Filtros : "ICB-BMB" "2013" Removidos: "Fundamenta Mathematicae" "TESTES DE HIPÓTESES" "Schonmann, Roberto Henrique" "Brasil" "dm" "sr" Limpar

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  • Source: Pharmacology, Biochemistry and Behavior. Unidade: ICB

    Assunto: FISIOLOGIA

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      GASPARINI, S. et al. Facilitation of sodium intake by combining noradrenaline into the lateral parabrachial nucleus with prazosin peripherally. Pharmacology, Biochemistry and Behavior, v. 111, p. 111-119, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.pbb.2013.08.017. Acesso em: 05 ago. 2024.
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      Gasparini, S., Gomide, J. M. C., Andrade-Franzé, G. M. F., Totola, L. T., De Luca Jr., L. A., Colombari, D. S. A., et al. (2013). Facilitation of sodium intake by combining noradrenaline into the lateral parabrachial nucleus with prazosin peripherally. Pharmacology, Biochemistry and Behavior, 111, 111-119. doi:10.1016/j.pbb.2013.08.017
    • NLM

      Gasparini S, Gomide JMC, Andrade-Franzé GMF, Totola LT, De Luca Jr. LA, Colombari DSA, De Paula PM, Moreira T dos S, Menani JV. Facilitation of sodium intake by combining noradrenaline into the lateral parabrachial nucleus with prazosin peripherally [Internet]. Pharmacology, Biochemistry and Behavior. 2013 ; 111 111-119.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.pbb.2013.08.017
    • Vancouver

      Gasparini S, Gomide JMC, Andrade-Franzé GMF, Totola LT, De Luca Jr. LA, Colombari DSA, De Paula PM, Moreira T dos S, Menani JV. Facilitation of sodium intake by combining noradrenaline into the lateral parabrachial nucleus with prazosin peripherally [Internet]. Pharmacology, Biochemistry and Behavior. 2013 ; 111 111-119.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.pbb.2013.08.017
  • Source: Hormone and Metabolic Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEHNEN, A. M. et al. Changes in renal glucose transporters in an animal model of metabolic syndrome. Hormone and Metabolic Research, v. 45, n. 11, p. 840-843 2013, 2013Tradução . . Disponível em: https://doi.org/10.1055/s-0033-1354381. Acesso em: 05 ago. 2024.
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      Lehnen, A. M., Leguisamo, N. M., Dias, L. D., Pinto, G. H., Okamoto, N. M., Machado, U. F., & Schaan, B. D. (2013). Changes in renal glucose transporters in an animal model of metabolic syndrome. Hormone and Metabolic Research, 45( 11), 840-843 2013. doi:10.1055/s-0033-1354381
    • NLM

      Lehnen AM, Leguisamo NM, Dias LD, Pinto GH, Okamoto NM, Machado UF, Schaan BD. Changes in renal glucose transporters in an animal model of metabolic syndrome [Internet]. Hormone and Metabolic Research. 2013 ; 45( 11): 840-843 2013.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1055/s-0033-1354381
    • Vancouver

      Lehnen AM, Leguisamo NM, Dias LD, Pinto GH, Okamoto NM, Machado UF, Schaan BD. Changes in renal glucose transporters in an animal model of metabolic syndrome [Internet]. Hormone and Metabolic Research. 2013 ; 45( 11): 840-843 2013.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1055/s-0033-1354381
  • Source: Islets. Unidade: ICB

    Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, INSULINA, SUPERÓXIDO DISMUTASE

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      GRACIANO, Maria Fernanda Rodrigues et al. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion. Islets, v. 5 n. 4, p. 139-148, 2013Tradução . . Disponível em: https://doi.org/10.4161/isl.25459. Acesso em: 05 ago. 2024.
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      Graciano, M. F. R., Valle, M. M. R., Curi, R., & Carpinelli, A. R. (2013). Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion. Islets, 5 n. 4, 139-148. doi:10.4161/isl.25459
    • NLM

      Graciano MFR, Valle MMR, Curi R, Carpinelli AR. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion [Internet]. Islets. 2013 ; 5 n. 4 139-148.[citado 2024 ago. 05 ] Available from: https://doi.org/10.4161/isl.25459
    • Vancouver

