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  • Artigo de periodico

    The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids (2019)

    Felizardo, Raphael José Ferreira; Watanabe, Ingrid Kazue Mizuno; Dardi, Patrizia; Rossoni, Luciana Venturini ; Câmara, Niels Olsen Saraiva

    Fonte: Pharmacological Research. Unidade: ICB

    Assuntos: FISIOLOGIA, IMUNOLOGIA, NEFROPATIAS, HIPERTENSÃO, INFLAMAÇÃO, ÁCIDOS GRAXOS, METABÓLITOS, TOXINAS, ENDOTOXINAS

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    • ABNT

      FELIZARDO, Raphael José Ferreira et al. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids. Pharmacological Research, v. 141, p. 366-377, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2019.01.019. Acesso em: 01 jul. 2024.

    • APA

      Felizardo, R. J. F., Watanabe, I. K. M., Dardi, P., Rossoni, L. V., & Câmara, N. O. S. (2019). The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids. Pharmacological Research, 141, 366-377. doi:10.1016/j.phrs.2019.01.019

    • NLM

      Felizardo RJF, Watanabe IKM, Dardi P, Rossoni LV, Câmara NOS. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids [Internet]. Pharmacological Research. 2019 ; 141 366-377.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.phrs.2019.01.019

    • Vancouver

      Felizardo RJF, Watanabe IKM, Dardi P, Rossoni LV, Câmara NOS. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids [Internet]. Pharmacological Research. 2019 ; 141 366-377.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.phrs.2019.01.019

  • Artigo de periodico

    Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons (2015)

    Leiria, Luiz O. S.; Arantes-Costa, Fernanda M.; Calixto, Marina C.; Alexandre, Eduardo C.; Moura, Rodrigo F.; Folli, Franco; Prado, Carla M.; Prado, Marco Antonio; Prado, Vania F.; Velloso, Licio A.; Donato Junior, José ; Antunes, Edson; Martins, Milton A.; Saad, Mario J. A.

    Fonte: Cell Reports. Unidades: FM, ICB

    Assuntos: TRANSTORNOS DO METABOLISMO DE GLUCOSE, NEURÔNIOS, OBESIDADE, ASMA, FATORES DE RISCO, TRONCO CEREBRAL, INSULINA

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    • ABNT

      LEIRIA, Luiz O. S. et al. Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons. Cell Reports, v. 11, n. 6, p. 934-943, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2015.04.012. Acesso em: 01 jul. 2024.

    • APA

      Leiria, L. O. S., Arantes-Costa, F. M., Calixto, M. C., Alexandre, E. C., Moura, R. F., Folli, F., et al. (2015). Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons. Cell Reports, 11( 6), 934-943. doi:10.1016/j.celrep.2015.04.012

    • NLM

      Leiria LOS, Arantes-Costa FM, Calixto MC, Alexandre EC, Moura RF, Folli F, Prado CM, Prado MA, Prado VF, Velloso LA, Donato Junior J, Antunes E, Martins MA, Saad MJA. Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons [Internet]. Cell Reports. 2015 ; 11( 6): 934-943.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.celrep.2015.04.012

    • Vancouver

      Leiria LOS, Arantes-Costa FM, Calixto MC, Alexandre EC, Moura RF, Folli F, Prado CM, Prado MA, Prado VF, Velloso LA, Donato Junior J, Antunes E, Martins MA, Saad MJA. Increased Airway Reactivity and Hyperinsulinemia in Obese Mice Are Linked by ERK Signaling in Brain Stem Cholinergic Neurons [Internet]. Cell Reports. 2015 ; 11( 6): 934-943.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.celrep.2015.04.012

  • Artigo de periodico

    The role of Nox2-derived ROS in the development of cognitive impairment after sepsis (2014)

    Hernandes, Marina S.; D'Ávila, Joana C.; Trevelin, Silvia C.; Reis, Patricia A.; Kinjo, Erika R.; Lopes, Lucia Rossetti ; Castro-Faria-Neto, Hugo C.; Cunha, Fernando de Queiroz; Britto, Luiz Roberto Giorgetti de ; Bozza, Fernando A.

