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Artigo de periodico
Low-Load Resistance Exercise with Blood Flow Restriction Increases Hypoxia-Induced Angiogenic Genes Expression (2022)
Fernandes, Rodrigo Volga; Tricoli, Valmor ; Soares, Antonio Garcia; Miyabara, Elen Haruka ; Aoki, Marcelo Saldanha ; Laurentino, Gilberto Cândido
Source: Journal of human kinetics. Unidades: EEFE, ICB, EACH
Subjects: TREINAMENTO DE FORÇA, PRESSÃO SANGUÍNEA, MÚSCULO ESQUELÉTICO, BIOMARCADORES, VASODILATAÇÃO
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FERNANDES, Rodrigo Volga et al. Low-Load Resistance Exercise with Blood Flow Restriction Increases Hypoxia-Induced Angiogenic Genes Expression. Journal of human kinetics, v. 84, n. 1, p. 82-91, 2022Tradução . . Disponível em: https://doi.org/10.2478/hukin-2022-0101. Acesso em: 10 nov. 2024.
APA
Fernandes, R. V., Tricoli, V., Soares, A. G., Miyabara, E. H., Aoki, M. S., & Laurentino, G. C. (2022). Low-Load Resistance Exercise with Blood Flow Restriction Increases Hypoxia-Induced Angiogenic Genes Expression. Journal of human kinetics, 84( 1), 82-91. doi:10.2478/hukin-2022-0101
NLM
Fernandes RV, Tricoli V, Soares AG, Miyabara EH, Aoki MS, Laurentino GC. Low-Load Resistance Exercise with Blood Flow Restriction Increases Hypoxia-Induced Angiogenic Genes Expression [Internet]. Journal of human kinetics. 2022 ; 84( 1): 82-91.[citado 2024 nov. 10 ] Available from: https://doi.org/10.2478/hukin-2022-0101
Vancouver
Fernandes RV, Tricoli V, Soares AG, Miyabara EH, Aoki MS, Laurentino GC. Low-Load Resistance Exercise with Blood Flow Restriction Increases Hypoxia-Induced Angiogenic Genes Expression [Internet]. Journal of human kinetics. 2022 ; 84( 1): 82-91.[citado 2024 nov. 10 ] Available from: https://doi.org/10.2478/hukin-2022-0101
Artigo de periodico-carta/editorial
Correction to: Muscle stem cell function is impaired in β2-adrenoceptor knockout mice (2022)
Koike, Tatiana Emy ; Fuziwar, Cesar Seigi ; Brum, Patrícia Chakur ; Kimura, Edna Teruko ; Rando, Thomas A. ; Miyabara, Elen Haruka
Source: Stem Cell Reviews and Reports. Unidades: EEFE, ICB
Subjects: ANATOMIA, BIOLOGIA CELULAR, CAMUNDONGOS, ADRENÉRGICOS, ADRENORECEPTORES, PROLIFERAÇÃO CELULAR, CÉLULAS-TRONCO, MÚSCULO ESQUELÉTICO, EXPRESSÃO GÊNICA
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KOIKE, Tatiana Emy et al. Correction to: Muscle stem cell function is impaired in β2-adrenoceptor knockout mice. Stem Cell Reviews and Reports. Heidelberg: Escola de Educação Física e Esporte, Universidade de São Paulo. Disponível em: https://doi.org/10.1007/s12015-022-10372-6. Acesso em: 10 nov. 2024. , 2022
APA
Koike, T. E., Fuziwar, C. S., Brum, P. C., Kimura, E. T., Rando, T. A., & Miyabara, E. H. (2022). Correction to: Muscle stem cell function is impaired in β2-adrenoceptor knockout mice. Stem Cell Reviews and Reports. Heidelberg: Escola de Educação Física e Esporte, Universidade de São Paulo. doi:10.1007/s12015-022-10372-6
NLM
Koike TE, Fuziwar CS, Brum PC, Kimura ET, Rando TA, Miyabara EH. Correction to: Muscle stem cell function is impaired in β2-adrenoceptor knockout mice [Internet]. Stem Cell Reviews and Reports. 2022 ;1 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12015-022-10372-6
Vancouver
Koike TE, Fuziwar CS, Brum PC, Kimura ET, Rando TA, Miyabara EH. Correction to: Muscle stem cell function is impaired in β2-adrenoceptor knockout mice [Internet]. Stem Cell Reviews and Reports. 2022 ;1 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12015-022-10372-6
Artigo de periodico
Urocortin 2 promotes hypertrophy and enhances skeletal muscle function through cAMP and insulin/IGF-1 signaling pathways (2022)
Silva, Natalia Lautherbach Ennes da ; Gonçalves, Dawit Albieiro Pinheiro; Silveira, Wilian de Assis ; Gomes, Sílvia de Paula; Valentim, Rafael Rossi ; Zanon, Neusa Maria ; Pereira, Marcelo Gomes ; Miyabara, Elen Haruka ; Navegantes, Luiz Carlos Carvalho ; Kettelhut, Isis do Carmo
Source: Molecular Metabolism. Unidades: FMRP, ICB, EEFE
Assunto: ANATOMIA
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SILVA, Natalia Lautherbach Ennes da et al. Urocortin 2 promotes hypertrophy and enhances skeletal muscle function through cAMP and insulin/IGF-1 signaling pathways. Molecular Metabolism, v. 60, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2022.101492. Acesso em: 10 nov. 2024.
