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Artigo de periodico
Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms (2024)
Glaser, Talita ; Martins, Patrícia; Beco, Renata; Bento, Carolina Adriane ; Cappellari, Angelica R; Oliveira, Sophia La Banca de; Merkel, Christian A; Sampaio, Vanessa Fernandes Arnaud ; Lameu, Claudiana ; Battastini, Ana Maria ; Ulrich, Henning
Source: Frontiers in Pharmacology. Unidades: FM, IQ
Subjects: ADENOSINA, CÉLULAS-TRONCO, CAFEÍNA
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ABNT
GLASER, Talita et al. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms. Frontiers in Pharmacology, v. 14, p. 1-16 art. 1328398, 2024Tradução . . Disponível em: https://dx.doi.org/10.3389/fphar.2023.1328398. Acesso em: 14 jun. 2024.
APA
Glaser, T., Martins, P., Beco, R., Bento, C. A., Cappellari, A. R., Oliveira, S. L. B. de, et al. (2024). Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms. Frontiers in Pharmacology, 14, 1-16 art. 1328398. doi:10.3389/fphar.2023.1328398
NLM
Glaser T, Martins P, Beco R, Bento CA, Cappellari AR, Oliveira SLB de, Merkel CA, Sampaio VFA, Lameu C, Battastini AM, Ulrich H. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms [Internet]. Frontiers in Pharmacology. 2024 ; 14 1-16 art. 1328398.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.3389/fphar.2023.1328398
Vancouver
Glaser T, Martins P, Beco R, Bento CA, Cappellari AR, Oliveira SLB de, Merkel CA, Sampaio VFA, Lameu C, Battastini AM, Ulrich H. Impairment of adenosine signaling disrupts early embryo development: unveiling the underlying mechanisms [Internet]. Frontiers in Pharmacology. 2024 ; 14 1-16 art. 1328398.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.3389/fphar.2023.1328398
Parte de monografia/livro
Purinergic signaling in brain physiology (2023)
Glaser, Talita ; Ulrich, Henning
Source: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Unidade: IQ
Assunto: FISIOLOGIA
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ABNT
GLASER, Talita e ULRICH, Henning. Purinergic signaling in brain physiology. Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Tradução . Cham: Springer, 2023. . . Acesso em: 14 jun. 2024.
APA
Glaser, T., & Ulrich, H. (2023). Purinergic signaling in brain physiology. In Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer.
NLM
Glaser T, Ulrich H. Purinergic signaling in brain physiology. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Vancouver
Glaser T, Ulrich H. Purinergic signaling in brain physiology. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Artigo de periodico
Bj-PRO-10c, as an allosteric regulator of argininosuccinate synthase, is a potential therapy for neuroblastoma metastasis (2023)
Coutinho, Fernanda; Guimarães, Lara Mendes Ferreira ; Seeger, Rodrigo; Santos, Ana Paula J; Glaser, Talita ; Yamamoto, Denise; Lacerda, Lucas; Sampaio, Vanessa Fernandes Arnaud ; Rossini, Caio Vinicius Teles ; Rabelo, Izadora; Medeiros, Nathália Miranda de ; Truzzi, Daniela Ramos ; Juliano, Maria Aparecida; Juliano, Luiz ; Ulrich, Henning ; Lameu, Claudiana
Source: Toxicon. Unidade: IQ
Subjects: METÁSTASE NEOPLÁSICA, NEUROBLASTOMA, ÓXIDO NÍTRICO, PEPTÍDEOS
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ABNT
COUTINHO, Fernanda et al. Bj-PRO-10c, as an allosteric regulator of argininosuccinate synthase, is a potential therapy for neuroblastoma metastasis. Toxicon, v. 233, p. 1-9 art. 107228, 2023Tradução . . Disponível em: https://dx.doi.org/10.1016/j.toxicon.2023.107228. Acesso em: 14 jun. 2024.
