ReP USP - Resultado da busca

Trabalho de evento-resumo periodico
Adaptation of uveal melanoma cells to MEK inhibition can be overcome through HDAC inhibition (2018)
Faião-Flores, Fernanda; Maria-Engler, Silvya Stuchi ; Eroglu, Zeynep; Smalley, Keiran S
Source: Cancer Research. Conference titles: Annual Meeting of the American Association for Cancer Research. Unidade: FCF
Subjects: MELANOMA, METÁSTASE NEOPLÁSICA
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ABNT
FAIÃO-FLORES, Fernanda; MARIA-ENGLER, Silvya Stuchi; EROGLU, Zeynep; SMALLEY, Keiran S. Adaptation of uveal melanoma cells to MEK inhibition can be overcome through HDAC inhibition. Cancer Research[S.l: s.n.], 2018.
APA
Faião-Flores, F., Maria-Engler, S. S., Eroglu, Z., & Smalley, K. S. (2018). Adaptation of uveal melanoma cells to MEK inhibition can be overcome through HDAC inhibition. Cancer Research. Philadelphia.
NLM
Faião-Flores F, Maria-Engler SS, Eroglu Z, Smalley KS. Adaptation of uveal melanoma cells to MEK inhibition can be overcome through HDAC inhibition. Cancer Research. 2018 ; 78( 13): 1232 res. 4814.
Vancouver
Faião-Flores F, Maria-Engler SS, Eroglu Z, Smalley KS. Adaptation of uveal melanoma cells to MEK inhibition can be overcome through HDAC inhibition. Cancer Research. 2018 ; 78( 13): 1232 res. 4814.
Trabalho de evento-resumo
4-NC efficacy in overcoming resistance to BRAF and MEK inhibitors in human melanoma by ER stress (2018)
Fernandes, Débora Kristina Alves; Oliveira, Erica Aparecida de; Faião-Flores, Fernanda; Rebecca, Gretchen Alicea; Turato, Walter Miguel; Weeraratna, Ashani T; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Resumos. Conference titles: International Symposium on Pathophysiology and Toxicology/ISPAT. Unidade: FCF
Subjects: MELANOMA, PROTEÍNAS QUINASES
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ABNT
FERNANDES, Débora Kristina Alves; OLIVEIRA, Erica Aparecida de; FAIÃO-FLORES, Fernanda; et al. 4-NC efficacy in overcoming resistance to BRAF and MEK inhibitors in human melanoma by ER stress. Anais.. São Paulo: FCF/USP, 2018.Disponível em: .
APA
Fernandes, D. K. A., Oliveira, E. A. de, Faião-Flores, F., Rebecca, G. A., Turato, W. M., Weeraratna, A. T., et al. (2018). 4-NC efficacy in overcoming resistance to BRAF and MEK inhibitors in human melanoma by ER stress. In Resumos. São Paulo: FCF/USP. Recuperado de http://simpac.wixsite.com/simpac/symposium-publications
NLM
Fernandes DKA, Oliveira EA de, Faião-Flores F, Rebecca GA, Turato WM, Weeraratna AT, Barros SB de M, Maria-Engler SS. 4-NC efficacy in overcoming resistance to BRAF and MEK inhibitors in human melanoma by ER stress [Internet]. Resumos. 2018 ;Available from: http://simpac.wixsite.com/simpac/symposium-publications
Vancouver
Fernandes DKA, Oliveira EA de, Faião-Flores F, Rebecca GA, Turato WM, Weeraratna AT, Barros SB de M, Maria-Engler SS. 4-NC efficacy in overcoming resistance to BRAF and MEK inhibitors in human melanoma by ER stress [Internet]. Resumos. 2018 ;Available from: http://simpac.wixsite.com/simpac/symposium-publications
Trabalho de evento-resumo periodico
Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of DM-1 compound toxicity in melanoma cells (2017)
Oliveira, Erica Aparecida de; Lima, Diógenes Saulo de; Cardozo, Lucas Esteves; Souza, Garcia Ferreira de; Souza, Nayane de; Alves-Fernandes, Débora Kristina; Faião-Flores, Fernanda; Quincoces, José Agustín Pablo; Barros, Silvia Berlanga de Moraes ; Nakaya, Helder Takashi Imoto ; Monteiro, Gisele ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Workshop on Melanoma Models. Unidade: FCF
Subjects: MELANOMA, BIOINFORMÁTICA
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ABNT
OLIVEIRA, Erica Aparecida de; LIMA, Diógenes Saulo de; CARDOZO, Lucas Esteves; et al. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of DM-1 compound toxicity in melanoma cells. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2017.