      Graciano MFR, Valle MMR, Curi R, Carpinelli AR. Evidence for the involvement of GPR40 and NADPH oxidase in palmitic acid-induced superoxide production and insulin secretion [Internet]. Islets. 2013 ; 5 n. 4 139-148.[citado 2024 ago. 05 ] Available from: https://doi.org/10.4161/isl.25459
  • Source: Natural Product Research. Unidade: ICB

    Subjects: FISIOLOGIA, PRODUTOS NATURAIS, INSULINA, FLAVONÓIDES

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      FELIPE, E. T. et al. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects. Natural Product Research, v. 27, n. 10, p. 925-928, 2013Tradução . . Disponível em: https://doi.org/10.1080/14786419.2012.671315. Acesso em: 05 ago. 2024.
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      Felipe, E. T., Maestri, J. S., Kanunfre, C. C., Curi, R., Newsholme, P., Carpinelli, A. R., & Oliveira-Emilio, H. R. (2013). Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects. Natural Product Research, 27( 10), 925-928. doi:10.1080/14786419.2012.671315
    • NLM

      Felipe ET, Maestri JS, Kanunfre CC, Curi R, Newsholme P, Carpinelli AR, Oliveira-Emilio HR. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects [Internet]. Natural Product Research. 2013 ; 27( 10): 925-928.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1080/14786419.2012.671315
    • Vancouver

      Felipe ET, Maestri JS, Kanunfre CC, Curi R, Newsholme P, Carpinelli AR, Oliveira-Emilio HR. Cytotoxicity and cytoprotective effects of citrus flavanoids on insulin-secreting cells BRIN-BD11: beneficial synergic effects [Internet]. Natural Product Research. 2013 ; 27( 10): 925-928.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1080/14786419.2012.671315
  • Source: Journal of Membrane Biology. Unidade: ICB

    Assunto: FISIOLOGIA

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      GEYER, R. Ryan et al. Relative CO2/NH3 Permeabilities of Human RhAG, RhBG and RhCG. Journal of Membrane Biology, v. 246, n. 12, p. 915-926, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00232-013-9593-0. Acesso em: 05 ago. 2024.
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      Geyer, R. R., Parker, M. D., Toye, A. M., Boron, W. F., & Musa-Aziz, R. (2013). Relative CO2/NH3 Permeabilities of Human RhAG, RhBG and RhCG. Journal of Membrane Biology, 246( 12), 915-926. doi:10.1007/s00232-013-9593-0
    • NLM

      Geyer RR, Parker MD, Toye AM, Boron WF, Musa-Aziz R. Relative CO2/NH3 Permeabilities of Human RhAG, RhBG and RhCG [Internet]. Journal of Membrane Biology. 2013 ; 246( 12): 915-926.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s00232-013-9593-0
    • Vancouver

      Geyer RR, Parker MD, Toye AM, Boron WF, Musa-Aziz R. Relative CO2/NH3 Permeabilities of Human RhAG, RhBG and RhCG [Internet]. Journal of Membrane Biology. 2013 ; 246( 12): 915-926.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s00232-013-9593-0
  • Source: Diabetology and Metabolic Syndrome. Unidade: ICB

    Assunto: FISIOLOGIA

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      SABINO-SILVA, Robinson et al. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, v. 5, n. 1, p. 1-5, 2013Tradução . . Disponível em: https://doi.org/10.1186/1758-5996-5-64. Acesso em: 05 ago. 2024.
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      Sabino-Silva, R., Okamoto, M. M., David-Silva, A., Mori, C. R., Freitas, H. S., & Machado, U. F. (2013). Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, 5( 1), 1-5. doi:10.1186/1758-5996-5-64
    • NLM

      Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1186/1758-5996-5-64
    • Vancouver

      Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1186/1758-5996-5-64
  • Source: Applied Physiology Nutrition and Metabolism - Physiologie Appliquee Nutrition et Metabolisme. Unidades: ICB, FCF

