Filtros : "Jancar, Sonia" Limpar

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  • Source: Frontiers in Oncology. Unidade: ICB

    Subjects: CÉLULAS CULTIVADAS DE TUMOR, PROSTAGLANDINAS, RADIOTERAPIA, NEOPLASIAS DO COLON, CÉLULAS MORTAS, CARCINOMA DE CÉLULAS ESCAMOSAS, PLAQUETAS SANGUÍNEAS

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      SILVA-JUNIOR, Ildefonso Alves et al. Platelet-activating factor receptor ligands protect tumor cells from radiation-induced cell death. Frontiers in Oncology, v. 8, p. 1-11, 2018Tradução . . Disponível em: http://dx.doi.org/10.3389/fonc.2018.00010. Acesso em: 29 jan. 2023.
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      Silva-Junior, I. A., Dalmaso, B., Herbster, S. da S. G., Ana Paula Lepique,, & Jancar, S. (2018). Platelet-activating factor receptor ligands protect tumor cells from radiation-induced cell death. Frontiers in Oncology, 8, 1-11. doi:10.3389/fonc.2018.00010
    • NLM

      Silva-Junior IA, Dalmaso B, Herbster S da SG, Ana Paula Lepique, Jancar S. Platelet-activating factor receptor ligands protect tumor cells from radiation-induced cell death [Internet]. Frontiers in Oncology. 2018 ; 8 1-11.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.3389/fonc.2018.00010
    • Vancouver

      Silva-Junior IA, Dalmaso B, Herbster S da SG, Ana Paula Lepique, Jancar S. Platelet-activating factor receptor ligands protect tumor cells from radiation-induced cell death [Internet]. Frontiers in Oncology. 2018 ; 8 1-11.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.3389/fonc.2018.00010
  • Source: Clinics. Unidade: FM

    Subjects: QUIMIOTERAPIA, RADIOTERAPIA, NEOPLASIAS OVARIANAS, FATOR DE ATIVAÇÃO DE PLAQUETAS

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      SILVA JUNIOR, Ildefonso Alves da et al. Platelet activating factor receptor antagonists improve the efficacy of experimental chemo- and radiotherapy. Clinics. Sao Paulo: Faculdade de Medicina, Universidade de São Paulo. Disponível em: http://dx.doi.org/10.6061/clinics/2018/e792s. Acesso em: 29 jan. 2023. , 2018
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      Silva Junior, I. A. da, Chammas, R., Andrade, L. N. de S., & Jancar, S. (2018). Platelet activating factor receptor antagonists improve the efficacy of experimental chemo- and radiotherapy. Clinics. Sao Paulo: Faculdade de Medicina, Universidade de São Paulo. doi:10.6061/clinics/2018/e792s
    • NLM

      Silva Junior IA da, Chammas R, Andrade LN de S, Jancar S. Platelet activating factor receptor antagonists improve the efficacy of experimental chemo- and radiotherapy [Internet]. Clinics. 2018 ; 73[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.6061/clinics/2018/e792s
    • Vancouver

      Silva Junior IA da, Chammas R, Andrade LN de S, Jancar S. Platelet activating factor receptor antagonists improve the efficacy of experimental chemo- and radiotherapy [Internet]. Clinics. 2018 ; 73[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.6061/clinics/2018/e792s
  • Source: Nature reviews cancer. Unidades: FM, ICB

    Subjects: APOPTOSE, NEOPLASIAS, ONCOGENES, CÉLULAS CULTIVADAS DE TUMOR

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      CHAMMAS, Roger e ANDRADE, Luciana Nogueira de Sousa e JANCAR, Sonia. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 29 jan. 2023. , 2017
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      Chammas, R., Andrade, L. N. de S., & Jancar, S. (2017). Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. London: Faculdade de Medicina, Universidade de São Paulo. doi:10.1038/nrc.2017.15
    • NLM

      Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. 2017 ; 17( 4):[citado 2023 jan. 29 ]
    • Vancouver

      Chammas R, Andrade LN de S, Jancar S. Oncogenic effects of PAFR ligands produced in tumours upon chemotherapy and radiotherapy. Nature reviews cancer. 2017 ; 17( 4):[citado 2023 jan. 29 ]
  • Source: Journal of Immunology Research. Unidade: ICB