APA
Oliveira, E. A. de, Lima, D. S. de, Cardozo, L. E., Souza, G. F. de, Souza, N. de, Alves-Fernandes, D. K., et al. (2017). Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of DM-1 compound toxicity in melanoma cells. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Oliveira EA de, Lima DS de, Cardozo LE, Souza GF de, Souza N de, Alves-Fernandes DK, Faião-Flores F, Quincoces JAP, Barros SB de M, Nakaya HTI, Monteiro G, Maria-Engler SS. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of DM-1 compound toxicity in melanoma cells. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53 9 res. WM-010.
Vancouver
Oliveira EA de, Lima DS de, Cardozo LE, Souza GF de, Souza N de, Alves-Fernandes DK, Faião-Flores F, Quincoces JAP, Barros SB de M, Nakaya HTI, Monteiro G, Maria-Engler SS. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of DM-1 compound toxicity in melanoma cells. Brazilian Journal of Pharmaceutical Sciences. 2017 ; 53 9 res. WM-010.
Artigo de periodico
Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of a curcumin-analogue DM-1 toxicity in melanoma cells (2017)
Oliveira, Érica Aparecida de; Lima, Diógenes Saulo de; Cardozo, Lucas Esteves; Souza, Garcia Ferreira de; Souza, Nayane de; Alves-Fernandes, Débora Kristina; Faião-Flores, Fernanda; Quincoces, José Agustín Pablo; Barros, Silvia Berlanga de Moraes ; Nakaya, Helder Takashi Imoto ; Monteiro, Gisele ; Maria-Engler, Silvya Stuchi
Source: Pharmacological Research. Unidade: FCF
Subjects: MELANOMA, BIOINFORMÁTICA
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ABNT
OLIVEIRA, Érica Aparecida de; LIMA, Diógenes Saulo de; CARDOZO, Lucas Esteves; et al. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of a curcumin-analogue DM-1 toxicity in melanoma cells. Pharmacological Research, London, v. 125 parte. B, p. 178-187, 2017. Disponível em: < http://dx.doi.org/10.1016/j.phrs.2017.08.018 > DOI: 10.1016/j.phrs.2017.08.018.
APA
Oliveira, É. A. de, Lima, D. S. de, Cardozo, L. E., Souza, G. F. de, Souza, N. de, Alves-Fernandes, D. K., et al. (2017). Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of a curcumin-analogue DM-1 toxicity in melanoma cells. Pharmacological Research, 125 parte. B, 178-187. doi:10.1016/j.phrs.2017.08.018
NLM
Oliveira ÉA de, Lima DS de, Cardozo LE, Souza GF de, Souza N de, Alves-Fernandes DK, Faião-Flores F, Quincoces JAP, Barros SB de M, Nakaya HTI, Monteiro G, Maria-Engler SS. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of a curcumin-analogue DM-1 toxicity in melanoma cells [Internet]. Pharmacological Research. 2017 ; 125 parte. B 178-187.Available from: http://dx.doi.org/10.1016/j.phrs.2017.08.018
Vancouver
Oliveira ÉA de, Lima DS de, Cardozo LE, Souza GF de, Souza N de, Alves-Fernandes DK, Faião-Flores F, Quincoces JAP, Barros SB de M, Nakaya HTI, Monteiro G, Maria-Engler SS. Toxicogenomic and bioinformatics platforms to identify key molecular mechanisms of a curcumin-analogue DM-1 toxicity in melanoma cells [Internet]. Pharmacological Research. 2017 ; 125 parte. B 178-187.Available from: http://dx.doi.org/10.1016/j.phrs.2017.08.018
Trabalho de evento-resumo periodico
Targeting hedgehog transcription factors GLl1 and GLl2 restores sensitivity to vemurafenib resistant human melanoma cells (2016)
Faião-Flores, Fernanda; Fernandes, Débora Kristina Magalhães Alves; Pennacchi, Paula Comune; Sandri, Silvana; Vicente, Anna Luiza Silva Almeida; Scapulatempo Neto, Cristovam; Vasquez, Vinicius de Lima; Reis, Rui Manuel Vieira; Smalley, Keiran S; Maria-Engler, Silvya Stuchi
Source: Cancer Research. Conference titles: Annual Meeting of the American Association for Cancer Research (AACR). Unidade: FCF
Assunto: MELANOMA
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ABNT
FAIÃO-FLORES, Fernanda; FERNANDES, Débora Kristina Magalhães Alves; PENNACCHI, Paula Comune; et al. Targeting hedgehog transcription factors GLl1 and GLl2 restores sensitivity to vemurafenib resistant human melanoma cells. Cancer Research[S.l: s.n.], 2016.Disponível em: .
