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Artigo de periodico
"Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " (2023)
Raquel, Hiviny de Ataides ; Perego, Sany M. ; Masson, Gustavo S.; Jensen, Leonardo ; Colquhoun, Alison ; Michelini, Lisete Compagno
Source: Clinical Science. Unidade: ICB
Subjects: BIOLOGIA CELULAR, FISIOLOGIA, INSUFICIÊNCIA CARDÍACA ANIMAL, BARREIRA HEMATO-ENCEFÁLICA, RATOS WISTAR, SISTEMA NERVOSO AUTÔNOMO, CIRCULAÇÃO ANIMAL, EXERCÍCIO FÍSICO, PROTEÍNAS DA MEMBRANA
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ABNT
RAQUEL, Hiviny de Ataides et al. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, v. 137, n. 15, p. 1049-1066, 2023Tradução . . Disponível em: https://doi.org/10.1042/CS20230489. Acesso em: 18 nov. 2024.
APA
Raquel, H. de A., Perego, S. M., Masson, G. S., Jensen, L., Colquhoun, A., & Michelini, L. C. (2023). "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training ". Clinical Science, 137( 15), 1049-1066. doi:10.1042/CS20230489
NLM
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20230489
Vancouver
Raquel H de A, Perego SM, Masson GS, Jensen L, Colquhoun A, Michelini LC. "Blood-brain barrier lesion - a novel determinant of autonomic imbalance in heart failure and the effects of exercise training " [Internet]. Clinical Science. 2023 ; 137( 15): 1049-1066.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20230489
Artigo de periodico
Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress (2021)
Dardi, Patrizia ; Perazza, Laís Rossi; Couto, Gisele Kruger ; Campos, Gianne Paul; Capettini, Luciano dos Santos Aggum ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, VEIA CAVA, ANTIOXIDANTES, INSUFICIÊNCIA CARDÍACA ANIMAL, AORTA TORÁCICA, AORTA DE ANIMAL, ENDOTÉLIO VASCULAR, PERÓXIDO DE HIDROGÊNIO, INFARTO DO MIOCÁRDIO, ÓXIDO NÍTRICO, RATOS
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DARDI, Patrizia et al. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress. Clinical Science, 2021Tradução . . Disponível em: https://doi.org/10.1042/CS20210810. Acesso em: 18 nov. 2024.
APA
Dardi, P., Perazza, L. R., Couto, G. K., Campos, G. P., Capettini, L. dos S. A., & Rossoni, L. V. (2021). Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress. Clinical Science. doi:10.1042/CS20210810
NLM
Dardi P, Perazza LR, Couto GK, Campos GP, Capettini L dos SA, Rossoni LV. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress [Internet]. Clinical Science. 2021 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20210810
Vancouver
Dardi P, Perazza LR, Couto GK, Campos GP, Capettini L dos SA, Rossoni LV. Vena cava presents endothelial dysfunction prior to thoracic aorta in heart failure: the pivotal role of nNOS uncoupling/ oxidative stress [Internet]. Clinical Science. 2021 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20210810
Artigo de periodico
Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure (2020)
Fontes, Milene Tavares ; Paula, Suliana Mesquita ; Lino, Caroline Antunes ; Senger, Nathalia ; Couto, Gisele Kruger; Barreto-Chaves, Maria Luiza Morais ; Mill, José Geraldo ; Rossoni, Luciana Venturini
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, SISTEMA RENINA-ANGIOTENSINA, TECIDO ADIPOSO, INSUFICIÊNCIA CARDÍACA ANIMAL, DOENÇAS CARDIOVASCULARES EM ANIMAL, ENDOTÉLIO VASCULAR, INFARTO DO MIOCÁRDIO, RATOS, ANGIOTENSINAS, AORTA DE ANIMAL, ESTRESSE OXIDATIVO, ÓXIDO NÍTRICO, AORTA TORÁCICA
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ABNT
FONTES, Milene Tavares et al. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20201099. Acesso em: 18 nov. 2024.