    Subjects: COMPOSIÇÃO CORPORAL, SUPLEMENTAÇÃO ALIMENTAR

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      PEDROSA, Rogerio Graça et al. Leucine supplementation increases serum insulin-like growth factor 1 concentration and liver protein/RNA ratio in rats after a period of nutritional recovery. Applied Physiology Nutrition and Metabolism - Physiologie Appliquee Nutrition et Metabolisme, v. 38, n. 6, p. 694-697, 2013Tradução . . Disponível em: https://doi.org/10.1139/apnm-2012-0440. Acesso em: 05 ago. 2024.
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      Pedrosa, R. G., Donato Junior, J., Pires, I. S. de O., & Tirapegui, J. (2013). Leucine supplementation increases serum insulin-like growth factor 1 concentration and liver protein/RNA ratio in rats after a period of nutritional recovery. Applied Physiology Nutrition and Metabolism - Physiologie Appliquee Nutrition et Metabolisme, 38( 6), 694-697. doi:10.1139/apnm-2012-0440
    • NLM

      Pedrosa RG, Donato Junior J, Pires IS de O, Tirapegui J. Leucine supplementation increases serum insulin-like growth factor 1 concentration and liver protein/RNA ratio in rats after a period of nutritional recovery [Internet]. Applied Physiology Nutrition and Metabolism - Physiologie Appliquee Nutrition et Metabolisme. 2013 ; 38( 6): 694-697.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1139/apnm-2012-0440
    • Vancouver

      Pedrosa RG, Donato Junior J, Pires IS de O, Tirapegui J. Leucine supplementation increases serum insulin-like growth factor 1 concentration and liver protein/RNA ratio in rats after a period of nutritional recovery [Internet]. Applied Physiology Nutrition and Metabolism - Physiologie Appliquee Nutrition et Metabolisme. 2013 ; 38( 6): 694-697.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1139/apnm-2012-0440
  • Source: Behavioural Brain Research. Unidade: ICB

    Subjects: ANATOMIA, FISIOLOGIA

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      FRANÇA, Nubia Regina Moreira et al. Antinociception induced by motor cortex stimulation: somatotopy of behavioral response and profile of neuronal activation. Behavioural Brain Research, v. 250, p. 211-221, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2013.05.019. Acesso em: 05 ago. 2024.
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      França, N. R. M., Toniolo, E. F., Franciosi, A. C., Alves, A. da S., Andrade, D. C. de, Fonoff, E. T., et al. (2013). Antinociception induced by motor cortex stimulation: somatotopy of behavioral response and profile of neuronal activation. Behavioural Brain Research, 250, 211-221. doi:10.1016/j.bbr.2013.05.019
    • NLM

      França NRM, Toniolo EF, Franciosi AC, Alves A da S, Andrade DC de, Fonoff ET, Britto LRG de, Dale CS. Antinociception induced by motor cortex stimulation: somatotopy of behavioral response and profile of neuronal activation [Internet]. Behavioural Brain Research. 2013 ; 250 211-221.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.bbr.2013.05.019
    • Vancouver

      França NRM, Toniolo EF, Franciosi AC, Alves A da S, Andrade DC de, Fonoff ET, Britto LRG de, Dale CS. Antinociception induced by motor cortex stimulation: somatotopy of behavioral response and profile of neuronal activation [Internet]. Behavioural Brain Research. 2013 ; 250 211-221.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.bbr.2013.05.019
  • Conference titles: Annual Congress of the European College of Sport Science (ECSS). Unidades: EEFE, EACH, ICB

    Subjects: FISIOLOGIA DO EXERCÍCIO, HIPOTENSÃO, HIPERTENSÃO, PRESSÃO SANGUÍNEA, TREINAMENTO AERÓBIO

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      BRITO, L et al. Post-exercise hypotension is greater in the morning than the evening in pre-hypertensive men. 2013, Anais.. Barcelona: Escola de Educação Física e Esporte, Universidade de São Paulo, 2013. Disponível em: http://www.ecss-congress.eu/2013/13/index.php/scientific-programme. Acesso em: 05 ago. 2024.
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      Brito, L., Rezende, R., Silva Junior, N. D. da, Menna Barreto, L. S., Tinucci, T., Cipolla Neto, J., & Forjaz, C. L. M. (2013). Post-exercise hypotension is greater in the morning than the evening in pre-hypertensive men. In . Barcelona: Escola de Educação Física e Esporte, Universidade de São Paulo. Recuperado de http://www.ecss-congress.eu/2013/13/index.php/scientific-programme
    • NLM