    Subjects: IMUNOLOGIA, MACRÓFAGOS, PLAQUETAS SANGUÍNEAS, CÉLULAS CULTIVADAS DE TUMOR, NEUTRÓFILOS, LINFÓCITOS

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      SILVA JUNIOR, Ildefonso Alves da et al. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, v. 2017, p. 1-10, 2017Tradução . . Disponível em: http://dx.doi.org/10.1155/2017/5482768. Acesso em: 29 jan. 2023.
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      Silva Junior, I. A. da, Stone, S. C., Rossetti, R. A. M., Jancar, S., & Lepique, A. P. (2017). Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor. Journal of Immunology Research, 2017, 1-10. doi:10.1155/2017/5482768
    • NLM

      Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2017/5482768
    • Vancouver

      Silva Junior IA da, Stone SC, Rossetti RAM, Jancar S, Lepique AP. Modulation of tumor-associated macrophages (TAM) phenotype by platelet-activating factor (PAF) receptor [Internet]. Journal of Immunology Research. 2017 ; 2017 1-10.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2017/5482768
  • Source: Proceedings. Conference titles: Brazilian Conference on Natural Products. Unidade: IQSC

    Subjects: FAUNA MARINHA, FUNGOS

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      NICACIO, Karen J et al. Biological activities of Phomactins from a marine-derived fungus Biatriospora sp. 2017, Anais.. Vitória: UFES, 2017. Disponível em: https://repositorio.usp.br/directbitstream/7c4ed098-030c-4a1f-acc1-cef44664577a/P17322.pdf. Acesso em: 29 jan. 2023.
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      Nicacio, K. J., Silva Junor, I. A., Gubiani, J. R., Ferreira, A. G., Sette, L. D., Jancar, S., & Berlinck, R. G. de S. (2017). Biological activities of Phomactins from a marine-derived fungus Biatriospora sp. In Proceedings. Vitória: UFES. Recuperado de https://repositorio.usp.br/directbitstream/7c4ed098-030c-4a1f-acc1-cef44664577a/P17322.pdf
    • NLM

      Nicacio KJ, Silva Junor IA, Gubiani JR, Ferreira AG, Sette LD, Jancar S, Berlinck RG de S. Biological activities of Phomactins from a marine-derived fungus Biatriospora sp [Internet]. Proceedings. 2017 ;[citado 2023 jan. 29 ] Available from: https://repositorio.usp.br/directbitstream/7c4ed098-030c-4a1f-acc1-cef44664577a/P17322.pdf
    • Vancouver

      Nicacio KJ, Silva Junor IA, Gubiani JR, Ferreira AG, Sette LD, Jancar S, Berlinck RG de S. Biological activities of Phomactins from a marine-derived fungus Biatriospora sp [Internet]. Proceedings. 2017 ;[citado 2023 jan. 29 ] Available from: https://repositorio.usp.br/directbitstream/7c4ed098-030c-4a1f-acc1-cef44664577a/P17322.pdf
  • Source: Journal of Immunobiology. Unidade: ICB

    Subjects: IMUNOLOGIA, INTERLEUCINA 10, CÉLULAS DENDRÍTICAS, PLAQUETAS SANGUÍNEAS, MACRÓFAGOS, PROSTAGLANDINAS

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      KOGA, Marianna Mainardi et al. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells. Journal of Immunobiology, v. 2, n. 2, p. 1-9, 2017Tradução . . Disponível em: http://dx.doi.org/10.4172/2476-1966.1000123. Acesso em: 29 jan. 2023.
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      Koga, M. M., Ribeiro Junior, L. F., Jancar, S., & Rios, F. J. (2017). Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells. Journal of Immunobiology, 2( 2), 1-9. doi:10.4172/2476-1966.1000123
    • NLM

      Koga MM, Ribeiro Junior LF, Jancar S, Rios FJ. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells [Internet]. Journal of Immunobiology. 2017 ; 2( 2): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.4172/2476-1966.1000123
    • Vancouver

