Filtros : "Serrano-Nascimento, Caroline" Limpar

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  • Source: Revista Brasileira de Farmacognosia. Unidade: ICB

    Subjects: PLANTAS MEDICINAIS, ESTRESSE OXIDATIVO, AROEIRA, ÁRVORES FLORESTAIS, TOXICIDADE DO SOLO, INFLAMAÇÃO, FISIOLOGIA, EXTRATOS (FORMAS FARMACÊUTICAS), GLÂNDULA TIREOIDE, FARMACOGNOSIA

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      SILVIA CRISTINA FIGUEIRA OLINTO,; CALIL-SILVEIRA, Jamile; DIAS, Rafael B. A.; et al. Antiproliferative and cytotoxic effects of Schinus terebinthifolia leaf extract on thyroid follicular cells. Revista Brasileira de Farmacognosia, João Pessoa, Sociedade Brasileira de Farmacognosia, v. 30, p. 693-700, 2020. Disponível em: < https://doi.org/10.1007/s43450-020-00098-2 > DOI: 10.1007/s43450-020-00098-2.
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      Silvia Cristina Figueira Olinto,, Calil-Silveira, J., Dias, R. B. A., Valle, M. M. R., Serrano-Nascimento, C., Rocha, P. S., et al. (2020). Antiproliferative and cytotoxic effects of Schinus terebinthifolia leaf extract on thyroid follicular cells. Revista Brasileira de Farmacognosia, 30, 693-700. doi:10.1007/s43450-020-00098-2
    • NLM

      Silvia Cristina Figueira Olinto, Calil-Silveira J, Dias RBA, Valle MMR, Serrano-Nascimento C, Rocha PS, Monteiro-Alfredo T, Santos EL dos, Souza K de P, Nunes MT. Antiproliferative and cytotoxic effects of Schinus terebinthifolia leaf extract on thyroid follicular cells [Internet]. Revista Brasileira de Farmacognosia. 2020 ; 30 693-700.Available from: https://doi.org/10.1007/s43450-020-00098-2
    • Vancouver

      Silvia Cristina Figueira Olinto, Calil-Silveira J, Dias RBA, Valle MMR, Serrano-Nascimento C, Rocha PS, Monteiro-Alfredo T, Santos EL dos, Souza K de P, Nunes MT. Antiproliferative and cytotoxic effects of Schinus terebinthifolia leaf extract on thyroid follicular cells [Internet]. Revista Brasileira de Farmacognosia. 2020 ; 30 693-700.Available from: https://doi.org/10.1007/s43450-020-00098-2
  • Source: Scientific Reports. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline; SALGUEIRO, Rafael Barrera; PANTALEÃO, Thiago; COSTA, Vânia Maria Corrêa da; NUNES, Maria Tereza. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring. Scientific Reports, London, Nature Publishing Group, v. 7, n. 1, p. 1-12, 2017. Disponível em: < http://dx.doi.org/10.1038/s41598-017-15529-9 > DOI: 10.1038/s41598-017-15529-9.
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      Serrano-Nascimento, C., Salgueiro, R. B., Pantaleão, T., Costa, V. M. C. da, & Nunes, M. T. (2017). Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring. Scientific Reports, 7( 1), 1-12. doi:10.1038/s41598-017-15529-9
    • NLM

      Serrano-Nascimento C, Salgueiro RB, Pantaleão T, Costa VMC da, Nunes MT. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring [Internet]. Scientific Reports. 2017 ; 7( 1): 1-12.Available from: http://dx.doi.org/10.1038/s41598-017-15529-9
    • Vancouver

      Serrano-Nascimento C, Salgueiro RB, Pantaleão T, Costa VMC da, Nunes MT. Maternal exposure to iodine excess throughout pregnancy and lactation induces hypothyroidism in adult male rat offspring [Internet]. Scientific Reports. 2017 ; 7( 1): 1-12.Available from: http://dx.doi.org/10.1038/s41598-017-15529-9
  • Source: Endocrine Connections. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline; SALGUEIRO, Rafael Barrera; VITZEL, Kaio Fernando; et al. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats. Endocrine Connections, Bristol, BioScientifica Ltd., v. 6, n. 7, p. 510-521, 2017. Disponível em: < http://dx.doi.org/10.1530/EC-17-0106 > DOI: 10.1530/EC-17-0106.
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      Serrano-Nascimento, C., Salgueiro, R. B., Vitzel, K. F., Pantaleão, T., Costa, V. M. C. da, & Nunes, M. T. (2017). Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats. Endocrine Connections, 6( 7), 510-521. doi:10.1530/EC-17-0106
    • NLM

