ReP USP - Resultado da busca

Artigo de periodico
Leveraging transcriptional dynamics to improve BRAF inhibitor responses in melanoma (2019)
Smalley, Inna; Kim, Eunjung; Li, Jiannong; Spence, Paige; Wyatt, Clayton; Eroglu, Zeynep; Sondak, Vernon K; Messina, Jane L; Babacan, Nalan Akgul ; Maria-Engler, Silvya Stuchi ; De Armas, Lesley; Williams, Sion L; Gatenby, Robert A; Chen, Y. Ann; Anderson, Alexander R. A; Smalley, Keiran S. M
Fonte: EBioMedicine. Unidade: FCF
Assuntos: HETEROGENEIDADE, MELANOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SMALLEY, Inna et al. Leveraging transcriptional dynamics to improve BRAF inhibitor responses in melanoma. EBioMedicine, v. 48, p. 178-190, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.ebiom.2019.09.023. Acesso em: 02 nov. 2024.
APA
Smalley, I., Kim, E., Li, J., Spence, P., Wyatt, C., Eroglu, Z., et al. (2019). Leveraging transcriptional dynamics to improve BRAF inhibitor responses in melanoma. EBioMedicine, 48, 178-190. doi:10.1016/j.ebiom.2019.09.023
NLM
Smalley I, Kim E, Li J, Spence P, Wyatt C, Eroglu Z, Sondak VK, Messina JL, Babacan NA, Maria-Engler SS, De Armas L, Williams SL, Gatenby RA, Chen YA, Anderson ARA, Smalley KSM. Leveraging transcriptional dynamics to improve BRAF inhibitor responses in melanoma [Internet]. EBioMedicine. 2019 ; 48 178-190.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ebiom.2019.09.023
Vancouver
Smalley I, Kim E, Li J, Spence P, Wyatt C, Eroglu Z, Sondak VK, Messina JL, Babacan NA, Maria-Engler SS, De Armas L, Williams SL, Gatenby RA, Chen YA, Anderson ARA, Smalley KSM. Leveraging transcriptional dynamics to improve BRAF inhibitor responses in melanoma [Internet]. EBioMedicine. 2019 ; 48 178-190.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1016/j.ebiom.2019.09.023
Artigo de periodico
Inhibition of autophagy enhances the effects of the AKT inhibitor MK-2206 when combined with paclitaxel and carboplatin in BRAF wild-type melanoma (2014)
Rebecca, Vito William; Massaro, Renato Ramos; Fedorenko, Inna V; Sondak, Vernon K; Anderson, Alexander R. A; Kim, Eunjung; Amaravadi, Ravi K; Maria-Engler, Silvya Stuchi ; Messina, Jane L; Gibney, Geoffrey T; Kudchadkar, Ragini R; Smalley, Keiran S. M
Fonte: Pigment Cell Melanoma Research. Unidade: FCF
Assuntos: QUIMIOTERAPIA, MELANOMA, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REBECCA, Vito William et al. Inhibition of autophagy enhances the effects of the AKT inhibitor MK-2206 when combined with paclitaxel and carboplatin in BRAF wild-type melanoma. Pigment Cell Melanoma Research, v. 27, n. 3, p. 465-478, 2014Tradução . . Disponível em: https://doi.org/10.1111/pcmr.12227. Acesso em: 02 nov. 2024.
