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  • Fonte: Immunology. Unidades: ICB, ESALQ

    Assuntos: AEDES, CAMUNDONGOS, CITOMETRIA DE FLUXO, FENÓTIPOS, PELE DE ANIMAL, IMUNIDADE, INFLAMAÇÃO, PICADAS DE INSETOS, SALIVA

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    • ABNT

      HENRIQUE, Maressa de Oliveira et al. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes. Immunology, v. 158, n. 1, p. 47-59, 2019Tradução . . Disponível em: https://doi.org/10.1111/imm.13096. Acesso em: 21 ago. 2024.
    • APA

      Henrique, M. de O., Santos Neto, L., Assis, J. B. de, Barros, M. S. de, Capurro, M. de L., Lepique, A. P., et al. (2019). Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes. Immunology, 158( 1), 47-59. doi:10.1111/imm.13096
    • NLM

      Henrique M de O, Santos Neto L, Assis JB de, Barros MS de, Capurro M de L, Lepique AP, Fonseca DM da, Nunes A de S. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes [Internet]. Immunology. 2019 ; 158( 1): 47-59.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/imm.13096
    • Vancouver

      Henrique M de O, Santos Neto L, Assis JB de, Barros MS de, Capurro M de L, Lepique AP, Fonseca DM da, Nunes A de S. Evaluation of inflammatory skin infiltrate following Aedes aegypti bites in sensitized and non‐sensitized mice reveals saliva‐dependent and immune‐dependent phenotypes [Internet]. Immunology. 2019 ; 158( 1): 47-59.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/imm.13096
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      RODRIGUES, Michele F. et al. Apoptosis of macrophages during pulmonary Mycobacterium bovis infection: correlation with intracellular bacillary load and cytokine levels. Immunology, v. 128, n. 1 pt. 2, p. e691-e699, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2009.03062.x. Acesso em: 21 ago. 2024.
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      Rodrigues, M. F., Barsante, M. M., Alves, C. C. S., Souza, M. A., Ferreira, A. P., Amarante-Mendes, J. G. P., & Teixeira, H. C. (2009). Apoptosis of macrophages during pulmonary Mycobacterium bovis infection: correlation with intracellular bacillary load and cytokine levels. Immunology, 128( 1 pt. 2), e691-e699. doi:10.1111/j.1365-2567.2009.03062.x
    • NLM

      Rodrigues MF, Barsante MM, Alves CCS, Souza MA, Ferreira AP, Amarante-Mendes JGP, Teixeira HC. Apoptosis of macrophages during pulmonary Mycobacterium bovis infection: correlation with intracellular bacillary load and cytokine levels [Internet]. Immunology. 2009 ; 128( 1 pt. 2): e691-e699.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2009.03062.x
    • Vancouver

      Rodrigues MF, Barsante MM, Alves CCS, Souza MA, Ferreira AP, Amarante-Mendes JGP, Teixeira HC. Apoptosis of macrophages during pulmonary Mycobacterium bovis infection: correlation with intracellular bacillary load and cytokine levels [Internet]. Immunology. 2009 ; 128( 1 pt. 2): e691-e699.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2009.03062.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      MARGUTI, Ivo et al. Expansion of CD4+ CD25+ Foxp3+ T cells by bone marrow-derived dendritic cells. Immunology, v. 127, n. 1, p. 50-61, 2009Tradução . . Acesso em: 21 ago. 2024.
    • APA

      Marguti, I., Yamamoto, G. L., Costa, T. B. da, Rizzo, L. V., & Moraes, L. V. (2009). Expansion of CD4+ CD25+ Foxp3+ T cells by bone marrow-derived dendritic cells. Immunology, 127( 1), 50-61.
    • NLM

      Marguti I, Yamamoto GL, Costa TB da, Rizzo LV, Moraes LV. Expansion of CD4+ CD25+ Foxp3+ T cells by bone marrow-derived dendritic cells. Immunology. 2009 ; 127( 1): 50-61.[citado 2024 ago. 21 ]
    • Vancouver