      Serrano-Nascimento C, Salgueiro RB, Vitzel KF, Pantaleão T, Costa VMC da, Nunes MT. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats [Internet]. Endocrine Connections. 2017 ; 6( 7): 510-521.Available from: http://dx.doi.org/10.1530/EC-17-0106
    • Vancouver

      Serrano-Nascimento C, Salgueiro RB, Vitzel KF, Pantaleão T, Costa VMC da, Nunes MT. Iodine excess exposure during pregnancy and lactation impairs maternal thyroid function in rats [Internet]. Endocrine Connections. 2017 ; 6( 7): 510-521.Available from: http://dx.doi.org/10.1530/EC-17-0106
  • Source: American Journal of Physiology. Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile; SERRANO-NASCIMENTO, Caroline; KOPP, Peter Andreas; NUNES, Maria Tereza. Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, Bethesda, American Physiological Society, v. 310, n. 7, p. C576-C582, 2016. Disponível em: < http://dx.doi.org/10.1152/ajpcell.00210.2015 > DOI: 10.1152/ajpcell.00210.2015.
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      Calil-Silveira, J., Serrano-Nascimento, C., Kopp, P. A., & Nunes, M. T. (2016). Iodide excess regulates its own efflux: a possible involvement of pendrin. American Journal of Physiology. Cell Physiology, 310( 7), C576-C582. doi:10.1152/ajpcell.00210.2015
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.Available from: http://dx.doi.org/10.1152/ajpcell.00210.2015
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Kopp PA, Nunes MT. Iodide excess regulates its own efflux: a possible involvement of pendrin [Internet]. American Journal of Physiology. Cell Physiology. 2016 ; 310( 7): C576-C582.Available from: http://dx.doi.org/10.1152/ajpcell.00210.2015
  • Source: Thyroid. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile; SERRANO-NASCIMENTO, Caroline; LACONCA, Raquel Cardoso; et al. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats. Thyroid, New York, Mary Ann Liebert Publishers, v. 26, n. 10, p. 1488-1498, 2016. Disponível em: < http://dx.doi.org/10.1089/thy.20150338 > DOI: 10.1089/thy.20150338.
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      Calil-Silveira, J., Serrano-Nascimento, C., Laconca, R. C., Schmiedecke, L., Salgueiro, R. B., Kondo, A. K., & Nunes, M. T. (2016). Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats. Thyroid, 26( 10), 1488-1498. doi:10.1089/thy.20150338
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Laconca RC, Schmiedecke L, Salgueiro RB, Kondo AK, Nunes MT. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats [Internet]. Thyroid. 2016 ; 26( 10): 1488-1498.Available from: http://dx.doi.org/10.1089/thy.20150338
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Laconca RC, Schmiedecke L, Salgueiro RB, Kondo AK, Nunes MT. Underlying mechanisms of pituitary-thyroid axis function disruption by chronic iodine excess in rats [Internet]. Thyroid. 2016 ; 26( 10): 1488-1498.Available from: http://dx.doi.org/10.1089/thy.20150338
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline; NICOLA, Juan Pablo; TEIXEIRA, Silvania da Silva; et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, Limerick, Elsevier, v. 426, p. 73-90, 2016. Disponível em: < http://dx.doi.org/10.1016/j.mce.2016.02.006 > DOI: 10.1016/j.mce.2016.02.006.
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      Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
    • NLM

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.Available from: http://dx.doi.org/10.1016/j.mce.2016.02.006
    • Vancouver

      Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.Available from: http://dx.doi.org/10.1016/j.mce.2016.02.006
  • Source: Endocrinology. Unidade: ICB

    Subjects: FISIOLOGIA, SODIO, IONS, HORMÔNIOS TIREOIDIANOS, IODO

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      SERRANO-NASCIMENTO, Caroline; TEIXEIRA, Silvania da Silva; NACHBAR, Renato Tadeu; et al. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, Chevy Chase, Endocrine Society, v. 155, n. 3, p. 1145-1156, 2014. Disponível em: < http://dx.doi.org/10.1210/en.2013-1665 > DOI: 10.1210/en.2013-1665.
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      Serrano-Nascimento, C., Teixeira, S. da S., Nachbar, R. T., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2014). The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, 155( 3), 1145-1156. doi:10.1210/en.2013-1665
    • NLM

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.Available from: http://dx.doi.org/10.1210/en.2013-1665
    • Vancouver

      Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.Available from: http://dx.doi.org/10.1210/en.2013-1665
  • Source: Journal of Molecular Histology. Unidade: ICB

    Subjects: FISIOLOGIA, HISTOLOGIA

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      SALGADO, Renato M.; COVARRUBIAS, Ambart C.; FAVARO, Rodolfo R.; et al. Estradiol induces transcriptional and posttranscriptional modifications in versican expression in the mouse uterus. Journal of Molecular Histology, Dordrecht, v. 44, n. 2, p. 221-229, 2013. Disponível em: < http://dx.doi.org/10.1007/s10735-012-9476-1 > DOI: 10.1007/s10735-012-9476-1.
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      Salgado, R. M., Covarrubias, A. C., Favaro, R. R., Serrano-Nascimento, C., Nunes, M. T., & Zorn, T. M. T. (2013). Estradiol induces transcriptional and posttranscriptional modifications in versican expression in the mouse uterus. Journal of Molecular Histology, 44( 2), 221-229. doi:10.1007/s10735-012-9476-1
    • NLM

      Salgado RM, Covarrubias AC, Favaro RR, Serrano-Nascimento C, Nunes MT, Zorn TMT. Estradiol induces transcriptional and posttranscriptional modifications in versican expression in the mouse uterus [Internet]. Journal of Molecular Histology. 2013 ; 44( 2): 221-229.Available from: http://dx.doi.org/10.1007/s10735-012-9476-1
    • Vancouver

      Salgado RM, Covarrubias AC, Favaro RR, Serrano-Nascimento C, Nunes MT, Zorn TMT. Estradiol induces transcriptional and posttranscriptional modifications in versican expression in the mouse uterus [Internet]. Journal of Molecular Histology. 2013 ; 44( 2): 221-229.Available from: http://dx.doi.org/10.1007/s10735-012-9476-1
  • Source: Experimental and Clinical Endocrinology Diabetes. Unidade: ICB

    Assunto: FISIOLOGIA

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      GOULART-SILVA, Francemilson; SERRANO-NASCIMENTO, Caroline; TEIXEIRA, S. S.; NUNES, Maria Tereza. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes, Heidelberg, v. 121, n. 1, p. 14-19, 2013.
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      Goulart-Silva, F., Serrano-Nascimento, C., Teixeira, S. S., & Nunes, M. T. (2013). Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes, 121( 1), 14-19.
    • NLM

      Goulart-Silva F, Serrano-Nascimento C, Teixeira SS, Nunes MT. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes. 2013 ; 121( 1): 14-19.
    • Vancouver

      Goulart-Silva F, Serrano-Nascimento C, Teixeira SS, Nunes MT. Triiodothyronine (T3) induces proinsulin gene expression by activating PI3K: possible roles for GSK-3β and the transcriptional factor PDX-1. Experimental and Clinical Endocrinology Diabetes. 2013 ; 121( 1): 14-19.
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      SERRANO-NASCIMENTO, Caroline; CALIL-SILVEIRA, Jamile; GOULART-SILVA, Francemilson; NUNES, Maria Tereza. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells. Molecular and Cellular Endocrinology, Limerick, v. 349, n. 2, p. 154-161, 2012. Disponível em: < http://dx.doi.org/doi: 10.1016/j.mce.2011.09.036 > DOI: 10.1016/j.mce.2011.09.036.
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      Serrano-Nascimento, C., Calil-Silveira, J., Goulart-Silva, F., & Nunes, M. T. (2012). New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells. Molecular and Cellular Endocrinology, 349( 2), 154-161. doi:10.1016/j.mce.2011.09.036
    • NLM

      Serrano-Nascimento C, Calil-Silveira J, Goulart-Silva F, Nunes MT. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells [Internet]. Molecular and Cellular Endocrinology. 2012 ; 349( 2): 154-161.Available from: http://dx.doi.org/doi: 10.1016/j.mce.2011.09.036
    • Vancouver

      Serrano-Nascimento C, Calil-Silveira J, Goulart-Silva F, Nunes MT. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells [Internet]. Molecular and Cellular Endocrinology. 2012 ; 349( 2): 154-161.Available from: http://dx.doi.org/doi: 10.1016/j.mce.2011.09.036
  • Source: Thyroid. Unidade: ICB