APA
Fontes, M. T., Paula, S. M., Lino, C. A., Senger, N., Couto, G. K., Barreto-Chaves, M. L. M., et al. (2020). Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure. Clinical Science. doi:10.1042/CS20201099
NLM
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20201099
Vancouver
Fontes MT, Paula SM, Lino CA, Senger N, Couto GK, Barreto-Chaves MLM, Mill JG, Rossoni LV. Renin-angiotensin system overactivation in perivascular adipose tissue contributes to vascular dysfunction in heart failure [Internet]. Clinical Science. 2020 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20201099
Artigo de periodico
Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure (2019)
Andrade, David Campos ; Toledo, Camilo ; Díaz, Hugo Sebastián ; Lucero, Claudia; Álvarez, Alexis Arce ; Santos, Luiz Marcelo Oliveira ; Takakura, Ana Carolina ; Moreira, Thiago dos Santos ; Schultz, Harold D. ; Marcus, Noah J. ; Alcayaga, Julio ; Rio, Rodrigo Del
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA, SISTEMA NERVOSO SIMPÁTICO, INSUFICIÊNCIA CARDÍACA ANIMAL, NEURÔNIOS, IMUNOTOXINAS, TOXINAS EM ANIMAL, RATOS, ARRITMIA, TRONCO DE ANIMAL, CATECOLAMINAS
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ABNT
ANDRADE, David Campos et al. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, v. 133, n. 3, p. 393-405, 2019Tradução . . Disponível em: https://doi.org/10.1042/CS20180589. Acesso em: 18 nov. 2024.
APA
Andrade, D. C., Toledo, C., Díaz, H. S., Lucero, C., Álvarez, A. A., Santos, L. M. O., et al. (2019). Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure. Clinical Science, 133( 3), 393-405. doi:10.1042/CS20180589
NLM
Andrade DC, Toledo C, Díaz HS, Lucero C, Álvarez AA, Santos LMO, Takakura AC, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20180589
Vancouver
Andrade DC, Toledo C, Díaz HS, Lucero C, Álvarez AA, Santos LMO, Takakura AC, Moreira T dos S, Schultz HD, Marcus NJ, Alcayaga J, Rio RD. Ablation of brainstem C1 neurons improves cardiac function in volume overload heart failure [Internet]. Clinical Science. 2019 ; 133( 3): 393-405.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20180589
Artigo de periodico
A past and present overview of macrophage metabolism and functional outcomes (2017)
Curi, Rui ; Mendes, Renata de Siqueira; Crispin, Luiz Aurélio de Campos ; Norata, Giuseppe Danilo ; Sampaio, Sandra Coccuzzo ; Newsholme, Philip
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, MACRÓFAGOS, METABOLISMO ENERGÉTICO, ÁCIDOS GRAXOS, GLICOSE, GLUTAMINA, INSULINA, HORMÔNIOS TIREOIDIANOS
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ABNT
CURI, Rui et al. A past and present overview of macrophage metabolism and functional outcomes. Clinical Science, v. 131, n. 12, p. 1329-1342, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20170220. Acesso em: 18 nov. 2024.
APA
Curi, R., Mendes, R. de S., Crispin, L. A. de C., Norata, G. D., Sampaio, S. C., & Newsholme, P. (2017). A past and present overview of macrophage metabolism and functional outcomes. Clinical Science, 131( 12), 1329-1342. doi:10.1042/CS20170220
NLM
Curi R, Mendes R de S, Crispin LA de C, Norata GD, Sampaio SC, Newsholme P. A past and present overview of macrophage metabolism and functional outcomes [Internet]. Clinical Science. 2017 ; 131( 12): 1329-1342.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20170220
Vancouver
Curi R, Mendes R de S, Crispin LA de C, Norata GD, Sampaio SC, Newsholme P. A past and present overview of macrophage metabolism and functional outcomes [Internet]. Clinical Science. 2017 ; 131( 12): 1329-1342.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20170220
Artigo de periodico
Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease (2017)
Festuccia, William Tadeu Lara
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FESTUCCIA, William Tadeu Lara. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, v. 131, n. 4, p. 327-328, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20160855. Acesso em: 18 nov. 2024.
APA
Festuccia, W. T. L. (2017). Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease. Clinical Science, 131( 4), 327-328. doi:10.1042/CS20160855
NLM
Festuccia WTL. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20160855
Vancouver
Festuccia WTL. Turning up the heat against metabolic syndrome and non-alcoholic fatty liver disease [Internet]. Clinical Science. 2017 ; 131( 4): 327-328.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20160855
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 18 nov. 2024.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line (2011)
Keane, Deirdre C.; Takahashi, Hilton Kenji; Dhayal, Shalinee; Morgan, Noel G.; Curi, Rui; Newsholme, Philip
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
KEANE, Deirdre C. et al. Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line. Clinical Science, v. 120, n. 5, p. 195-206, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20100282. Acesso em: 18 nov. 2024.