APA
Faião-Flores, F., Fernandes, D. K. M. A., Pennacchi, P. C., Sandri, S., Vicente, A. L. S. A., Scapulatempo Neto, C., et al. (2016). Targeting hedgehog transcription factors GLl1 and GLl2 restores sensitivity to vemurafenib resistant human melanoma cells. Cancer Research. Philadelphia. Recuperado de http://cancerres.aacrjournals.org/content/76/14_supplement/4669.short
NLM
Faião-Flores F, Fernandes DKMA, Pennacchi PC, Sandri S, Vicente ALSA, Scapulatempo Neto C, Vasquez V de L, Reis RMV, Smalley KS, Maria-Engler SS. Targeting hedgehog transcription factors GLl1 and GLl2 restores sensitivity to vemurafenib resistant human melanoma cells [Internet]. Cancer Research. 2016 ; 76( 14):Available from: http://cancerres.aacrjournals.org/content/76/14_supplement/4669.short
Vancouver
Faião-Flores F, Fernandes DKMA, Pennacchi PC, Sandri S, Vicente ALSA, Scapulatempo Neto C, Vasquez V de L, Reis RMV, Smalley KS, Maria-Engler SS. Targeting hedgehog transcription factors GLl1 and GLl2 restores sensitivity to vemurafenib resistant human melanoma cells [Internet]. Cancer Research. 2016 ; 76( 14):Available from: http://cancerres.aacrjournals.org/content/76/14_supplement/4669.short
Trabalho de evento-resumo periodico
Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1 (2016)
Oliveira, Erica Aparecida de; Souza, Garcia Ferreira de; Lima, Diógenes Saulo de; Cardozo, Lucas Esteves; Faião-Flores, Fernanda; Suárez, José Agustín Pablo Quincoces; Nakaya, Helder Takashi Imoto ; Monteiro, Gisele ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Sciences and Technology Meeting of the Faculty of Pharmaceutical Sciences, University of São Paulo. Unidade: FCF
Subjects: MELANOMA, BIOLOGIA
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ABNT
OLIVEIRA, Erica Aparecida de; SOUZA, Garcia Ferreira de; LIMA, Diógenes Saulo de; et al. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2016.
APA
Oliveira, E. A. de, Souza, G. F. de, Lima, D. S. de, Cardozo, L. E., Faião-Flores, F., Suárez, J. A. P. Q., et al. (2016). Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Oliveira EA de, Souza GF de, Lima DS de, Cardozo LE, Faião-Flores F, Suárez JAPQ, Nakaya HTI, Monteiro G, Maria-Engler SS. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 14 res. FCF024.
Vancouver
Oliveira EA de, Souza GF de, Lima DS de, Cardozo LE, Faião-Flores F, Suárez JAPQ, Nakaya HTI, Monteiro G, Maria-Engler SS. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 14 res. FCF024.
Trabalho de evento-resumo periodico
The curcumin analog DM-1 inhibits migration and downregulates MMP-2 in vemurafenib resistant melanoma cell lines (2016)
Souza, Nayane de; Oliveira, Érica Aparecida de; Faião-Flores, Fernanda; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Sciences and Technology Meeting of the Faculty of Pharmaceutical Sciences, University of São Paulo. Unidade: FCF
Assunto: MELANOMA
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ABNT
SOUZA, Nayane de; OLIVEIRA, Érica Aparecida de; FAIÃO-FLORES, Fernanda; MARIA-ENGLER, Silvya Stuchi. The curcumin analog DM-1 inhibits migration and downregulates MMP-2 in vemurafenib resistant melanoma cell lines. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2016.
APA
Souza, N. de, Oliveira, É. A. de, Faião-Flores, F., & Maria-Engler, S. S. (2016). The curcumin analog DM-1 inhibits migration and downregulates MMP-2 in vemurafenib resistant melanoma cell lines. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Souza N de, Oliveira ÉA de, Faião-Flores F, Maria-Engler SS. The curcumin analog DM-1 inhibits migration and downregulates MMP-2 in vemurafenib resistant melanoma cell lines. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 40 res. FCF076.
Vancouver
Souza N de, Oliveira ÉA de, Faião-Flores F, Maria-Engler SS. The curcumin analog DM-1 inhibits migration and downregulates MMP-2 in vemurafenib resistant melanoma cell lines. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 40 res. FCF076.