    Assunto: FISIOLOGIA

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      TEIXEIRA, Silvania Silva; TAMRAKAR, Akhilesh K.; GOULART-SILVA, Francemilson; et al. Triiodothyronine acutely stimulates glucose transport into L6 muscle cells without increasing surface GLUT4, GLUT1, or GLUT3. Thyroid, New York, v. 22, n. 7, p. 747-754, 2012. Disponível em: < http://dx.doi.org/10.1089/thy.2011.0422 > DOI: 10.1089/thy.2011.0422.
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      Teixeira, S. S., Tamrakar, A. K., Goulart-Silva, F., Serrano-Nascimento, C., Klip, A., & Nunes, M. T. (2012). Triiodothyronine acutely stimulates glucose transport into L6 muscle cells without increasing surface GLUT4, GLUT1, or GLUT3. Thyroid, 22( 7), 747-754. doi:10.1089/thy.2011.0422
    • NLM

      Teixeira SS, Tamrakar AK, Goulart-Silva F, Serrano-Nascimento C, Klip A, Nunes MT. Triiodothyronine acutely stimulates glucose transport into L6 muscle cells without increasing surface GLUT4, GLUT1, or GLUT3 [Internet]. Thyroid. 2012 ; 22( 7): 747-754.Available from: http://dx.doi.org/10.1089/thy.2011.0422
    • Vancouver

      Teixeira SS, Tamrakar AK, Goulart-Silva F, Serrano-Nascimento C, Klip A, Nunes MT. Triiodothyronine acutely stimulates glucose transport into L6 muscle cells without increasing surface GLUT4, GLUT1, or GLUT3 [Internet]. Thyroid. 2012 ; 22( 7): 747-754.Available from: http://dx.doi.org/10.1089/thy.2011.0422
  • Source: Molecular and Cellular Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

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      CALIL-SILVEIRA, Jamile; SERRANO-NASCIMENTO, Caroline; NUNES, Maria Tereza. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, Amsterdam, v. 350, n. 1, p. 118-124, 2012. Disponível em: < http://dx.doi.org/10.1016/j.mce.2011.12.002 > DOI: 10.1016/j.mce.2011.12.002.
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      Calil-Silveira, J., Serrano-Nascimento, C., & Nunes, M. T. (2012). Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms. Molecular and Cellular Endocrinology, 350( 1), 118-124. doi:10.1016/j.mce.2011.12.002
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.Available from: http://dx.doi.org/10.1016/j.mce.2011.12.002
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Nunes MT. Iodide treatment acutely increases pendrin (SLC26A4) mRNA expression in the rat thyroid and the PCCl3 thyroid cell line by transcriptional mechanisms [Internet]. Molecular and Cellular Endocrinology. 2012 ; 350( 1): 118-124.Available from: http://dx.doi.org/10.1016/j.mce.2011.12.002
  • Source: Abstracts. Conference titles: Thematic Symposium: new frontiers in applied physiology. Unidade: ICB

    Assunto: FISIOLOGIA HUMANA

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      SERRANO-NASCIMENTO, Caroline; NICOLA, Juan Pablo; MASINI-REPISO, Ana Maria; NUNES, Maria Tereza. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. Anais.. São Paulo: Dep/Physiol/Biophys/ICB/USP, 2012.
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      Serrano-Nascimento, C., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2012). Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. In Abstracts. São Paulo: Dep/Physiol/Biophys/ICB/USP.
    • NLM

      Serrano-Nascimento C, Nicola JP, Masini-Repiso AM, Nunes MT. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. Abstracts. 2012 ;
    • Vancouver

      Serrano-Nascimento C, Nicola JP, Masini-Repiso AM, Nunes MT. Inhibitory effects of iodide excess on sodium-iodide symporter (NIS) transcription rate involves Pax8 and p65. Abstracts. 2012 ;
  • Source: The Journal of Clinical Endocrinology and Metabolism. Unidade: ICB

    Assunto: FISIOLOGIA

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      NICOLA, Juan Pablo; NAZAR, Magalí; SERRANO-NASCIMENTO, Caroline; et al. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, Philadelphia, v. 96, n. 7, p. E1100-E1107, 2011. Disponível em: < http://dx.doi.org/doi:10.1210/jc.2011-0349 > DOI: 10.1210/jc.2011-0349.
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      Nicola, J. P., Nazar, M., Serrano-Nascimento, C., Goulart-Silva, F., Sobrero, G., Testa, G., et al. (2011). Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism. The Journal of Clinical Endocrinology and Metabolism, 96( 7), E1100-E1107. doi:10.1210/jc.2011-0349
    • NLM