APA
Keane, D. C., Takahashi, H. K., Dhayal, S., Morgan, N. G., Curi, R., & Newsholme, P. (2011). Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line. Clinical Science, 120( 5), 195-206. doi:10.1042/CS20100282
NLM
Keane DC, Takahashi HK, Dhayal S, Morgan NG, Curi R, Newsholme P. Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line [Internet]. Clinical Science. 2011 ; 120( 5): 195-206.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20100282
Vancouver
Keane DC, Takahashi HK, Dhayal S, Morgan NG, Curi R, Newsholme P. Arachidonic acid actions on functional integrity and attenuation of the negative effects of palmitic acid in a clonal pancreatic β-cell line [Internet]. Clinical Science. 2011 ; 120( 5): 195-206.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20100282
Artigo de periodico
Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries (2011)
Rossoni, Luciana Venturini ; Wareing, Mark; Wenceslau, Camilla Ferreira; Al-Abri, Mahmood; Cobb, Chris; Austin, Clare
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSONI, Luciana Venturini et al. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, v. 121, n. 10, p. 449-458, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20110259. Acesso em: 18 nov. 2024.
APA
Rossoni, L. V., Wareing, M., Wenceslau, C. F., Al-Abri, M., Cobb, C., & Austin, C. (2011). Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, 121( 10), 449-458. doi:10.1042/CS20110259
NLM
Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20110259
Vancouver
Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/CS20110259
Artigo de periodico
Short-chain fatty acids stimulate the migration of neutrophils to inflammatory sites (2009)
Vinolo, Marco Aurélio Ramirez; Rodrigues, Hosana Gomes; Hatanaka, Elaine; Hebeda, Cristina B.; Farsky, Sandra Helena Poliselli ; Curi, Rui
Source: Clinical Science. Unidades: FCF, ICB
Assunto: FISIOLOGIA
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ABNT
VINOLO, Marco Aurélio Ramirez et al. Short-chain fatty acids stimulate the migration of neutrophils to inflammatory sites. Clinical Science, v. 117, n. 9, p. 331-338, 2009Tradução . . Disponível em: https://doi.org/10.1042/cs20080642. Acesso em: 18 nov. 2024.
APA
Vinolo, M. A. R., Rodrigues, H. G., Hatanaka, E., Hebeda, C. B., Farsky, S. H. P., & Curi, R. (2009). Short-chain fatty acids stimulate the migration of neutrophils to inflammatory sites. Clinical Science, 117( 9), 331-338. doi:10.1042/cs20080642
NLM
Vinolo MAR, Rodrigues HG, Hatanaka E, Hebeda CB, Farsky SHP, Curi R. Short-chain fatty acids stimulate the migration of neutrophils to inflammatory sites [Internet]. Clinical Science. 2009 ; 117( 9): 331-338.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20080642
Vancouver
Vinolo MAR, Rodrigues HG, Hatanaka E, Hebeda CB, Farsky SHP, Curi R. Short-chain fatty acids stimulate the migration of neutrophils to inflammatory sites [Internet]. Clinical Science. 2009 ; 117( 9): 331-338.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20080642
Artigo de periodico
Induction of apoptosis in rat lymphocytes by starvation (2007)
Pires, Juliana; Curi, Rui; Otton, Rosemari
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
PIRES, Juliana e CURI, Rui e OTTON, Rosemari. Induction of apoptosis in rat lymphocytes by starvation. Clinical Science, v. 112, n. 1, p. 59-67, 2007Tradução . . Disponível em: https://doi.org/10.1042/cs20060212. Acesso em: 18 nov. 2024.
APA
Pires, J., Curi, R., & Otton, R. (2007). Induction of apoptosis in rat lymphocytes by starvation. Clinical Science, 112( 1), 59-67. doi:10.1042/cs20060212
NLM
Pires J, Curi R, Otton R. Induction of apoptosis in rat lymphocytes by starvation [Internet]. Clinical Science. 2007 ; 112( 1): 59-67.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20060212
Vancouver
Pires J, Curi R, Otton R. Induction of apoptosis in rat lymphocytes by starvation [Internet]. Clinical Science. 2007 ; 112( 1): 59-67.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20060212
Artigo de periodico
Mechanisms by which fatty acids regulate leucocyte function (2007)
Lima, Thais Martins de; Gorjão, Renata; Hatanaka, Elaine; Cury-Boaventura, Maria Fernanda; Portioli Silva, Erica Paula; Procópio, Joaquim; Curi, Rui
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
LIMA, Thais Martins de et al. Mechanisms by which fatty acids regulate leucocyte function. Clinical Science, v. 113, n. 2, p. 65-77, 2007Tradução . . Acesso em: 18 nov. 2024.