    Fonte: Journal of Neuroinflammation. Unidades: ICB, FMRP

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      HERNANDES, Marina S. et al. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, v. 11, n. 01, p. 01-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-36. Acesso em: 01 jul. 2024.

    • APA

      Hernandes, M. S., D'Ávila, J. C., Trevelin, S. C., Reis, P. A., Kinjo, E. R., Lopes, L. R., et al. (2014). The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, 11( 01), 01-12. doi:10.1186/1742-2094-11-36

    • NLM

      Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1186/1742-2094-11-36

    • Vancouver

      Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1186/1742-2094-11-36

  • Artigo de periodico

    The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure (2014)

    Ruchaya, P. J.; Antunes, Vagner Roberto ; Paton, J. F. R.; Murphy, D.; Yao, S. T.

    Fonte: Experimental Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      RUCHAYA, P. J. et al. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, v. 99, n. 1, p. 111-122, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.075432. Acesso em: 01 jul. 2024.

    • APA

      Ruchaya, P. J., Antunes, V. R., Paton, J. F. R., Murphy, D., & Yao, S. T. (2014). The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, 99( 1), 111-122. doi:10.1113/expphysiol.2013.075432

    • NLM

      Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432

    • Vancouver

      Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432

  • Artigo de periodico

    Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats (2014)

    Fontes, M. T.; Silva, T. L. B. T.; Mota, M. M.; Barreto, A. S.; Rossoni, Luciana Venturini ; Santos, M. R. V.

    Fonte: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      FONTES, M. T. et al. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, v. 94, n. 1, p. 24-29, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.017. Acesso em: 01 jul. 2024.

    • APA

      Fontes, M. T., Silva, T. L. B. T., Mota, M. M., Barreto, A. S., Rossoni, L. V., & Santos, M. R. V. (2014). Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, 94( 1), 24-29. doi:10.1016/j.lfs.2013.11.017

    • NLM

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017

    • Vancouver

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017

  • Artigo de periodico

    Possible crosstalk between leptin and proclactin during pregnancy (2014)

    Nagaishi, V. S.; Cardinali, L. I.; Zampieri, Thais Tessari; Furigo, Isadora Clivatti; Metzger, Martin Andreas ; Donato Junior, José

    Fonte: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      NAGAISHI, V. S. et al. Possible crosstalk between leptin and proclactin during pregnancy. Neuroscience, v. 259, p. 71-83, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.11.050. Acesso em: 01 jul. 2024.

    • APA

      Nagaishi, V. S., Cardinali, L. I., Zampieri, T. T., Furigo, I. C., Metzger, M. A., & Donato Junior, J. (2014). Possible crosstalk between leptin and proclactin during pregnancy. Neuroscience, 259, 71-83. doi:10.1016/j.neuroscience.2013.11.050

    • NLM

      Nagaishi VS, Cardinali LI, Zampieri TT, Furigo IC, Metzger MA, Donato Junior J. Possible crosstalk between leptin and proclactin during pregnancy [Internet]. Neuroscience. 2014 ; 259 71-83.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.11.050

    • Vancouver

      Nagaishi VS, Cardinali LI, Zampieri TT, Furigo IC, Metzger MA, Donato Junior J. Possible crosstalk between leptin and proclactin during pregnancy [Internet]. Neuroscience. 2014 ; 259 71-83.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.11.050

  • Artigo de periodico

    Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats (2013)

    Sabino-Silva, Robinson; Okamoto, Maristela Mitiko; David-Silva, Aline; Mori, Cristina Rosana; Freitas, Helayne Soares; Machado, Ubiratan Fabres

    Fonte: Diabetology and Metabolic Syndrome. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      SABINO-SILVA, Robinson et al. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, v. 5, n. 1, p. 1-5, 2013Tradução . . Disponível em: https://doi.org/10.1186/1758-5996-5-64. Acesso em: 01 jul. 2024.