      Brito L, Rezende R, Silva Junior ND da, Menna Barreto LS, Tinucci T, Cipolla Neto J, Forjaz CLM. Post-exercise hypotension is greater in the morning than the evening in pre-hypertensive men [Internet]. 2013 ;[citado 2024 ago. 05 ] Available from: http://www.ecss-congress.eu/2013/13/index.php/scientific-programme
    • Vancouver

      Brito L, Rezende R, Silva Junior ND da, Menna Barreto LS, Tinucci T, Cipolla Neto J, Forjaz CLM. Post-exercise hypotension is greater in the morning than the evening in pre-hypertensive men [Internet]. 2013 ;[citado 2024 ago. 05 ] Available from: http://www.ecss-congress.eu/2013/13/index.php/scientific-programme
  • Source: The FASEB journal. Conference titles: ASPET Annual Meeting at EB (Experimental Biology). Unidades: EEFE, FM, ICB

    Subjects: FISIOLOGIA DO EXERCÍCIO, INSUFICIÊNCIA CARDÍACA

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      CORREA, Ligia M Antunes et al. Exercise training restores muscle mechano and metaboreflex sensitivity in heart failure patients. The FASEB journal. Bethesda: Escola de Educação Física e Esporte, Universidade de São Paulo. . Acesso em: 05 ago. 2024. , 2013
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      Correa, L. M. A., Nobre, T. S., Groehs, R. V., Alves, M. J. N. N., Rondon, M. U., Mady, C., et al. (2013). Exercise training restores muscle mechano and metaboreflex sensitivity in heart failure patients. The FASEB journal. Bethesda: Escola de Educação Física e Esporte, Universidade de São Paulo.
    • NLM

      Correa LMA, Nobre TS, Groehs RV, Alves MJNN, Rondon MU, Mady C, Almeida DR, Oliveira P, Lima MF, Mathias W, Brum PC, Rossoni LV, Oliveira EM, Middlekauff HR, Negrão CE. Exercise training restores muscle mechano and metaboreflex sensitivity in heart failure patients. The FASEB journal. 2013 ; 27[citado 2024 ago. 05 ]
    • Vancouver

      Correa LMA, Nobre TS, Groehs RV, Alves MJNN, Rondon MU, Mady C, Almeida DR, Oliveira P, Lima MF, Mathias W, Brum PC, Rossoni LV, Oliveira EM, Middlekauff HR, Negrão CE. Exercise training restores muscle mechano and metaboreflex sensitivity in heart failure patients. The FASEB journal. 2013 ; 27[citado 2024 ago. 05 ]
  • Source: International Journal of Cardiology. Unidades: ICB, FM

    Subjects: CÉLULAS-TRONCO (USO TERAPÊUTICO), BIOLOGIA MOLECULAR, MIOCARDIOPATIA HIPERTRÓFICA, MIOCÁRDIO, HEMODINÂMICA

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      SANTOS, Leonardo dos et al. Cell therapy prevents structural, functional and molecular remodeling of remote non-infarcted myocardium. International Journal of Cardiology, v. 168, n. 4, p. 3829-3836, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2013.06.026. Acesso em: 05 ago. 2024.
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      Santos, L. dos, Gonçalves, G. A., Davel, A. P., Santos, A. A., Krieger, J. E., Rossoni, L. V., & Tucci, P. J. F. (2013). Cell therapy prevents structural, functional and molecular remodeling of remote non-infarcted myocardium. International Journal of Cardiology, 168( 4), 3829-3836. doi:10.1016/j.ijcard.2013.06.026
    • NLM

      Santos L dos, Gonçalves GA, Davel AP, Santos AA, Krieger JE, Rossoni LV, Tucci PJF. Cell therapy prevents structural, functional and molecular remodeling of remote non-infarcted myocardium [Internet]. International Journal of Cardiology. 2013 ; 168( 4): 3829-3836.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2013.06.026
    • Vancouver