Trabalho de evento-resumo
Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1 (2016)
Oliveira, Erica Aparecida de; Souza, Garcia Ferreira de; Lima, Diógenes Saulo de; Cardozo, Lucas Esteves; Faião-Flores, Fernanda; Suárez, José Agustín Pablo Quincoces; Nakaya, Helder Takashi Imoto ; Monteiro, Gisele ; Maria-Engler, Silvya Stuchi
Source: Resumos. Conference titles: Congresso da Sociedade Brasileira de Biologia Celular. Unidade: FCF
Subjects: MELANOMA, SACCHAROMYCES
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ABNT
OLIVEIRA, Erica Aparecida de; SOUZA, Garcia Ferreira de; LIMA, Diógenes Saulo de; et al. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Anais.. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC), 2016.
APA
Oliveira, E. A. de, Souza, G. F. de, Lima, D. S. de, Cardozo, L. E., Faião-Flores, F., Suárez, J. A. P. Q., et al. (2016). Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. In Resumos. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC).
NLM
Oliveira EA de, Souza GF de, Lima DS de, Cardozo LE, Faião-Flores F, Suárez JAPQ, Nakaya HTI, Monteiro G, Maria-Engler SS. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Resumos. 2016 ;
Vancouver
Oliveira EA de, Souza GF de, Lima DS de, Cardozo LE, Faião-Flores F, Suárez JAPQ, Nakaya HTI, Monteiro G, Maria-Engler SS. Integrative toxicogenomic and system biology platforms to target melanoma genes by DM-1. Resumos. 2016 ;
Trabalho de evento-resumo periodico
Antitumoral activity of 4-nerolidylcathecol (4-NC) in naïve and vemurafenib-resistant melanoma cells (2016)
Alves-Fernandes, Débora Kristina; Faião-Flores, Fernanda; Sandri, Silvana; Oliveira, Érica Aparecida de; Turato, Walter Miguel; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Sciences and Technology Meeting of the Faculty of Pharmaceutical Sciences, University of São Paulo. Unidade: FCF
Subjects: CÉLULAS, MELANOMA
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ABNT
ALVES-FERNANDES, Débora Kristina; FAIÃO-FLORES, Fernanda; SANDRI, Silvana; et al. Antitumoral activity of 4-nerolidylcathecol (4-NC) in naïve and vemurafenib-resistant melanoma cells. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2016.
APA
Alves-Fernandes, D. K., Faião-Flores, F., Sandri, S., Oliveira, É. A. de, Turato, W. M., Barros, S. B. de M., & Maria-Engler, S. S. (2016). Antitumoral activity of 4-nerolidylcathecol (4-NC) in naïve and vemurafenib-resistant melanoma cells. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Alves-Fernandes DK, Faião-Flores F, Sandri S, Oliveira ÉA de, Turato WM, Barros SB de M, Maria-Engler SS. Antitumoral activity of 4-nerolidylcathecol (4-NC) in naïve and vemurafenib-resistant melanoma cells. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 17 res. FCF030.
Vancouver
Alves-Fernandes DK, Faião-Flores F, Sandri S, Oliveira ÉA de, Turato WM, Barros SB de M, Maria-Engler SS. Antitumoral activity of 4-nerolidylcathecol (4-NC) in naïve and vemurafenib-resistant melanoma cells. Brazilian Journal of Pharmaceutical Sciences. 2016 ; 52 17 res. FCF030.
Trabalho de evento-resumo
4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model (2016)
Fernandes, Débora Kristina Alves; Faião-Flores, Fernanda; Sandri, Silvana; Oliveira, Erica Aparecida de; Turato, Walter Miguel; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Resumos. Conference titles: Congresso da Sociedade Brasileira de Biologia Celular. Unidade: FCF
Subjects: MELANOMA, DANO AO DNA
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ABNT
FERNANDES, Débora Kristina Alves; FAIÃO-FLORES, Fernanda; SANDRI, Silvana; et al. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. Anais.. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC), 2016.
APA
Fernandes, D. K. A., Faião-Flores, F., Sandri, S., Oliveira, E. A. de, Turato, W. M., Barros, S. B. de M., & Maria-Engler, S. S. (2016). 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. In Resumos. São Paulo: Sociedade Brasileira de Biologia Celular (SBBC).
NLM
Fernandes DKA, Faião-Flores F, Sandri S, Oliveira EA de, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. Resumos. 2016 ;
Vancouver
Fernandes DKA, Faião-Flores F, Sandri S, Oliveira EA de, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces antiproliferative effects in naïve and vemurafenib-resistant melanoma cells in vitro in xenograft model. Resumos. 2016 ;
Trabalho de evento-resumo periodico
4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma (2015)
Alves, Débora Kristina Magalhães; Faião-Flores, Fernanda; Turato, Walter Miguel; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Science and Technology Meeting of the Faculty of Pharmaceutical Sciences, University of São Paulo. Unidade: FCF
Subjects: MELANOMA, DANO AO DNA
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ABNT
ALVES, Débora Kristina Magalhães; FAIÃO-FLORES, Fernanda; TURATO, Walter Miguel; BARROS, Silvia Berlanga de Moraes; MARIA-ENGLER, Silvya Stuchi. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2015.