APA
Coutinho, F., Guimarães, L. M. F., Seeger, R., Santos, A. P. J., Glaser, T., Yamamoto, D., et al. (2023). Bj-PRO-10c, as an allosteric regulator of argininosuccinate synthase, is a potential therapy for neuroblastoma metastasis. Toxicon, 233, 1-9 art. 107228. doi:10.1016/j.toxicon.2023.107228
NLM
Coutinho F, Guimarães LMF, Seeger R, Santos APJ, Glaser T, Yamamoto D, Lacerda L, Sampaio VFA, Rossini CVT, Rabelo I, Medeiros NM de, Truzzi DR, Juliano MA, Juliano L, Ulrich H, Lameu C. Bj-PRO-10c, as an allosteric regulator of argininosuccinate synthase, is a potential therapy for neuroblastoma metastasis [Internet]. Toxicon. 2023 ; 233 1-9 art. 107228.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.1016/j.toxicon.2023.107228
Vancouver
Coutinho F, Guimarães LMF, Seeger R, Santos APJ, Glaser T, Yamamoto D, Lacerda L, Sampaio VFA, Rossini CVT, Rabelo I, Medeiros NM de, Truzzi DR, Juliano MA, Juliano L, Ulrich H, Lameu C. Bj-PRO-10c, as an allosteric regulator of argininosuccinate synthase, is a potential therapy for neuroblastoma metastasis [Internet]. Toxicon. 2023 ; 233 1-9 art. 107228.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.1016/j.toxicon.2023.107228
Artigo de periodico
Differentiated embryonic Neurospheres from familial alzheimer’s disease model show innate Immune and glial cell responses (2023)
Pillat, Micheli Mainardi ; Ayupe, Ana Carolina ; Juvenal, Guilherme Araújo ; Meinerz, Carine; Glaser, Talita ; Pellegrina, Diogo Vieira da Silva ; Paiva, Daisylea Souza; Mello, Carlos Fernando ; Longo, Beatriz Monteiro ; Reis, Eduardo Moraes ; Ulrich, Henning
Source: Stem Cell Reviews and Reports. Unidade: IQ
Subjects: DOENÇA DE ALZHEIMER, EXPRESSÃO GÊNICA, DOENÇAS DEGENERATIVAS
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ABNT
PILLAT, Micheli Mainardi et al. Differentiated embryonic Neurospheres from familial alzheimer’s disease model show innate Immune and glial cell responses. Stem Cell Reviews and Reports, v. 19, p. 1800-1811, 2023Tradução . . Disponível em: https://dx.doi.org/10.1007/s12015-023-10542-0. Acesso em: 14 jun. 2024.
APA
Pillat, M. M., Ayupe, A. C., Juvenal, G. A., Meinerz, C., Glaser, T., Pellegrina, D. V. da S., et al. (2023). Differentiated embryonic Neurospheres from familial alzheimer’s disease model show innate Immune and glial cell responses. Stem Cell Reviews and Reports, 19, 1800-1811. doi:10.1007/s12015-023-10542-0
NLM
Pillat MM, Ayupe AC, Juvenal GA, Meinerz C, Glaser T, Pellegrina DV da S, Paiva DS, Mello CF, Longo BM, Reis EM, Ulrich H. Differentiated embryonic Neurospheres from familial alzheimer’s disease model show innate Immune and glial cell responses [Internet]. Stem Cell Reviews and Reports. 2023 ; 19 1800-1811.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.1007/s12015-023-10542-0
Vancouver
Pillat MM, Ayupe AC, Juvenal GA, Meinerz C, Glaser T, Pellegrina DV da S, Paiva DS, Mello CF, Longo BM, Reis EM, Ulrich H. Differentiated embryonic Neurospheres from familial alzheimer’s disease model show innate Immune and glial cell responses [Internet]. Stem Cell Reviews and Reports. 2023 ; 19 1800-1811.[citado 2024 jun. 14 ] Available from: https://dx.doi.org/10.1007/s12015-023-10542-0
Monografia/livro
Purinergic Signaling in Neurodevelopment, Neuroinflammation and Neurodegeneration (2023)
Ulrich, Henning; Illes, Peter; Glaser, Talita
Unidade: IQ
Subjects: RECEPTORES, NUCLEOTÍDEOS
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ABNT
ULRICH, Henning e ILLES, Peter e GLASER, Talita. Purinergic Signaling in Neurodevelopment, Neuroinflammation and Neurodegeneration. . Cham: Springer. . Acesso em: 14 jun. 2024. , 2023
APA
Ulrich, H., Illes, P., & Glaser, T. (2023). Purinergic Signaling in Neurodevelopment, Neuroinflammation and Neurodegeneration. Cham: Springer.
NLM
Ulrich H, Illes P, Glaser T. Purinergic Signaling in Neurodevelopment, Neuroinflammation and Neurodegeneration. 2023 ;[citado 2024 jun. 14 ]
Vancouver
Ulrich H, Illes P, Glaser T. Purinergic Signaling in Neurodevelopment, Neuroinflammation and Neurodegeneration. 2023 ;[citado 2024 jun. 14 ]
Artigo de periodico
Diferentiated embryonic neurospheres from familial Alzheimer’s Disease model show innate immune and glial cell responses (2023)
Pillat, Micheli Mainardi ; Ayupe, Ana Carolina; Juvenal, Guilherme Araújo ; Meinerz, Carine; Glaser, Talita ; Pellegrina, Diogo Vieira da Silva ; Paiva, Daisylea Souza; Mello, Carlos Fernando ; Longo, Beatriz Monteiro ; Reis, Eduardo Moraes ; Ulrich, Henning
Source: Stem Cell Reviews and Reports. Unidade: IQ
Subjects: DOENÇAS NEURODEGENERATIVAS, EXPRESSÃO GÊNICA, GENOMAS, CITOCINAS
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ABNT
PILLAT, Micheli Mainardi et al. Diferentiated embryonic neurospheres from familial Alzheimer’s Disease model show innate immune and glial cell responses. Stem Cell Reviews and Reports, v. 19, n. 6, p. 1800-1811, 2023Tradução . . Disponível em: https://doi.org/10.1007/s12015-023-10542-0. Acesso em: 14 jun. 2024.