APA
Rebecca, V. W., Massaro, R. R., Fedorenko, I. V., Sondak, V. K., Anderson, A. R. A., Kim, E., et al. (2014). Inhibition of autophagy enhances the effects of the AKT inhibitor MK-2206 when combined with paclitaxel and carboplatin in BRAF wild-type melanoma. Pigment Cell Melanoma Research, 27( 3), 465-478. doi:10.1111/pcmr.12227
NLM
Rebecca VW, Massaro RR, Fedorenko IV, Sondak VK, Anderson ARA, Kim E, Amaravadi RK, Maria-Engler SS, Messina JL, Gibney GT, Kudchadkar RR, Smalley KSM. Inhibition of autophagy enhances the effects of the AKT inhibitor MK-2206 when combined with paclitaxel and carboplatin in BRAF wild-type melanoma [Internet]. Pigment Cell Melanoma Research. 2014 ; 27( 3): 465-478.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/pcmr.12227
Vancouver
Rebecca VW, Massaro RR, Fedorenko IV, Sondak VK, Anderson ARA, Kim E, Amaravadi RK, Maria-Engler SS, Messina JL, Gibney GT, Kudchadkar RR, Smalley KSM. Inhibition of autophagy enhances the effects of the AKT inhibitor MK-2206 when combined with paclitaxel and carboplatin in BRAF wild-type melanoma [Internet]. Pigment Cell Melanoma Research. 2014 ; 27( 3): 465-478.[citado 2024 nov. 02 ] Available from: https://doi.org/10.1111/pcmr.12227
Trabalho de evento-resumo periodico
BRAF wild-type melanoma survival hinges upon AKT activity and adaptive autophagic reprogramming in response to chemotherapy (2013)
Rebecca, Vito William; Massaro, Raquel; Fedorenko, Inna V; Gibney, Geoffrey T; Sondak, Vernon K; Amaravadi, Ravi K; Kin, S; Maria-Engler, Silvya Stuchi ; Kudchadkar, Ragini; Smalley, K
Fonte: Pigment Cell & Melanoma Research. Nome do evento: International Congress of the Society for Melanoma Research. Unidade: FCF
Assuntos: MELANOMA, QUIMIOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REBECCA, Vito William et al. BRAF wild-type melanoma survival hinges upon AKT activity and adaptive autophagic reprogramming in response to chemotherapy. Pigment Cell & Melanoma Research. Hoboken: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo. . Acesso em: 02 nov. 2024. , 2013
APA
Rebecca, V. W., Massaro, R., Fedorenko, I. V., Gibney, G. T., Sondak, V. K., Amaravadi, R. K., et al. (2013). BRAF wild-type melanoma survival hinges upon AKT activity and adaptive autophagic reprogramming in response to chemotherapy. Pigment Cell & Melanoma Research. Hoboken: Faculdade de Ciências Farmacêuticas, Universidade de São Paulo.
NLM
Rebecca VW, Massaro R, Fedorenko IV, Gibney GT, Sondak VK, Amaravadi RK, Kin S, Maria-Engler SS, Kudchadkar R, Smalley K. BRAF wild-type melanoma survival hinges upon AKT activity and adaptive autophagic reprogramming in response to chemotherapy. Pigment Cell & Melanoma Research. 2013 ; 26( 6): 991.[citado 2024 nov. 02 ]
Vancouver
Rebecca VW, Massaro R, Fedorenko IV, Gibney GT, Sondak VK, Amaravadi RK, Kin S, Maria-Engler SS, Kudchadkar R, Smalley K. BRAF wild-type melanoma survival hinges upon AKT activity and adaptive autophagic reprogramming in response to chemotherapy. Pigment Cell & Melanoma Research. 2013 ; 26( 6): 991.[citado 2024 nov. 02 ]
Trabalho de evento-resumo
Exhaustion of autophagy-mediated cell survival underlies the pre-clinical and clinical activity of chemotherapy combined with the AKT inhibitor MK-2206 in BRAF wild-type melanoma (2013)
Rebecca, Vito W; Massaro, Renato Ramos; Maria-Engler, Silvya Stuchi ; Messina, Jane L; Gibney, Geoffrey T; Sondak, Vernon K; Kudchadkar, Ragini; Smalley, Keiran S
Fonte: Abstracts. Nome do evento: Annual Meeting of the American Association for Cancer Research. Unidade: FCF
Assuntos: MELANOMA, QUIMIOTERAPIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REBECCA, Vito W et al. Exhaustion of autophagy-mediated cell survival underlies the pre-clinical and clinical activity of chemotherapy combined with the AKT inhibitor MK-2206 in BRAF wild-type melanoma. 2013, Anais.. Philadelphia: American Association for Cancer Research, 2013. . Acesso em: 02 nov. 2024.
APA
Rebecca, V. W., Massaro, R. R., Maria-Engler, S. S., Messina, J. L., Gibney, G. T., Sondak, V. K., et al. (2013). Exhaustion of autophagy-mediated cell survival underlies the pre-clinical and clinical activity of chemotherapy combined with the AKT inhibitor MK-2206 in BRAF wild-type melanoma. In Abstracts. Philadelphia: American Association for Cancer Research.
NLM
Rebecca VW, Massaro RR, Maria-Engler SS, Messina JL, Gibney GT, Sondak VK, Kudchadkar R, Smalley KS. Exhaustion of autophagy-mediated cell survival underlies the pre-clinical and clinical activity of chemotherapy combined with the AKT inhibitor MK-2206 in BRAF wild-type melanoma. Abstracts. 2013 ;[citado 2024 nov. 02 ]
Vancouver
Rebecca VW, Massaro RR, Maria-Engler SS, Messina JL, Gibney GT, Sondak VK, Kudchadkar R, Smalley KS. Exhaustion of autophagy-mediated cell survival underlies the pre-clinical and clinical activity of chemotherapy combined with the AKT inhibitor MK-2206 in BRAF wild-type melanoma. Abstracts. 2013 ;[citado 2024 nov. 02 ]

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