APA
Alves, D. K. M., Faião-Flores, F., Turato, W. M., Barros, S. B. de M., & Maria-Engler, S. S. (2015). 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Alves DKM, Faião-Flores F, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51 44 res. FCF083.
Vancouver
Alves DKM, Faião-Flores F, Turato WM, Barros SB de M, Maria-Engler SS. 4-Nerolidylcathecol (4-NC) induces tumoral growth inhibition in melanoma. Brazilian Journal of Pharmaceutical Sciences. 2015 ; 51 44 res. FCF083.
Artigo de periodico
MMP-9/RECK Imbalance: a mechanism associated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis (2015)
Discacciati, Michelle G; Gimenes, Fabricia; Pennacchi, Paula Comune; Faião-Flores, Fernanda; Zeferino, Luiz Carlos; Derchain, Sophie Françoise Mauricette; Teixiera, Júlio C; Costa, Maria C; Zonta, Marco Antonio; Termini, Lara; Boccardo, Enrique; Longatto Filho, Adhemar; Consolaro, Marcia Edilaine Lopes; Villa, Luisa Lina; Maria-Engler, Silvya Stuchi
Source: Cancer Epidemiology Biomarkers & Prevention. Unidades: FCF, FM, ICB
Subjects: HPV, CHLAMYDIA
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ABNT
DISCACCIATI, Michelle G; GIMENES, Fabricia; PENNACCHI, Paula Comune; et al. MMP-9/RECK Imbalance: a mechanism associated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis. Cancer Epidemiology Biomarkers & Prevention, Philadelphia, v. 24, n. 10, p. 1539-1547, 2015. Disponível em: < http://dx.doi.org/10.1158/1055-9965.EPI-15-0420 > DOI: 10.1158/1055-9965.EPI-15-0420.
APA
Discacciati, M. G., Gimenes, F., Pennacchi, P. C., Faião-Flores, F., Zeferino, L. C., Derchain, S. F. M., et al. (2015). MMP-9/RECK Imbalance: a mechanism associated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis. Cancer Epidemiology Biomarkers & Prevention, 24( 10), 1539-1547. doi:10.1158/1055-9965.EPI-15-0420
NLM
Discacciati MG, Gimenes F, Pennacchi PC, Faião-Flores F, Zeferino LC, Derchain SFM, Teixiera JC, Costa MC, Zonta MA, Termini L, Boccardo E, Longatto Filho A, Consolaro MEL, Villa LL, Maria-Engler SS. MMP-9/RECK Imbalance: a mechanism associated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis [Internet]. Cancer Epidemiology Biomarkers & Prevention. 2015 ; 24( 10): 1539-1547.Available from: http://dx.doi.org/10.1158/1055-9965.EPI-15-0420
Vancouver
Discacciati MG, Gimenes F, Pennacchi PC, Faião-Flores F, Zeferino LC, Derchain SFM, Teixiera JC, Costa MC, Zonta MA, Termini L, Boccardo E, Longatto Filho A, Consolaro MEL, Villa LL, Maria-Engler SS. MMP-9/RECK Imbalance: a mechanism associated with high-grade cervical lesions and genital infection by human papillomavirus and Chlamydia trachomatis [Internet]. Cancer Epidemiology Biomarkers & Prevention. 2015 ; 24( 10): 1539-1547.Available from: http://dx.doi.org/10.1158/1055-9965.EPI-15-0420
Trabalho de evento-resumo periodico
RECK is required for migration and invasiveness in melanoma after vemurafenib treatment (2014)
Tiago, Manoela; Faião-Flores, Fernanda; Sandri, Silvana; Pennacchi, Paula Comune; Smalley, Keiram; Maria-Engler, Silvya Stuchi
Source: Pigment Cell & Melanoma. Conference titles: Society for Melanoma Research Congress. Unidade: FCF
Assunto: MELANOMA
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ABNT
TIAGO, Manoela; FAIÃO-FLORES, Fernanda; SANDRI, Silvana; et al. RECK is required for migration and invasiveness in melanoma after vemurafenib treatment. Pigment Cell & Melanoma[S.l: s.n.], 2014.
APA
Tiago, M., Faião-Flores, F., Sandri, S., Pennacchi, P. C., Smalley, K., & Maria-Engler, S. S. (2014). RECK is required for migration and invasiveness in melanoma after vemurafenib treatment. Pigment Cell & Melanoma. Hoboken.
