Filtros : "Toledo, S. P. A." "2000" Limpar

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  • Fonte: Fertility & Sterility. Unidade: FM

    Assunto: CLÍNICA GERAL

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    • ABNT

      TOLEDO, S. P. A. e MARINO, Filomena. Inactivating mutations in the LH receptor: as rare as "a hen with teeth" or as frequent as polycystic ovary syndrome?. Fertility & Sterility, v. 73, p. 655-656, 2000Tradução . . Acesso em: 01 out. 2024.
    • APA

      Toledo, S. P. A., & Marino, F. (2000). Inactivating mutations in the LH receptor: as rare as "a hen with teeth" or as frequent as polycystic ovary syndrome? Fertility & Sterility, 73, 655-656.
    • NLM

      Toledo SPA, Marino F. Inactivating mutations in the LH receptor: as rare as "a hen with teeth" or as frequent as polycystic ovary syndrome? Fertility & Sterility. 2000 ; 73 655-656.[citado 2024 out. 01 ]
    • Vancouver

      Toledo SPA, Marino F. Inactivating mutations in the LH receptor: as rare as "a hen with teeth" or as frequent as polycystic ovary syndrome? Fertility & Sterility. 2000 ; 73 655-656.[citado 2024 out. 01 ]
  • Fonte: The Journal of Clinical Endocrinology & Metabolism. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      PERNASETTI, Flavia et al. Impaired adrenocorticotropin-adrenal axis in combined pituytary hormone deficiency caused by a two-base pair deletion (301-302delAG) in the prophet of Pit-1 gene. The Journal of Clinical Endocrinology & Metabolism, v. 85, n. 01, p. 390-397, 2000Tradução . . Acesso em: 01 out. 2024.
    • APA

      Pernasetti, F., Toledo, S. P. A., Vasilyev, V. V., Hayashida, C. Y., Cogan, J. D., Ferrari, C., et al. (2000). Impaired adrenocorticotropin-adrenal axis in combined pituytary hormone deficiency caused by a two-base pair deletion (301-302delAG) in the prophet of Pit-1 gene. The Journal of Clinical Endocrinology & Metabolism, 85( 01), 390-397.
    • NLM

      Pernasetti F, Toledo SPA, Vasilyev VV, Hayashida CY, Cogan JD, Ferrari C, Lourenço DM, Mellon PL. Impaired adrenocorticotropin-adrenal axis in combined pituytary hormone deficiency caused by a two-base pair deletion (301-302delAG) in the prophet of Pit-1 gene. The Journal of Clinical Endocrinology & Metabolism. 2000 ; 85( 01): 390-397.[citado 2024 out. 01 ]
    • Vancouver

      Pernasetti F, Toledo SPA, Vasilyev VV, Hayashida CY, Cogan JD, Ferrari C, Lourenço DM, Mellon PL. Impaired adrenocorticotropin-adrenal axis in combined pituytary hormone deficiency caused by a two-base pair deletion (301-302delAG) in the prophet of Pit-1 gene. The Journal of Clinical Endocrinology & Metabolism. 2000 ; 85( 01): 390-397.[citado 2024 out. 01 ]
  • Fonte: European Journal of Endocrinology. Unidade: FM

    Assunto: ENDOCRINOLOGIA

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    • ABNT

      HAYASHIDA, Cesar Yoiti et al. Familial growth hormone deficiency with mutated GHRH receptor gene: clinical and hormonal findings in homozygous and heterozygous individuals from Itabaianinha. European Journal of Endocrinology, v. 142, n. 06, p. 557-563, 2000Tradução . . Disponível em: https://doi.org/10.1530/eje.0.1420557. Acesso em: 01 out. 2024.
    • APA

      Hayashida, C. Y., Gondo, R. G., Ferrari, C., Toledo, S. P. A., Salvatori, R., Levine, M. A., et al. (2000). Familial growth hormone deficiency with mutated GHRH receptor gene: clinical and hormonal findings in homozygous and heterozygous individuals from Itabaianinha. European Journal of Endocrinology, 142( 06), 557-563. doi:10.1530/eje.0.1420557
    • NLM

      Hayashida CY, Gondo RG, Ferrari C, Toledo SPA, Salvatori R, Levine MA, Ezabella MCL, Abelin N, Gianella Neto D, Wajchenberg BL. Familial growth hormone deficiency with mutated GHRH receptor gene: clinical and hormonal findings in homozygous and heterozygous individuals from Itabaianinha [Internet]. European Journal of Endocrinology. 2000 ; 142( 06): 557-563.[citado 2024 out. 01 ] Available from: https://doi.org/10.1530/eje.0.1420557
    • Vancouver

      Hayashida CY, Gondo RG, Ferrari C, Toledo SPA, Salvatori R, Levine MA, Ezabella MCL, Abelin N, Gianella Neto D, Wajchenberg BL. Familial growth hormone deficiency with mutated GHRH receptor gene: clinical and hormonal findings in homozygous and heterozygous individuals from Itabaianinha [Internet]. European Journal of Endocrinology. 2000 ; 142( 06): 557-563.[citado 2024 out. 01 ] Available from: https://doi.org/10.1530/eje.0.1420557
  • Fonte: American Journal of Human Genetics. Unidade: FM

    Assunto: MEDICINA INTERNA

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    • ABNT

      IANAKIEV, P et al. Acheiropodia is caused by a genomic deletion in C7orf2, the human orthologue of the Lmbr1 gene. American Journal of Human Genetics, v. 68, p. 01-08, 2000Tradução . . Acesso em: 01 out. 2024.
    • APA

      Ianakiev, P., van Baren, M. J., Daly, M. J., Toledo, S. P. A., Cavalcanti, M. G., Correia Neto, J., et al. (2000). Acheiropodia is caused by a genomic deletion in C7orf2, the human orthologue of the Lmbr1 gene. American Journal of Human Genetics, 68, 01-08.
    • NLM

      Ianakiev P, van Baren MJ, Daly MJ, Toledo SPA, Cavalcanti MG, Correia Neto J, Silveira EL, Maia AF, Heutink P, Kilpatrick MW, Tsipouras P. Acheiropodia is caused by a genomic deletion in C7orf2, the human orthologue of the Lmbr1 gene. American Journal of Human Genetics. 2000 ; 68 01-08.[citado 2024 out. 01 ]
    • Vancouver

      Ianakiev P, van Baren MJ, Daly MJ, Toledo SPA, Cavalcanti MG, Correia Neto J, Silveira EL, Maia AF, Heutink P, Kilpatrick MW, Tsipouras P. Acheiropodia is caused by a genomic deletion in C7orf2, the human orthologue of the Lmbr1 gene. American Journal of Human Genetics. 2000 ; 68 01-08.[citado 2024 out. 01 ]

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