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Artigo de periodico
AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity (2023)
Leite, Jefferson Antonio ; Menezes, Luísa ; Martins, Eloisa ; Rodrigues, Tamara Silva; Tavares, Lucas ; Ebering, Anna ; Schelmbauer, Carsten ; Cebinelli, Guilherme Cesar Martelossi ; Zinina, Valeriya ; Golden, Artemy ; Soshnikova, Natalia ; Zamboni, Dario S. ; Cunha, Fernando Q. ; Magdalena Huber ; Waisman, Ari ; Silva, João Santana da ; Carlos, Daniela ; Câmara, Niels Olsen Saraiva
Source: iScience. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, CAMUNDONGOS, PROTEÍNAS, DIFERENCIAÇÃO CELULAR, LINFÓCITOS T, CÉLULAS SANGUÍNEAS
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LEITE, Jefferson Antonio et al. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, v. 26, n. 11, p. art. 108134. 19 , 2023Tradução . . Disponível em: https://doi.org/10.1016/j.isci.2023.108134. Acesso em: 18 nov. 2024.
APA
Leite, J. A., Menezes, L., Martins, E., Rodrigues, T. S., Tavares, L., Ebering, A., et al. (2023). AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity. iScience, 26( 11), art. 108134. 19 . doi:10.1016/j.isci.2023.108134
NLM
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Vancouver
Leite JA, Menezes L, Martins E, Rodrigues TS, Tavares L, Ebering A, Schelmbauer C, Cebinelli GCM, Zinina V, Golden A, Soshnikova N, Zamboni DS, Cunha FQ, Magdalena Huber, Waisman A, Silva JS da, Carlos D, Câmara NOS. AIM2 promotes TH17 cells differentiation by regulating RORγt transcription activity [Internet]. iScience. 2023 ; 26( 11): art. 108134. 19 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.isci.2023.108134
Artigo de periodico
Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein (2022)
Castro, Julia T.; Azevedo, Patrick; Fumagalli, Marcílio Jorge; Hojo-Souza, Natalia S.; Salazar, Natalia; Almeida, Gregório G.; Oliveira, Livia I.; Faustino, Lídia; Antonelli, Lis R.; Marçal, Tomas G.; Augusto, Marconi; Valiate, Bruno; Fiorini, Alex; Rattis, Bruna Amanda da Cruz; Ramos, Simone Gusmão; Piccin, Mariela Pires Cabral; Nonato, Osvaldo Campos dos Santos; Benevides, Luciana; Magalhães, Rubens; Cassaro, Bruno; Burle, Gabriela; Doro, Daniel; Kalil, Jorge; Durigon, Edison Luiz ; Salazar, Andrés; Caballero, Otávia; Santiago, Helton; Machado, Alexandre; Silva, João Santana da ; Fonseca, Flávio da; Fernandes, Ana Paula; Teixeira, Santuza R.; Gazzinelli, Ricardo Tostes
Source: Nature Communications. Unidades: FMRP, ICB
Subjects: ANTICORPOS, LINFÓCITOS T, VACINAS, COVID-19, GLICOPROTEÍNAS, CORONAVIRUS
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CASTRO, Julia T. et al. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein. Nature Communications, v. 13, n. 1, p. 1-16, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41467-022-32547-y. Acesso em: 18 nov. 2024.
