Filtros : "Pinto Junior, Danilo Correa" Limpar

Filtros



Refine with date range


  • Source: Molecular and Cellular Biochemistry. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GERLINGER-ROMERO, Frederico et al. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p. Molecular and Cellular Biochemistry, v. 427, n. 1-2, p. 187-199, 2017Tradução . . Disponível em: https://doi.org/10.1007/s11010-016-2910-z. Acesso em: 06 out. 2024.
    • APA

      Gerlinger-Romero, F., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., & Machado, U. F. (2017). Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p. Molecular and Cellular Biochemistry, 427( 1-2), 187-199. doi:10.1007/s11010-016-2910-z
    • NLM

      Gerlinger-Romero F, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p [Internet]. Molecular and Cellular Biochemistry. 2017 ; 427( 1-2): 187-199.[citado 2024 out. 06 ] Available from: https://doi.org/10.1007/s11010-016-2910-z
    • Vancouver

      Gerlinger-Romero F, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF. Dysregulation between TRIM63/FBXO32 expression and soleus muscle wasting in diabetic rats: potential role of miR-1-3p, -29a/b-3p, and -133a/b-3p [Internet]. Molecular and Cellular Biochemistry. 2017 ; 427( 1-2): 187-199.[citado 2024 out. 06 ] Available from: https://doi.org/10.1007/s11010-016-2910-z
  • Source: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      MORAES, Paulo Alexandre et al. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, v. 114, n. 1, p. 36-44, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2014.07.040. Acesso em: 06 out. 2024.
    • APA

      Moraes, P. A., Yonamine, C. Y., Pinto Junior, D. C., Esteves, J. V. D. C., Machado, U. F., & Mori, R. C. (2014). Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites. Life Sciences, 114( 1), 36-44. doi:10.1016/j.lfs.2014.07.040
    • NLM

      Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 out. 06 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040
    • Vancouver

      Moraes PA, Yonamine CY, Pinto Junior DC, Esteves JVDC, Machado UF, Mori RC. Insulin acutely triggers transcription of Slc2a4 gene: participation of the AT-rich, E-box and NFKB-binding sites [Internet]. Life Sciences. 2014 ; 114( 1): 36-44.[citado 2024 out. 06 ] Available from: https://doi.org/10.1016/j.lfs.2014.07.040

Digital Library of Intellectual Production of Universidade de São Paulo     2012 - 2024