APA
Pillat, M. M., Ayupe, A. C., Juvenal, G. A., Meinerz, C., Glaser, T., Pellegrina, D. V. da S., et al. (2023). Diferentiated embryonic neurospheres from familial Alzheimer’s Disease model show innate immune and glial cell responses. Stem Cell Reviews and Reports, 19( 6), 1800-1811. doi:10.1007/s12015-023-10542-0
NLM
Pillat MM, Ayupe AC, Juvenal GA, Meinerz C, Glaser T, Pellegrina DV da S, Paiva DS, Mello CF, Longo BM, Reis EM, Ulrich H. Diferentiated embryonic neurospheres from familial Alzheimer’s Disease model show innate immune and glial cell responses [Internet]. Stem Cell Reviews and Reports. 2023 ; 19( 6): 1800-1811.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1007/s12015-023-10542-0
Vancouver
Pillat MM, Ayupe AC, Juvenal GA, Meinerz C, Glaser T, Pellegrina DV da S, Paiva DS, Mello CF, Longo BM, Reis EM, Ulrich H. Diferentiated embryonic neurospheres from familial Alzheimer’s Disease model show innate immune and glial cell responses [Internet]. Stem Cell Reviews and Reports. 2023 ; 19( 6): 1800-1811.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1007/s12015-023-10542-0
Parte de monografia/livro
Purinergic signaling in Parkinson’s disease (2023)
Silva, Jean Bezerra ; Ferreira, Ana Flávia Fernandes ; Glaser, Talita ; Ulrich, Henning ; Britto, Luiz Roberto Giorgetti de
Source: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Unidades: IQ, ICB
Assunto: DOENÇA DE PARKINSON
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ABNT
SILVA, Jean Bezerra et al. Purinergic signaling in Parkinson’s disease. Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Tradução . Cham: Springer, 2023. . . Acesso em: 14 jun. 2024.
APA
Silva, J. B., Ferreira, A. F. F., Glaser, T., Ulrich, H., & Britto, L. R. G. de. (2023). Purinergic signaling in Parkinson’s disease. In Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer.
NLM
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Vancouver
Silva JB, Ferreira AFF, Glaser T, Ulrich H, Britto LRG de. Purinergic signaling in Parkinson’s disease. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Parte de monografia/livro
Purinergic signaling: an overview (2023)
Glaser, Talita ; Ulrich, Henning
Source: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Unidade: IQ
Subjects: PURINAS, MOLÉCULA
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ABNT
GLASER, Talita e ULRICH, Henning. Purinergic signaling: an overview. Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Tradução . Cham: Springer, 2023. . . Acesso em: 14 jun. 2024.
APA
Glaser, T., & Ulrich, H. (2023). Purinergic signaling: an overview. In Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer.
NLM
Glaser T, Ulrich H. Purinergic signaling: an overview. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Vancouver
Glaser T, Ulrich H. Purinergic signaling: an overview. In: Purinergic signaling in neurodevelopment, neuroinflammation and neurodegeneration. Cham: Springer; 2023. [citado 2024 jun. 14 ]
Artigo de periodico
Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease (2023)
Ribeiro, Deidiane Elisa ; Petiz, Lyvia Lintzmaier ; Glaser, Talita ; Giacomelli, Ágatha Oliveira ; Andrejew, Roberta ; Saab, Fernando de Azevedo Ribeiro; Milanis, Milena da Silva ; Campos, Henrique Correia; Sampaio, Vanessa Fernandes Arnaud ; La Banca, Sophia; Longo, Beatriz Monteiro ; Lameu, Claudiana ; Tang, Yong; Resende, Rodrigo Ribeiro ; Ferreira, Sergio T ; Ulrich, Henning
Source: Neuropharmacology. Unidade: IQ
Subjects: NEUROPSIQUIATRIA, DOENÇA DE ALZHEIMER, DEPRESSÃO, TRANSTORNOS DO SONO
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ABNT
RIBEIRO, Deidiane Elisa et al. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease. Neuropharmacology, v. 226, p. 1-16 art. 109371, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2022.109371. Acesso em: 14 jun. 2024.