NLM
Tiago M, Faião-Flores F, Sandri S, Pennacchi PC, Smalley K, Maria-Engler SS. RECK is required for migration and invasiveness in melanoma after vemurafenib treatment. Pigment Cell & Melanoma. 2014 ; 27( 6): 1234.
Vancouver
Tiago M, Faião-Flores F, Sandri S, Pennacchi PC, Smalley K, Maria-Engler SS. RECK is required for migration and invasiveness in melanoma after vemurafenib treatment. Pigment Cell & Melanoma. 2014 ; 27( 6): 1234.
Trabalho de evento-resumo periodico
Epithelial mesenchymal transition as an alternative pathway in vemurafenib and vemurafenib/trametinib chemoresistant melanoma (2014)
Faião-Flores, Fernanda; Alves, D. K; Sandri, Silvana; Maria-Engler, Silvya Stuchi
Source: Pigment Cell & Melanoma. Conference titles: Society for Melanoma Research Congress. Unidade: FCF
Subjects: CÉLULAS EPITELIAIS, MELANOMA
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ABNT
FAIÃO-FLORES, Fernanda; ALVES, D. K; SANDRI, Silvana; MARIA-ENGLER, Silvya Stuchi. Epithelial mesenchymal transition as an alternative pathway in vemurafenib and vemurafenib/trametinib chemoresistant melanoma. Pigment Cell & Melanoma[S.l: s.n.], 2014.
APA
Faião-Flores, F., Alves, D. K., Sandri, S., & Maria-Engler, S. S. (2014). Epithelial mesenchymal transition as an alternative pathway in vemurafenib and vemurafenib/trametinib chemoresistant melanoma. Pigment Cell & Melanoma. Hoboken.
NLM
Faião-Flores F, Alves DK, Sandri S, Maria-Engler SS. Epithelial mesenchymal transition as an alternative pathway in vemurafenib and vemurafenib/trametinib chemoresistant melanoma. Pigment Cell & Melanoma. 2014 ; 27( 6): 1190.
Vancouver
Faião-Flores F, Alves DK, Sandri S, Maria-Engler SS. Epithelial mesenchymal transition as an alternative pathway in vemurafenib and vemurafenib/trametinib chemoresistant melanoma. Pigment Cell & Melanoma. 2014 ; 27( 6): 1190.
Trabalho de evento-resumo periodico
Evaluation of antimetastatic activity in vemurafenib resistant melanoma cell lines after DM-1 treatment (2014)
Souza, Nayane de; Suárez, José Quincoces; Maria-Engler, Silvya Stuchi ; Ikeda, Gustavo Takashi; Faião-Flores, Fernanda; Pennacchi, Paula Comune
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Science and Technology Meeting of the Faculty of Pharmaceutical Sciences. Unidade: FCF
Subjects: MELANOMA, RESISTÊNCIA MICROBIANA ÀS DROGAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Nayane de; SUÁREZ, José Quincoces; MARIA-ENGLER, Silvya Stuchi; et al. Evaluation of antimetastatic activity in vemurafenib resistant melanoma cell lines after DM-1 treatment. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2014.
APA
Souza, N. de, Suárez, J. Q., Maria-Engler, S. S., Ikeda, G. T., Faião-Flores, F., & Pennacchi, P. C. (2014). Evaluation of antimetastatic activity in vemurafenib resistant melanoma cell lines after DM-1 treatment. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Souza N de, Suárez JQ, Maria-Engler SS, Ikeda GT, Faião-Flores F, Pennacchi PC. Evaluation of antimetastatic activity in vemurafenib resistant melanoma cell lines after DM-1 treatment. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50 4 res. FCF003.
Vancouver
Souza N de, Suárez JQ, Maria-Engler SS, Ikeda GT, Faião-Flores F, Pennacchi PC. Evaluation of antimetastatic activity in vemurafenib resistant melanoma cell lines after DM-1 treatment. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50 4 res. FCF003.
Trabalho de evento-resumo periodico
Apoptosis expression in vemurafenib-resistant melanoma cells induced by curcumin analog DM-1 (2014)
Ikeda, Gustavo Takashi; Souza, Nayane de; Pennacchi, Paula Comune; Faião-Flores, Fernanda; Suárez, José Quincoces; Maria-Engler, Silvya Stuchi
Source: Brazilian Journal of Pharmaceutical Sciences. Conference titles: Pharmaceutical Science and Technology Meeting of the Faculty of Pharmaceutical Sciences. Unidade: FCF
Subjects: MELANOMA, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
IKEDA, Gustavo Takashi; SOUZA, Nayane de; PENNACCHI, Paula Comune; et al. Apoptosis expression in vemurafenib-resistant melanoma cells induced by curcumin analog DM-1. Brazilian Journal of Pharmaceutical Sciences[S.l: s.n.], 2014.