APA
Castro, J. T., Azevedo, P., Fumagalli, M. J., Hojo-Souza, N. S., Salazar, N., Almeida, G. G., et al. (2022). Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein. Nature Communications, 13( 1), 1-16. doi:10.1038/s41467-022-32547-y
NLM
Castro JT, Azevedo P, Fumagalli MJ, Hojo-Souza NS, Salazar N, Almeida GG, Oliveira LI, Faustino L, Antonelli LR, Marçal TG, Augusto M, Valiate B, Fiorini A, Rattis BA da C, Ramos SG, Piccin MPC, Nonato OC dos S, Benevides L, Magalhães R, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Santiago H, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein [Internet]. Nature Communications. 2022 ; 13( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41467-022-32547-y
Vancouver
Castro JT, Azevedo P, Fumagalli MJ, Hojo-Souza NS, Salazar N, Almeida GG, Oliveira LI, Faustino L, Antonelli LR, Marçal TG, Augusto M, Valiate B, Fiorini A, Rattis BA da C, Ramos SG, Piccin MPC, Nonato OC dos S, Benevides L, Magalhães R, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Santiago H, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Promotion of neutralizing antibody-independent immunity to wild-type and SARS-CoV-2 variants of concern using an RBD-Nucleocapsid fusion protein [Internet]. Nature Communications. 2022 ; 13( 1): 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/s41467-022-32547-y
Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
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COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 18 nov. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
Interleukin-6 and the gut microbiota influence melanoma progression in obese mice (2021)
Pereira, Felipe Valença ; Melo, Amanda C. L.; Silva, Marina B.; Melo, Filipe Menegatti de ; Terra, Fernanda Fernandes ; Castro, Iris A.; Donato Junior, José ; Festuccia, William Tadeu Lara ; Silva, João Santana da ; Câmara, Niels Olsen Saraiva
Source: Nutrition and Cancer. Unidades: ICB, FMRP
Subjects: FISIOLOGIA, IMUNOLOGIA, LEPTINA, INTERLEUCINA 6, CAMUNDONGOS, MELANOMA ANIMAL, OBESIDADE, DIETA ANIMAL, GORDURAS NA DIETA, CÉLULAS CULTIVADAS DE TUMOR, MODELOS ANIMAIS DE DOENÇAS
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PEREIRA, Felipe Valença et al. Interleukin-6 and the gut microbiota influence melanoma progression in obese mice. Nutrition and Cancer, v. 73, n. 4, p. 1-10, 2021Tradução . . Disponível em: https://doi.org/10.1080/01635581.2020.1764982. Acesso em: 18 nov. 2024.
APA
Pereira, F. V., Melo, A. C. L., Silva, M. B., Melo, F. M. de, Terra, F. F., Castro, I. A., et al. (2021). Interleukin-6 and the gut microbiota influence melanoma progression in obese mice. Nutrition and Cancer, 73( 4), 1-10. doi:10.1080/01635581.2020.1764982
NLM
Pereira FV, Melo ACL, Silva MB, Melo FM de, Terra FF, Castro IA, Donato Junior J, Festuccia WTL, Silva JS da, Câmara NOS. Interleukin-6 and the gut microbiota influence melanoma progression in obese mice [Internet]. Nutrition and Cancer. 2021 ; 73( 4): 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/01635581.2020.1764982
Vancouver
Pereira FV, Melo ACL, Silva MB, Melo FM de, Terra FF, Castro IA, Donato Junior J, Festuccia WTL, Silva JS da, Câmara NOS. Interleukin-6 and the gut microbiota influence melanoma progression in obese mice [Internet]. Nutrition and Cancer. 2021 ; 73( 4): 1-10.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1080/01635581.2020.1764982
Artigo de periodico
Neutralizing antibody-independent immunity to SARS-CoV-2 in hamsters and hACE-2 transgenic mice immunized with a RBD/Nucleocapsid fusion protein (2021)
Castro, Julia T.; Fumagalli, Marcílio Jorge; Hojo-Souza, Nathalia Satchiko; Azevedo, Patrick; Salazar, Natalia; Rattis, Bruna Amanda Cruz; Ramos, Simone Gusmão; Faustino, Lídia; Almeida, Gregório G.; Oliveira, Livia I.; Marçal, Tomas G.; Augusto, Marconi; Magalhães, Rubens Daniel Miserani; Cassaro, Bruno; Burle, Gabriela; Doro, Daniel; Kalil, Jorge ; Durigon, Edison Luiz ; Salazar, Andrés; Caballero, Otávia; Machado, Alexandre; Silva, João Santana da ; Fonseca, Flavio da; Fernandes, Ana Paula; Teixeira, Santuza R.; Gazzinelli, Ricardo Tostes
Source: BioRxiv. Unidades: FMRP, ICB
Subjects: IMUNIDADE, ÁCIDOS NUCLEICOS, VÍRUS, COVID-19, ANGIOTENSINAS
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ABNT
CASTRO, Julia T. et al. Neutralizing antibody-independent immunity to SARS-CoV-2 in hamsters and hACE-2 transgenic mice immunized with a RBD/Nucleocapsid fusion protein. BioRxiv, 2021Tradução . . Disponível em: https://doi.org/10.1101/2021.09.16.460663. Acesso em: 18 nov. 2024.