APA
Silva, N. L. E. da, Gonçalves, D. A. P., Silveira, W. de A., Gomes, S. de P., Valentim, R. R., Zanon, N. M., et al. (2022). Urocortin 2 promotes hypertrophy and enhances skeletal muscle function through cAMP and insulin/IGF-1 signaling pathways. Molecular Metabolism, 60, 1-16. doi:10.1016/j.molmet.2022.101492
NLM
Silva NLE da, Gonçalves DAP, Silveira W de A, Gomes S de P, Valentim RR, Zanon NM, Pereira MG, Miyabara EH, Navegantes LCC, Kettelhut I do C. Urocortin 2 promotes hypertrophy and enhances skeletal muscle function through cAMP and insulin/IGF-1 signaling pathways [Internet]. Molecular Metabolism. 2022 ; 60 1-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.molmet.2022.101492
Vancouver
Silva NLE da, Gonçalves DAP, Silveira W de A, Gomes S de P, Valentim RR, Zanon NM, Pereira MG, Miyabara EH, Navegantes LCC, Kettelhut I do C. Urocortin 2 promotes hypertrophy and enhances skeletal muscle function through cAMP and insulin/IGF-1 signaling pathways [Internet]. Molecular Metabolism. 2022 ; 60 1-16.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.molmet.2022.101492
Artigo de periodico
Aerobic exercise training and in vivo akt activation counteract cancer cachexia by inducing a hypertrophic profile through eIF-2α modulation (2022)
Pereira, Marcelo Gomes ; Voltarelli, Vanessa Azevedo ; Tobias, Gabriel Cardial ; Souza, Lara de; Borges, Gabriela Silva ; Paixão, Ailma Oliveira da ; Bowen, Thomas Scott; Demasi, Marilene ; Miyabara, Elen Haruka ; Brum, Patricia Chakur
Source: Cancers. Unidades: EEFE, ICB
Subjects: ANATOMIA, EXERCÍCIO FÍSICO, CAQUEXIA, MÚSCULO ESQUELÉTICO, PERDA DE MASSA, PERDA DE PESO, COMORBIDADE
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PEREIRA, Marcelo Gomes et al. Aerobic exercise training and in vivo akt activation counteract cancer cachexia by inducing a hypertrophic profile through eIF-2α modulation. Cancers, v. 14, n. 1, p. 1-20, 2022Tradução . . Disponível em: https://doi.org/10.3390/cancers14010028. Acesso em: 10 nov. 2024.
APA
Pereira, M. G., Voltarelli, V. A., Tobias, G. C., Souza, L. de, Borges, G. S., Paixão, A. O. da, et al. (2022). Aerobic exercise training and in vivo akt activation counteract cancer cachexia by inducing a hypertrophic profile through eIF-2α modulation. Cancers, 14( 1), 1-20. doi:10.3390/cancers14010028
NLM
Pereira MG, Voltarelli VA, Tobias GC, Souza L de, Borges GS, Paixão AO da, Bowen TS, Demasi M, Miyabara EH, Brum PC. Aerobic exercise training and in vivo akt activation counteract cancer cachexia by inducing a hypertrophic profile through eIF-2α modulation [Internet]. Cancers. 2022 ; 14( 1): 1-20.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cancers14010028
Vancouver
Pereira MG, Voltarelli VA, Tobias GC, Souza L de, Borges GS, Paixão AO da, Bowen TS, Demasi M, Miyabara EH, Brum PC. Aerobic exercise training and in vivo akt activation counteract cancer cachexia by inducing a hypertrophic profile through eIF-2α modulation [Internet]. Cancers. 2022 ; 14( 1): 1-20.[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cancers14010028
Artigo de periodico
Muscle stem cell function is impaired in β2-Adrenoceptor knockout mice (2022)
Koike, Tatiana Emy ; Fuziwara, Cesar Seigi ; Brum, Patrícia Chakur ; Kimura, Edna Teruko ; Rando, Thomas A. ; Miyabara, Elen Haruka
Source: Stem Cell Reviews and Reports. Unidades: ICB, EEFE
Subjects: ANATOMIA, BIOLOGIA CELULAR, ADRENORECEPTORES, DIFERENCIAÇÃO CELULAR, FIBRAS MUSCULARES, MIOBLASTOS, IMUNOHISTOQUÍMICA, EXPERIMENTOS ANIMAIS, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, CICLO CELULAR, MÚSCULO ESQUELÉTICO
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KOIKE, Tatiana Emy et al. Muscle stem cell function is impaired in β2-Adrenoceptor knockout mice. Stem Cell Reviews and Reports, v. 18, p. 2431-2443, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12015-022-10334-y. Acesso em: 10 nov. 2024.