      Koga MM, Ribeiro Junior LF, Jancar S, Rios FJ. Platelet-activation factor receptor induces interleukin 10 production through STAT3 activation in dendritic cells [Internet]. Journal of Immunobiology. 2017 ; 2( 2): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.4172/2476-1966.1000123
  • Source: Journal of Diabetes and its Complications. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FILGUEIRAS, Luciano Ribeiro et al. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, v. 31, n. 2, p. 334-339, 2017Tradução . . Disponível em: http://dx.doi.org/10.1016/j.jdiacomp.2016.08.001. Acesso em: 29 jan. 2023.
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      Filgueiras, L. R., Brandt, S. L., Ramalho, T. R. de O., Jancar, S., & Serezani, C. H. (2017). Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice. Journal of Diabetes and its Complications, 31( 2), 334-339. doi:10.1016/j.jdiacomp.2016.08.001
    • NLM

      Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.jdiacomp.2016.08.001
    • Vancouver

      Filgueiras LR, Brandt SL, Ramalho TR de O, Jancar S, Serezani CH. Imbalance between HDAC and HAT activities drives aberrante STAT1/MyD88 expression in macrophages from type 1 diabetic mice [Internet]. Journal of Diabetes and its Complications. 2017 ; 31( 2): 334-339.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.jdiacomp.2016.08.001
  • Source: Clinical Science. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FILGUEIRAS, Luciano Ribeiro et al. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, v. 130, n. 8, p. 601-612, 2016Tradução . . Disponível em: http://dx.doi.org/10.1042/CS20150538. Acesso em: 29 jan. 2023.
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      Filgueiras, L. R., Koga, M. M., Quaresma, P. G., Ishizuka, E. K., Montes, M. B. A., Prada, P. O., et al. (2016). PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis. Clinical Science, 130( 8), 601-612. doi:10.1042/CS20150538
    • NLM

      Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1042/CS20150538
    • Vancouver

      Filgueiras LR, Koga MM, Quaresma PG, Ishizuka EK, Montes MBA, Prada PO, Saad MJ, Jancar S, Rios FJ. PAFR in adipose tissue macrophages is associated with anti-inflammatory phenotype and metabolic homoeostasis [Internet]. Clinical Science. 2016 ; 130( 8): 601-612.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1042/CS20150538
  • Source: Scientific Reports. Unidade: ICB

    Assunto: IMUNOLOGIA

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      KOGA, Marianna M. et al. Boosting adaptive immunity:: a new role for PAFR antagonists. Scientific Reports, v. 6, n. 39146, p. 1-9, 2016Tradução . . Disponível em: http://dx.doi.org/10.1038/srep39146. Acesso em: 29 jan. 2023.
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      Koga, M. M., Rios, F. J., Jancar, S., Bizarro, B., & Sá-Nunes, A. (2016). Boosting adaptive immunity:: a new role for PAFR antagonists. Scientific Reports, 6( 39146), 1-9. doi:10.1038/srep39146
    • NLM

      Koga MM, Rios FJ, Jancar S, Bizarro B, Sá-Nunes A. Boosting adaptive immunity:: a new role for PAFR antagonists [Internet]. Scientific Reports. 2016 ; 6( 39146): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1038/srep39146
    • Vancouver

      Koga MM, Rios FJ, Jancar S, Bizarro B, Sá-Nunes A. Boosting adaptive immunity:: a new role for PAFR antagonists [Internet]. Scientific Reports. 2016 ; 6( 39146): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1038/srep39146
  • Source: Science Signaling. Unidade: ICB

    Subjects: IMUNOLOGIA, LEUCOTRIENOS B, INFLAMAÇÃO, SEPSE, CAMUNDONGOS

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      FILGUEIRAS, Luciano Ribeiro et al. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes. Science Signaling, v. 8, n. 361, p. 1-9, 2015Tradução . . Disponível em: http://dx.doi.org/10.1126/scisignal.2005568. Acesso em: 29 jan. 2023.
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      Filgueiras, L. R., Brandt, S. L., Wang, S., Wang, Z., Morris, D. L., Evans-Molina, C., et al. (2015). Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes. Science Signaling, 8( 361), 1-9. doi:10.1126/scisignal.2005568
    • NLM

      Filgueiras LR, Brandt SL, Wang S, Wang Z, Morris DL, Evans-Molina C, Mirmira RG, Jancar S, Serezani CHC. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes [Internet]. Science Signaling. 2015 ; 8( 361): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1126/scisignal.2005568
    • Vancouver

