Filtros : "Grace, Anthony A." "Neuropsychopharmacology" Removido: "Mathis, Alice de" Limpar

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  • Source: Neuropsychopharmacology. Unidade: FMRP

    Subjects: MODELOS ANIMAIS DE DOENÇAS, HIPOCAMPO, ESQUIZOFRENIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      KIEMES, Amanda et al. GABAA and NMDA receptor density alterations and their behavioral correlates in the gestational methylazoxymethanol acetate model for schizophrenia. Neuropsychopharmacology, v. 47, n. 3, p. 687-695, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41386-021-01213-0. Acesso em: 03 jul. 2024.
    • APA

      Kiemes, A., Gomes, F. V., Cash, D., Uliana, D. L., Simmons, C., Singh, N., et al. (2022). GABAA and NMDA receptor density alterations and their behavioral correlates in the gestational methylazoxymethanol acetate model for schizophrenia. Neuropsychopharmacology, 47( 3), 687-695. doi:10.1038/s41386-021-01213-0
    • NLM

      Kiemes A, Gomes FV, Cash D, Uliana DL, Simmons C, Singh N, Vernon AC, Turkheimer F, Davies C, Stone JM, Grace AA, Modinos G. GABAA and NMDA receptor density alterations and their behavioral correlates in the gestational methylazoxymethanol acetate model for schizophrenia [Internet]. Neuropsychopharmacology. 2022 ; 47( 3): 687-695.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-021-01213-0
    • Vancouver

      Kiemes A, Gomes FV, Cash D, Uliana DL, Simmons C, Singh N, Vernon AC, Turkheimer F, Davies C, Stone JM, Grace AA, Modinos G. GABAA and NMDA receptor density alterations and their behavioral correlates in the gestational methylazoxymethanol acetate model for schizophrenia [Internet]. Neuropsychopharmacology. 2022 ; 47( 3): 687-695.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-021-01213-0
  • Source: Neuropsychopharmacology. Unidade: FMRP

    Subjects: HIPOCAMPO, NÚCLEOS TALÂMICOS, PLASTICIDADE NEURONAL, NEURÔNIOS

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    • ABNT

      ULIANA, Daniela L. e GOMES, Felipe Villela e GRACE, Anthony A. Nucleus reuniens inactivation reverses stress-induced hypodopaminergic state and altered hippocampal-accumbens synaptic plasticity. Neuropsychopharmacology, v. 47 , n. 8, p. 1513-1522, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41386-022-01333-1. Acesso em: 03 jul. 2024.
    • APA

      Uliana, D. L., Gomes, F. V., & Grace, A. A. (2022). Nucleus reuniens inactivation reverses stress-induced hypodopaminergic state and altered hippocampal-accumbens synaptic plasticity. Neuropsychopharmacology, 47 ( 8), 1513-1522. doi:10.1038/s41386-022-01333-1
    • NLM

      Uliana DL, Gomes FV, Grace AA. Nucleus reuniens inactivation reverses stress-induced hypodopaminergic state and altered hippocampal-accumbens synaptic plasticity [Internet]. Neuropsychopharmacology. 2022 ;47 ( 8): 1513-1522.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-022-01333-1
    • Vancouver

      Uliana DL, Gomes FV, Grace AA. Nucleus reuniens inactivation reverses stress-induced hypodopaminergic state and altered hippocampal-accumbens synaptic plasticity [Internet]. Neuropsychopharmacology. 2022 ;47 ( 8): 1513-1522.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-022-01333-1
  • Source: Neuropsychopharmacology. Unidade: FMRP

    Subjects: NEURÔNIOS, CÓRTEX PRÉ-FRONTAL, ESTRESSE, CÉREBRO

    PrivadoAcesso à fonteDOIHow to cite
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    • ABNT

      ULIANA, Daniela L. e GOMES, Felipe Villela e GRACE, Anthony A. Stress impacts corticoamygdalar connectivity in an age-dependent manner. Neuropsychopharmacology, v. 46, n. 4, p. 731-740, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41386-020-00886-3. Acesso em: 03 jul. 2024.
    • APA

      Uliana, D. L., Gomes, F. V., & Grace, A. A. (2021). Stress impacts corticoamygdalar connectivity in an age-dependent manner. Neuropsychopharmacology, 46( 4), 731-740. doi:10.1038/s41386-020-00886-3
    • NLM

      Uliana DL, Gomes FV, Grace AA. Stress impacts corticoamygdalar connectivity in an age-dependent manner [Internet]. Neuropsychopharmacology. 2021 ; 46( 4): 731-740.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-020-00886-3
    • Vancouver

      Uliana DL, Gomes FV, Grace AA. Stress impacts corticoamygdalar connectivity in an age-dependent manner [Internet]. Neuropsychopharmacology. 2021 ; 46( 4): 731-740.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-020-00886-3
  • Source: Neuropsychopharmacology. Unidade: FMRP

    Subjects: MEDO, ESQUIZOFRENIA, ROEDORES, NEUROFARMACOLOGIA

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    • ABNT

      ULIANA, Daniela L. e RESSTEL, Leonardo Barbosa Moraes e GRACE, Anthony A. Fear extinction disruption in a developmental rodent model of schizophrenia correlates with an impairment in basolateral amygdala-medial prefrontal cortex plasticity. Neuropsychopharmacology, v. 43, n. 12, p. 2459-2467, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41386-018-0128-3. Acesso em: 03 jul. 2024.
    • APA

      Uliana, D. L., Resstel, L. B. M., & Grace, A. A. (2018). Fear extinction disruption in a developmental rodent model of schizophrenia correlates with an impairment in basolateral amygdala-medial prefrontal cortex plasticity. Neuropsychopharmacology, 43( 12), 2459-2467. doi:10.1038/s41386-018-0128-3
    • NLM

      Uliana DL, Resstel LBM, Grace AA. Fear extinction disruption in a developmental rodent model of schizophrenia correlates with an impairment in basolateral amygdala-medial prefrontal cortex plasticity [Internet]. Neuropsychopharmacology. 2018 ; 43( 12): 2459-2467.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-018-0128-3
    • Vancouver

      Uliana DL, Resstel LBM, Grace AA. Fear extinction disruption in a developmental rodent model of schizophrenia correlates with an impairment in basolateral amygdala-medial prefrontal cortex plasticity [Internet]. Neuropsychopharmacology. 2018 ; 43( 12): 2459-2467.[citado 2024 jul. 03 ] Available from: https://doi.org/10.1038/s41386-018-0128-3

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