APA
Koike, T. E., Fuziwara, C. S., Brum, P. C., Kimura, E. T., Rando, T. A., & Miyabara, E. H. (2022). Muscle stem cell function is impaired in β2-Adrenoceptor knockout mice. Stem Cell Reviews and Reports, 18, 2431-2443. doi:10.1007/s12015-022-10334-y
NLM
Koike TE, Fuziwara CS, Brum PC, Kimura ET, Rando TA, Miyabara EH. Muscle stem cell function is impaired in β2-Adrenoceptor knockout mice [Internet]. Stem Cell Reviews and Reports. 2022 ; 18 2431-2443.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12015-022-10334-y
Vancouver
Koike TE, Fuziwara CS, Brum PC, Kimura ET, Rando TA, Miyabara EH. Muscle stem cell function is impaired in β2-Adrenoceptor knockout mice [Internet]. Stem Cell Reviews and Reports. 2022 ; 18 2431-2443.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s12015-022-10334-y
Artigo de periodico
Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity (2021)
Alves, Christiano Robles Rodrigues ; Eichelberger, Eric J. ; Silva, Willian das Neves ; Ribeiro, Márcio Augusto Campos; Bechara, Luiz Roberto Grassmann ; Voltarelli, Vanessa Azevedo ; Almeida, Ney Robson Ferreira de; Hagen, Lars ; Sharma, Animesh ; Ferreira, Julio Cesar Batista ; Swoboda, Kathryn J. ; Slupphaug, Geir ; Brum, Patrícia Chakur
Source: FASEB Journal. Unidades: EEFE, ICB, FM
Subjects: ANATOMIA, ATROFIA MUSCULAR, PERDA DE MASSA, AEROBIOSE, CAQUEXIA, DOENÇAS MUSCULOSQUELÉTICAS, ESTRESSE OXIDATIVO, FIBRAS MUSCULARES
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ALVES, Christiano Robles Rodrigues et al. Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity. FASEB Journal, v. 35, n. 7, p. e21714, 2021Tradução . . Disponível em: https://doi.org/10.1096/fj.202100263R. Acesso em: 10 nov. 2024.
APA
Alves, C. R. R., Eichelberger, E. J., Silva, W. das N., Ribeiro, M. A. C., Bechara, L. R. G., Voltarelli, V. A., et al. (2021). Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity. FASEB Journal, 35( 7), e21714. doi:10.1096/fj.202100263R
NLM
Alves CRR, Eichelberger EJ, Silva W das N, Ribeiro MAC, Bechara LRG, Voltarelli VA, Almeida NRF de, Hagen L, Sharma A, Ferreira JCB, Swoboda KJ, Slupphaug G, Brum PC. Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity [Internet]. FASEB Journal. 2021 ; 35( 7): e21714.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1096/fj.202100263R
Vancouver
Alves CRR, Eichelberger EJ, Silva W das N, Ribeiro MAC, Bechara LRG, Voltarelli VA, Almeida NRF de, Hagen L, Sharma A, Ferreira JCB, Swoboda KJ, Slupphaug G, Brum PC. Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity [Internet]. FASEB Journal. 2021 ; 35( 7): e21714.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1096/fj.202100263R
Artigo de periodico
Guidelines for the use and interpretation of assays for monitoring autophagy (2021)
Klionsky, Daniel J ; Baptista, Maurício da Silva ; Martins, Joilson de Oliveira ; Brum, Patrícia Chakur ; Ferreira, Julio Cesar Batista ; Palmisano, Giuseppe ; Câmara, Niels Olsen Saraiva ; Leopoldino, Andréia Machado ; Araújo, Thaís Fenz
Source: Autophagy. Unidades: IQ, FCF, EEFE, ICB, FCFRP
Subjects: NEOPLASIAS, LISOSSOMOS, CÉLULAS, BIOLOGIA CELULAR, ANATOMIA, PARASITOLOGIA, IMUNOLOGIA
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KLIONSKY, Daniel J et al. Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy, v. 17, n. 1, p. 1-382, 2021Tradução . . Disponível em: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280. Acesso em: 10 nov. 2024.
APA
Klionsky, D. J., Baptista, M. da S., Martins, J. de O., Brum, P. C., Ferreira, J. C. B., Palmisano, G., et al. (2021). Guidelines for the use and interpretation of assays for monitoring autophagy. Autophagy, 17( 1), 1-382. Recuperado de https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
NLM
Klionsky DJ, Baptista M da S, Martins J de O, Brum PC, Ferreira JCB, Palmisano G, Câmara NOS, Leopoldino AM, Araújo TF. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2021 ; 17( 1): 1-382.[citado 2024 nov. 10 ] Available from: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
Vancouver
Klionsky DJ, Baptista M da S, Martins J de O, Brum PC, Ferreira JCB, Palmisano G, Câmara NOS, Leopoldino AM, Araújo TF. Guidelines for the use and interpretation of assays for monitoring autophagy [Internet]. Autophagy. 2021 ; 17( 1): 1-382.[citado 2024 nov. 10 ] Available from: https://www.tandfonline.com/doi/full/10.1080/15548627.2020.1797280
Artigo de periodico
β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise (2021)
Voltarelli, Vanessa Azevedo ; Coronado, Michael ; Fernandes, Larissa Gonçalves ; Campos, Juliane Cruz ; Jannig, Paulo Roberto; Ferreira, Julio Cesar Batista ; Fajardo, Giovanni ; Brum, Patrícia Chakur ; Bernstein, Daniel
Source: Molecular Diversity Preservation International. Unidades: ICB, EEFE
Subjects: EXERCÍCIO FÍSICO, SISTEMA NERVOSO SIMPÁTICO, MÚSCULO ESQUELÉTICO, ADRENORECEPTORES, ADRENÉRGICOS, ANATOMIA, MITOCÔNDRIAS, JUNÇÃO NEUROMUSCULAR, CAMUNDONGOS, PROTEÍNAS QUINASES
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ABNT
VOLTARELLI, Vanessa Azevedo et al. β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise. Molecular Diversity Preservation International, v. 10, n. 1, 2021Tradução . . Disponível em: https://doi.org/10.3390/cells10010146. Acesso em: 10 nov. 2024.