APA
Castro, J. T., Fumagalli, M. J., Hojo-Souza, N. S., Azevedo, P., Salazar, N., Rattis, B. A. C., et al. (2021). Neutralizing antibody-independent immunity to SARS-CoV-2 in hamsters and hACE-2 transgenic mice immunized with a RBD/Nucleocapsid fusion protein. BioRxiv. doi:10.1101/2021.09.16.460663
NLM
Castro JT, Fumagalli MJ, Hojo-Souza NS, Azevedo P, Salazar N, Rattis BAC, Ramos SG, Faustino L, Almeida GG, Oliveira LI, Marçal TG, Augusto M, Magalhães RDM, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Neutralizing antibody-independent immunity to SARS-CoV-2 in hamsters and hACE-2 transgenic mice immunized with a RBD/Nucleocapsid fusion protein [Internet]. BioRxiv. 2021 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1101/2021.09.16.460663
Vancouver
Castro JT, Fumagalli MJ, Hojo-Souza NS, Azevedo P, Salazar N, Rattis BAC, Ramos SG, Faustino L, Almeida GG, Oliveira LI, Marçal TG, Augusto M, Magalhães RDM, Cassaro B, Burle G, Doro D, Kalil J, Durigon EL, Salazar A, Caballero O, Machado A, Silva JS da, Fonseca F da, Fernandes AP, Teixeira SR, Gazzinelli RT. Neutralizing antibody-independent immunity to SARS-CoV-2 in hamsters and hACE-2 transgenic mice immunized with a RBD/Nucleocapsid fusion protein [Internet]. BioRxiv. 2021 ;[citado 2024 nov. 18 ] Available from: https://doi.org/10.1101/2021.09.16.460663
Artigo de periodico
The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway (2020)
Leite, Jefferson Antonio; Pessenda, Gabriela ; Gomes, Isabel Cristina Guerra ; Santana, Alynne Karen Mendonça de; Pereira, Camila André; Costa, Frederico Ribeiro Campos; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Faria, Ana Maria Caetano ; Almeida, Danilo Candido de ; Câmara, Niels Olsen Saraiva ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Sartori, Daniela Carlos
Source: Cells. Unidades: FMRP, ICB
Assunto: IMUNOLOGIA
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ABNT
LEITE, Jefferson Antonio et al. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, v. 9, n. 4, p. 21 , 2020Tradução . . Disponível em: https://doi.org/10.3390/cells9040959. Acesso em: 18 nov. 2024.
APA
Leite, J. A., Pessenda, G., Gomes, I. C. G., Santana, A. K. M. de, Pereira, C. A., Costa, F. R. C., et al. (2020). The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway. Cells, 9( 4), 21 . doi:10.3390/cells9040959
NLM
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells9040959
Vancouver
Leite JA, Pessenda G, Gomes ICG, Santana AKM de, Pereira CA, Costa FRC, Ramos SG, Zamboni DS, Faria AMC, Almeida DC de, Câmara NOS, Passaglia R de CAT, Silva JS da, Sartori DC. The DNA Sensor AIM2 Protects against Streptozotocin-Induced Type 1 Diabetes by Regulating Intestinal Homeostasis via the IL-18 Pathway [Internet]. Cells. 2020 ; 9( 4): 21 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3390/cells9040959
Artigo de periodico
NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model (2020)
Sartori, Daniela Carlos ; Pérez, Malena Martínez ; Leite, Jefferson Antonio; Rocha, Fernanda Agostini; Martins, Larissa Marfori Sampaio; Pereira, Camila André; Silva, Thais Fernanda de Campos Fraga da ; Pucci, Taís Andrade; Ramos, Simone Gusmão ; Câmara, Niels Olsen Saraiva ; Bonato, Vânia Luiza Deperon ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB, FCFRP
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIABETES MELLITUS, INFLAMAÇÃO, OBESIDADE, ADIPOSIDADE, CAMUNDONGOS, RESISTÊNCIA À INSULINA, HIPERGLICEMIA, LINFONODOS DE ANIMAL, EXPRESSÃO GÊNICA, BACTÉRIAS GRAM-NEGATIVAS, BACTEROIDES
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ABNT
SARTORI, Daniela Carlos et al. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, v. 11, p. 16 , 2020Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2020.01265. Acesso em: 18 nov. 2024.