      Filgueiras LR, Brandt SL, Wang S, Wang Z, Morris DL, Evans-Molina C, Mirmira RG, Jancar S, Serezani CHC. Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes [Internet]. Science Signaling. 2015 ; 8( 361): 1-9.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1126/scisignal.2005568
  • Source: Mediators of Inflammation. Unidade: ICB

    Subjects: IMUNOLOGIA, PARACOCCIDIOIDES BRASILIENSIS, MACRÓFAGOS, INFECÇÕES RESPIRATÓRIAS

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      RIBEIRO, Laura Rossetti Rios et al. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, v. 2015, p. 1-17, 2015Tradução . . Disponível em: http://dx.doi.org/10.1155/2015/852574. Acesso em: 29 jan. 2023.
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      Ribeiro, L. R. R., Loures, F. V., Araújo, E. F. de, Feriotti, C., Costa, T. A. da, Serezani, C. H., et al. (2015). Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis. Mediators of Inflammation, 2015, 1-17. doi:10.1155/2015/852574
    • NLM

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2015/852574
    • Vancouver

      Ribeiro LRR, Loures FV, Araújo EF de, Feriotti C, Costa TA da, Serezani CH, Jancar S, Calich VLG. Lipoxin inhibits fungal uptake by macrophages and reduces the severity of acute pulmonary infection caused by Paracoccidioides brasiliensis [Internet]. Mediators of Inflammation. 2015 ; 2015 1-17.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2015/852574
  • Source: Microbes and Infection. Unidade: ICB

    Subjects: IMUNOLOGIA, LEISHMANIA BRASILIENSIS, MACRÓFAGOS, LEUCOTRIENOS

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      MORATO, Camila I. et al. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, v. 16, n. 11, p. 945-953, 2014Tradução . . Disponível em: http://dx.doi.org/10.1016/j.micinf.2014.08.015. Acesso em: 29 jan. 2023.
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      Morato, C. I., Silva Jr, I. A. da, Borges, A. F., Dorta, M. L., Oliveira, M. A. P., Jancar, S., et al. (2014). Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages. Microbes and Infection, 16( 11), 945-953. doi:10.1016/j.micinf.2014.08.015
    • NLM

      Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.micinf.2014.08.015
    • Vancouver

      Morato CI, Silva Jr IA da, Borges AF, Dorta ML, Oliveira MAP, Jancar S, Serezani CH, Ribeiro-Dias F. Essential role of leukotriene B4 on Leishmania (Viannia) braziliensis killing by human macrophages [Internet]. Microbes and Infection. 2014 ; 16( 11): 945-953.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.micinf.2014.08.015
  • Source: Current Drug Targets. Unidades: ICB, FM

    Subjects: IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, PLAQUETAS SANGUÍNEAS

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      JANCAR, Sonia e CHAMMAS, Roger. PAF receptor and tumor growth. Current Drug Targets, v. 15, n. 10, p. 982-987, 2014Tradução . . Acesso em: 29 jan. 2023.
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      Jancar, S., & Chammas, R. (2014). PAF receptor and tumor growth. Current Drug Targets, 15( 10), 982-987.
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      Jancar S, Chammas R. PAF receptor and tumor growth. Current Drug Targets. 2014 ; 15( 10): 982-987.[citado 2023 jan. 29 ]
    • Vancouver

      Jancar S, Chammas R. PAF receptor and tumor growth. Current Drug Targets. 2014 ; 15( 10): 982-987.[citado 2023 jan. 29 ]
  • Source: BMC Pulmonary Medicine. Unidades: FM, FCF, ICB

    Subjects: RATOS, SEPSE (ETIOLOGIA), DIABETES MELLITUS (COMPLICAÇÕES), FIBROBLASTOS

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      FILGUEIRAS, Luciano et al. Sepsis-induced lung inflammation is modulated by insulin. BMC Pulmonary Medicine, v. 14, n. 177, p. 8 2014, 2014Tradução . . Disponível em: http://www.biomedcentral.com/1471-2466/14/177. Acesso em: 29 jan. 2023.
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      Filgueiras, L., Capelozzi, V. L., Martins, J. de O., & Jancar, S. (2014). Sepsis-induced lung inflammation is modulated by insulin. BMC Pulmonary Medicine, 14( 177), 8 2014. doi:10.1186/1471-2466-14-177
    • NLM

