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Artigo de periodico
Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway (2017)
Roversi, Fernanda Marconi; Pericole, Fernando Vieira; Machado Neto, João Agostinho ; Duarte, Adriana da Silva Santos; Longhini, Ana Leda; Corrocher, Flavia Adolfo; Palodetto, Bruna; Ferro, Karla Priscila; Rosa, Renata Giardini; Baratti, Mariana Ozello; Verjovski-Almeida, Sergio ; Traina, Fabiola; Molinari, Alessio; Botta, Maurizio; Saad, Sara Teresinha Olalla
Source: Biochimica et Biophysica Acta. Unidades: IQ, FMRP
Subjects: LEUCEMIA MIELOIDE AGUDA, CÉLULAS-TRONCO
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ABNT
ROVERSI, Fernanda Marconi et al. Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway. Biochimica et Biophysica Acta, v. 1863, n. 2, p. 450-461, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2016.11.013. Acesso em: 04 nov. 2024.
APA
Roversi, F. M., Pericole, F. V., Machado Neto, J. A., Duarte, A. da S. S., Longhini, A. L., Corrocher, F. A., et al. (2017). Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway. Biochimica et Biophysica Acta, 1863( 2), 450-461. doi:10.1016/j.bbadis.2016.11.013
NLM
Roversi FM, Pericole FV, Machado Neto JA, Duarte A da SS, Longhini AL, Corrocher FA, Palodetto B, Ferro KP, Rosa RG, Baratti MO, Verjovski-Almeida S, Traina F, Molinari A, Botta M, Saad STO. Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway [Internet]. Biochimica et Biophysica Acta. 2017 ; 1863( 2): 450-461.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bbadis.2016.11.013
Vancouver
Roversi FM, Pericole FV, Machado Neto JA, Duarte A da SS, Longhini AL, Corrocher FA, Palodetto B, Ferro KP, Rosa RG, Baratti MO, Verjovski-Almeida S, Traina F, Molinari A, Botta M, Saad STO. Hematopoietic cell kinase (HCK) is a potential therapeutic target for dysplastic and leukemic cells due to integration of erythropoietin/PI3K pathway and regulation of erythropoiesis: HCK in erythropoietin/PI3K pathway [Internet]. Biochimica et Biophysica Acta. 2017 ; 1863( 2): 450-461.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.bbadis.2016.11.013
Artigo de periodico
Serine protease inhibitor kunitz-type 2 is downregulated in myelodysplastic syndromes and modulates cell–cell adhesion (2014)
Roversi, Fernanda Marconi; Lopes, Matheus Rodrigues; Machado Neto, João Agostinho ; Longhini, Ana Leda F.; Duarte, Adriana Silva Santos; Baratti, Mariana O.; Palodetto, Bruna; Corrocher, Flávia Adolfo; Pericole, Fernando V.; Campos, Paula de Melo; Favaro, Patricia; Traina, Fabíola; Saad, Sara Teresinha Olalla
Source: Stem Cells and Development. Unidade: FMRP
Subjects: DOENÇAS HEMATOLÓGICAS, HEMATOPOESE, INIBIDORES DE ENZIMAS
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ABNT
ROVERSI, Fernanda Marconi et al. Serine protease inhibitor kunitz-type 2 is downregulated in myelodysplastic syndromes and modulates cell–cell adhesion. Stem Cells and Development, v. 23, n. 10, p. 1109-1120, 2014Tradução . . Disponível em: https://doi.org/10.1089/scd.2013.0441. Acesso em: 04 nov. 2024.
APA
Roversi, F. M., Lopes, M. R., Machado Neto, J. A., Longhini, A. L. F., Duarte, A. S. S., Baratti, M. O., et al. (2014). Serine protease inhibitor kunitz-type 2 is downregulated in myelodysplastic syndromes and modulates cell–cell adhesion. Stem Cells and Development, 23( 10), 1109-1120. doi:10.1089/scd.2013.0441
NLM
Roversi FM, Lopes MR, Machado Neto JA, Longhini ALF, Duarte ASS, Baratti MO, Palodetto B, Corrocher FA, Pericole FV, Campos P de M, Favaro P, Traina F, Saad STO. Serine protease inhibitor kunitz-type 2 is downregulated in myelodysplastic syndromes and modulates cell–cell adhesion [Internet]. Stem Cells and Development. 2014 ; 23( 10): 1109-1120.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1089/scd.2013.0441
Vancouver
Roversi FM, Lopes MR, Machado Neto JA, Longhini ALF, Duarte ASS, Baratti MO, Palodetto B, Corrocher FA, Pericole FV, Campos P de M, Favaro P, Traina F, Saad STO. Serine protease inhibitor kunitz-type 2 is downregulated in myelodysplastic syndromes and modulates cell–cell adhesion [Internet]. Stem Cells and Development. 2014 ; 23( 10): 1109-1120.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1089/scd.2013.0441
Trabalho de evento-resumo
Downregulation of SPINT2 expression in stromal cells from myelodysplastic syndrome patients increases HGF and SDF-1 secretion and promotes cell-cell adhesion by alteration in integrin expression (2013)
Roversi, Fernanda Marconi; Lopes, Matheus Rodrigues; Machado Neto, João Agostinho ; Longhini, Ana Leda; Duarte, Adriana Silva Santos; Palodetto, Bruna; Corrocher, Flavia Adolfo; Campos, Paula de Melo; Baratti, Mariana O.; Pericole, Fernando V.; Favaro, Patricia; Traina, Fabíola; Saad, Sara Teresinha Ollala
Source: Abstracts, Program and Personal Scheduler. Conference titles: Annual Meeting and Exposition of American Society of Hematology (ASH). Unidade: FMRP
Subjects: HEMATOPOESE, DOENÇAS HEMATOLÓGICAS, GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROVERSI, Fernanda Marconi et al. Downregulation of SPINT2 expression in stromal cells from myelodysplastic syndrome patients increases HGF and SDF-1 secretion and promotes cell-cell adhesion by alteration in integrin expression. 2013, Anais.. New Orleans: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo, 2013. Disponível em: https://ash.confex.com/ash/2013/webprogram/Paper57527.html. Acesso em: 04 nov. 2024.