      Marguti I, Yamamoto GL, Costa TB da, Rizzo LV, Moraes LV. Expansion of CD4+ CD25+ Foxp3+ T cells by bone marrow-derived dendritic cells. Immunology. 2009 ; 127( 1): 50-61.[citado 2024 ago. 21 ]
  • Fonte: Immunology. Unidades: ICB, FM

    Assuntos: CÉLULAS DENDRÍTICAS, IMUNOHISTOQUÍMICA, RATOS, PLASMODIUM (PARASITOLOGIA), CITOMETRIA DE FLUXO, HEMATOPOESE, MALÁRIA (PARASITOLOGIA)

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    • ABNT

      GARNICA, Margoth Ramos et al. Supplementation of CXCL12 (CXCL12) induces homing of CD11c+ dendritic cells to the spleen and enhances control of Plasmodium berghei malaria in BALB/c mice. Immunology, v. 115, n. 3, p. 399-406, 2005Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2005.02178.x. Acesso em: 21 ago. 2024.
    • APA

      Garnica, M. R., Moraes, L. V. de, Rizzo, L. V., & Andrade Júnior, H. F. de. (2005). Supplementation of CXCL12 (CXCL12) induces homing of CD11c+ dendritic cells to the spleen and enhances control of Plasmodium berghei malaria in BALB/c mice. Immunology, 115( 3), 399-406. doi:10.1111/j.1365-2567.2005.02178.x
    • NLM

      Garnica MR, Moraes LV de, Rizzo LV, Andrade Júnior HF de. Supplementation of CXCL12 (CXCL12) induces homing of CD11c+ dendritic cells to the spleen and enhances control of Plasmodium berghei malaria in BALB/c mice [Internet]. Immunology. 2005 ; 115( 3): 399-406.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2005.02178.x
    • Vancouver

      Garnica MR, Moraes LV de, Rizzo LV, Andrade Júnior HF de. Supplementation of CXCL12 (CXCL12) induces homing of CD11c+ dendritic cells to the spleen and enhances control of Plasmodium berghei malaria in BALB/c mice [Internet]. Immunology. 2005 ; 115( 3): 399-406.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2005.02178.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      BASTOS, Karina R. B. et al. Analysis of the activation profile of dendritic cells derived from the bone marrow of interleukin-12/interleukin-23-deficient mice. Immunology, v. 114, p. 499-506, 2005Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2567.2005.02118.x. Acesso em: 21 ago. 2024.
    • APA

      Bastos, K. R. B., Moraes, L. de D. V. de, Zago, C. A., Marinho, C. R. F., Russo, M., Alvarez, J. M., & Lima, M. R. D. 'I. (2005). Analysis of the activation profile of dendritic cells derived from the bone marrow of interleukin-12/interleukin-23-deficient mice. Immunology, 114, 499-506. doi:10.1111/j.1365-2567.2005.02118.x
    • NLM

      Bastos KRB, Moraes L de DV de, Zago CA, Marinho CRF, Russo M, Alvarez JM, Lima MRD'I. Analysis of the activation profile of dendritic cells derived from the bone marrow of interleukin-12/interleukin-23-deficient mice [Internet]. Immunology. 2005 ; 114 499-506.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2005.02118.x
    • Vancouver

      Bastos KRB, Moraes L de DV de, Zago CA, Marinho CRF, Russo M, Alvarez JM, Lima MRD'I. Analysis of the activation profile of dendritic cells derived from the bone marrow of interleukin-12/interleukin-23-deficient mice [Internet]. Immunology. 2005 ; 114 499-506.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1111/j.1365-2567.2005.02118.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      SILVA, Ana Paula Galvão da e JACYSYN, Jacqueline F. e ABRAHAMSOHN, Ises de Almeida. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response. Immunology, v. 108, p. 230-237, 2003Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2003.01571.x. Acesso em: 21 ago. 2024.
    • APA

      Silva, A. P. G. da, Jacysyn, J. F., & Abrahamsohn, I. de A. (2003). Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response. Immunology, 108, 230-237. doi:10.1046/j.1365-2567.2003.01571.x
    • NLM

      Silva APG da, Jacysyn JF, Abrahamsohn I de A. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response [Internet]. Immunology. 2003 ; 108 230-237.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2003.01571.x
    • Vancouver

