Filtros : "Simões, Daniel" Limpar

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  • Source: Journal of Endocrinology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      SIMÕES, Daniel et al. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, v. 231, n. 3, p. 235-244, 2016Tradução . . Disponível em: https://doi.org/10.1530/JOE-16-0259. Acesso em: 23 nov. 2025.
    • APA

      Simões, D., Riva, P., Peliciari-Garcia, R. A., Cruzat, V. F., Graciano, M. fernanda, Munhoz, A. C., et al. (2016). Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase. Journal of Endocrinology, 231( 3), 235-244. doi:10.1530/JOE-16-0259
    • NLM

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1530/JOE-16-0259
    • Vancouver

      Simões D, Riva P, Peliciari-Garcia RA, Cruzat VF, Graciano M fernanda, Munhoz AC, Taneda M, Cipolla-Neto J, Carpinelli AR. Melatonin modifies basal and stimulated insulin secretion via NADPH oxidase [Internet]. Journal of Endocrinology. 2016 ; 231( 3): 235-244.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1530/JOE-16-0259
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      MUNHOZ, Ana Cláudia et al. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, v. 11, n. 6, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0158166. Acesso em: 23 nov. 2025.
    • APA

      Munhoz, A. C., Riva, P., Simões, D., Curi, R., & Carpinelli, A. R. (2016). Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, 11( 6), 1-17. doi:10.1371/journal.pone.0158166
    • NLM

      Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1371/journal.pone.0158166
    • Vancouver

      Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1371/journal.pone.0158166
  • Source: Pancreas. Unidade: ICB

    Subjects: FISIOLOGIA, ILHOTAS DE LANGERHANS, SUPLEMENTOS ALIMENTARES PARA ANIMAIS, ÓLEOS DE PEIUXE

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      LUCENA, Camila Ferraz et al. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, v. 44, n. 2, p. 287-295, 2015Tradução . . Disponível em: https://doi.org/10.1097/MPA.0000000000000249. Acesso em: 23 nov. 2025.
    • APA

      Lucena, C. F., Roma, L. P., Graciano, M. F. R., Veras, K., Simões, D., Curi, R., & Carpinelli, A. R. (2015). Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, 44( 2), 287-295. doi:10.1097/MPA.0000000000000249
    • NLM

      Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
    • Vancouver

      Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
  • Source: American Journal of Physiology. Endocrinology and Metabolism. Unidade: ICB

    Subjects: FISIOLOGIA, RATOS, GLIC0SE, MONOSSACARIDEOS

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      FESTUCCIA, William Tadeu Lara et al. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, v. 9, p. E1046-E1054, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00683.2013. Acesso em: 23 nov. 2025.
    • APA

      Festuccia, W. T. L., Blanchard, P. -G., Belchior, T., Chimin, P., Paschoal, V. A., Magdalon, J., et al. (2014). PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, 9, E1046-E1054. doi:10.1152/ajpendo.00683.2013
    • NLM

      Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013
    • Vancouver

      Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2025 nov. 23 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013

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