      Santos L dos, Gonçalves GA, Davel AP, Santos AA, Krieger JE, Rossoni LV, Tucci PJF. Cell therapy prevents structural, functional and molecular remodeling of remote non-infarcted myocardium [Internet]. International Journal of Cardiology. 2013 ; 168( 4): 3829-3836.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.ijcard.2013.06.026
  • Source: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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      REAL, Caroline Cristiano et al. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, v. 237, p. 118-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.01.060. Acesso em: 05 ago. 2024.
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      Real, C. C., Ferreira, A. F. B., Chaves-Kirsten, G. P., Torrão, A. da S., Pires, R. S., & Britto, L. R. G. de. (2013). BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, 237, 118-129. doi:10.1016/j.neuroscience.2013.01.060
    • NLM

      Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060
    • Vancouver

      Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060
  • Source: American Journal of Physiology, Endocrinology and Methabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      FARIA, Juliana A. et al. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, v. 305, n. 2, p. E230-E242, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2013. Acesso em: 05 ago. 2024.
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      Faria, J. A., Kinote, A., Ignacio-Souza, L. M., Araújo, T. M. de, Razolli, D. S., Doneda, D. L., et al. (2013). Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, 305( 2), E230-E242. doi:10.1152/ajpendo.00094.2013
    • NLM

      Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
    • Vancouver

      Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
  • Source: Neuropharmacology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 05 ago. 2024.
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      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Source: Journal of Neurotrauma. Unidades: FO, ICB

    Subjects: LASER NÃO CIRÚRGICO, NERVO TRIGÊMEO

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      MARTINS, Daniel de Oliveira et al. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins. Journal of Neurotrauma, v. 30, n. 6, p. 480-486, 2013Tradução . . Disponível em: https://doi.org/10.1089/neu.2012.2603. Acesso em: 05 ago. 2024.
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      Martins, D. de O., Santos, F. M. dos, Oliveira, M. E. de, Britto, L. R. G. de, Lemos, J. B. D., & Chacur, M. (2013). Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins. Journal of Neurotrauma, 30( 6), 480-486. doi:10.1089/neu.2012.2603
    • NLM

      Martins D de O, Santos FM dos, Oliveira ME de, Britto LRG de, Lemos JBD, Chacur M. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins [Internet]. Journal of Neurotrauma. 2013 ; 30( 6): 480-486.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1089/neu.2012.2603
    • Vancouver

      Martins D de O, Santos FM dos, Oliveira ME de, Britto LRG de, Lemos JBD, Chacur M. Laser therapy and the pain-related behavior after injury of the inferior alveolar nerve: possible involvement of neurotrophins [Internet]. Journal of Neurotrauma. 2013 ; 30( 6): 480-486.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1089/neu.2012.2603
  • Source: Experimental Brain Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      RIGHI, Luana Lira e RIBEIRO-DO-VALLE, Luiz Eduardo. Which factors are important for crossmodal attentional effect?. Experimental Brain Research, v. 225, n. 4, p. 491-498, 2013Tradução . . Disponível em: https://doi.org/10.1007/s00221-012-3389-9. Acesso em: 05 ago. 2024.
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      Righi, L. L., & Ribeiro-do-Valle, L. E. (2013). Which factors are important for crossmodal attentional effect? Experimental Brain Research, 225( 4), 491-498. doi:10.1007/s00221-012-3389-9
    • NLM

      Righi LL, Ribeiro-do-Valle LE. Which factors are important for crossmodal attentional effect? [Internet]. Experimental Brain Research. 2013 ; 225( 4): 491-498.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s00221-012-3389-9
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      Righi LL, Ribeiro-do-Valle LE. Which factors are important for crossmodal attentional effect? [Internet]. Experimental Brain Research. 2013 ; 225( 4): 491-498.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s00221-012-3389-9
  • Source: Cellular and Molecular Neurobiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      LEONELLI, Mauro e MARTINS, Daniel O. e BRITTO, Luiz Roberto Giorgetti de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases. Cellular and Molecular Neurobiology, v. 33, n. 3, p. 379-392, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10571-012-9904-5. Acesso em: 05 ago. 2024.
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      Leonelli, M., Martins, D. O., & Britto, L. R. G. de. (2013). Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases. Cellular and Molecular Neurobiology, 33( 3), 379-392. doi:10.1007/s10571-012-9904-5
    • NLM