      Nicola JP, Nazar M, Serrano-Nascimento C, Goulart-Silva F, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.Available from: http://dx.doi.org/doi:10.1210/jc.2011-0349
    • Vancouver

      Nicola JP, Nazar M, Serrano-Nascimento C, Goulart-Silva F, Sobrero G, Testa G, Nunes MT, Muñoz L, Miras M, Masini-Repiso AM. Iodide transport defect: functional characterization of a novel mutation in the Na+/I- symporter 5' -untranslated region in a patient with congenital hypothyroidism [Internet]. The Journal of Clinical Endocrinology and Metabolism. 2011 ; 96( 7): E1100-E1107.Available from: http://dx.doi.org/doi:10.1210/jc.2011-0349
  • Source: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Assunto: FISIOLOGIA

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      GOULART-SILVA, F.; SERRANO-NASCIMENTO, Caroline; NUNES, Maria Tereza. Hypothyroidism decreases proinsulin gene expression and the attachment of its mRNA and eEF1A protein to the actin cytoskeleton of INS-1E cells. Brazilian Journal of Medical and Biological Research, São Paulo, v. 44, n. 10, p. 1060-1067, 2011. Disponível em: < http://dx.doi.org/10.1590/S0100-879X2011007500121 > DOI: 10.1590/S0100-879X2011007500121.
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      Goulart-Silva, F., Serrano-Nascimento, C., & Nunes, M. T. (2011). Hypothyroidism decreases proinsulin gene expression and the attachment of its mRNA and eEF1A protein to the actin cytoskeleton of INS-1E cells. Brazilian Journal of Medical and Biological Research, 44( 10), 1060-1067. doi:10.1590/S0100-879X2011007500121
    • NLM

      Goulart-Silva F, Serrano-Nascimento C, Nunes MT. Hypothyroidism decreases proinsulin gene expression and the attachment of its mRNA and eEF1A protein to the actin cytoskeleton of INS-1E cells [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 10): 1060-1067.Available from: http://dx.doi.org/10.1590/S0100-879X2011007500121
    • Vancouver

      Goulart-Silva F, Serrano-Nascimento C, Nunes MT. Hypothyroidism decreases proinsulin gene expression and the attachment of its mRNA and eEF1A protein to the actin cytoskeleton of INS-1E cells [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 10): 1060-1067.Available from: http://dx.doi.org/10.1590/S0100-879X2011007500121
  • Source: American Journal of Physiology Cell Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

    DOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      SERRANO-NASCIMENTO, Caroline; SILVEIRA, Jamile Calil; NUNES, Maria Tereza. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology, Bethesda, v. 298, p. C893-C899, 2010. DOI: 10.1152/ajpcell.00224.2009.
    • APA

      Serrano-Nascimento, C., Silveira, J. C., & Nunes, M. T. (2010). Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology, 298, C893-C899. doi:10.1152/ajpcell.00224.2009
    • NLM

      Serrano-Nascimento C, Silveira JC, Nunes MT. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology. 2010 ; 298 C893-C899.
    • Vancouver

      Serrano-Nascimento C, Silveira JC, Nunes MT. Posttranscriptional regulation of sodium-iodide symporter mRNA expression in the rat thyroid gland by acute iodide administration. American Journal of Physiology Cell Physiology. 2010 ; 298 C893-C899.
  • Source: Programa. Conference titles: Reunião Anual da Federação de Sociedades de Biologia Experimental - FeSBE. Unidade: ICB

    Assunto: FISIOLOGIA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      CALIL-SILVEIRA, Jamile; SERRANO-NASCIMENTO, Caroline; POYARES, Leonice Lourenço; NUNES, Maria Tereza. Efeito da administração aguda de iodo sobre a expressão dos MRNAS de NIS e pendrina. Anais.. Águas de Lindóia: [s.n.], 2008.
    • APA

      Calil-Silveira, J., Serrano-Nascimento, C., Poyares, L. L., & Nunes, M. T. (2008). Efeito da administração aguda de iodo sobre a expressão dos MRNAS de NIS e pendrina. In Programa. Águas de Lindóia.
    • NLM

      Calil-Silveira J, Serrano-Nascimento C, Poyares LL, Nunes MT. Efeito da administração aguda de iodo sobre a expressão dos MRNAS de NIS e pendrina. Programa. 2008 ;
    • Vancouver

      Calil-Silveira J, Serrano-Nascimento C, Poyares LL, Nunes MT. Efeito da administração aguda de iodo sobre a expressão dos MRNAS de NIS e pendrina. Programa. 2008 ;

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