Filho RD, Yamamoto GL, Silva TJ, Rocha LA, Linnenkamp BDW, Castro MAA, Bartholdi D, Schaller A, Leeb T, Kelmann S, Utagawa CY, Steiner CE, Steinmetz L, Honjo RS, Kim CA, Wang L, Abourjaili-Bilodeau R, Campeau PM, Warma M, Passos-Bueno MR, Hoch NC, Bertola DR. Biallelic variants in DNA2 cause poikiloderma with congenital cataracts and severe growth failure reminiscent of Rothmund-Thomson syndrome [Internet]. Journal of Medical Genetics. 2023 ;[citado 2024 jun. 18 ] Available from: https://doi.org/10.1136/jmg-2022-109119
Vancouver
Lazzaro Filho RD, Yamamoto GL, Silva TJ, Rocha LA, Linnenkamp BDW, Castro MAA, Bartholdi D, Schaller A, Leeb T, Kelmann S, Utagawa CY, Steiner CE, Steinmetz L, Honjo RS, Kim CA, Wang L, Abourjaili-Bilodeau R, Campeau PM, Warma M, Passos-Bueno MR, Hoch NC, Bertola DR. Biallelic variants in DNA2 cause poikiloderma with congenital cataracts and severe growth failure reminiscent of Rothmund-Thomson syndrome [Internet]. Journal of Medical Genetics. 2023 ;[citado 2024 jun. 18 ] Available from: https://doi.org/10.1136/jmg-2022-109119
Artigo de periodico
Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study (2023)
Toyoshima, Marcos Tadashi Kakitani ; Santana, Monique de Fatima Mello; Silva, Amanda R. M; Mello, Gabriela B; Bezerra, Daniele P. Santos; Góes, Marisa F. S; Bosco, Adriana A; Caramelli, Bruno ; Ronsein, Graziella Eliza ; Giannella, Maria Lucia Correa ; Passarelli, Marisa
Fonte: Diabetology and Metabolic Syndrome. Unidades: FM, IQ
Assuntos: PROTEÔMICA, DIABETES MELLITUS, ARTERIOSCLEROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TOYOSHIMA, Marcos Tadashi Kakitani et al. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study. Diabetology and Metabolic Syndrome, v. 15, p. 1-14 art. 42, 2023Tradução . . Disponível em: https://doi.org/10.1186/s13098-023-01007-y. Acesso em: 18 jun. 2024.
APA
Toyoshima, M. T. K., Santana, M. de F. M., Silva, A. R. M., Mello, G. B., Bezerra, D. P. S., Góes, M. F. S., et al. (2023). Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study. Diabetology and Metabolic Syndrome, 15, 1-14 art. 42. doi:10.1186/s13098-023-01007-y
NLM
Toyoshima MTK, Santana M de FM, Silva ARM, Mello GB, Bezerra DPS, Góes MFS, Bosco AA, Caramelli B, Ronsein GE, Giannella MLC, Passarelli M. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study [Internet]. Diabetology and Metabolic Syndrome. 2023 ; 15 1-14 art. 42.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1186/s13098-023-01007-y
Vancouver
Toyoshima MTK, Santana M de FM, Silva ARM, Mello GB, Bezerra DPS, Góes MFS, Bosco AA, Caramelli B, Ronsein GE, Giannella MLC, Passarelli M. Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study [Internet]. Diabetology and Metabolic Syndrome. 2023 ; 15 1-14 art. 42.[citado 2024 jun. 18 ] Available from: https://doi.org/10.1186/s13098-023-01007-y

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