APA
Sartori, D. C., Pérez, M. M., Leite, J. A., Rocha, F. A., Martins, L. M. S., Pereira, C. A., et al. (2020). NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model. Frontiers in Immunology, 11, 16 . doi:10.3389/fimmu.2020.01265
NLM
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Vancouver
Sartori DC, Pérez MM, Leite JA, Rocha FA, Martins LMS, Pereira CA, Silva TF de CF da, Pucci TA, Ramos SG, Câmara NOS, Bonato VLD, Passaglia R de CAT, Silva JS da. NOD2 deficiency promotes intestinal CD4+ T lymphocyte imbalance, metainflammation, and aggravates Type 2 Diabetes in murine model [Internet]. Frontiers in Immunology. 2020 ; 11 16 .[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2020.01265
Artigo de periodico
B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation (2019)
Benevides, Luciana ; Costa, Renata Sesti ; Tavares, Lucas Alves; Russo, Momtchilo ; Martins, Gislâine A.; Silva, Luis Lamberti Pinto da ; Arruda, Luisa Karla de Paula ; Cunha, Fernando de Queiroz ; Carregaro, Vanessa ; Silva, João Santana da
Source: Journal of Allergy and Clinical Immunology. Unidades: ICB, FMRP
Subjects: ALERGIA E IMUNOLOGIA, DIFERENCIAÇÃO CELULAR, LINFÓCITOS B, INFLAMAÇÃO, CITOMETRIA DE FLUXO, ELISA, REAÇÃO EM CADEIA POR POLIMERASE, CAMUNDONGOS
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ABNT
BENEVIDES, Luciana et al. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, v. 143, n. 3, p. 1119-1130, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.jaci.2018.06.046. Acesso em: 18 nov. 2024.
APA
Benevides, L., Costa, R. S., Tavares, L. A., Russo, M., Martins, G. A., Silva, L. L. P. da, et al. (2019). B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation. Journal of Allergy and Clinical Immunology, 143( 3), 1119-1130. doi:10.1016/j.jaci.2018.06.046
NLM
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Vancouver
Benevides L, Costa RS, Tavares LA, Russo M, Martins GA, Silva LLP da, Arruda LK de P, Cunha F de Q, Carregaro V, Silva JS da. B lymphocyte–induced maturation protein 1 controls TH9 cell development, IL-9 production, and allergic inflammation [Internet]. Journal of Allergy and Clinical Immunology. 2019 ; 143( 3): 1119-1130.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.jaci.2018.06.046
Artigo de periodico
The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression (2018)
Amano, Mariane Tami ; Castoldi, Angela ; Andrade-Oliveira, Vinicius ; Latância, Marcela Teatin ; Terra, Fernanda Fernandes ; Correa-Costa, Matheus ; Breda, Cristiane Naffah Souza ; Felizardo, Raphael José Ferreira ; Pereira, Welbert Oliveira de ; Silva, Marina Burgos da ; Miyagi, Mariana Yasue Saito ; Aguiar, Cristhiane Favero ; Hiyane, Meire Ioshie ; Silva, João Santana da ; Moura, Ivan Cruz ; Câmara, Niels Olsen Saraiva
Source: International Immunopharmacology. Unidades: ICB, FMRP, FCF, BIOTECNOLOGIA
Subjects: INSUFICIÊNCIA RENAL AGUDA, NEFROPATIAS, MACRÓFAGOS, IMUNOLOGIA
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ABNT
AMANO, Mariane Tami et al. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, v. 64, p. 151-161, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2018.08.020. Acesso em: 18 nov. 2024.
APA
Amano, M. T., Castoldi, A., Andrade-Oliveira, V., Latância, M. T., Terra, F. F., Correa-Costa, M., et al. (2018). The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression. International Immunopharmacology, 64, 151-161. doi:10.1016/j.intimp.2018.08.020
NLM
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Vancouver
Amano MT, Castoldi A, Andrade-Oliveira V, Latância MT, Terra FF, Correa-Costa M, Breda CNS, Felizardo RJF, Pereira WO de, Silva MB da, Miyagi MYS, Aguiar CF, Hiyane MI, Silva JS da, Moura IC, Câmara NOS. The lack of PI3Kγ favors M1 macrophage polarization and does not prevent kidney diseases progression [Internet]. International Immunopharmacology. 2018 ; 64 151-161.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.intimp.2018.08.020
Artigo de periodico
CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development (2018)
Braga, Tárcio Teodoro; Costa, Matheus Correa; Silva, Reinaldo Correia; Cruz, Mário Costa; Hiyane, Meire Ioshie; Silva, João Santana da; Perez, Katia Regina; Cuccovia, Iolanda Midea ; Câmara, Niels Olsen Saraiva
Source: Inflammopharmacology. Unidades: ICB, FMRP, IQ
Subjects: MONÓCITOS, QUIMIOCINAS, INFLAMAÇÃO, TÉCNICAS DE DIAGNÓSTICO UROLÓGICO
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ABNT
BRAGA, Tárcio Teodoro et al. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, v. 26, p. 403-411, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10787-017-0317-4. Acesso em: 18 nov. 2024.