    • APA

      Sabino-Silva, R., Okamoto, M. M., David-Silva, A., Mori, C. R., Freitas, H. S., & Machado, U. F. (2013). Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, 5( 1), 1-5. doi:10.1186/1758-5996-5-64

    • NLM

      Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1186/1758-5996-5-64

    • Vancouver

      Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1186/1758-5996-5-64

  • Artigo de periodico

    BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease (2013)

    Real, Caroline Cristiano; Ferreira, A. F. B.; Chaves-Kirsten, Gabriela Pena; Torrão, Andréa da Silva ; Pires, R. S.; Britto, Luiz Roberto Giorgetti de

    Fonte: Neuroscience. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      REAL, Caroline Cristiano et al. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, v. 237, p. 118-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.01.060. Acesso em: 01 jul. 2024.

    • APA

      Real, C. C., Ferreira, A. F. B., Chaves-Kirsten, G. P., Torrão, A. da S., Pires, R. S., & Britto, L. R. G. de. (2013). BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, 237, 118-129. doi:10.1016/j.neuroscience.2013.01.060

    • NLM

      Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060

    • Vancouver

      Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060

  • Artigo de periodico

    Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells (2013)

    Lima, Larissa de Sá; Kawamoto, Elisa Mitiko ; Munhoz, Carolina Demarchi ; Kinoshita, Paula Fernanda; Orellana, Ana Maria Marques; Curi, Rui; Rossoni, Luciana Venturini ; Avellar, M. C. W.; Scavone, Cristoforo

    Fonte: Neuropharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 01 jul. 2024.

    • APA

      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006

    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006

    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006

  • Artigo de periodico

    Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats (2013)

    Silva, Ariel S. da; Dias, Lucinara D.; Borges, Júlia F.; Markoski, Melissa M.; Souza, Martina S. de; Irigoyen, Maria C.; Machado, Ubiratan Fabres ; Schaan, Beatriz D.

    Fonte: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      SILVA, Ariel S. da et al. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, v. 91, n. 24-26, p. 1174-1179, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.05.001. Acesso em: 01 jul. 2024.

    • APA

      Silva, A. S. da, Dias, L. D., Borges, J. F., Markoski, M. M., Souza, M. S. de, Irigoyen, M. C., et al. (2013). Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, 91( 24-26), 1174-1179. doi:10.1016/j.lfs.2013.05.001

    • NLM

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001

    • Vancouver

      Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001

  • Artigo de periodico-carta/editorial

    SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] (2013)

    Corrêa-Giannella, Maria Lucia; Machado, Ubiratan Fabres

    Fonte: Pharmacogenomics. Unidades: FM, ICB

    Assuntos: INSULINA (RESISTÊNCIA), DIABETES MELLITUS, FARMACOLOGIA CLÍNICA, GENES

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    • ABNT

      CORRÊA-GIANNELLA, Maria Lucia e MACHADO, Ubiratan Fabres. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial]. Pharmacogenomics. London: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45. Acesso em: 01 jul. 2024. , 2013

    • APA

      Corrêa-Giannella, M. L., & Machado, U. F. (2013). SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial]. Pharmacogenomics. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.2217/pgs.10.93

    • NLM

      Corrêa-Giannella ML, Machado UF. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] [Internet]. Pharmacogenomics. 2013 ; 14( 8): 847-850.[citado 2024 jul. 01 ] Available from: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45

    • Vancouver

      Corrêa-Giannella ML, Machado UF. SLC2A4gene: a promising target for pharmacogenomics of insulin resistance [Editorial] [Internet]. Pharmacogenomics. 2013 ; 14( 8): 847-850.[citado 2024 jul. 01 ] Available from: http://www.futuremedicine.com/doi/pdf/10.2217/pgs.13.45

  • Artigo de periodico

    Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats (2013)

    Blanco-Rivero, Javier; Roque, Fernanda R.; Sastre, Esther; Caracuel, Laura; Couto, Gisele Kruger; Avendaño, María Soledad; Paula, Suliana Mesquita; Rossoni, Luciana Venturini ; Salaices, Mercedes; Balfagón, Gloria

    Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      BLANCO-RIVERO, Javier et al. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, v. 31, n. 5, p. 916-926, 2013Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32835f749c. Acesso em: 01 jul. 2024.