APA
Ribeiro, D. E., Petiz, L. L., Glaser, T., Giacomelli, Á. O., Andrejew, R., Saab, F. de A. R., et al. (2023). Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease. Neuropharmacology, 226, 1-16 art. 109371. doi:10.1016/j.neuropharm.2022.109371
NLM
Ribeiro DE, Petiz LL, Glaser T, Giacomelli ÁO, Andrejew R, Saab F de AR, Milanis M da S, Campos HC, Sampaio VFA, La Banca S, Longo BM, Lameu C, Tang Y, Resende RR, Ferreira ST, Ulrich H. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease [Internet]. Neuropharmacology. 2023 ; 226 1-16 art. 109371.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.neuropharm.2022.109371
Vancouver
Ribeiro DE, Petiz LL, Glaser T, Giacomelli ÁO, Andrejew R, Saab F de AR, Milanis M da S, Campos HC, Sampaio VFA, La Banca S, Longo BM, Lameu C, Tang Y, Resende RR, Ferreira ST, Ulrich H. Purinergic signaling in cognitive impairment and neuropsychiatric symptoms of Alzheimer’s disease [Internet]. Neuropharmacology. 2023 ; 226 1-16 art. 109371.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.neuropharm.2022.109371
Artigo de periodico
Organoids as a novel tool in modelling infectious diseases (2023)
Oliveira, Lucas Felipe de; Mendes Filho, Daniel ; Marques, Bruno Lemes; Maciel, Giovana Figueiredo; Parreira, Ricardo Cambraia; Carmo Neto, José Rodrigues do ; Silva, Priscilla Elias Ferreira da; Guerra, Rhanoica Oliveira ; Silva, Marcos Vinícius da ; Santiago, Helton C ; Birbrair, Alexander ; Kihara, Alexandre Hiroaki ; Silva, Valdo José Dias da; Glaser, Talita ; Resende, Rodrigo R; Ulrich, Henning
Source: Seminars in Cell and Developmental Biology. Unidades: IQ, FMRP
Subjects: ORGANELAS CELULARES, DOENÇAS INFECCIOSAS, BIOENGENHARIA, CÉLULAS-TRONCO
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ABNT
OLIVEIRA, Lucas Felipe de et al. Organoids as a novel tool in modelling infectious diseases. Seminars in Cell and Developmental Biology, v. 144, p. 87-96, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.semcdb.2022.09.003. Acesso em: 14 jun. 2024.
APA
Oliveira, L. F. de, Mendes Filho, D., Marques, B. L., Maciel, G. F., Parreira, R. C., Carmo Neto, J. R. do, et al. (2023). Organoids as a novel tool in modelling infectious diseases. Seminars in Cell and Developmental Biology, 144, 87-96. doi:10.1016/j.semcdb.2022.09.003
NLM
Oliveira LF de, Mendes Filho D, Marques BL, Maciel GF, Parreira RC, Carmo Neto JR do, Silva PEF da, Guerra RO, Silva MV da, Santiago HC, Birbrair A, Kihara AH, Silva VJD da, Glaser T, Resende RR, Ulrich H. Organoids as a novel tool in modelling infectious diseases [Internet]. Seminars in Cell and Developmental Biology. 2023 ; 144 87-96.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.semcdb.2022.09.003
Vancouver
Oliveira LF de, Mendes Filho D, Marques BL, Maciel GF, Parreira RC, Carmo Neto JR do, Silva PEF da, Guerra RO, Silva MV da, Santiago HC, Birbrair A, Kihara AH, Silva VJD da, Glaser T, Resende RR, Ulrich H. Organoids as a novel tool in modelling infectious diseases [Internet]. Seminars in Cell and Developmental Biology. 2023 ; 144 87-96.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.semcdb.2022.09.003
Artigo de periodico
Neuroprotective effects of resistance physical exercise on the APP/PS1 mouse model of Alzheimer’s disease (2023)
Campos, Henrique Correia; Ribeiro, Deidiane Elisa; Hashiguchi, Debora; Glaser, Talita ; Milanis, Milena da Silva ; Gimenes, Christiane ; Suchecki, Deborah ; Arida, Ricardo Mario ; Ulrich, Henning ; Longo, Beatriz Monteiro
Source: Frontiers in Neuroscience. Unidade: IQ
Subjects: EXERCÍCIO FÍSICO, DOENÇA DE ALZHEIMER
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ABNT
CAMPOS, Henrique Correia et al. Neuroprotective effects of resistance physical exercise on the APP/PS1 mouse model of Alzheimer’s disease. Frontiers in Neuroscience, v. 17, p. 1-11 art. 1132825, 2023Tradução . . Disponível em: https://doi.org/10.3389/fnins.2023.1132825. Acesso em: 14 jun. 2024.