      Silva APG da, Jacysyn JF, Abrahamsohn I de A. Resistant mice lacking interleukin-12 become susceptible to trypanosoma cruzi infection but fail to mount a T helper type 2 response [Internet]. Immunology. 2003 ; 108 230-237.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2003.01571.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      GRISOTTO, M G et al. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules. Immunology, v. 102, p. 209-217, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01170.x. Acesso em: 21 ago. 2024.
    • APA

      Grisotto, M. G., Lima, M. R. D. 'I., Marinho, C. R. F., Tadokoro, C. E., Abrahamsohn, I. de A., & Alvarez, J. M. (2001). Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules. Immunology, 102, 209-217. doi:10.1046/j.1365-2567.2001.01170.x
    • NLM

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules [Internet]. Immunology. 2001 ;102 209-217.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
    • Vancouver

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8+ cells in trypanosoma cruzi-infected chronic mice are down-rgulated for T-cell receptor-'alfa''Beta' and CD8 molecules [Internet]. Immunology. 2001 ;102 209-217.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      GRISOTTO, Marcos Grigolin et al. Most parasite-specific CD8 + cells in Trypanossoma cruzi infected chronic mice are down-regulated for T-cell receptor alpha, beta and CD8 molecules. Immunology, v. 102, p. 1-14, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01170.x. Acesso em: 21 ago. 2024.
    • APA

      Grisotto, M. G., Lima, M. R. D. 'I., Marinho, C. R. F., Tadokoro, C. E., Abrahamsohn, I. de A., & Alvarez, J. M. (2001). Most parasite-specific CD8 + cells in Trypanossoma cruzi infected chronic mice are down-regulated for T-cell receptor alpha, beta and CD8 molecules. Immunology, 102, 1-14. doi:10.1046/j.1365-2567.2001.01170.x
    • NLM

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8 + cells in Trypanossoma cruzi infected chronic mice are down-regulated for T-cell receptor alpha, beta and CD8 molecules [Internet]. Immunology. 2001 ;102 1-14.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
    • Vancouver

      Grisotto MG, Lima MRD'I, Marinho CRF, Tadokoro CE, Abrahamsohn I de A, Alvarez JM. Most parasite-specific CD8 + cells in Trypanossoma cruzi infected chronic mice are down-regulated for T-cell receptor alpha, beta and CD8 molecules [Internet]. Immunology. 2001 ;102 1-14.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01170.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      JACYSYN, J F e ABRAHAMSOHN, Ises de Almeida e MACEDO, Mahasti Sahihi de. Modulation of delayed-type hypersensitivity during the time course of immune response to a protein antigen. Immunology, v. 102 p. 373-379, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01181.x. Acesso em: 21 ago. 2024.
    • APA

      Jacysyn, J. F., Abrahamsohn, I. de A., & Macedo, M. S. de. (2001). Modulation of delayed-type hypersensitivity during the time course of immune response to a protein antigen. Immunology, 102 p. 373-379. doi:10.1046/j.1365-2567.2001.01181.x
    • NLM

      Jacysyn JF, Abrahamsohn I de A, Macedo MS de. Modulation of delayed-type hypersensitivity during the time course of immune response to a protein antigen [Internet]. Immunology. 2001 ;102 p. 373-379[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01181.x
    • Vancouver

      Jacysyn JF, Abrahamsohn I de A, Macedo MS de. Modulation of delayed-type hypersensitivity during the time course of immune response to a protein antigen [Internet]. Immunology. 2001 ;102 p. 373-379[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01181.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      JACYSYN, Jacqueline de Fátima e ABRAHAMSOHN, Ises de Almeida e MACEDO, Mahasti Sahihi de. Modulation of delayed-type hypersensitivity during the time courses of imune response to a protein antigen. Immunology, v. 102, p. 373-379, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01181.x. Acesso em: 21 ago. 2024.
    • APA

      Jacysyn, J. de F., Abrahamsohn, I. de A., & Macedo, M. S. de. (2001). Modulation of delayed-type hypersensitivity during the time courses of imune response to a protein antigen. Immunology, 102, 373-379. doi:10.1046/j.1365-2567.2001.01181.x
    • NLM