    • APA

      Blanco-Rivero, J., Roque, F. R., Sastre, E., Caracuel, L., Couto, G. K., Avendaño, M. S., et al. (2013). Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, 31( 5), 916-926. doi:10.1097/HJH.0b013e32835f749c

    • NLM

      Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c

    • Vancouver

      Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c

  • Artigo de periodico

    Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland (2013)

    Villela, Darine; Lima, Larissa de Sá; Peres, Rafael; Peliciari-Garcia, Rodrigo Antonio; Amaral, Fernanda Gaspar do; Cipolla-Neto, José; Scavone, Cristoforo ; Afeche, Solange Castro

    Fonte: Life Sciences. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      VILLELA, Darine et al. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, v. 94, n. 2, p. 122-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.004. Acesso em: 01 jul. 2024.

    • APA

      Villela, D., Lima, L. de S., Peres, R., Peliciari-Garcia, R. A., Amaral, F. G. do, Cipolla-Neto, J., et al. (2013). Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, 94( 2), 122-129. doi:10.1016/j.lfs.2013.11.004

    • NLM

      Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004

    • Vancouver

      Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004

  • Artigo de periodico

    Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine (2013)

    Totola, Leonardo Tedesco; Alves, T. B.; Takakura, Ana Carolina ; Ferreira-Neto, Hildebrando Candido; Antunes, Vagner Roberto ; Menani, J. V.; Colombari, E.; Moreira, Thiago dos Santos

    Fonte: Neuroscience. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      TOTOLA, Leonardo Tedesco et al. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine. Neuroscience, v. 250, p. 80-91, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.06.065. Acesso em: 01 jul. 2024.

    • APA

      Totola, L. T., Alves, T. B., Takakura, A. C., Ferreira-Neto, H. C., Antunes, V. R., Menani, J. V., et al. (2013). Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine. Neuroscience, 250, 80-91. doi:10.1016/j.neuroscience.2013.06.065

    • NLM

      Totola LT, Alves TB, Takakura AC, Ferreira-Neto HC, Antunes VR, Menani JV, Colombari E, Moreira T dos S. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine [Internet]. Neuroscience. 2013 ; 250 80-91.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.06.065

    • Vancouver

      Totola LT, Alves TB, Takakura AC, Ferreira-Neto HC, Antunes VR, Menani JV, Colombari E, Moreira T dos S. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine [Internet]. Neuroscience. 2013 ; 250 80-91.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.06.065

  • Artigo de periodico

    Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets (2013)

    Marçal, Anderson Carlos; Camporez, João Paulo Gabriel; Lima-Salgado, Thais M.; Cintra, D. E.; Akamine, Eliana Hiromi ; Ribeiro, L. M.; Almeida, F. N.; Zanuto, R. P.; Curi, Rui; Boldrini, Silvia de Campos; Liberti, Edson Aparecido; Fiamoncini, Jarlei ; Hirabara, S. M.; Deschamps, F. C.; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira

    Fonte: British Journal of Nutrition. Unidade: ICB

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      MARÇAL, Anderson Carlos et al. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, v. 109, n. 12, p. 2154-2165, 2013Tradução . . Disponível em: https://doi.org/10.1017/S0007114512004576. Acesso em: 01 jul. 2024.

    • APA

      Marçal, A. C., Camporez, J. P. G., Lima-Salgado, T. M., Cintra, D. E., Akamine, E. H., Ribeiro, L. M., et al. (2013). Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, 109( 12), 2154-2165. doi:10.1017/S0007114512004576

    • NLM

      Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1017/S0007114512004576

    • Vancouver

      Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1017/S0007114512004576

  • Artigo de periodico

    Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats (2013)

    David-Silva, Aline; Freitas, Helayne Soares de; Okamoto, Maristela M.; Sabino-Silva, Robinson; Schaan, Beatriz D.; Machado, Ubiratan Fabres

    Fonte: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      DAVID-SILVA, Aline et al. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, v. 93, n. 22, p. 805-813, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.10.011. Acesso em: 01 jul. 2024.