APA
Braga, T. T., Costa, M. C., Silva, R. C., Cruz, M. C., Hiyane, M. I., Silva, J. S. da, et al. (2018). CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, 26, 403-411. doi:10.1007/s10787-017-0317-4
NLM
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Vancouver
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Artigo de periodico
Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven Inflammation (2017)
Medina, Tiago Silva; Frade, Amanda Farage; Oliveira, Gabriela Gonçalves; Silva, Maria Claúdia; David, Bruna Araujo; Silva, Grace Kelly da; Fonseca, Denise Morais da ; Sesti-Costa, Renata; Baron, Monique Andrade; Ianni, Bárbara Maria; Pereira, Alexandre Costa; Chevillard, Christophe; Cunha-Neto, Edécio; Marin Neto, José Antonio ; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FM, FMRP, ICB
Subjects: TRYPANOSOMA CRUZI, INFLAMAÇÃO, MIOCARDITE, DOENÇA DE CHAGAS, LINFÓCITOS
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ABNT
MEDINA, Tiago Silva et al. Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven Inflammation. Frontiers in Immunology, v. 8, p. art. 1213 1-17, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.01213. Acesso em: 18 nov. 2024.
APA
Medina, T. S., Frade, A. F., Oliveira, G. G., Silva, M. C., David, B. A., Silva, G. K. da, et al. (2017). Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven Inflammation. Frontiers in Immunology, 8, art. 1213 1-17. doi:10.3389/fimmu.2017.01213
NLM
Medina TS, Frade AF, Oliveira GG, Silva MC, David BA, Silva GK da, Fonseca DM da, Sesti-Costa R, Baron MA, Ianni BM, Pereira AC, Chevillard C, Cunha-Neto E, Marin Neto JA, Silva JS da. Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven Inflammation [Internet]. Frontiers in Immunology. 2017 ; 8 art. 1213 1-17.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.01213
Vancouver
Medina TS, Frade AF, Oliveira GG, Silva MC, David BA, Silva GK da, Fonseca DM da, Sesti-Costa R, Baron MA, Ianni BM, Pereira AC, Chevillard C, Cunha-Neto E, Marin Neto JA, Silva JS da. Ebi3 Prevents Trypanosoma cruzi-Induced Myocarditis by Dampening IFN-γ-Driven Inflammation [Internet]. Frontiers in Immunology. 2017 ; 8 art. 1213 1-17.[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.01213
Artigo de periodico
Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model (2017)
Carlos, Daniela ; Costa, Frederico Ribeiro Campos ; Pereira, Camila André; Rocha, Fernanda Agostini; Yaochite, Juliana Navarro Ueda; Oliveira, Gabriela G.; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo ; Ramos, Simone Gusmão ; Zamboni, Dario Simões ; Câmara, Niels Olsen Saraiva ; Ryffel, Bernhard; Silva, João Santana da
Source: Frontiers in Immunology. Unidades: FMRP, ICB
Subjects: CITOCINAS, DIABETES MELLITUS, DNA MITOCONDRIAL
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ABNT
CARLOS, Daniela et al. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2017.00164. Acesso em: 18 nov. 2024.
APA
Carlos, D., Costa, F. R. C., Pereira, C. A., Rocha, F. A., Yaochite, J. N. U., Oliveira, G. G., et al. (2017). Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model. Frontiers in Immunology, 8. doi:10.3389/fimmu.2017.00164
NLM
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Vancouver
Carlos D, Costa FRC, Pereira CA, Rocha FA, Yaochite JNU, Oliveira GG, Carneiro FS, Tostes R de CA, Ramos SG, Zamboni DS, Câmara NOS, Ryffel B, Silva JS da. Mitochondrial DNA activates the NLRP3 inflammasome and predisposes to type 1 diabetes in murine model [Internet]. Frontiers in Immunology. 2017 ; 8[citado 2024 nov. 18 ] Available from: https://doi.org/10.3389/fimmu.2017.00164
Artigo de periodico
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset (2016)
Costa, Frederico R. C.; Françozo, Marcela C. S.; Oliveira, Gabriela G. de; Ignacio, Aline; Castoldi, Angela; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Zoete, Marcel R. de; Palm, Noah W.; Flavell, Richard A.; Silva, João Santana da; Carlos, Daniela
Source: Journal of Experimental Medicine. Unidades: FMRP, ICB
Subjects: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS, DOENÇAS AUTOIMUNES, SISTEMA IMUNE, PÂNCREAS
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ABNT
COSTA, Frederico R. C. et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, v. 213, n. 7, p. 1223-1239, 2016Tradução . . Disponível em: https://doi.org/10.1084/jem.20150744. Acesso em: 18 nov. 2024.