APA
Roversi, F. M., Lopes, M. R., Machado Neto, J. A., Longhini, A. L., Duarte, A. S. S., Palodetto, B., et al. (2013). Downregulation of SPINT2 expression in stromal cells from myelodysplastic syndrome patients increases HGF and SDF-1 secretion and promotes cell-cell adhesion by alteration in integrin expression. In Abstracts, Program and Personal Scheduler. New Orleans: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Recuperado de https://ash.confex.com/ash/2013/webprogram/Paper57527.html
NLM
Roversi FM, Lopes MR, Machado Neto JA, Longhini AL, Duarte ASS, Palodetto B, Corrocher FA, Campos P de M, Baratti MO, Pericole FV, Favaro P, Traina F, Saad STO. Downregulation of SPINT2 expression in stromal cells from myelodysplastic syndrome patients increases HGF and SDF-1 secretion and promotes cell-cell adhesion by alteration in integrin expression [Internet]. Abstracts, Program and Personal Scheduler. 2013 ;[citado 2024 nov. 04 ] Available from: https://ash.confex.com/ash/2013/webprogram/Paper57527.html
Vancouver
Roversi FM, Lopes MR, Machado Neto JA, Longhini AL, Duarte ASS, Palodetto B, Corrocher FA, Campos P de M, Baratti MO, Pericole FV, Favaro P, Traina F, Saad STO. Downregulation of SPINT2 expression in stromal cells from myelodysplastic syndrome patients increases HGF and SDF-1 secretion and promotes cell-cell adhesion by alteration in integrin expression [Internet]. Abstracts, Program and Personal Scheduler. 2013 ;[citado 2024 nov. 04 ] Available from: https://ash.confex.com/ash/2013/webprogram/Paper57527.html
Artigo de periodico
MDR-1 and GST polymorphisms are involved in myelodysplasia progression (2013)
Palodetto, Bruna; Campos, Paula de Melo; Benites, Bruno Deltreggia; Chauffaille, Maria de Lourdes Lopes Ferrari; Velloso, Elvira Deolinda Rodrigues Pereira; Traina, Fabiola; Saad, Sara Teresinha Olalla
Source: Leukemia Research. Unidade: FMRP
Subjects: IDOSOS, DOENÇAS HEMATOLÓGICAS, POLIMORFISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PALODETTO, Bruna et al. MDR-1 and GST polymorphisms are involved in myelodysplasia progression. Leukemia Research, v. 37, n. 8, p. 970-973, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.leukres.2013.04.024. Acesso em: 04 nov. 2024.
APA
Palodetto, B., Campos, P. de M., Benites, B. D., Chauffaille, M. de L. L. F., Velloso, E. D. R. P., Traina, F., & Saad, S. T. O. (2013). MDR-1 and GST polymorphisms are involved in myelodysplasia progression. Leukemia Research, 37( 8), 970-973. doi:10.1016/j.leukres.2013.04.024
NLM
Palodetto B, Campos P de M, Benites BD, Chauffaille M de LLF, Velloso EDRP, Traina F, Saad STO. MDR-1 and GST polymorphisms are involved in myelodysplasia progression [Internet]. Leukemia Research. 2013 ; 37( 8): 970-973.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.leukres.2013.04.024
Vancouver
Palodetto B, Campos P de M, Benites BD, Chauffaille M de LLF, Velloso EDRP, Traina F, Saad STO. MDR-1 and GST polymorphisms are involved in myelodysplasia progression [Internet]. Leukemia Research. 2013 ; 37( 8): 970-973.[citado 2024 nov. 04 ] Available from: https://doi.org/10.1016/j.leukres.2013.04.024

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