APA
Campos, H. C., Ribeiro, D. E., Hashiguchi, D., Glaser, T., Milanis, M. da S., Gimenes, C., et al. (2023). Neuroprotective effects of resistance physical exercise on the APP/PS1 mouse model of Alzheimer’s disease. Frontiers in Neuroscience, 17, 1-11 art. 1132825. doi:10.3389/fnins.2023.1132825
NLM
Campos HC, Ribeiro DE, Hashiguchi D, Glaser T, Milanis M da S, Gimenes C, Suchecki D, Arida RM, Ulrich H, Longo BM. Neuroprotective effects of resistance physical exercise on the APP/PS1 mouse model of Alzheimer’s disease [Internet]. Frontiers in Neuroscience. 2023 ; 17 1-11 art. 1132825.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fnins.2023.1132825
Vancouver
Campos HC, Ribeiro DE, Hashiguchi D, Glaser T, Milanis M da S, Gimenes C, Suchecki D, Arida RM, Ulrich H, Longo BM. Neuroprotective effects of resistance physical exercise on the APP/PS1 mouse model of Alzheimer’s disease [Internet]. Frontiers in Neuroscience. 2023 ; 17 1-11 art. 1132825.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fnins.2023.1132825
Artigo de periodico
Huntingtin protein maintains balanced energetics in mouse cardiomyocytes (2022)
Tomczyk, Marta; Glaser, Talita ; Ulrich, Henning ; Slominska, Ewa M; Smolenski, Ryszard T
Source: Nucleosides, Nucleotides & Nucleic Acids. Unidade: IQ
Subjects: NUCLEOTÍDEOS, METABOLISMO ENERGÉTICO
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ABNT
TOMCZYK, Marta et al. Huntingtin protein maintains balanced energetics in mouse cardiomyocytes. Nucleosides, Nucleotides & Nucleic Acids, v. 41, n. 3, p. 231-238, 2022Tradução . . Disponível em: https://doi.org/10.1080/15257770.2020.1815769. Acesso em: 14 jun. 2024.
APA
Tomczyk, M., Glaser, T., Ulrich, H., Slominska, E. M., & Smolenski, R. T. (2022). Huntingtin protein maintains balanced energetics in mouse cardiomyocytes. Nucleosides, Nucleotides & Nucleic Acids, 41( 3), 231-238. doi:10.1080/15257770.2020.1815769
NLM
Tomczyk M, Glaser T, Ulrich H, Slominska EM, Smolenski RT. Huntingtin protein maintains balanced energetics in mouse cardiomyocytes [Internet]. Nucleosides, Nucleotides & Nucleic Acids. 2022 ; 41( 3): 231-238.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1080/15257770.2020.1815769
Vancouver
Tomczyk M, Glaser T, Ulrich H, Slominska EM, Smolenski RT. Huntingtin protein maintains balanced energetics in mouse cardiomyocytes [Internet]. Nucleosides, Nucleotides & Nucleic Acids. 2022 ; 41( 3): 231-238.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1080/15257770.2020.1815769
Artigo de periodico
Therapeutic effects of cannabinoids and their applications in COVID-19 treatment (2022)
Pérez, Rebeca; Glaser, Talita ; Villegas, Cecilia ; Burgos, Viviana; Ulrich, Henning ; Paz, Cristian
Source: Life. Unidade: IQ
Subjects: CANABINOIDES, COVID-19
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ABNT
PÉREZ, Rebeca et al. Therapeutic effects of cannabinoids and their applications in COVID-19 treatment. Life, v. 12, n. 12, p. 1-19, 2022Tradução . . Disponível em: https://doi.org/10.3390/life12122117. Acesso em: 14 jun. 2024.
APA
Pérez, R., Glaser, T., Villegas, C., Burgos, V., Ulrich, H., & Paz, C. (2022). Therapeutic effects of cannabinoids and their applications in COVID-19 treatment. Life, 12( 12), 1-19. doi:10.3390/life12122117
NLM
Pérez R, Glaser T, Villegas C, Burgos V, Ulrich H, Paz C. Therapeutic effects of cannabinoids and their applications in COVID-19 treatment [Internet]. Life. 2022 ; 12( 12): 1-19.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3390/life12122117
Vancouver
Pérez R, Glaser T, Villegas C, Burgos V, Ulrich H, Paz C. Therapeutic effects of cannabinoids and their applications in COVID-19 treatment [Internet]. Life. 2022 ; 12( 12): 1-19.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3390/life12122117
Artigo de periodico
P2X7 receptor isoform B is a key drug resistance mediator for neuroblastoma (2022)
Sampaio, Vanessa Fernandes Arnaud ; Bento, Carolina Adriane ; Glaser, Talita ; Adinolfi, Elena ; Ulrich, Henning ; Lameu, Claudiana
Source: Frontiers in Oncology. Unidade: IQ
Subjects: RECEPTORES, NEUROBLASTOMA, RESISTÊNCIA MICROBIANA ÀS DROGAS
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ABNT
SAMPAIO, Vanessa Fernandes Arnaud et al. P2X7 receptor isoform B is a key drug resistance mediator for neuroblastoma. Frontiers in Oncology, v. 12, p. 1-19 art. 966404, 2022Tradução . . Disponível em: https://doi.org/10.3389/fonc.2022.966404. Acesso em: 14 jun. 2024.