      Filgueiras L, Capelozzi VL, Martins J de O, Jancar S. Sepsis-induced lung inflammation is modulated by insulin [Internet]. BMC Pulmonary Medicine. 2014 ; 14( 177): 8 2014.[citado 2023 jan. 29 ] Available from: http://www.biomedcentral.com/1471-2466/14/177
    • Vancouver

      Filgueiras L, Capelozzi VL, Martins J de O, Jancar S. Sepsis-induced lung inflammation is modulated by insulin [Internet]. BMC Pulmonary Medicine. 2014 ; 14( 177): 8 2014.[citado 2023 jan. 29 ] Available from: http://www.biomedcentral.com/1471-2466/14/177
  • Source: Journal of Immunology. Unidade: ICB

    Subjects: IMUNOLOGIA, LEUCOTRIENOS B, MACRÓFAGOS

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      WANG, Zhuo et al. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, v. 192, n. 5, p. 2349-2356, 2014Tradução . . Disponível em: http://dx.doi.org/10.4049/jimmunol.1302982. Acesso em: 29 jan. 2023.
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      Wang, Z., Filgueiras, L. R., Wang, S., Serezani, A. P. M., Peters-Golden, M., Jancar, S., & Serezani, C. H. (2014). Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation. Journal of Immunology, 192( 5), 2349-2356. doi:10.4049/jimmunol.1302982
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      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.4049/jimmunol.1302982
    • Vancouver

      Wang Z, Filgueiras LR, Wang S, Serezani APM, Peters-Golden M, Jancar S, Serezani CH. Leukotriene B4 enhances the generation of proinflammatory MicroRNAs to promote MyD88-dependent macrophage activation [Internet]. Journal of Immunology. 2014 ; 192( 5): 2349-2356.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.4049/jimmunol.1302982
  • Source: Laboratory Investigation. Unidades: ICB, FM

    Subjects: IMUNOLOGIA, PLAQUETAS SANGUINEAS, INFLAMAÇÃO

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      CORREA-COSTA, Matheus et al. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis. Laboratory Investigation, v. 94, n. 4, p. 455-466, 2014Tradução . . Disponível em: http://dx.doi.org/10.1038/labinvest.2013.155. Acesso em: 29 jan. 2023.
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      Correa-Costa, M., Andrade-Oliveira, V., Braga, T. T., Castoldi, A., Aguiar, C. F. de, Origassa, C. S. T., et al. (2014). Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis. Laboratory Investigation, 94( 4), 455-466. doi:10.1038/labinvest.2013.155
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      Correa-Costa M, Andrade-Oliveira V, Braga TT, Castoldi A, Aguiar CF de, Origassa CST, Rodas ACD, Hiyane MI, Malheiros DMAC, Rios FJO, Jancar S, Câmara NOS. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis [Internet]. Laboratory Investigation. 2014 ; 94( 4): 455-466.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1038/labinvest.2013.155
    • Vancouver

      Correa-Costa M, Andrade-Oliveira V, Braga TT, Castoldi A, Aguiar CF de, Origassa CST, Rodas ACD, Hiyane MI, Malheiros DMAC, Rios FJO, Jancar S, Câmara NOS. Activation of platelet-activating factor receptor exacerbates renal inflammation and promotes fibrosis [Internet]. Laboratory Investigation. 2014 ; 94( 4): 455-466.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1038/labinvest.2013.155
  • Source: Mediators of Inflammation. Unidade: ICB

    Assunto: IMUNOLOGIA

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      FERRACINI, Matheus et al. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor. Mediators of Inflammation, v. 2013, p. 1-8, 2013Tradução . . Disponível em: http://dx.doi.org/10.1155/2013/950273. Acesso em: 29 jan. 2023.
    • APA

      Ferracini, M., Rios, F. J. O., Pecenin, M., & Jancar, S. (2013). Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor. Mediators of Inflammation, 2013, 1-8. doi:10.1155/2013/950273
    • NLM

      Ferracini M, Rios FJO, Pecenin M, Jancar S. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2013/950273
    • Vancouver

