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Artigo de periodico
Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria (2012)
Francischetti, Ivo M. B.; Oliveira, Carlo J.; Ostera, Graciela R.; Yager, Stephanie B.; Debierre-Grockiego, Françoise; Carregaro, Vanessa; Jaramillo-Gutierrez, Giovanna; Hume, Jen C. C.; Jiang, Lubin; Moretz, Samuel E.; Lin, Christina K.; Ribeiro, José M. C.; Long, Carole A.; Vickers, Brandi K.; Schwarz, Ralph T.; Seydel, Karl B.; Iacobelli, Massimo; Ackerman, Hans C.; Srinivasan, Prakash; Gomes, Regis B.; Wang, Xunde; Monteiro, Robson Q.; Kotsyfakis, Michail; Nunes, Anderson de Sá ; Waisberg, Michael
Fonte: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
FRANCISCHETTI, Ivo M. B. et al. Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 32, p. 786-798, 2012Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.112.250761. Acesso em: 02 nov. 2025.
APA
Francischetti, I. M. B., Oliveira, C. J., Ostera, G. R., Yager, S. B., Debierre-Grockiego, F., Carregaro, V., et al. (2012). Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria. Arteriosclerosis, Thrombosis, and Vascular Biology, 32, 786-798. doi:10.1161/ATVBAHA.112.250761
NLM
Francischetti IMB, Oliveira CJ, Ostera GR, Yager SB, Debierre-Grockiego F, Carregaro V, Jaramillo-Gutierrez G, Hume JCC, Jiang L, Moretz SE, Lin CK, Ribeiro JMC, Long CA, Vickers BK, Schwarz RT, Seydel KB, Iacobelli M, Ackerman HC, Srinivasan P, Gomes RB, Wang X, Monteiro RQ, Kotsyfakis M, Nunes A de S, Waisberg M. Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2012 ; 32 786-798.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1161/ATVBAHA.112.250761
Vancouver
Francischetti IMB, Oliveira CJ, Ostera GR, Yager SB, Debierre-Grockiego F, Carregaro V, Jaramillo-Gutierrez G, Hume JCC, Jiang L, Moretz SE, Lin CK, Ribeiro JMC, Long CA, Vickers BK, Schwarz RT, Seydel KB, Iacobelli M, Ackerman HC, Srinivasan P, Gomes RB, Wang X, Monteiro RQ, Kotsyfakis M, Nunes A de S, Waisberg M. Defibrotide interferes with several steps of the coagulation-inflammation cycle and exhibits therapeutic potential to treat severe malaria [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2012 ; 32 786-798.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1161/ATVBAHA.112.250761
Artigo de periodico
Lufaxin, a novel factor Xa inhibitor from the salivary gland of the sand fly Lutzomyia longipalpis, blocks PAR2 activation and inhibits inflammation and thrombosis in vivo (2012)
Collin, Nicolas; Assunpção, Teresa C. F.; Mizurini, Daniella M.; Gilmore, Dana; Dutra-Oliveira, Angélica; Kotsyfakis, Michalis; Nunes, Anderson de Sá ; Teixeira, Clarissa; Ribeiro, José M. C.; Monteiro, Robson Q.; Valenzuela, Jesus G.; Francischetti, Ivo M. B.
Fonte: Arteriosclerosis, Thrombosis Vascular Biology. Unidade: ICB
Assunto: IMUNOLOGIA
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ABNT
COLLIN, Nicolas et al. Lufaxin, a novel factor Xa inhibitor from the salivary gland of the sand fly Lutzomyia longipalpis, blocks PAR2 activation and inhibits inflammation and thrombosis in vivo. Arteriosclerosis, Thrombosis Vascular Biology, v. 32, n. 9, p. 2185-2198, 2012Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.112.253906. Acesso em: 02 nov. 2025.
APA
Collin, N., Assunpção, T. C. F., Mizurini, D. M., Gilmore, D., Dutra-Oliveira, A., Kotsyfakis, M., et al. (2012). Lufaxin, a novel factor Xa inhibitor from the salivary gland of the sand fly Lutzomyia longipalpis, blocks PAR2 activation and inhibits inflammation and thrombosis in vivo. Arteriosclerosis, Thrombosis Vascular Biology, 32( 9), 2185-2198. doi:10.1161/ATVBAHA.112.253906
NLM
Collin N, Assunpção TCF, Mizurini DM, Gilmore D, Dutra-Oliveira A, Kotsyfakis M, Nunes A de S, Teixeira C, Ribeiro JMC, Monteiro RQ, Valenzuela JG, Francischetti IMB. Lufaxin, a novel factor Xa inhibitor from the salivary gland of the sand fly Lutzomyia longipalpis, blocks PAR2 activation and inhibits inflammation and thrombosis in vivo [Internet]. Arteriosclerosis, Thrombosis Vascular Biology. 2012 ; 32( 9): 2185-2198.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1161/ATVBAHA.112.253906
Vancouver
Collin N, Assunpção TCF, Mizurini DM, Gilmore D, Dutra-Oliveira A, Kotsyfakis M, Nunes A de S, Teixeira C, Ribeiro JMC, Monteiro RQ, Valenzuela JG, Francischetti IMB. Lufaxin, a novel factor Xa inhibitor from the salivary gland of the sand fly Lutzomyia longipalpis, blocks PAR2 activation and inhibits inflammation and thrombosis in vivo [Internet]. Arteriosclerosis, Thrombosis Vascular Biology. 2012 ; 32( 9): 2185-2198.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1161/ATVBAHA.112.253906
Artigo de periodico
Malignant transformation in melanocytes is associated with increased production of procoagulant microvesicles (2011)
Lima, Luize G.; Oliveira, Andreia S.; Campos, Luiza C.; Bonamino, Martin; Chammas, Roger; Werneck, Claudio C.; Vicente, Cristina P.; Barcinski, Marcello A.; Petersen, Lars C.; Monteiro, Robson Q.