APA
Voltarelli, V. A., Coronado, M., Fernandes, L. G., Campos, J. C., Jannig, P. R., Ferreira, J. C. B., et al. (2021). β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise. Molecular Diversity Preservation International, 10( 1). doi:10.3390/cells10010146
NLM
Voltarelli VA, Coronado M, Fernandes LG, Campos JC, Jannig PR, Ferreira JCB, Fajardo G, Brum PC, Bernstein D. β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise [Internet]. Molecular Diversity Preservation International. 2021 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cells10010146
Vancouver
Voltarelli VA, Coronado M, Fernandes LG, Campos JC, Jannig PR, Ferreira JCB, Fajardo G, Brum PC, Bernstein D. β2-Adrenergic signaling modulates mitochondrial function and morphology in skeletal muscle in response to aerobic exercise [Internet]. Molecular Diversity Preservation International. 2021 ; 10( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.3390/cells10010146
Artigo de periodico
Histidine dipeptides are key regulators of excitation-contraction coupling in cardiac muscle: evidence from a novel CARNS1 knockout rat model (2021)
Gonçalves, Lívia de Souza; Sales, Lucas Peixoto; Saito, Tiemi Raquel de Moraes; Campos, Juliane Cruz ; Fernandes, Alan Lins ; Natali, José Eduardo Soubhia ; Jensen, Leonardo ; Arnold, Alexandre José Tavolari ; Ramalho, Lisley Santos ; Bechara, Luiz Roberto Grassmann ; Esteca, Marcos Vinicius ; Correa, Isis Paloppi ; Sant'Anna, Diogo; Ceroni, Alexandre; Michelini, Lisete Compagno ; Gualano, Bruno ; Teodoro, Walcy Paganelli Rosolia ; Carvalho, Victor Henrique ; Artioli, Guilherme Giannini ; Ferreira, Julio Cesar Batista
Source: Redox Biology. Unidades: ICB, FM, EEFE, IQ
Subjects: FISIOLOGIA, MÚSCULO ESQUELÉTICO, SISTEMA CARDIOVASCULAR, CONTRAÇÃO MUSCULAR, MODELOS ANIMAIS DE DOENÇAS, RATOS, ESTRESSE OXIDATIVO
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GONÇALVES, Lívia de Souza et al. Histidine dipeptides are key regulators of excitation-contraction coupling in cardiac muscle: evidence from a novel CARNS1 knockout rat model. Redox Biology, v. 44, p. 1-12, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2021.102016. Acesso em: 10 nov. 2024.
APA
Gonçalves, L. de S., Sales, L. P., Saito, T. R. de M., Campos, J. C., Fernandes, A. L., Natali, J. E. S., et al. (2021). Histidine dipeptides are key regulators of excitation-contraction coupling in cardiac muscle: evidence from a novel CARNS1 knockout rat model. Redox Biology, 44, 1-12. doi:10.1016/j.redox.2021.102016
NLM
Gonçalves L de S, Sales LP, Saito TR de M, Campos JC, Fernandes AL, Natali JES, Jensen L, Arnold AJT, Ramalho LS, Bechara LRG, Esteca MV, Correa IP, Sant'Anna D, Ceroni A, Michelini LC, Gualano B, Teodoro WPR, Carvalho VH, Artioli GG, Ferreira JCB. Histidine dipeptides are key regulators of excitation-contraction coupling in cardiac muscle: evidence from a novel CARNS1 knockout rat model [Internet]. Redox Biology. 2021 ; 44 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.redox.2021.102016
Vancouver
Gonçalves L de S, Sales LP, Saito TR de M, Campos JC, Fernandes AL, Natali JES, Jensen L, Arnold AJT, Ramalho LS, Bechara LRG, Esteca MV, Correa IP, Sant'Anna D, Ceroni A, Michelini LC, Gualano B, Teodoro WPR, Carvalho VH, Artioli GG, Ferreira JCB. Histidine dipeptides are key regulators of excitation-contraction coupling in cardiac muscle: evidence from a novel CARNS1 knockout rat model [Internet]. Redox Biology. 2021 ; 44 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.redox.2021.102016
Artigo de periodico
Thyrotoxicosis involves β2-adrenoceptor signaling to negatively affect microarchitecture and biomechanical properties of the femur (2019)
Papi, Bianca Neofiti ; Albuquerque, Rudá Prestes e ; Rodrigues, Manuela Miranda ; Gonçalves, Natalia Juliana Nardelli; Jorgetti, Vanda ; Brum, Patrícia Chakur ; Ferreira, Julio Cesar Batista ; Gouveia, Cecília Helena
Source: Thyroid. Unidades: EEFE, ICB, FM
Subjects: HORMÔNIOS TIREOIDIANOS, SISTEMA NERVOSO SIMPÁTICO, SISTEMA NERVOSO, CÉLULAS DO OSSO, ADRENORECEPTORES, CAMUNDONGOS, ANATOMIA, DOENÇAS ÓSSEAS, REMODELAÇÃO ÓSSEA, FÊMUR, ESTRADIOL, ESTRÓGENOS
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ABNT
PAPI, Bianca Neofiti et al. Thyrotoxicosis involves β2-adrenoceptor signaling to negatively affect microarchitecture and biomechanical properties of the femur. Thyroid, v. 29, n. 8, p. 1060-1072, 2019Tradução . . Disponível em: https://doi.org/10.1089/thy.2018.0259. Acesso em: 10 nov. 2024.