APA
Costa, F. R. C., Françozo, M. C. S., Oliveira, G. G. de, Ignacio, A., Castoldi, A., Zamboni, D. S., et al. (2016). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset. Journal of Experimental Medicine, 213( 7), 1223-1239. doi:10.1084/jem.20150744
NLM
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1084/jem.20150744
Vancouver
Costa FRC, Françozo MCS, Oliveira GG de, Ignacio A, Castoldi A, Zamboni DS, Ramos SG, Câmara NOS, Zoete MR de, Palm NW, Flavell RA, Silva JS da, Carlos D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to T1D onset [Internet]. Journal of Experimental Medicine. 2016 ; 213( 7): 1223-1239.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1084/jem.20150744
Artigo de periodico
Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice (2015)
Carlos, Daniela; Yaochite, Juliana Navarro Ueda; Rocha, Fernanda Agostini; Toso, Vanina Danuza; Malmegrim, Kelen C. R.; Ramos, Simone Gusmão; Jamur, Maria Célia; Oliver, Constance; Câmara, Niels Olsen Saraiva ; Andrade, Marcus Vinicius Melo de; Cunha, Fernando de Queiróz; Silva, João Santana da
Source: European Journal of Immunology. Unidades: FMRP, ICB
Subjects: MASTÓCITOS, DIABETES MELLITUS, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARLOS, Daniela et al. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice. European Journal of Immunology, v. 45, n. 10, p. 2873–2885, 2015Tradução . . Disponível em: https://doi.org/10.1002/eji.201545498. Acesso em: 18 nov. 2024.
APA
Carlos, D., Yaochite, J. N. U., Rocha, F. A., Toso, V. D., Malmegrim, K. C. R., Ramos, S. G., et al. (2015). Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice. European Journal of Immunology, 45( 10), 2873–2885. doi:10.1002/eji.201545498
NLM
Carlos D, Yaochite JNU, Rocha FA, Toso VD, Malmegrim KCR, Ramos SG, Jamur MC, Oliver C, Câmara NOS, Andrade MVM de, Cunha F de Q, Silva JS da. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice [Internet]. European Journal of Immunology. 2015 ; 45( 10): 2873–2885.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/eji.201545498
Vancouver
Carlos D, Yaochite JNU, Rocha FA, Toso VD, Malmegrim KCR, Ramos SG, Jamur MC, Oliver C, Câmara NOS, Andrade MVM de, Cunha F de Q, Silva JS da. Mast cells control insulitis and increase Treg cells to confer protection against STZ-induced type 1 diabetes in mice [Internet]. European Journal of Immunology. 2015 ; 45( 10): 2873–2885.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1002/eji.201545498
Trabalho de evento-resumo
Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset (2015)
Ribeiro, Frederico; Françozo, Marcela Cristina Santiago; Zamboni, Dario Simões; Ramos, Simone Gusmão; Câmara, Niels Olsen Saraiva ; Silva, João Santana da; Sartori, Daniela
Source: Abstracts. Conference titles: INNATE IMMUNITY 2015. Unidades: FMRP, ICB
Subjects: DIABETES MELLITUS, INTESTINOS, BACTÉRIAS, PROTEÍNAS
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ABNT
RIBEIRO, Frederico et al. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset . 2015, Anais.. Guarujá: SBI, 2015. Disponível em: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I. Acesso em: 18 nov. 2024.
APA
Ribeiro, F., Françozo, M. C. S., Zamboni, D. S., Ramos, S. G., Câmara, N. O. S., Silva, J. S. da, & Sartori, D. (2015). Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset . In Abstracts. Guarujá: SBI. Recuperado de https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
NLM
Ribeiro F, Françozo MCS, Zamboni DS, Ramos SG, Câmara NOS, Silva JS da, Sartori D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset [Internet]. Abstracts. 2015 ;[citado 2024 nov. 18 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
Vancouver
Ribeiro F, Françozo MCS, Zamboni DS, Ramos SG, Câmara NOS, Silva JS da, Sartori D. Gut microbiota translocation to the pancreatic lymph nodes triggers NOD2 activation and contributes to type 1 diabetes onset [Internet]. Abstracts. 2015 ;[citado 2024 nov. 18 ] Available from: https://icongresso.itarget.com.br/useradm/anais/?clt=imu.3&lng=I
Artigo de periodico
Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation (2014)
Faustino, L.; Fonseca, Denise Morais da ; Florsheim, Esther Borges; Resende, R. R.; Lepique, Ana Paula ; Faquim-Mauro, E. L.; Gomes, E.; Silva, João Santana da; Yagita, H.; Russo, Momtchilo
Source: Mucosal Immunology. Unidades: ICB, FMRP
Subjects: ASMA, INFECÇÕES RESPIRATÓRIAS, INFLAMAÇÃO (EXPERIMENTAÇÃO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FAUSTINO, L. et al. Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation. Mucosal Immunology, v. 7, n. 5, p. 1199–1208, 2014Tradução . . Disponível em: https://doi.org/10.1038/mi.2014.9. Acesso em: 18 nov. 2024.