APA
Sampaio, V. F. A., Bento, C. A., Glaser, T., Adinolfi, E., Ulrich, H., & Lameu, C. (2022). P2X7 receptor isoform B is a key drug resistance mediator for neuroblastoma. Frontiers in Oncology, 12, 1-19 art. 966404. doi:10.3389/fonc.2022.966404
NLM
Sampaio VFA, Bento CA, Glaser T, Adinolfi E, Ulrich H, Lameu C. P2X7 receptor isoform B is a key drug resistance mediator for neuroblastoma [Internet]. Frontiers in Oncology. 2022 ; 12 1-19 art. 966404.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fonc.2022.966404
Vancouver
Sampaio VFA, Bento CA, Glaser T, Adinolfi E, Ulrich H, Lameu C. P2X7 receptor isoform B is a key drug resistance mediator for neuroblastoma [Internet]. Frontiers in Oncology. 2022 ; 12 1-19 art. 966404.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fonc.2022.966404
Artigo de periodico
Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patients (2022)
Pietrobon, Anna Julia ; Andrejew, Roberta ; Custodio, Ricardo Wesley Alberca ; Oliveira, Luana de Mendonça ; Scholl, Juliete Nathali ; Teixeira, Franciane Mouradian Emidio ; Brito, Cyro Alves de ; Glaser, Talita ; Kazmierski, Julia ; Goffinet, Christine ; Turdo, Anna Claudia; Yendo, Tatiana Mina ; Aoki, Valeria; Figueiró, Fabrício; Battastini, Ana Maria ; Ulrich, Henning ; Benard, Gil ; Duarte, Alberto José da Silva; Sato, Maria Notomi
Source: Frontiers in Immunology. Unidades: FM, IQ, ICB
Subjects: COVID-19, PURINAS, ADENOSINA
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ABNT
PIETROBON, Anna Julia et al. Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patients. Frontiers in Immunology, v. 13, p. 1-12 art. 1012027, 2022Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2022.1012027. Acesso em: 14 jun. 2024.
APA
Pietrobon, A. J., Andrejew, R., Custodio, R. W. A., Oliveira, L. de M., Scholl, J. N., Teixeira, F. M. E., et al. (2022). Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patients. Frontiers in Immunology, 13, 1-12 art. 1012027. doi:10.3389/fimmu.2022.1012027
NLM
Pietrobon AJ, Andrejew R, Custodio RWA, Oliveira L de M, Scholl JN, Teixeira FME, Brito CA de, Glaser T, Kazmierski J, Goffinet C, Turdo AC, Yendo TM, Aoki V, Figueiró F, Battastini AM, Ulrich H, Benard G, Duarte AJ da S, Sato MN. Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patients [Internet]. Frontiers in Immunology. 2022 ; 13 1-12 art. 1012027.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fimmu.2022.1012027
Vancouver
Pietrobon AJ, Andrejew R, Custodio RWA, Oliveira L de M, Scholl JN, Teixeira FME, Brito CA de, Glaser T, Kazmierski J, Goffinet C, Turdo AC, Yendo TM, Aoki V, Figueiró F, Battastini AM, Ulrich H, Benard G, Duarte AJ da S, Sato MN. Dysfunctional purinergic signaling correlates with disease severity in COVID-19 patients [Internet]. Frontiers in Immunology. 2022 ; 13 1-12 art. 1012027.[citado 2024 jun. 14 ] Available from: https://doi.org/10.3389/fimmu.2022.1012027
Artigo de periodico
Ginkgolides and Huperzine A for complementary treatment of Alzheimer's disease (2022)
Villegas, Cecilia ; Perez, Rebeca; Petiz, Lyvia Lintzmaier ; Glaser, Talita ; Ulrich, Henning ; Paz, Cristian
Source: IUBMB Life. Unidade: IQ
Subjects: DOENÇA DE ALZHEIMER, TERAPIAS COMPLEMENTARES
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ABNT
VILLEGAS, Cecilia et al. Ginkgolides and Huperzine A for complementary treatment of Alzheimer's disease. IUBMB Life, v. 74, n. 8, p. 763-779, 2022Tradução . . Disponível em: https://doi.org/10.1002/iub.2613. Acesso em: 14 jun. 2024.