APA
Papi, B. N., Albuquerque, R. P. e, Rodrigues, M. M., Gonçalves, N. J. N., Jorgetti, V., Brum, P. C., et al. (2019). Thyrotoxicosis involves β2-adrenoceptor signaling to negatively affect microarchitecture and biomechanical properties of the femur. Thyroid, 29( 8), 1060-1072. doi:10.1089/thy.2018.0259
NLM
Papi BN, Albuquerque RP e, Rodrigues MM, Gonçalves NJN, Jorgetti V, Brum PC, Ferreira JCB, Gouveia CH. Thyrotoxicosis involves β2-adrenoceptor signaling to negatively affect microarchitecture and biomechanical properties of the femur [Internet]. Thyroid. 2019 ; 29( 8): 1060-1072.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1089/thy.2018.0259
Vancouver
Papi BN, Albuquerque RP e, Rodrigues MM, Gonçalves NJN, Jorgetti V, Brum PC, Ferreira JCB, Gouveia CH. Thyrotoxicosis involves β2-adrenoceptor signaling to negatively affect microarchitecture and biomechanical properties of the femur [Internet]. Thyroid. 2019 ; 29( 8): 1060-1072.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1089/thy.2018.0259
Artigo de periodico
Low-intensity resistance training with partial blood flow restriction and high-intensity resistance training induce similar changes in skeletal muscle transcriptome in elderly humans (2019)
Vechin, Felipe C.; Libardi, Cleiton A.; Conceição, Miguel S.; Damas, Felipe; Cavaglieri, Claudia R.; Chacon-Mikahil, Mara Patricia T.; Coutinho, Luiz Lehmann; Andrade, Sónia C. S ; Neves Júnior, Manoel T.; Roschel, Hamilton ; Tricoli, Valmor; Baptista, Igor L.; Moriscot, Anselmo Sigari ; Ugrinowitsch, Carlos
Source: Applied Physiology, Nutrition, and Metabolism. Unidades: EEFE, ESALQ, IB, ICB
Subjects: ENVELHECIMENTO, EXPRESSÃO GÊNICA, TRANSCRIÇÃO GÊNICA, FLUXO SANGUÍNEO, MÚSCULO ESQUELÉTICO, IDOSOS
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ABNT
VECHIN, Felipe C. et al. Low-intensity resistance training with partial blood flow restriction and high-intensity resistance training induce similar changes in skeletal muscle transcriptome in elderly humans. Applied Physiology, Nutrition, and Metabolism, v. 44, n. 2, p. 216-220, 2019Tradução . . Disponível em: https://doi.org/10.1139/apnm-2018-0146. Acesso em: 10 nov. 2024.
APA
Vechin, F. C., Libardi, C. A., Conceição, M. S., Damas, F., Cavaglieri, C. R., Chacon-Mikahil, M. P. T., et al. (2019). Low-intensity resistance training with partial blood flow restriction and high-intensity resistance training induce similar changes in skeletal muscle transcriptome in elderly humans. Applied Physiology, Nutrition, and Metabolism, 44( 2), 216-220. doi:10.1139/apnm-2018-0146
NLM
Vechin FC, Libardi CA, Conceição MS, Damas F, Cavaglieri CR, Chacon-Mikahil MPT, Coutinho LL, Andrade SCS, Neves Júnior MT, Roschel H, Tricoli V, Baptista IL, Moriscot AS, Ugrinowitsch C. Low-intensity resistance training with partial blood flow restriction and high-intensity resistance training induce similar changes in skeletal muscle transcriptome in elderly humans [Internet]. Applied Physiology, Nutrition, and Metabolism. 2019 ; 44( 2): 216-220.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1139/apnm-2018-0146
Vancouver
Vechin FC, Libardi CA, Conceição MS, Damas F, Cavaglieri CR, Chacon-Mikahil MPT, Coutinho LL, Andrade SCS, Neves Júnior MT, Roschel H, Tricoli V, Baptista IL, Moriscot AS, Ugrinowitsch C. Low-intensity resistance training with partial blood flow restriction and high-intensity resistance training induce similar changes in skeletal muscle transcriptome in elderly humans [Internet]. Applied Physiology, Nutrition, and Metabolism. 2019 ; 44( 2): 216-220.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1139/apnm-2018-0146
Artigo de periodico
Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy (2018)
Campos, Juliane Cruz; Baehr, Leslie M; Gomes, Kátia Maria Sampaio; Bechara, Luiz Roberto Grassmann; Voltarelli, Vanessa Azevedo; Bozi, Luiz Henrique Marchesi; Ribeiro, Márcio Augusto Campos; Ferreira, Nikolas Dresch; Moreira, José Bianco Nascimento; Brum, Patrícia Chakur ; Bodine, Sue C; Ferreira, Julio Cesar Batista
Source: Scientific Reports. Unidades: EEFE, ICB
Subjects: DOENÇAS NEUROMUSCULARES, EXERCÍCIO FÍSICO, TREINAMENTO AERÓBIO, MÚSCULO ESQUELÉTICO, ATROFIA MUSCULAR, NEUROBIOLOGIA, RATOS
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ABNT
CAMPOS, Juliane Cruz et al. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy. Scientific Reports, v. 8, n. 1, p. 14 , 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-30365-1. Acesso em: 10 nov. 2024.
