Fonte: PLOS ONE. Unidades: IQ, FMVZ
Assuntos: SUPERÓXIDO DISMUTASE, RADICAIS LIVRES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LINARES, Edlaine et al. Tempol moderately extends survival in a 'HSOD1 POT. G93A' ALS rat model by inhibiting neuronal cell loss, oxidative damage and levels of non-native 'HSOD1 POT. G93A' forms. PLOS ONE, v. 8, n. 2, p. 1-12, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0055868. Acesso em: 06 out. 2024.APA
Linares, E., Seixas, L. V., Prazeres, J. N. dos, Ladd, F. V. L., Ladd, A. A. B. L., Ribeiro, A. A. C. M., & Augusto, O. (2013). Tempol moderately extends survival in a 'HSOD1 POT. G93A' ALS rat model by inhibiting neuronal cell loss, oxidative damage and levels of non-native 'HSOD1 POT. G93A' forms. PLOS ONE, 8( 2), 1-12. doi:10.1371/journal.pone.0055868NLM
Linares E, Seixas LV, Prazeres JN dos, Ladd FVL, Ladd AABL, Ribeiro AACM, Augusto O. Tempol moderately extends survival in a 'HSOD1 POT. G93A' ALS rat model by inhibiting neuronal cell loss, oxidative damage and levels of non-native 'HSOD1 POT. G93A' forms [Internet]. PLOS ONE. 2013 ; 8( 2): 1-12.[citado 2024 out. 06 ] Available from: https://doi.org/10.1371/journal.pone.0055868Vancouver
Linares E, Seixas LV, Prazeres JN dos, Ladd FVL, Ladd AABL, Ribeiro AACM, Augusto O. Tempol moderately extends survival in a 'HSOD1 POT. G93A' ALS rat model by inhibiting neuronal cell loss, oxidative damage and levels of non-native 'HSOD1 POT. G93A' forms [Internet]. PLOS ONE. 2013 ; 8( 2): 1-12.[citado 2024 out. 06 ] Available from: https://doi.org/10.1371/journal.pone.0055868