    • APA

      David-Silva, A., Freitas, H. S. de, Okamoto, M. M., Sabino-Silva, R., Schaan, B. D., & Machado, U. F. (2013). Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, 93( 22), 805-813. doi:10.1016/j.lfs.2013.10.011

    • NLM

      David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011

    • Vancouver

      David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011

  • Artigo de periodico-carta/editorial

    Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? (2013)

    Moreira, Thiago dos Santos ; Menani, Jose V.; Colombari, Eduardo; Takakura, Ana Carolina

    Fonte: Journal of Physiology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      MOREIRA, Thiago dos Santos et al. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat?. Journal of Physiology. Londres: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1113/jphysiol.2012.244640. Acesso em: 01 jul. 2024. , 2013

    • APA

      Moreira, T. dos S., Menani, J. V., Colombari, E., & Takakura, A. C. (2013). Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? Journal of Physiology. Londres: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1113/jphysiol.2012.244640

    • NLM

      Moreira T dos S, Menani JV, Colombari E, Takakura AC. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? [Internet]. Journal of Physiology. 2013 ; 591( 3): 745-746.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/jphysiol.2012.244640

    • Vancouver

      Moreira T dos S, Menani JV, Colombari E, Takakura AC. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? [Internet]. Journal of Physiology. 2013 ; 591( 3): 745-746.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/jphysiol.2012.244640

  • Artigo de periodico

    Control of breathing and blood pressure by parafacial neurons in conscious rats (2013)

    Takakura, Ana Carolina ; Moreira, Thiago dos Santos ; De Paula, Patrícia M.; Menani, José V.; Colombari, Eduardo

    Fonte: Experimental Physiology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      TAKAKURA, Ana Carolina et al. Control of breathing and blood pressure by parafacial neurons in conscious rats. Experimental Physiology, v. 98, n. 1, p. 304-315, 2013Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2012.065128. Acesso em: 01 jul. 2024.

    • APA

      Takakura, A. C., Moreira, T. dos S., De Paula, P. M., Menani, J. V., & Colombari, E. (2013). Control of breathing and blood pressure by parafacial neurons in conscious rats. Experimental Physiology, 98( 1), 304-315. doi:10.1113/expphysiol.2012.065128

    • NLM

      Takakura AC, Moreira T dos S, De Paula PM, Menani JV, Colombari E. Control of breathing and blood pressure by parafacial neurons in conscious rats [Internet]. Experimental Physiology. 2013 ; 98( 1): 304-315.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/expphysiol.2012.065128

    • Vancouver

      Takakura AC, Moreira T dos S, De Paula PM, Menani JV, Colombari E. Control of breathing and blood pressure by parafacial neurons in conscious rats [Internet]. Experimental Physiology. 2013 ; 98( 1): 304-315.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1113/expphysiol.2012.065128

  • Artigo de periodico

    Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells (2013)

    Costa, E. S.; Faiad, O. J.; Landgraf, R. G.; Ferreira, A. K.; Brigatte, P.; Curi, Rui; Cury, Y.

    Fonte: Toxicon. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      COSTA, E. S. et al. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, v. 74, p. 167-178, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2013.08.057. Acesso em: 01 jul. 2024.

    • APA

      Costa, E. S., Faiad, O. J., Landgraf, R. G., Ferreira, A. K., Brigatte, P., Curi, R., & Cury, Y. (2013). Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, 74, 167-178. doi:10.1016/j.toxicon.2013.08.057

    • NLM

      Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057

    • Vancouver

      Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057

  • Artigo de periodico

    Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition (2013)

    Donato Junior, José ; Lee. C; Ratra, D. V.; Franci, Celso Rodrigues; Canteras, Newton Sabino ; Elias, C. F.

    Fonte: Neuroscience. Unidades: ICB, FMRP

    Assuntos: ANATOMIA, FISIOLOGIA

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    • ABNT

      DONATO JUNIOR, José et al. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, v. 241, p. 67-79, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.03.013. Acesso em: 01 jul. 2024.

    • APA

      Donato Junior, J., Lee. C,, Ratra, D. V., Franci, C. R., Canteras, N. S., & Elias, C. F. (2013). Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition. Neuroscience, 241, 67-79. doi:10.1016/j.neuroscience.2013.03.013

    • NLM

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013

    • Vancouver

      Donato Junior J, Lee. C, Ratra DV, Franci CR, Canteras NS, Elias CF. Lesions of the ventral premammillary nucleus disrupt the dynamic changes in kiss1 and gnrh expression characteristic of the proestrus–estrus transition [Internet]. Neuroscience. 2013 ; 241 67-79.[citado 2024 jul. 01 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.03.013

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