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Artigo de periodico
Tumor glycolytic profiling through 18F-FDG PET/CT predicts immune checkpoint inhibitor efficacy in advanced NSCLC (2022)
Silva, Saulo Brito; Wanderley, Carlos Wagner Souza; Marin, José Flavio Gomes; Macedo, Mariana Petaccia de; Nascimento, Ellen Caroline Toledo do; Antonacio, Fernanda Fronzoni; Figueiredo, Caroline Sales; Cunha, Mateus Trinconi ; Cunha, Fernando de Queiroz ; Castro Junior, Gilberto de
Source: Therapeutic Advances in Medical Oncology. Unidades: FMRP, FM
Subjects: BIOMARCADORES, GLICOSE, IMUNOTERAPIA, NEOPLASIAS PULMONARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Saulo Brito et al. Tumor glycolytic profiling through 18F-FDG PET/CT predicts immune checkpoint inhibitor efficacy in advanced NSCLC. Therapeutic Advances in Medical Oncology, v. 14, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1177/17588359221138386. Acesso em: 11 nov. 2024.
APA
Silva, S. B., Wanderley, C. W. S., Marin, J. F. G., Macedo, M. P. de, Nascimento, E. C. T. do, Antonacio, F. F., et al. (2022). Tumor glycolytic profiling through 18F-FDG PET/CT predicts immune checkpoint inhibitor efficacy in advanced NSCLC. Therapeutic Advances in Medical Oncology, 14, 1-13. doi:10.1177/17588359221138386
NLM
Silva SB, Wanderley CWS, Marin JFG, Macedo MP de, Nascimento ECT do, Antonacio FF, Figueiredo CS, Cunha MT, Cunha F de Q, Castro Junior G de. Tumor glycolytic profiling through 18F-FDG PET/CT predicts immune checkpoint inhibitor efficacy in advanced NSCLC [Internet]. Therapeutic Advances in Medical Oncology. 2022 ; 14 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1177/17588359221138386
Vancouver
Silva SB, Wanderley CWS, Marin JFG, Macedo MP de, Nascimento ECT do, Antonacio FF, Figueiredo CS, Cunha MT, Cunha F de Q, Castro Junior G de. Tumor glycolytic profiling through 18F-FDG PET/CT predicts immune checkpoint inhibitor efficacy in advanced NSCLC [Internet]. Therapeutic Advances in Medical Oncology. 2022 ; 14 1-13.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1177/17588359221138386
Artigo de periodico
Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation (2021)
Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner Souza; Veras, Flávio Protásio ; Sônego, Fabiane; Nascimento, Daniele C.; Gonçalves, Augusto Velozo ; Martins, Timna Varela; Morelo, David Fernando Colon ; Borges, Vanessa F.; Brauer, Verônica Soares; Damasceno, Luis Eduardo Alves ; Silva, Katiussia P.; Toller-kawahisa, Juliana Escher; Batah, Sabrina Setembre ; Souza, Ana Letícia Júlio de; Monteiro, Valter Vinícius Silva; Oliveira, Antonio Edson Rocha; Donate, Paula Barbim; Zoppi, Daniel; Borges, Marcos de Carvalho ; Almeida, Fausto Bruno dos Reis ; Nakaya, Helder Takashi Imoto ; Fabro, Alexandre Todorovic ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Zamboni, Dario Simões ; Cunha, Fernando de Queiroz
Source: Blood. Unidades: FCFRP, FMRP, FCF
Subjects: FAGÓCITOS, GRANULÓCITOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Camila Meirelles de Souza et al. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation. Blood, v. 138, n. 25, p. 2702-2713, 2021Tradução . . Disponível em: https://doi.org/10.1182/blood.2021011525. Acesso em: 11 nov. 2024.
APA
Silva, C. M. de S., Wanderley, C. W. S., Veras, F. P., Sônego, F., Nascimento, D. C., Gonçalves, A. V., et al. (2021). Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation. Blood, 138( 25), 2702-2713. doi:10.1182/blood.2021011525
NLM
Silva CM de S, Wanderley CWS, Veras FP, Sônego F, Nascimento DC, Gonçalves AV, Martins TV, Morelo DFC, Borges VF, Brauer VS, Damasceno LEA, Silva KP, Toller-kawahisa JE, Batah SS, Souza ALJ de, Monteiro VVS, Oliveira AER, Donate PB, Zoppi D, Borges M de C, Almeida FB dos R, Nakaya HTI, Fabro AT, Cunha TM, Alves Filho JCF, Zamboni DS, Cunha F de Q. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation [Internet]. Blood. 2021 ; 138( 25): 2702-2713.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1182/blood.2021011525
Vancouver
Silva CM de S, Wanderley CWS, Veras FP, Sônego F, Nascimento DC, Gonçalves AV, Martins TV, Morelo DFC, Borges VF, Brauer VS, Damasceno LEA, Silva KP, Toller-kawahisa JE, Batah SS, Souza ALJ de, Monteiro VVS, Oliveira AER, Donate PB, Zoppi D, Borges M de C, Almeida FB dos R, Nakaya HTI, Fabro AT, Cunha TM, Alves Filho JCF, Zamboni DS, Cunha F de Q. Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation [Internet]. Blood. 2021 ; 138( 25): 2702-2713.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1182/blood.2021011525
Artigo de periodico
Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide (2018)
Dornelas-Filho, Amílcar Figueiredo; Pereira, Venúcia Bruna Magalhães; Wong, Deysi Viviana T.; Nobre, Lívia Maria Soares; Melo, Anielle Torres; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner Souza; Nour, Mariana Lima; Araújo, Lis Caetano Nobrega Costa; Silva, Renan Oliveira; Pinto, Francisco Maxwell Martins; Bingana, Rudy Diavila; Souza, Marcellus Henrique Loiola Ponte de; Alencar, Nylane Maria Nunes; Silva, Paulo Goberlânio B.; Alves, Ana Paula Negreiros Nunes; Almeida, Paulo Roberto Carvalho; Cunha, Fernando de Queiroz; Lima Júnior, Roberto César P.
Source: International Immunopharmacology. Unidade: FMRP
Subjects: ANTINEOPLÁSICOS, CISTITE, HEMORRAGIA, NEUTRÓFILOS, POLISSACARÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DORNELAS-FILHO, Amílcar Figueiredo et al. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, v. 62, p. 96-108, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.06.031. Acesso em: 11 nov. 2024.
APA
Dornelas-Filho, A. F., Pereira, V. B. M., Wong, D. V. T., Nobre, L. M. S., Melo, A. T., Silva, C. M. de S., et al. (2018). Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide. International Immunopharmacology, 62, 96-108. doi:10.1016/j.intimp.2018.06.031
NLM
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031
Vancouver
Dornelas-Filho AF, Pereira VBM, Wong DVT, Nobre LMS, Melo AT, Silva CM de S, Wanderley CWS, Nour ML, Araújo LCNC, Silva RO, Pinto FMM, Bingana RD, Souza MHLP de, Alencar NMN, Silva PGB, Alves APNN, Almeida PRC, Cunha F de Q, Lima Júnior RCP. Neutrophils contribute to the pathogenesis of hemorrhagic cystitis induced by ifosfamide [Internet]. International Immunopharmacology. 2018 ; 62 96-108.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1016/j.intimp.2018.06.031

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