Fonte: Thrombosis and Haemostasis. Unidades: FM, ICB
Assuntos: MELANOMA, DOENÇAS VASCULARES, TROMBOSE, CULTURA DE CELULAS, CITOMETRIA DE FLUXO
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ABNT
LIMA, Luize G. et al. Malignant transformation in melanocytes is associated with increased production of procoagulant microvesicles. Thrombosis and Haemostasis, v. 106, n. 4, p. 712-723, 2011Tradução . . Disponível em: https://doi.org/10.1160/th11-03-0143. Acesso em: 02 nov. 2025.
APA
Lima, L. G., Oliveira, A. S., Campos, L. C., Bonamino, M., Chammas, R., Werneck, C. C., et al. (2011). Malignant transformation in melanocytes is associated with increased production of procoagulant microvesicles. Thrombosis and Haemostasis, 106( 4), 712-723. doi:10.1160/th11-03-0143
NLM
Lima LG, Oliveira AS, Campos LC, Bonamino M, Chammas R, Werneck CC, Vicente CP, Barcinski MA, Petersen LC, Monteiro RQ. Malignant transformation in melanocytes is associated with increased production of procoagulant microvesicles [Internet]. Thrombosis and Haemostasis. 2011 ; 106( 4): 712-723.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1160/th11-03-0143
Vancouver
Lima LG, Oliveira AS, Campos LC, Bonamino M, Chammas R, Werneck CC, Vicente CP, Barcinski MA, Petersen LC, Monteiro RQ. Malignant transformation in melanocytes is associated with increased production of procoagulant microvesicles [Internet]. Thrombosis and Haemostasis. 2011 ; 106( 4): 712-723.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1160/th11-03-0143
Artigo de periodico
Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias (2011)
Veiga, Camilla de S. B.; Carneiro-Lobo, Tatiana C.; Coelho, Cláudia J. B. P.; Carvalho, Silvia M. F.; Maia, Raquel C.; Vasconcelos, Flávia C.; Abdelhay, Eliana; Mencalha, André L.; Ferreira, Aline F.; Castro, Fabíola Attié de; Monteiro, Robson Q.
Fonte: Blood Cells, Molecules, and Diseases. Unidade: FCFRP
Assuntos: LEUCEMIA MIELOIDE AGUDA, RECEPTORES
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ABNT
VEIGA, Camilla de S. B. et al. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias. Blood Cells, Molecules, and Diseases, v. 46, n. 3, p. 230-234, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.bcmd.2010.12.005. Acesso em: 02 nov. 2025.
APA
Veiga, C. de S. B., Carneiro-Lobo, T. C., Coelho, C. J. B. P., Carvalho, S. M. F., Maia, R. C., Vasconcelos, F. C., et al. (2011). Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias. Blood Cells, Molecules, and Diseases, 46( 3), 230-234. doi:10.1016/j.bcmd.2010.12.005
NLM
Veiga C de SB, Carneiro-Lobo TC, Coelho CJBP, Carvalho SMF, Maia RC, Vasconcelos FC, Abdelhay E, Mencalha AL, Ferreira AF, Castro FA de, Monteiro RQ. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 3): 230-234.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1016/j.bcmd.2010.12.005
Vancouver
Veiga C de SB, Carneiro-Lobo TC, Coelho CJBP, Carvalho SMF, Maia RC, Vasconcelos FC, Abdelhay E, Mencalha AL, Ferreira AF, Castro FA de, Monteiro RQ. Increased expression of protease-activated receptor 1 (PAR-1) in human leukemias [Internet]. Blood Cells, Molecules, and Diseases. 2011 ; 46( 3): 230-234.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1016/j.bcmd.2010.12.005
Artigo de periodico
Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner (2009)
Lima, Luize G.; Chammas, Roger; Monteiro, Robson Q.; Moreira, Maria Elisabete C.; Barcinski, Marcello André
Fonte: Cancer Letters. Unidades: FM, ICB
Assuntos: MELANOMA, CULTURA DE CÉLULAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Luize G. et al. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner. Cancer Letters, v. 283, n. 2, p. 168-175, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.canlet.2009.03.041. Acesso em: 02 nov. 2025.
APA
Lima, L. G., Chammas, R., Monteiro, R. Q., Moreira, M. E. C., & Barcinski, M. A. (2009). Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner. Cancer Letters, 283( 2), 168-175. doi:10.1016/j.canlet.2009.03.041
NLM
Lima LG, Chammas R, Monteiro RQ, Moreira MEC, Barcinski MA. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner [Internet]. Cancer Letters. 2009 ; 283( 2): 168-175.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1016/j.canlet.2009.03.041
Vancouver
Lima LG, Chammas R, Monteiro RQ, Moreira MEC, Barcinski MA. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner [Internet]. Cancer Letters. 2009 ; 283( 2): 168-175.[citado 2025 nov. 02 ] Available from: https://doi.org/10.1016/j.canlet.2009.03.041

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