      Ferracini M, Rios FJO, Pecenin M, Jancar S. Clearance of apoptotic cells by macrophages induces regulatory phenotype and involves stimulation of CD36 and platelet-activating factor receptor [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2013/950273
  • Source: Mediators of Inflammation. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      RIOS, Francisco J. et al. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR. Mediators of Inflammation, v. 2013, p. 1-8, 2013Tradução . . Disponível em: http://dx.doi.org/10.1155/2013/198193. Acesso em: 29 jan. 2023.
    • APA

      Rios, F. J., Koga, M. M., Pecenin, M., Ferracini, M., Gidlund, M. A., & Jancar, S. (2013). Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR. Mediators of Inflammation, 2013, 1-8. doi:10.1155/2013/198193
    • NLM

      Rios FJ, Koga MM, Pecenin M, Ferracini M, Gidlund MA, Jancar S. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2013/198193
    • Vancouver

      Rios FJ, Koga MM, Pecenin M, Ferracini M, Gidlund MA, Jancar S. Oxidized LDL induces alternative macrophage phenotype through activation of CD36 and PAFR [Internet]. Mediators of Inflammation. 2013 ; 2013 1-8.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1155/2013/198193
  • Source: Prostaglandins, Leukotrienes and Essential Fatty Acids. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      KOGA, Marianna Mainardi et al. Activation of PAF-receptor induces regulatory dendritic cells through PGE2 and IL-10. Prostaglandins, Leukotrienes and Essential Fatty Acids, v. 89, n. 05, p. 319-326, 2013Tradução . . Disponível em: http://dx.doi.org/10.1016/j.plefa.2013.09.003. Acesso em: 29 jan. 2023.
    • APA

      Koga, M. M., Bizzarro, B., Sá-Nunes, A., Rios, F. J. O., & Jancar, S. (2013). Activation of PAF-receptor induces regulatory dendritic cells through PGE2 and IL-10. Prostaglandins, Leukotrienes and Essential Fatty Acids, 89( 05), 319-326. doi:10.1016/j.plefa.2013.09.003
    • NLM

      Koga MM, Bizzarro B, Sá-Nunes A, Rios FJO, Jancar S. Activation of PAF-receptor induces regulatory dendritic cells through PGE2 and IL-10 [Internet]. Prostaglandins, Leukotrienes and Essential Fatty Acids. 2013 ; 89( 05): 319-326.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.plefa.2013.09.003
    • Vancouver

      Koga MM, Bizzarro B, Sá-Nunes A, Rios FJO, Jancar S. Activation of PAF-receptor induces regulatory dendritic cells through PGE2 and IL-10 [Internet]. Prostaglandins, Leukotrienes and Essential Fatty Acids. 2013 ; 89( 05): 319-326.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1016/j.plefa.2013.09.003
  • Source: PLOS ONE. Unidade: ICB

    Assunto: IMUNOLOGIA

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    • ABNT

      FRANCISCO J. O. RIOS, et al. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts. PLOS ONE, v. 8, n. 10, p. 1-13, 2013Tradução . . Disponível em: http://dx.doi.org/10.1371/journal.pone.0076893. Acesso em: 29 jan. 2023.
    • APA

      Francisco J. O. Rios,, Ferracini, M., Pecenin, M., Koga, M. M., Wang, Y., Ketelhuth, D. F. J., & Jancar, S. (2013). Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts. PLOS ONE, 8( 10), 1-13. doi:10.1371/journal.pone.0076893
    • NLM

      Francisco J. O. Rios, Ferracini M, Pecenin M, Koga MM, Wang Y, Ketelhuth DFJ, Jancar S. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts [Internet]. PLOS ONE. 2013 ;8( 10): 1-13.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1371/journal.pone.0076893
    • Vancouver

      Francisco J. O. Rios, Ferracini M, Pecenin M, Koga MM, Wang Y, Ketelhuth DFJ, Jancar S. Uptake of oxLDL and IL-10 production by macrophages requires PAFR and CD36 recruitment into the same lipid rafts [Internet]. PLOS ONE. 2013 ;8( 10): 1-13.[citado 2023 jan. 29 ] Available from: http://dx.doi.org/10.1371/journal.pone.0076893

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