Fonte: Biochemical and Biophysical Research Communications. Unidade: FM
Assuntos: ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS, RECEPTORES DE ANGIOTENSINA, CISALHAMENTO
ABNT
BARAUNA, Valério G. et al. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177. Biochemical and Biophysical Research Communications, v. 441, n. 4, p. 713-719, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2013.10.108. Acesso em: 18 ago. 2024.APA
Barauna, V. G., Mantuan, P. R., Magalhães, F. C., Campos, L. C. G., & Krieger, J. E. (2013). AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177. Biochemical and Biophysical Research Communications, 441( 4), 713-719. doi:10.1016/j.bbrc.2013.10.108NLM
Barauna VG, Mantuan PR, Magalhães FC, Campos LCG, Krieger JE. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177 [Internet]. Biochemical and Biophysical Research Communications. 2013 ; 441( 4): 713-719.[citado 2024 ago. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2013.10.108Vancouver
Barauna VG, Mantuan PR, Magalhães FC, Campos LCG, Krieger JE. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177 [Internet]. Biochemical and Biophysical Research Communications. 2013 ; 441( 4): 713-719.[citado 2024 ago. 18 ] Available from: https://doi.org/10.1016/j.bbrc.2013.10.108