APA
Lima, T. M. de, Gorjão, R., Hatanaka, E., Cury-Boaventura, M. F., Portioli Silva, E. P., Procópio, J., & Curi, R. (2007). Mechanisms by which fatty acids regulate leucocyte function. Clinical Science, 113( 2), 65-77.
NLM
Lima TM de, Gorjão R, Hatanaka E, Cury-Boaventura MF, Portioli Silva EP, Procópio J, Curi R. Mechanisms by which fatty acids regulate leucocyte function. Clinical Science. 2007 ; 113( 2): 65-77.[citado 2024 nov. 18 ]
Vancouver
Lima TM de, Gorjão R, Hatanaka E, Cury-Boaventura MF, Portioli Silva EP, Procópio J, Curi R. Mechanisms by which fatty acids regulate leucocyte function. Clinical Science. 2007 ; 113( 2): 65-77.[citado 2024 nov. 18 ]
Artigo de periodico
Comparative toxicity of fatty acids on a macrophage cell line (J774) (2006)
Lima, Thais Martins de; Cury-Boaventura, Maria Fernanda; Giannocco, Gisele; Nunes, Maria Tereza ; Curi, Rui
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Thais Martins de et al. Comparative toxicity of fatty acids on a macrophage cell line (J774). Clinical Science, v. 111, p. 307-317, 2006Tradução . . Disponível em: https://doi.org/10.1042/cs20060064. Acesso em: 18 nov. 2024.
APA
Lima, T. M. de, Cury-Boaventura, M. F., Giannocco, G., Nunes, M. T., & Curi, R. (2006). Comparative toxicity of fatty acids on a macrophage cell line (J774). Clinical Science, 111, 307-317. doi:10.1042/cs20060064
NLM
Lima TM de, Cury-Boaventura MF, Giannocco G, Nunes MT, Curi R. Comparative toxicity of fatty acids on a macrophage cell line (J774) [Internet]. Clinical Science. 2006 ; 111 307-317.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20060064
Vancouver
Lima TM de, Cury-Boaventura MF, Giannocco G, Nunes MT, Curi R. Comparative toxicity of fatty acids on a macrophage cell line (J774) [Internet]. Clinical Science. 2006 ; 111 307-317.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20060064
Artigo de periodico
Regulation of reactive oxygen species (ROS) production by C18 fatty acids in Jurkat and Raji cells (2005)
Cury-Boaventura, Maria Fernanda; Curi, Rui
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CURY-BOAVENTURA, Maria Fernanda e CURI, Rui. Regulation of reactive oxygen species (ROS) production by C18 fatty acids in Jurkat and Raji cells. Clinical Science, v. 108, p. 245-253, 2005Tradução . . Disponível em: https://doi.org/10.1042/cs20040281. Acesso em: 18 nov. 2024.
APA
Cury-Boaventura, M. F., & Curi, R. (2005). Regulation of reactive oxygen species (ROS) production by C18 fatty acids in Jurkat and Raji cells. Clinical Science, 108, 245-253. doi:10.1042/cs20040281
NLM
Cury-Boaventura MF, Curi R. Regulation of reactive oxygen species (ROS) production by C18 fatty acids in Jurkat and Raji cells [Internet]. Clinical Science. 2005 ; 108 245-253.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20040281
Vancouver
Cury-Boaventura MF, Curi R. Regulation of reactive oxygen species (ROS) production by C18 fatty acids in Jurkat and Raji cells [Internet]. Clinical Science. 2005 ; 108 245-253.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs20040281
Artigo de periodico
Glutamine plays a role in superoxide production and the expression of p47 phox, p22phox and gp91phox in rat neutrophils (2002)
Pithon-Curi, Tania Cristina; Levada, Adriana C.; Lopes, Lucia Rossetti ; Doi, Sonia Q.; Curi, Rui
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PITHON-CURI, Tania Cristina et al. Glutamine plays a role in superoxide production and the expression of p47 phox, p22phox and gp91phox in rat neutrophils. Clinical Science, v. 103, p. 403-408, 2002Tradução . . Disponível em: https://doi.org/10.1042/cs1030403. Acesso em: 18 nov. 2024.