APA
Campos, J. C., Baehr, L. M., Gomes, K. M. S., Bechara, L. R. G., Voltarelli, V. A., Bozi, L. H. M., et al. (2018). Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy. Scientific Reports, 8( 1), 14 . doi:10.1038/s41598-018-30365-1
NLM
Campos JC, Baehr LM, Gomes KMS, Bechara LRG, Voltarelli VA, Bozi LHM, Ribeiro MAC, Ferreira ND, Moreira JBN, Brum PC, Bodine SC, Ferreira JCB. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy [Internet]. Scientific Reports. 2018 ; 8( 1): 14 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-018-30365-1
Vancouver
Campos JC, Baehr LM, Gomes KMS, Bechara LRG, Voltarelli VA, Bozi LHM, Ribeiro MAC, Ferreira ND, Moreira JBN, Brum PC, Bodine SC, Ferreira JCB. Exercise prevents impaired autophagy and proteostasis in a model of neurogenic myopathy [Internet]. Scientific Reports. 2018 ; 8( 1): 14 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-018-30365-1
Artigo de periodico
Global disruption of α2A adrenoceptor barely affects bone tissue but minimizes the detrimental effects of thyrotoxicosis on cortical bone (2018)
Beber, Gisele Miyamura Martins ; Teixeira, Marília Bianca Cruz Grecco ; Silva, Marcos Vinicius da ; Papi, Bianca Neofiti ; Rodrigues, Manuela Miranda ; Brum, Patrícia Chakur ; Gouveia, Cecília Helena
Source: Frontiers in physiology. Unidades: EEFE, ICB, FM
Subjects: OSSO E OSSOS, SISTEMA NERVOSO SIMPÁTICO, HORMÔNIOS TIREOIDIANOS, CÉLULAS DO OSSO, ADRENORECEPTORES, CAMUNDONGOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BEBER, Gisele Miyamura Martins et al. Global disruption of α2A adrenoceptor barely affects bone tissue but minimizes the detrimental effects of thyrotoxicosis on cortical bone. Frontiers in physiology, v. 9, p. 11 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00486. Acesso em: 10 nov. 2024.
APA
Beber, G. M. M., Teixeira, M. B. C. G., Silva, M. V. da, Papi, B. N., Rodrigues, M. M., Brum, P. C., & Gouveia, C. H. (2018). Global disruption of α2A adrenoceptor barely affects bone tissue but minimizes the detrimental effects of thyrotoxicosis on cortical bone. Frontiers in physiology, 9, 11 . doi:10.3389/fendo.2018.00486
NLM
Beber GMM, Teixeira MBCG, Silva MV da, Papi BN, Rodrigues MM, Brum PC, Gouveia CH. Global disruption of α2A adrenoceptor barely affects bone tissue but minimizes the detrimental effects of thyrotoxicosis on cortical bone [Internet]. Frontiers in physiology. 2018 ; 9 11 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00486
Vancouver
Beber GMM, Teixeira MBCG, Silva MV da, Papi BN, Rodrigues MM, Brum PC, Gouveia CH. Global disruption of α2A adrenoceptor barely affects bone tissue but minimizes the detrimental effects of thyrotoxicosis on cortical bone [Internet]. Frontiers in physiology. 2018 ; 9 11 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.3389/fendo.2018.00486
Artigo de periodico
Effect of eccentric action velocity on expression of genes related to myostatin signaling pathway in human skeletal muscle (2018)
Roschel, Hamilton; Ugrinowistch, Carlos; Santos, Audrei Reis; Barbosa, Wesley Pereira; Miyabara, Elen Haruka ; Tricoli, Valmor Alberto Augusto; Aoki, Marcelo Saldanha
Source: Biology of sport. Unidades: EEFE, ICB, EACH
Subjects: EXPRESSÃO GÊNICA, TREINAMENTO DE FORÇA, MÚSCULO ESQUELÉTICO, EXERCÍCIO FÍSICO, REAÇÃO EM CADEIA POR POLIMERASE, FATORES DE CRESCIMENTO, MÚSCULOS, BIÓPSIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSCHEL, Hamilton et al. Effect of eccentric action velocity on expression of genes related to myostatin signaling pathway in human skeletal muscle. Biology of sport, v. 35, n. 2, p. 111-119, 2018Tradução . . Disponível em: https://doi.org/10.5114/biolsport.2018.71600. Acesso em: 10 nov. 2024.
APA
Roschel, H., Ugrinowistch, C., Santos, A. R., Barbosa, W. P., Miyabara, E. H., Tricoli, V. A. A., & Aoki, M. S. (2018). Effect of eccentric action velocity on expression of genes related to myostatin signaling pathway in human skeletal muscle. Biology of sport, 35( 2), 111-119. doi:10.5114/biolsport.2018.71600
NLM
Roschel H, Ugrinowistch C, Santos AR, Barbosa WP, Miyabara EH, Tricoli VAA, Aoki MS. Effect of eccentric action velocity on expression of genes related to myostatin signaling pathway in human skeletal muscle [Internet]. Biology of sport. 2018 ; 35( 2): 111-119.[citado 2024 nov. 10 ] Available from: https://doi.org/10.5114/biolsport.2018.71600
Vancouver
Roschel H, Ugrinowistch C, Santos AR, Barbosa WP, Miyabara EH, Tricoli VAA, Aoki MS. Effect of eccentric action velocity on expression of genes related to myostatin signaling pathway in human skeletal muscle [Internet]. Biology of sport. 2018 ; 35( 2): 111-119.[citado 2024 nov. 10 ] Available from: https://doi.org/10.5114/biolsport.2018.71600
Artigo de periodico
Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 (2018)
Bozi, Luiz Henrique Marchesi; Takano, Ana Paula Cremasco ; Campos, Juliane Cruz; Rolim, Natale Pinheiro Lage; Dourado, Paulo Magno Martins; Voltarelli, Vanessa Azevedo; Wisløff, Ulrik; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Brum, Patricia Chakur
Source: International journal of cardiology. Unidades: ICB, EEFE
Subjects: RETÍCULO ENDOPLASMÁTICO, INFARTO DO MIOCÁRDIO, DOENÇAS CARDIOVASCULARES, PROTEÍNAS, RATOS WISTAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BOZI, Luiz Henrique Marchesi et al. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, v. 272, p. 194-201, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2018.08.070. Acesso em: 10 nov. 2024.