APA
Ikeda, G. T., Souza, N. de, Pennacchi, P. C., Faião-Flores, F., Suárez, J. Q., & Maria-Engler, S. S. (2014). Apoptosis expression in vemurafenib-resistant melanoma cells induced by curcumin analog DM-1. Brazilian Journal of Pharmaceutical Sciences. São Paulo.
NLM
Ikeda GT, Souza N de, Pennacchi PC, Faião-Flores F, Suárez JQ, Maria-Engler SS. Apoptosis expression in vemurafenib-resistant melanoma cells induced by curcumin analog DM-1. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50 51 res. FCF098.
Vancouver
Ikeda GT, Souza N de, Pennacchi PC, Faião-Flores F, Suárez JQ, Maria-Engler SS. Apoptosis expression in vemurafenib-resistant melanoma cells induced by curcumin analog DM-1. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50 51 res. FCF098.
Artigo de periodico
Basic Red 51, a permitted semi-permanent hair dye, is cytotoxic to human skin cells: studies in monolayer and 3D skin model using human keratinocytes (HaCaT) (2014)
Zanoni, Thalita B.; Tiago, Manoela; Faião-Flores, Fernanda; Barros, Silvia Berlanga de Moraes ; Bast, Aalt; Hageman, Geja; Oliveira, Danielle Palma de; Maria-Engler, Silvya Stuchi
Source: Toxicology Letters. Unidades: FCF, FCFRP
Subjects: TINTURAS (ANÁLISE), CABELO (ANÁLISE), TEMPO DE REAÇÃO, TOXICOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ZANONI, Thalita B.; TIAGO, Manoela; FAIÃO-FLORES, Fernanda; et al. Basic Red 51, a permitted semi-permanent hair dye, is cytotoxic to human skin cells: studies in monolayer and 3D skin model using human keratinocytes (HaCaT). Toxicology Letters, County Clare, v. 227 , p. 139-149, 2014. Disponível em: < http://dx.doi.org/10.1016/j.toxlet.2014.03.007 > DOI: 10.1016/j.toxlet.2014.03.007.
APA
Zanoni, T. B., Tiago, M., Faião-Flores, F., Barros, S. B. de M., Bast, A., Hageman, G., et al. (2014). Basic Red 51, a permitted semi-permanent hair dye, is cytotoxic to human skin cells: studies in monolayer and 3D skin model using human keratinocytes (HaCaT). Toxicology Letters, 227 , 139-149. doi:10.1016/j.toxlet.2014.03.007
NLM
Zanoni TB, Tiago M, Faião-Flores F, Barros SB de M, Bast A, Hageman G, Oliveira DP de, Maria-Engler SS. Basic Red 51, a permitted semi-permanent hair dye, is cytotoxic to human skin cells: studies in monolayer and 3D skin model using human keratinocytes (HaCaT) [Internet]. Toxicology Letters. 2014 ; 227 139-149.Available from: http://dx.doi.org/10.1016/j.toxlet.2014.03.007
Vancouver
Zanoni TB, Tiago M, Faião-Flores F, Barros SB de M, Bast A, Hageman G, Oliveira DP de, Maria-Engler SS. Basic Red 51, a permitted semi-permanent hair dye, is cytotoxic to human skin cells: studies in monolayer and 3D skin model using human keratinocytes (HaCaT) [Internet]. Toxicology Letters. 2014 ; 227 139-149.Available from: http://dx.doi.org/10.1016/j.toxlet.2014.03.007
Trabalho de evento-resumo periodico
Tumoral growth inhibition by 4-nerolidylcathecol (4-NC) in a melanoma tumor model in vivo (2014)
Alves, D. K; Faião-Flores, Fernanda; Barros, Silvia Berlanga de Moraes ; Maria-Engler, Silvya Stuchi
Source: Pigment Cell & Melanoma. Conference titles: Society for Melanoma Research Congress. Unidade: FCF
Assunto: MELANOMA
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ABNT
ALVES, D. K; FAIÃO-FLORES, Fernanda; BARROS, Silvia Berlanga de Moraes; MARIA-ENGLER, Silvya Stuchi. Tumoral growth inhibition by 4-nerolidylcathecol (4-NC) in a melanoma tumor model in vivo. Pigment Cell & Melanoma[S.l: s.n.], 2014.
APA
Alves, D. K., Faião-Flores, F., Barros, S. B. de M., & Maria-Engler, S. S. (2014). Tumoral growth inhibition by 4-nerolidylcathecol (4-NC) in a melanoma tumor model in vivo. Pigment Cell & Melanoma. Hoboken.
