Source: American Journal of Physiology. Endocrinology and Metabolism. Unidade: ICB
Subjects: FISIOLOGIA, RATOS, GLIC0SE, MONOSSACARIDEOS
ABNT
FESTUCCIA, William Tadeu Lara et al. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, v. 9, p. E1046-E1054, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00683.2013. Acesso em: 03 nov. 2024.APA
Festuccia, W. T. L., Blanchard, P. -G., Belchior, T., Chimin, P., Paschoal, V. A., Magdalon, J., et al. (2014). PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats. American Journal of Physiology. Endocrinology and Metabolism, 9, E1046-E1054. doi:10.1152/ajpendo.00683.2013NLM
Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013Vancouver
Festuccia WTL, Blanchard P-G, Belchior T, Chimin P, Paschoal VA, Magdalon J, Hirabara SM, Simões D, St-Pierre P, Marette A, Deshaies Y, Carpinelli AR. PPARγ activation attenuates glucose intolerance induced by mTOR inhibition with rapamycin in rats [Internet]. American Journal of Physiology. Endocrinology and Metabolism. 2014 ; 9 E1046-E1054.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1152/ajpendo.00683.2013