APA
Bozi, L. H. M., Takano, A. P. C., Campos, J. C., Rolim, N. P. L., Dourado, P. M. M., Voltarelli, V. A., et al. (2018). Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3. International journal of cardiology, 272, 194-201. doi:10.1016/j.ijcard.2018.08.070
NLM
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Vancouver
Bozi LHM, Takano APC, Campos JC, Rolim NPL, Dourado PMM, Voltarelli VA, Wisløff U, Ferreira JCB, Barreto-Chaves MLM, Brum PC. Endoplasmic reticulum stress impairs cardiomyocyte contractility through JNK-dependent upregulation of BNIP3 [Internet]. International journal of cardiology. 2018 ; 272 194-201.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.ijcard.2018.08.070
Artigo de periodico
Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats (2017)
Cunha, Telma F; Bechara, Luiz R. G; Bacurau, Aline V. N; Jannig, Paulo R; Voltarelli, Vanessa A; Dourado, Paulo M; Vasconcelos, Andrea R; Scavone, Cristoforo ; Ferreira, Julio Cesar Batista ; Brum, Patrícia Chakur
Source: Journal of applied physiology. Unidades: ICB, EEFE
Subjects: INSUFICIÊNCIA CARDÍACA, MÚSCULO ESQUELÉTICO, EXERCÍCIO FÍSICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CUNHA, Telma F et al. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats. Journal of applied physiology, v. 122, n. 4, p. 817-827, 2017Tradução . . Disponível em: https://doi.org/10.1152/japplphysiol.00182.2016. Acesso em: 10 nov. 2024.
APA
Cunha, T. F., Bechara, L. R. G., Bacurau, A. V. N., Jannig, P. R., Voltarelli, V. A., Dourado, P. M., et al. (2017). Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats. Journal of applied physiology, 122( 4), 817-827. doi:10.1152/japplphysiol.00182.2016
NLM
Cunha TF, Bechara LRG, Bacurau AVN, Jannig PR, Voltarelli VA, Dourado PM, Vasconcelos AR, Scavone C, Ferreira JCB, Brum PC. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats [Internet]. Journal of applied physiology. 2017 ; 122( 4): 817-827.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/japplphysiol.00182.2016
Vancouver
Cunha TF, Bechara LRG, Bacurau AVN, Jannig PR, Voltarelli VA, Dourado PM, Vasconcelos AR, Scavone C, Ferreira JCB, Brum PC. Exercise training decreases NADPH oxidase activity and restores skeletal muscle mass in heart failure rats [Internet]. Journal of applied physiology. 2017 ; 122( 4): 817-827.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1152/japplphysiol.00182.2016
Artigo de periodico
Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure (2017)
Campos, Juliane Cruz; Queliconi, Bruno Barros; Bozi, Luiz Henrique Marchesi; Bechara, Luiz Roberto Grassmann; Dourado, Paulo Magno Martins; Andres, Allen M; Jannig, Paulo Roberto; Gomes, Katia Maria Sampaio; Zambelli, Vanessa Olzon; Resende, Cibele Rocha; Fonseca, Silvia Carolina Guatimosim; Brum, Patricia Chakur; Mochly-Rosen, Daria; Gottlieb, Roberta A; Kowaltowski, Alicia Juliana ; Ferreira, Julio Cesar Batista
Source: Autophagy. Unidades: EEFE, IQ, ICB
Subjects: DOENÇAS CARDIOVASCULARES, EXERCÍCIO FÍSICO, MITOCÔNDRIAS, BIOENERGÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMPOS, Juliane Cruz et al. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure. Autophagy, v. 13, n. 8, p. 1304-1317, 2017Tradução . . Disponível em: https://doi.org/10.1080/15548627.2017.1325062. Acesso em: 10 nov. 2024.
APA
Campos, J. C., Queliconi, B. B., Bozi, L. H. M., Bechara, L. R. G., Dourado, P. M. M., Andres, A. M., et al. (2017). Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure. Autophagy, 13( 8), 1304-1317. doi:10.1080/15548627.2017.1325062
NLM
Campos JC, Queliconi BB, Bozi LHM, Bechara LRG, Dourado PMM, Andres AM, Jannig PR, Gomes KMS, Zambelli VO, Resende CR, Fonseca SCG, Brum PC, Mochly-Rosen D, Gottlieb RA, Kowaltowski AJ, Ferreira JCB. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure [Internet]. Autophagy. 2017 ; 13( 8): 1304-1317.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/15548627.2017.1325062
Vancouver
Campos JC, Queliconi BB, Bozi LHM, Bechara LRG, Dourado PMM, Andres AM, Jannig PR, Gomes KMS, Zambelli VO, Resende CR, Fonseca SCG, Brum PC, Mochly-Rosen D, Gottlieb RA, Kowaltowski AJ, Ferreira JCB. Exercise reestablishes autophagic flux and mitochondrial quality control in heart failure [Internet]. Autophagy. 2017 ; 13( 8): 1304-1317.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/15548627.2017.1325062
Artigo de periodico
Lack of α2C-adrenoceptor results in contrasting phenotypes of long bones and vertebra and prevents the thyrotoxicosis-induced osteopenia (2016)
Teixeira, Marilia Bianca Cruz Grecco; Martins, Gisele Miyamura; Miranda-Rodrigues, Manuela; Araújo, Iasmin Ferreira de; Oliveira, Ricardo; Brum, Patricia Chakur; Gouveia, Cecília Helena
Source: PLOS ONE. Unidades: EEFE, ICB
Assunto: OSTEOPATIAS METABÓLICAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TEIXEIRA, Marilia Bianca Cruz Grecco et al. Lack of α2C-adrenoceptor results in contrasting phenotypes of long bones and vertebra and prevents the thyrotoxicosis-induced osteopenia. PLOS ONE, v. 11, n. ja 2016, p. 17 , 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0146795. Acesso em: 10 nov. 2024.