APA
Pithon-Curi, T. C., Levada, A. C., Lopes, L. R., Doi, S. Q., & Curi, R. (2002). Glutamine plays a role in superoxide production and the expression of p47 phox, p22phox and gp91phox in rat neutrophils. Clinical Science, 103, 403-408. doi:10.1042/cs1030403
NLM
Pithon-Curi TC, Levada AC, Lopes LR, Doi SQ, Curi R. Glutamine plays a role in superoxide production and the expression of p47 phox, p22phox and gp91phox in rat neutrophils [Internet]. Clinical Science. 2002 ;103 403-408.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs1030403
Vancouver
Pithon-Curi TC, Levada AC, Lopes LR, Doi SQ, Curi R. Glutamine plays a role in superoxide production and the expression of p47 phox, p22phox and gp91phox in rat neutrophils [Internet]. Clinical Science. 2002 ;103 403-408.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs1030403
Artigo de periodico
Effects of parathyroid hormone and calcium and their interrelationship on urinary acidification in the rat (1992)
Gil, F Z; Silva, V L C; Cavanal, M F; Malnic, Gerhard
Source: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GIL, F Z et al. Effects of parathyroid hormone and calcium and their interrelationship on urinary acidification in the rat. Clinical Science, v. 83, p. 711-5, 1992Tradução . . Disponível em: https://doi.org/10.1042/cs0830711. Acesso em: 18 nov. 2024.
APA
Gil, F. Z., Silva, V. L. C., Cavanal, M. F., & Malnic, G. (1992). Effects of parathyroid hormone and calcium and their interrelationship on urinary acidification in the rat. Clinical Science, 83, 711-5. doi:10.1042/cs0830711
NLM
Gil FZ, Silva VLC, Cavanal MF, Malnic G. Effects of parathyroid hormone and calcium and their interrelationship on urinary acidification in the rat [Internet]. Clinical Science. 1992 ;83 711-5.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs0830711
Vancouver
Gil FZ, Silva VLC, Cavanal MF, Malnic G. Effects of parathyroid hormone and calcium and their interrelationship on urinary acidification in the rat [Internet]. Clinical Science. 1992 ;83 711-5.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs0830711
Artigo de periodico
Bicarbonate reabsorption and electrophysiology of proximal tubules in uninephrectomized rats (1991)
Limongi, D M Z; Cassola, Antônio Carlos; Woronik, Viktória; Malnic, Gerhard
Source: Clinical Science. Unidades: ICB, FM
Subjects: FISIOLOGIA, FISIOLOGIA ANIMAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMONGI, D M Z et al. Bicarbonate reabsorption and electrophysiology of proximal tubules in uninephrectomized rats. Clinical Science, v. 81, n. 2 , p. 141-6, 1991Tradução . . Disponível em: https://doi.org/10.1042/cs0810141. Acesso em: 18 nov. 2024.
APA
Limongi, D. M. Z., Cassola, A. C., Woronik, V., & Malnic, G. (1991). Bicarbonate reabsorption and electrophysiology of proximal tubules in uninephrectomized rats. Clinical Science, 81( 2 ), 141-6. doi:10.1042/cs0810141
NLM
Limongi DMZ, Cassola AC, Woronik V, Malnic G. Bicarbonate reabsorption and electrophysiology of proximal tubules in uninephrectomized rats [Internet]. Clinical Science. 1991 ;81( 2 ): 141-6.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs0810141
Vancouver
Limongi DMZ, Cassola AC, Woronik V, Malnic G. Bicarbonate reabsorption and electrophysiology of proximal tubules in uninephrectomized rats [Internet]. Clinical Science. 1991 ;81( 2 ): 141-6.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1042/cs0810141
Artigo de periodico
Renal acidification defect induced by lithium in control and acidotic rats (1990)
Maclaughlin, M; Aires, Margarida de Mello
Source: Clinical Science. Unidade: ICB
Subjects: FISIOLOGIA, SISTEMA URINARIO (FISIOLOGIA)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACLAUGHLIN, M e AIRES, Margarida de Mello. Renal acidification defect induced by lithium in control and acidotic rats. Clinical Science, v. 79, p. 23-7, 1990Tradução . . Acesso em: 18 nov. 2024.
APA
Maclaughlin, M., & Aires, M. de M. (1990). Renal acidification defect induced by lithium in control and acidotic rats. Clinical Science, 79, 23-7.
NLM
Maclaughlin M, Aires M de M. Renal acidification defect induced by lithium in control and acidotic rats. Clinical Science. 1990 ;79 23-7.[citado 2024 nov. 18 ]
Vancouver
Maclaughlin M, Aires M de M. Renal acidification defect induced by lithium in control and acidotic rats. Clinical Science. 1990 ;79 23-7.[citado 2024 nov. 18 ]

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