NLM
Alves DK, Faião-Flores F, Barros SB de M, Maria-Engler SS. Tumoral growth inhibition by 4-nerolidylcathecol (4-NC) in a melanoma tumor model in vivo. Pigment Cell & Melanoma. 2014 ; 27( 6): 1170.
Vancouver
Alves DK, Faião-Flores F, Barros SB de M, Maria-Engler SS. Tumoral growth inhibition by 4-nerolidylcathecol (4-NC) in a melanoma tumor model in vivo. Pigment Cell & Melanoma. 2014 ; 27( 6): 1170.
Artigo de periodico
Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy (2013)
Faião-Flores, Fernanda; Coelho, Paulo Rogério Pinto; Arruda-Neto, João Dias Toledo; Maria-Engler, Silvya Stuchi ; Tiago, Manoela; Capelozzi, Vera Luiza; Giorgi, Ricardo Rodrigues; Maria, Durvanei Augusto
Source: PLOS ONE. Unidades: IF, FCF, FM
Subjects: APOPTOSE, CULTURA DE CÉLULAS, MELANOMA, CITOMETRIA DE FLUXO, REAÇÃO EM CADEIA POR POLIMERASE, ELETROFORESE EM GEL, IMUNOHISTOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAIÃO-FLORES, Fernanda; COELHO, Paulo Rogério Pinto; ARRUDA-NETO, João Dias Toledo; et al. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy. PLOS ONE, San Francisco, v. 8, n. 3, p. 12 , 2013. Disponível em: < http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0059639 > DOI: 10.1371/journal.pone.0059639.
APA
Faião-Flores, F., Coelho, P. R. P., Arruda-Neto, J. D. T., Maria-Engler, S. S., Tiago, M., Capelozzi, V. L., et al. (2013). Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy. PLOS ONE, 8( 3), 12 . doi:10.1371/journal.pone.0059639
NLM
Faião-Flores F, Coelho PRP, Arruda-Neto JDT, Maria-Engler SS, Tiago M, Capelozzi VL, Giorgi RR, Maria DA. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy [Internet]. PLOS ONE. 2013 ; 8( 3): 12 .Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0059639
Vancouver
Faião-Flores F, Coelho PRP, Arruda-Neto JDT, Maria-Engler SS, Tiago M, Capelozzi VL, Giorgi RR, Maria DA. Apoptosis through Bcl-2/Bax and Cleaved Caspase Up-Regulation in Melanoma Treated by Boron Neutron Capture Therapy [Internet]. PLOS ONE. 2013 ; 8( 3): 12 .Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0059639
Artigo de periodico
The curcumin analog DM-1 induces apoptotic cell death in melanoma (2013)
Faião-Flores, Fernanda; Suárez, José Agustín Quincoces; Maria-Engler, Silvya Stuchi ; Soto-Cerrato, Vanessa; Perez-Tomás, Ricardo; Maria, Durvanei Augusto
Source: Tumor Biology. Unidade: FCF
Subjects: MELANOMA, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAIÃO-FLORES, Fernanda; SUÁREZ, José Agustín Quincoces; MARIA-ENGLER, Silvya Stuchi; et al. The curcumin analog DM-1 induces apoptotic cell death in melanoma. Tumor Biology, Dordrecht, v. 34, n. 2, p. 1119-1129, 2013. Disponível em: < http://dx.doi.org/10.1007/s13277-013-0653-y > DOI: 10.1007/s13277-013-0653-y.
APA
Faião-Flores, F., Suárez, J. A. Q., Maria-Engler, S. S., Soto-Cerrato, V., Perez-Tomás, R., & Maria, D. A. (2013). The curcumin analog DM-1 induces apoptotic cell death in melanoma. Tumor Biology, 34( 2), 1119-1129. doi:10.1007/s13277-013-0653-y
NLM
Faião-Flores F, Suárez JAQ, Maria-Engler SS, Soto-Cerrato V, Perez-Tomás R, Maria DA. The curcumin analog DM-1 induces apoptotic cell death in melanoma [Internet]. Tumor Biology. 2013 ; 34( 2): 1119-1129.Available from: http://dx.doi.org/10.1007/s13277-013-0653-y
Vancouver
Faião-Flores F, Suárez JAQ, Maria-Engler SS, Soto-Cerrato V, Perez-Tomás R, Maria DA. The curcumin analog DM-1 induces apoptotic cell death in melanoma [Internet]. Tumor Biology. 2013 ; 34( 2): 1119-1129.Available from: http://dx.doi.org/10.1007/s13277-013-0653-y

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