APA
Teixeira, M. B. C. G., Martins, G. M., Miranda-Rodrigues, M., Araújo, I. F. de, Oliveira, R., Brum, P. C., & Gouveia, C. H. (2016). Lack of α2C-adrenoceptor results in contrasting phenotypes of long bones and vertebra and prevents the thyrotoxicosis-induced osteopenia. PLOS ONE, 11( ja 2016), 17 . doi:10.1371/journal.pone.0146795
NLM
Teixeira MBCG, Martins GM, Miranda-Rodrigues M, Araújo IF de, Oliveira R, Brum PC, Gouveia CH. Lack of α2C-adrenoceptor results in contrasting phenotypes of long bones and vertebra and prevents the thyrotoxicosis-induced osteopenia [Internet]. PLOS ONE. 2016 ; 11( ja 2016): 17 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0146795
Vancouver
Teixeira MBCG, Martins GM, Miranda-Rodrigues M, Araújo IF de, Oliveira R, Brum PC, Gouveia CH. Lack of α2C-adrenoceptor results in contrasting phenotypes of long bones and vertebra and prevents the thyrotoxicosis-induced osteopenia [Internet]. PLOS ONE. 2016 ; 11( ja 2016): 17 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0146795
Artigo de periodico
β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9 (2016)
Silva, Meiricris T; Nascimento, Tábata L; Pereira, Marcelo G; Siqueira, Adriane S; Brum, Patricia Chakur; Jaeger, Ruy Gastaldoni ; Miyabara, Elen Haruka
Source: Cell and Tissue Research. Unidades: EEFE, ICB
Subjects: ADRENÉRGICOS, TECIDO CONJUNTIVO, MÚSCULO ESQUELÉTICO, METALOPROTEINASES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Meiricris T et al. β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9. Cell and Tissue Research, v. 365, n. 1, p. 173-186, 2016Tradução . . Disponível em: https://doi.org/10.1007/s00441-016-2373-2. Acesso em: 10 nov. 2024.
APA
Silva, M. T., Nascimento, T. L., Pereira, M. G., Siqueira, A. S., Brum, P. C., Jaeger, R. G., & Miyabara, E. H. (2016). β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9. Cell and Tissue Research, 365( 1), 173-186. doi:10.1007/s00441-016-2373-2
NLM
Silva MT, Nascimento TL, Pereira MG, Siqueira AS, Brum PC, Jaeger RG, Miyabara EH. β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9 [Internet]. Cell and Tissue Research. 2016 ; 365( 1): 173-186.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00441-016-2373-2
Vancouver
Silva MT, Nascimento TL, Pereira MG, Siqueira AS, Brum PC, Jaeger RG, Miyabara EH. β2-Adrenoceptor is involved in connective tissue remodeling in regenerating muscles by decreasing the activity of MMP-9 [Internet]. Cell and Tissue Research. 2016 ; 365( 1): 173-186.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1007/s00441-016-2373-2
Artigo de periodico
Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats (2015)
Gabriel-Costa, Daniele; Cunha, Telma Fatima da; Bechara, Luiz Roberto Grassmann; Fortunato, Rodrigo Soares; Bozi, Luiz Henrique Marchesi; Coelho, Marcele de Almeida; Barreto-Chaves, Maria Luiza Morais ; Brum, Patricia Chakur
Source: PLOS ONE. Unidades: EEFE, ICB
Subjects: CORAÇÃO, OXIDAÇÃO, SUPERÓXIDO DISMUTASE, MITOCÔNDRIAS, ANTIOXIDANTES, VENTRÍCULO CARDÍACO, FOSFATOS, MÚSCULO ESQUELÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GABRIEL-COSTA, Daniele et al. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats. PLOS ONE, v. 10, n. 5, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0127843. Acesso em: 10 nov. 2024.
APA
Gabriel-Costa, D., Cunha, T. F. da, Bechara, L. R. G., Fortunato, R. S., Bozi, L. H. M., Coelho, M. de A., et al. (2015). Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats. PLOS ONE, 10( 5). doi:10.1371/journal.pone.0127843
NLM
Gabriel-Costa D, Cunha TF da, Bechara LRG, Fortunato RS, Bozi LHM, Coelho M de A, Barreto-Chaves MLM, Brum PC. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats [Internet]. PLOS ONE. 2015 ; 10( 5):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0127843
Vancouver
Gabriel-Costa D, Cunha TF da, Bechara LRG, Fortunato RS, Bozi LHM, Coelho M de A, Barreto-Chaves MLM, Brum PC. Lactate up-regulates the expression of lactate oxidation complex-related genes in left ventricular cardiac tissue of rats [Internet]. PLOS ONE. 2015 ; 10( 5):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1371/journal.pone.0127843

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