APA
Villegas, C., Perez, R., Petiz, L. L., Glaser, T., Ulrich, H., & Paz, C. (2022). Ginkgolides and Huperzine A for complementary treatment of Alzheimer's disease. IUBMB Life, 74( 8), 763-779. doi:10.1002/iub.2613
NLM
Villegas C, Perez R, Petiz LL, Glaser T, Ulrich H, Paz C. Ginkgolides and Huperzine A for complementary treatment of Alzheimer's disease [Internet]. IUBMB Life. 2022 ; 74( 8): 763-779.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1002/iub.2613
Vancouver
Villegas C, Perez R, Petiz LL, Glaser T, Ulrich H, Paz C. Ginkgolides and Huperzine A for complementary treatment of Alzheimer's disease [Internet]. IUBMB Life. 2022 ; 74( 8): 763-779.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1002/iub.2613
Artigo de periodico
Rare CACNA1H and RELN variants interact through mTORC1 pathway in oligogenic autism spectrum disorder (2022)
Silva, André Luíz Teles e ; Glaser, Talita ; Oliveira, Karina Griesi ; Velloso, Juliana da Cruz Corrêa ; Wang, Jaqueline Yu Ting; Campos, Gabriele da Silva ; Ulrich, Henning ; Balan, Andrea ; Zarrei, Mehdi; Higginbotham, Edward J.; Scherer, Stephen W. ; Passos-Bueno, Maria Rita ; Sertié, Andrea Laurato
Source: Translational Psychiatry. Unidades: IQ, ICB, IB
Subjects: MICROBIOLOGIA, FÁRMACOS IMUNOSSUPRESSORES, TRANSTORNO DO ESPECTRO AUTISTA, CANAIS DE CÁLCIO, DISTÚRBIOS PSICOLÓGICOS, HERANÇA GENÉTICA, VARIAÇÃO GENÉTICA
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ABNT
SILVA, André Luíz Teles e et al. Rare CACNA1H and RELN variants interact through mTORC1 pathway in oligogenic autism spectrum disorder. Translational Psychiatry, v. 12, p. 1-11, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41398-022-01997-9. Acesso em: 14 jun. 2024.
APA
Silva, A. L. T. e, Glaser, T., Oliveira, K. G., Velloso, J. da C. C., Wang, J. Y. T., Campos, G. da S., et al. (2022). Rare CACNA1H and RELN variants interact through mTORC1 pathway in oligogenic autism spectrum disorder. Translational Psychiatry, 12, 1-11. doi:10.1038/s41398-022-01997-9
NLM
Silva ALT e, Glaser T, Oliveira KG, Velloso J da CC, Wang JYT, Campos G da S, Ulrich H, Balan A, Zarrei M, Higginbotham EJ, Scherer SW, Passos-Bueno MR, Sertié AL. Rare CACNA1H and RELN variants interact through mTORC1 pathway in oligogenic autism spectrum disorder [Internet]. Translational Psychiatry. 2022 ; 12 1-11.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1038/s41398-022-01997-9
Vancouver
Silva ALT e, Glaser T, Oliveira KG, Velloso J da CC, Wang JYT, Campos G da S, Ulrich H, Balan A, Zarrei M, Higginbotham EJ, Scherer SW, Passos-Bueno MR, Sertié AL. Rare CACNA1H and RELN variants interact through mTORC1 pathway in oligogenic autism spectrum disorder [Internet]. Translational Psychiatry. 2022 ; 12 1-11.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1038/s41398-022-01997-9
Artigo de periodico
Fmr1 exon 14 skipping in late embryonic development of the rat forebrain (2022)
Velloso, Juliana da Cruz Corrêa ; Linardi, Alessandra M.; Glaser, Talita ; Velloso, Fernando Janczur ; Rivas, Maria Prates ; Leite, Renata Elaine Paraizo; Grinberg, Lea Tenenholz ; Ulrich, Henning ; Akins, Michael R. ; Chiavegatto, Silvana ; Haddad, Luciana Amaral
Source: BMC Neuroscience. Unidades: FM, IQ, ICB, IB
Subjects: FARMACOLOGIA, PROSENCÉFALO, SISTEMA NERVOSO CENTRAL, VENTRÍCULOS CEREBRAIS, EMBRIÃO DE ANIMAL, RNA MENSAGEIRO, RATOS, EXONS
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ABNT
VELLOSO, Juliana da Cruz Corrêa et al. Fmr1 exon 14 skipping in late embryonic development of the rat forebrain. BMC Neuroscience, v. 23, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1186/s12868-022-00711-1. Acesso em: 14 jun. 2024.