      Leonelli M, Martins DO, Britto LRG de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases [Internet]. Cellular and Molecular Neurobiology. 2013 ; 33( 3): 379-392.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s10571-012-9904-5
    • Vancouver

      Leonelli M, Martins DO, Britto LRG de. Retinal cell death induced by TRPV1 activation involves NMDA signaling and upregulation of nitric oxide synthases [Internet]. Cellular and Molecular Neurobiology. 2013 ; 33( 3): 379-392.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1007/s10571-012-9904-5
  • Source: Pathophysiology. Unidades: ICB, FM

    Subjects: LEUCÓCITOS, MODELOS ANIMAIS, FALÊNCIA MÚLTIPLA DOS ÓRGÃOS, PULMÃO, FÍGADO, ESTRESSE OXIDATIVO, RATOS WISTAR

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      SGARBI, Mauricio Wanderley Moral et al. Leukocyte infiltration in lung, muscle, and liver after limb compression in rats. Pathophysiology, v. 20, n. 2, p. 111-116, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.pathophys.2012.12.001. Acesso em: 05 ago. 2024.
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      Sgarbi, M. W. M., Silva Jr., B. A., Peres, C. M., Alba, T. C., Curi, R., Soriano, F. G., et al. (2013). Leukocyte infiltration in lung, muscle, and liver after limb compression in rats. Pathophysiology, 20( 2), 111-116. doi:10.1016/j.pathophys.2012.12.001
    • NLM

      Sgarbi MWM, Silva Jr. BA, Peres CM, Alba TC, Curi R, Soriano FG, Ribeiro DA, Velasco IT. Leukocyte infiltration in lung, muscle, and liver after limb compression in rats [Internet]. Pathophysiology. 2013 ; 20( 2): 111-116.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.pathophys.2012.12.001
    • Vancouver

      Sgarbi MWM, Silva Jr. BA, Peres CM, Alba TC, Curi R, Soriano FG, Ribeiro DA, Velasco IT. Leukocyte infiltration in lung, muscle, and liver after limb compression in rats [Internet]. Pathophysiology. 2013 ; 20( 2): 111-116.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.pathophys.2012.12.001
  • Source: PLOS ONE. Unidades: FCF, ICB

    Subjects: AMINOÁCIDOS, MÚSCULO ESQUELÉTICO, EXPRESSÃO GÊNICA, SUPLEMENTAÇÃO ALIMENTAR

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      ZAMPIEIR, Thais T et al. Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus. PLOS ONE, v. 8, n. 12, p. 1-13 art. e84094, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0084094. Acesso em: 05 ago. 2024.
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      Zampieir, T. T., Pedroso, J. A. B., Furigo, I. C., Tirapegui, J., & Donato Junior, J. (2013). Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus. PLOS ONE, 8( 12), 1-13 art. e84094. doi:10.1371/journal.pone.0084094
    • NLM

      Zampieir TT, Pedroso JAB, Furigo IC, Tirapegui J, Donato Junior J. Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus [Internet]. PLOS ONE. 2013 ; 8( 12): 1-13 art. e84094.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1371/journal.pone.0084094
    • Vancouver

      Zampieir TT, Pedroso JAB, Furigo IC, Tirapegui J, Donato Junior J. Oral leucine supplementation is sensed by the brain but neither reduces food intake nor induces an anorectic pattern of gene expression in the hypothalamus [Internet]. PLOS ONE. 2013 ; 8( 12): 1-13 art. e84094.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1371/journal.pone.0084094
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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      SILVA, Ariel S. da et al. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, v. 91, n. 24-26, p. 1174-1179, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.05.001. Acesso em: 05 ago. 2024.
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      Silva, A. S. da, Dias, L. D., Borges, J. F., Markoski, M. M., Souza, M. S. de, Irigoyen, M. C., et al. (2013). Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, 91( 24-26), 1174-1179. doi:10.1016/j.lfs.2013.05.001
    • NLM

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001
    • Vancouver

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 ago. 05 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001

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