APA
Faustino, L., Fonseca, D. M. da, Florsheim, E. B., Resende, R. R., Lepique, A. P., Faquim-Mauro, E. L., et al. (2014). Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation. Mucosal Immunology, 7( 5), 1199–1208. doi:10.1038/mi.2014.9
NLM
Faustino L, Fonseca DM da, Florsheim EB, Resende RR, Lepique AP, Faquim-Mauro EL, Gomes E, Silva JS da, Yagita H, Russo M. Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation [Internet]. Mucosal Immunology. 2014 ; 7( 5): 1199–1208.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/mi.2014.9
Vancouver
Faustino L, Fonseca DM da, Florsheim EB, Resende RR, Lepique AP, Faquim-Mauro EL, Gomes E, Silva JS da, Yagita H, Russo M. Tumor necrosis factor-related apoptosis-inducing ligand mediates the resolution of allergic airway inflammation induced by chronic allergen inhalation [Internet]. Mucosal Immunology. 2014 ; 7( 5): 1199–1208.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1038/mi.2014.9
Artigo de periodico
Macrophage trafficking as key mediator of adenine-induced kidney injury (2014)
Correa-Costa, Matheus; Braga, Tárcio Teodoro; Felizardo, Raphael José Ferreira; Andrade-Oliveira, Vinicius; Perez, Katia Regina; Cuccovia, Iolanda Midea ; Hiyane, Meire Ioshie; Silva, João Santana da; Câmara, Niels Olsen Saraiva
Source: Mediators of Inflammation. Unidades: IQ, ICB, FMRP
Subjects: MACRÓFAGOS, BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORREA-COSTA, Matheus et al. Macrophage trafficking as key mediator of adenine-induced kidney injury. Mediators of Inflammation, p. 1-12 art. 291024, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/291024. Acesso em: 18 nov. 2024.
APA
Correa-Costa, M., Braga, T. T., Felizardo, R. J. F., Andrade-Oliveira, V., Perez, K. R., Cuccovia, I. M., et al. (2014). Macrophage trafficking as key mediator of adenine-induced kidney injury. Mediators of Inflammation, 1-12 art. 291024. doi:10.1155/2014/291024
NLM
Correa-Costa M, Braga TT, Felizardo RJF, Andrade-Oliveira V, Perez KR, Cuccovia IM, Hiyane MI, Silva JS da, Câmara NOS. Macrophage trafficking as key mediator of adenine-induced kidney injury [Internet]. Mediators of Inflammation. 2014 ; 1-12 art. 291024.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1155/2014/291024
Vancouver
Correa-Costa M, Braga TT, Felizardo RJF, Andrade-Oliveira V, Perez KR, Cuccovia IM, Hiyane MI, Silva JS da, Câmara NOS. Macrophage trafficking as key mediator of adenine-induced kidney injury [Internet]. Mediators of Inflammation. 2014 ; 1-12 art. 291024.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1155/2014/291024
Artigo de periodico
Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice (2011)
Fonseca, Denise Morais da ; Trombone, Ana Paula Fávaro; Repeke, Carlos Eduardo; Avila-Campos, Mario Julio; Coelho-Castelo, Arlete A. M.; Silva, João Santana da; Campanelli, Ana Paula; Bonato, Vânia Luiza Deperon; Garlet, Gustavo Pompermaier
Source: Journal of Clinical Periodontology. Unidades: FOB, FMRP, ICB
Subjects: INFLAMAÇÃO, IMUNOLOGIA, CAMUNDONGOS, CITOCINAS, DOENÇAS PERIODONTAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FONSECA, Denise Morais da et al. Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice. Journal of Clinical Periodontology, v. 38, n. 2, p. 131-141, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1600-051X.2010.01660.x. Acesso em: 18 nov. 2024.