APA
Velloso, J. da C. C., Linardi, A. M., Glaser, T., Velloso, F. J., Rivas, M. P., Leite, R. E. P., et al. (2022). Fmr1 exon 14 skipping in late embryonic development of the rat forebrain. BMC Neuroscience, 23, 1-15. doi:10.1186/s12868-022-00711-1
NLM
Velloso J da CC, Linardi AM, Glaser T, Velloso FJ, Rivas MP, Leite REP, Grinberg LT, Ulrich H, Akins MR, Chiavegatto S, Haddad LA. Fmr1 exon 14 skipping in late embryonic development of the rat forebrain [Internet]. BMC Neuroscience. 2022 ; 23 1-15.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s12868-022-00711-1
Vancouver
Velloso J da CC, Linardi AM, Glaser T, Velloso FJ, Rivas MP, Leite REP, Grinberg LT, Ulrich H, Akins MR, Chiavegatto S, Haddad LA. Fmr1 exon 14 skipping in late embryonic development of the rat forebrain [Internet]. BMC Neuroscience. 2022 ; 23 1-15.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1186/s12868-022-00711-1
Artigo de periodico
Complex diseases demand novel treatment strategies: understanding drug combination (2022)
Mello, Renata Siqueira de ; Sampaio, Vanessa Fernandes Arnaud ; Maciel, Lucas Ferreira; Sá, Vanessa de ; Glaser, Talita ; Ulrich, Henning ; Lameu, Claudiana
Source: Drug Combination Therapy. Unidade: IQ
Subjects: MEDICAMENTO, TERAPIA COMBINADA
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ABNT
MELLO, Renata Siqueira de et al. Complex diseases demand novel treatment strategies: understanding drug combination. Drug Combination Therapy, v. 4, n. 2, p. 1-12 art. 6, 2022Tradução . . Disponível em: https://doi.org/10.53388/DCT2022006. Acesso em: 14 jun. 2024.
APA
Mello, R. S. de, Sampaio, V. F. A., Maciel, L. F., Sá, V. de, Glaser, T., Ulrich, H., & Lameu, C. (2022). Complex diseases demand novel treatment strategies: understanding drug combination. Drug Combination Therapy, 4( 2), 1-12 art. 6. doi:10.53388/DCT2022006
NLM
Mello RS de, Sampaio VFA, Maciel LF, Sá V de, Glaser T, Ulrich H, Lameu C. Complex diseases demand novel treatment strategies: understanding drug combination [Internet]. Drug Combination Therapy. 2022 ; 4( 2): 1-12 art. 6.[citado 2024 jun. 14 ] Available from: https://doi.org/10.53388/DCT2022006
Vancouver
Mello RS de, Sampaio VFA, Maciel LF, Sá V de, Glaser T, Ulrich H, Lameu C. Complex diseases demand novel treatment strategies: understanding drug combination [Internet]. Drug Combination Therapy. 2022 ; 4( 2): 1-12 art. 6.[citado 2024 jun. 14 ] Available from: https://doi.org/10.53388/DCT2022006
Artigo de periodico
P2Y2 receptor activation promotes esophageal cancer cells proliferation via ERK1/2 pathway (2021)
Zaparte, Aline ; Cappellari, Angélica Regina; Brandão, Caroline A; Souza, Julia Bazana de; Borges, Thiago J; Kist, Luiza Wilges; Bogo, Maurício R; Zerbini, Luiz F; Pinto, Luis Felipe Ribeiro; Glaser, Talita ; Gonçalves, Maria Carolina Bittencourt; Naaldijk, Yahaira; Ulrich, Henning ; Morrone, Fernanda B
Source: European Journal of Pharmacology. Unidade: IQ
Subjects: NEOPLASIAS ESOFÁGICAS, NUCLEOTÍDEOS, PROLIFERAÇÃO CELULAR, RECEPTORES
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ABNT
ZAPARTE, Aline et al. P2Y2 receptor activation promotes esophageal cancer cells proliferation via ERK1/2 pathway. European Journal of Pharmacology, v. 891, p. 1-12 art. 173687, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2020.173687. Acesso em: 14 jun. 2024.
APA
Zaparte, A., Cappellari, A. R., Brandão, C. A., Souza, J. B. de, Borges, T. J., Kist, L. W., et al. (2021). P2Y2 receptor activation promotes esophageal cancer cells proliferation via ERK1/2 pathway. European Journal of Pharmacology, 891, 1-12 art. 173687. doi:10.1016/j.ejphar.2020.173687
NLM
Zaparte A, Cappellari AR, Brandão CA, Souza JB de, Borges TJ, Kist LW, Bogo MR, Zerbini LF, Pinto LFR, Glaser T, Gonçalves MCB, Naaldijk Y, Ulrich H, Morrone FB. P2Y2 receptor activation promotes esophageal cancer cells proliferation via ERK1/2 pathway [Internet]. European Journal of Pharmacology. 2021 ; 891 1-12 art. 173687.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2020.173687
Vancouver
Zaparte A, Cappellari AR, Brandão CA, Souza JB de, Borges TJ, Kist LW, Bogo MR, Zerbini LF, Pinto LFR, Glaser T, Gonçalves MCB, Naaldijk Y, Ulrich H, Morrone FB. P2Y2 receptor activation promotes esophageal cancer cells proliferation via ERK1/2 pathway [Internet]. European Journal of Pharmacology. 2021 ; 891 1-12 art. 173687.[citado 2024 jun. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2020.173687

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