      Jacysyn J de F, Abrahamsohn I de A, Macedo MS de. Modulation of delayed-type hypersensitivity during the time courses of imune response to a protein antigen [Internet]. Immunology. 2001 ;102 373-379.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01181.x
    • Vancouver

      Jacysyn J de F, Abrahamsohn I de A, Macedo MS de. Modulation of delayed-type hypersensitivity during the time courses of imune response to a protein antigen [Internet]. Immunology. 2001 ;102 373-379.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01181.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      SILVA, Ana Paula Galvão da e ABRAHAMSOHN, Ises de Almeida. Interleukin-12 stimulation of lymphoproliferative response in Trypanossoma cruzi infection. Immunology, 2001Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.2001.01311.x. Acesso em: 21 ago. 2024.
    • APA

      Silva, A. P. G. da, & Abrahamsohn, I. de A. (2001). Interleukin-12 stimulation of lymphoproliferative response in Trypanossoma cruzi infection. Immunology. doi:10.1046/j.1365-2567.2001.01311.x
    • NLM

      Silva APG da, Abrahamsohn I de A. Interleukin-12 stimulation of lymphoproliferative response in Trypanossoma cruzi infection [Internet]. Immunology. 2001 ;[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01311.x
    • Vancouver

      Silva APG da, Abrahamsohn I de A. Interleukin-12 stimulation of lymphoproliferative response in Trypanossoma cruzi infection [Internet]. Immunology. 2001 ;[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.2001.01311.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TORRECILHAS, A. C. T. et al. Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to trypanosoma cruzi. Immunology, v. 96, p. 381-388, 1999Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.1999.00719.x. Acesso em: 21 ago. 2024.
    • APA

      Torrecilhas, A. C. T., Faquim, E. M., Silva, A. V. da, & Abrahamsohn, I. de A. (1999). Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to trypanosoma cruzi. Immunology, 96, 381-388. doi:10.1046/j.1365-2567.1999.00719.x
    • NLM

      Torrecilhas ACT, Faquim EM, Silva AV da, Abrahamsohn I de A. Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to trypanosoma cruzi [Internet]. Immunology. 1999 ; 96 381-388.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.1999.00719.x
    • Vancouver

      Torrecilhas ACT, Faquim EM, Silva AV da, Abrahamsohn I de A. Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to trypanosoma cruzi [Internet]. Immunology. 1999 ; 96 381-388.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.1999.00719.x
  • Fonte: Immunology. Unidade: ICB

    Assunto: IMUNOLOGIA

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      TADOKORO, C E e MACEDO, Mahasti Sahihi de e ABRAHAMSOHN, Ises de Almeida. Saponin adjuvant primes for a dominant interleukin-10 production to ovalbumin and to trypanosoma cruzi antigen. Immunology, v. 89, p. 368-74, 1996Tradução . . Disponível em: https://doi.org/10.1046/j.1365-2567.1996.d01-767.x. Acesso em: 21 ago. 2024.
    • APA

      Tadokoro, C. E., Macedo, M. S. de, & Abrahamsohn, I. de A. (1996). Saponin adjuvant primes for a dominant interleukin-10 production to ovalbumin and to trypanosoma cruzi antigen. Immunology, 89, 368-74. doi:10.1046/j.1365-2567.1996.d01-767.x
    • NLM

      Tadokoro CE, Macedo MS de, Abrahamsohn I de A. Saponin adjuvant primes for a dominant interleukin-10 production to ovalbumin and to trypanosoma cruzi antigen [Internet]. Immunology. 1996 ;89 368-74.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.1996.d01-767.x
    • Vancouver

      Tadokoro CE, Macedo MS de, Abrahamsohn I de A. Saponin adjuvant primes for a dominant interleukin-10 production to ovalbumin and to trypanosoma cruzi antigen [Internet]. Immunology. 1996 ;89 368-74.[citado 2024 ago. 21 ] Available from: https://doi.org/10.1046/j.1365-2567.1996.d01-767.x

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