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Artigo de periodico
Intermittent fasting induces hypothalamic modifications resulting inlow feeding efficiency, low body mass and overeating (2014)
Chausse, Bruno; Solon, Carina; Silva, Camille Cristine Caldeira da; Reis, Ivan Masselli dos; Manchado Gobatto, Fúlvia de Barros; Gobatto, Cláudio Alexandre; Velloso, Lício Augusto; Kowaltowski, Alicia Juliana
Source: Endocrinology. Unidade: IQ
Subjects: MITOCÔNDRIAS, OXIDAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHAUSSE, Bruno et al. Intermittent fasting induces hypothalamic modifications resulting inlow feeding efficiency, low body mass and overeating. Endocrinology, v. 155, n. 7, p. 2456-2466, 2014Tradução . . Disponível em: https://doi.org/10.1210/en.2013-2057. Acesso em: 05 nov. 2024.
APA
Chausse, B., Solon, C., Silva, C. C. C. da, Reis, I. M. dos, Manchado Gobatto, F. de B., Gobatto, C. A., et al. (2014). Intermittent fasting induces hypothalamic modifications resulting inlow feeding efficiency, low body mass and overeating. Endocrinology, 155( 7), 2456-2466. doi:10.1210/en.2013-2057
NLM
Chausse B, Solon C, Silva CCC da, Reis IM dos, Manchado Gobatto F de B, Gobatto CA, Velloso LA, Kowaltowski AJ. Intermittent fasting induces hypothalamic modifications resulting inlow feeding efficiency, low body mass and overeating [Internet]. Endocrinology. 2014 ; 155( 7): 2456-2466.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1210/en.2013-2057
Vancouver
Chausse B, Solon C, Silva CCC da, Reis IM dos, Manchado Gobatto F de B, Gobatto CA, Velloso LA, Kowaltowski AJ. Intermittent fasting induces hypothalamic modifications resulting inlow feeding efficiency, low body mass and overeating [Internet]. Endocrinology. 2014 ; 155( 7): 2456-2466.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1210/en.2013-2057
Artigo de periodico
Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels (2012)
Kinote, Andrezza; Faria, Juliana A.; Roman, Erika A.; Solon, Carina; Razolli, Daniela S.; Ignacio-Souza, Letícia M.; Sollon, Carolina S.; Nascimento, Lucas F.; Araújo, Thiago M. de; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Velloso, Licio A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KINOTE, Andrezza et al. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, v. 153, n. 8, p. 3633-3645, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1341. Acesso em: 05 nov. 2024.
APA
Kinote, A., Faria, J. A., Roman, E. A., Solon, C., Razolli, D. S., Ignacio-Souza, L. M., et al. (2012). Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, 153( 8), 3633-3645. doi:10.1210/en.2012-1341
NLM
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1210/en.2012-1341
Vancouver
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1210/en.2012-1341
Artigo de periodico
Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia (2010)
Arruda, Ana Paula; Milanski, Marciane; Romanatto, Talita; Solon, Carina; Coope, Andressa; Festuccia, William Tadeu Lara ; Velloso, Licio Augusto; Hirabara, Sandro Massao; Ropelle, Eduardo; Cavalheira, José B.; Vercesi, Anibal Eugenio; Curi, Rui
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARRUDA, Ana Paula et al. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology, v. 151, n. 2, p. 683-694, 2010Tradução . . Acesso em: 05 nov. 2024.
APA
Arruda, A. P., Milanski, M., Romanatto, T., Solon, C., Coope, A., Festuccia, W. T. L., et al. (2010). Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology, 151( 2), 683-694.
NLM
Arruda AP, Milanski M, Romanatto T, Solon C, Coope A, Festuccia WTL, Velloso LA, Hirabara SM, Ropelle E, Cavalheira JB, Vercesi AE, Curi R. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology. 2010 ; 151( 2): 683-694.[citado 2024 nov. 05 ]
Vancouver
Arruda AP, Milanski M, Romanatto T, Solon C, Coope A, Festuccia WTL, Velloso LA, Hirabara SM, Ropelle E, Cavalheira JB, Vercesi AE, Curi R. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology. 2010 ; 151( 2): 683-694.[citado 2024 nov. 05 ]
Artigo de periodico
Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis (2009)
Romanatto, Talita; Roman, Erika A.; Denis, Raphael G.; Solon, Carina; Milanski, Marciane; Moraes, Juliana C.; Bonfleur, Maria L.; Degasperi, Giovanna R.; Picardi, Paty K.; Hirabara, Sandro Massao; Boschero, Antonio C.; Velloso, Licio; Curi, Rui
Source: Journal of Biological Chemistry. Unidades: FZEA, ICB
Subjects: FISIOLOGIA, NECROSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROMANATTO, Talita et al. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis. Journal of Biological Chemistry, v. 284, n. 52, p. 36213-36222, 2009Tradução . . Disponível em: https://doi.org/10.1074/jbc.a109.030874. Acesso em: 05 nov. 2024.
APA
Romanatto, T., Roman, E. A., Denis, R. G., Solon, C., Milanski, M., Moraes, J. C., et al. (2009). Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis. Journal of Biological Chemistry, 284( 52), 36213-36222. doi:10.1074/jbc.a109.030874
NLM
Romanatto T, Roman EA, Denis RG, Solon C, Milanski M, Moraes JC, Bonfleur ML, Degasperi GR, Picardi PK, Hirabara SM, Boschero AC, Velloso L, Curi R. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis [Internet]. Journal of Biological Chemistry. 2009 ; 284( 52): 36213-36222.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1074/jbc.a109.030874
Vancouver
Romanatto T, Roman EA, Denis RG, Solon C, Milanski M, Moraes JC, Bonfleur ML, Degasperi GR, Picardi PK, Hirabara SM, Boschero AC, Velloso L, Curi R. Deletion of tumor necrosis factor-α receptor 1 (TNFR1) Protects against diet-induced obesity by means of Increased thermogenesis [Internet]. Journal of Biological Chemistry. 2009 ; 284( 52): 36213-36222.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1074/jbc.a109.030874

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