APA
Fonseca, D. M. da, Trombone, A. P. F., Repeke, C. E., Avila-Campos, M. J., Coelho-Castelo, A. A. M., Silva, J. S. da, et al. (2011). Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice. Journal of Clinical Periodontology, 38( 2), 131-141. doi:10.1111/j.1600-051X.2010.01660.x
NLM
Fonseca DM da, Trombone APF, Repeke CE, Avila-Campos MJ, Coelho-Castelo AAM, Silva JS da, Campanelli AP, Bonato VLD, Garlet GP. Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice [Internet]. Journal of Clinical Periodontology. 2011 ; 38( 2): 131-141.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1600-051X.2010.01660.x
Vancouver
Fonseca DM da, Trombone APF, Repeke CE, Avila-Campos MJ, Coelho-Castelo AAM, Silva JS da, Campanelli AP, Bonato VLD, Garlet GP. Functional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in mice [Internet]. Journal of Clinical Periodontology. 2011 ; 38( 2): 131-141.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1111/j.1600-051X.2010.01660.x
Artigo de periodico
Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions (2011)
Carregaro, Vanessa; Nunes, Anderson de Sá ; Cunha, Thiago Mattar; Grespan, Renata; Oliveira, Carlos J. F.; Lima-Junior, Djalma S.; Costa, Diego L.; Verri Jr, Waldiceu A.; Milanezi, Cristiane M.; Pham, Van My; Brand, David D.; Venezuela, Jesus G.; Silva, João Santana da; Ribeiro, José M. C.; Cunha, Fernando Queiroz
Source: Journal of Immunology. Unidades: ICB, FMRP
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARREGARO, Vanessa et al. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, v. 187, n. 8, p. 4347-4359, 2011Tradução . . Disponível em: https://doi.org/10.4049/jimmunol.1003404. Acesso em: 18 nov. 2024.
APA
Carregaro, V., Nunes, A. de S., Cunha, T. M., Grespan, R., Oliveira, C. J. F., Lima-Junior, D. S., et al. (2011). Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions. Journal of Immunology, 187( 8), 4347-4359. doi:10.4049/jimmunol.1003404
NLM
Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1003404
Vancouver
Carregaro V, Nunes A de S, Cunha TM, Grespan R, Oliveira CJF, Lima-Junior DS, Costa DL, Verri Jr WA, Milanezi CM, Pham VM, Brand DD, Venezuela JG, Silva JS da, Ribeiro JMC, Cunha FQ. Nucleosides from Phlebotomus papatasi salivary gland ameliorate murine collagen-induced arthritis by impairing dendritic cell functions [Internet]. Journal of Immunology. 2011 ; 187( 8): 4347-4359.[citado 2024 nov. 18 ] Available from: https://doi.org/10.4049/jimmunol.1003404
Artigo de periodico
Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties (2011)
Oliveira, Carlo José Freire de; Nunes, Anderson de Sá ; Francischetti, Ivo M. B.; Carregaro, Vanessa; Anatriello, Elen; Silva, João Santana da; Santos, Isabel Kinney Ferreira de Miranda; Ribeiro, José M. C.; Ferreira, Beatriz Rossetti
Source: Journal of Biological Chemistry. Unidades: ICB, EERP, FMRP
Subjects: IMUNOLOGIA, PROTEINAS, ADJUVANTES IMUNOLÓGICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Carlo José Freire de et al. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties. Journal of Biological Chemistry, v. 286, n. 13, p. 10960-10969, 2011Tradução . . Disponível em: https://doi.org/10.1074/jbc.M110.205047. Acesso em: 18 nov. 2024.
APA
Oliveira, C. J. F. de, Nunes, A. de S., Francischetti, I. M. B., Carregaro, V., Anatriello, E., Silva, J. S. da, et al. (2011). Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties. Journal of Biological Chemistry, 286( 13), 10960-10969. doi:10.1074/jbc.M110.205047
NLM
Oliveira CJF de, Nunes A de S, Francischetti IMB, Carregaro V, Anatriello E, Silva JS da, Santos IKF de M, Ribeiro JMC, Ferreira BR. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties [Internet]. Journal of Biological Chemistry. 2011 ; 286( 13): 10960-10969.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1074/jbc.M110.205047
Vancouver
Oliveira CJF de, Nunes A de S, Francischetti IMB, Carregaro V, Anatriello E, Silva JS da, Santos IKF de M, Ribeiro JMC, Ferreira BR. Deconstructing tick saliva: Non-protein molecules with potent immunomodulatory properties [Internet]. Journal of Biological Chemistry. 2011 ; 286( 13): 10960-10969.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1074/jbc.M110.205047

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