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Artigo de periodico
Dual SGLT1/SGLT2 Inhibitor Phlorizin Ameliorates Non-Alcoholic Fatty Liver Disease and Hepatic Glucose Production in Type 2 Diabetic Mice (2020)
David-Silva, Aline; Esteves, Joao Victor; Morais, Mychel Raony P. T; Freitas, Helayne Soares; Zorn, Telma Maria; Giannella, Maria Lucia Cardillo Correa ; Machado, Ubiratan Fabres
Fonte: Diabetes metabolic syndrome and obesity-targets and therapy. Unidade: FM
Assuntos: DIABETES MELLITUS, GLICOSÍDEOS, RATOS, FÍGADO GORDUROSO
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ABNT
DAVID-SILVA, Aline et al. Dual SGLT1/SGLT2 Inhibitor Phlorizin Ameliorates Non-Alcoholic Fatty Liver Disease and Hepatic Glucose Production in Type 2 Diabetic Mice. Diabetes metabolic syndrome and obesity-targets and therapy, v. 13, p. 739-751, 2020Tradução . . Disponível em: https://doi.org/10.2147/DMSO.S242282. Acesso em: 23 jul. 2024.
APA
David-Silva, A., Esteves, J. V., Morais, M. R. P. T., Freitas, H. S., Zorn, T. M., Giannella, M. L. C. C., & Machado, U. F. (2020). Dual SGLT1/SGLT2 Inhibitor Phlorizin Ameliorates Non-Alcoholic Fatty Liver Disease and Hepatic Glucose Production in Type 2 Diabetic Mice. Diabetes metabolic syndrome and obesity-targets and therapy, 13, 739-751. doi:10.2147/DMSO.S242282
NLM
David-Silva A, Esteves JV, Morais MRPT, Freitas HS, Zorn TM, Giannella MLCC, Machado UF. Dual SGLT1/SGLT2 Inhibitor Phlorizin Ameliorates Non-Alcoholic Fatty Liver Disease and Hepatic Glucose Production in Type 2 Diabetic Mice [Internet]. Diabetes metabolic syndrome and obesity-targets and therapy. 2020 ; 13 739-751.[citado 2024 jul. 23 ] Available from: https://doi.org/10.2147/DMSO.S242282
Vancouver
David-Silva A, Esteves JV, Morais MRPT, Freitas HS, Zorn TM, Giannella MLCC, Machado UF. Dual SGLT1/SGLT2 Inhibitor Phlorizin Ameliorates Non-Alcoholic Fatty Liver Disease and Hepatic Glucose Production in Type 2 Diabetic Mice [Internet]. Diabetes metabolic syndrome and obesity-targets and therapy. 2020 ; 13 739-751.[citado 2024 jul. 23 ] Available from: https://doi.org/10.2147/DMSO.S242282
Artigo de periodico
Intensive insulin treatment induces insulin resistance in diabetic rats by impairing glucose metabolism-related mechanisms in muscle and liver (2011)
Okamoto, Maristela Mitiko; Anhê, Gabriel Forato; Sabino-Silva, Robinson; Marques, Milano Felipe dos Santos Ferreira; Freitas, Helayne Soares; Mori, Rosana Cristina Tieko; Melo, Karla Fabiana S.; Machado, Ubiratan Fabres
Fonte: The Journal of Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
OKAMOTO, Maristela Mitiko et al. Intensive insulin treatment induces insulin resistance in diabetic rats by impairing glucose metabolism-related mechanisms in muscle and liver. The Journal of Endocrinology, v. 211, n. 1, p. 55-64, 2011Tradução . . Disponível em: https://doi.org/10.1530/JOE-11-0105. Acesso em: 23 jul. 2024.
APA
Okamoto, M. M., Anhê, G. F., Sabino-Silva, R., Marques, M. F. dos S. F., Freitas, H. S., Mori, R. C. T., et al. (2011). Intensive insulin treatment induces insulin resistance in diabetic rats by impairing glucose metabolism-related mechanisms in muscle and liver. The Journal of Endocrinology, 211( 1), 55-64. doi:10.1530/JOE-11-0105
NLM
Okamoto MM, Anhê GF, Sabino-Silva R, Marques MF dos SF, Freitas HS, Mori RCT, Melo KFS, Machado UF. Intensive insulin treatment induces insulin resistance in diabetic rats by impairing glucose metabolism-related mechanisms in muscle and liver [Internet]. The Journal of Endocrinology. 2011 ; 211( 1): 55-64.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1530/JOE-11-0105
Vancouver
Okamoto MM, Anhê GF, Sabino-Silva R, Marques MF dos SF, Freitas HS, Mori RCT, Melo KFS, Machado UF. Intensive insulin treatment induces insulin resistance in diabetic rats by impairing glucose metabolism-related mechanisms in muscle and liver [Internet]. The Journal of Endocrinology. 2011 ; 211( 1): 55-64.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1530/JOE-11-0105
Artigo de periodico
SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats (2010)
Silva, Robinson Sabino da; Wagner, Ana Bárbara Teixeira; Burgi, Katia; Okamoto, Maristela Mitiko; Alves, Adilson da Silva; Lima, Guilherme Alves de; Freitas, Helayne Soares; Antunes, Vagner Roberto ; Machado, Ubiratan Fabres
Fonte: American Journal of Physiology: Endocrinology and Metabolism. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SILVA, Robinson Sabino da et al. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats. American Journal of Physiology: Endocrinology and Metabolism, v. 299, n. 6, p. E1028-E1037, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00395.2010. Acesso em: 23 jul. 2024.
APA
Silva, R. S. da, Wagner, A. B. T., Burgi, K., Okamoto, M. M., Alves, A. da S., Lima, G. A. de, et al. (2010). SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats. American Journal of Physiology: Endocrinology and Metabolism, 299( 6), E1028-E1037. doi:10.1152/ajpendo.00395.2010
NLM
Silva RS da, Wagner ABT, Burgi K, Okamoto MM, Alves A da S, Lima GA de, Freitas HS, Antunes VR, Machado UF. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1028-E1037.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1152/ajpendo.00395.2010
Vancouver
Silva RS da, Wagner ABT, Burgi K, Okamoto MM, Alves A da S, Lima GA de, Freitas HS, Antunes VR, Machado UF. SGLT1 protein expression in plasma membrane of acinar cells correlates with the sympathetic outflow to salivary glands in diabetic and hypertensive rats [Internet]. American Journal of Physiology: Endocrinology and Metabolism. 2010 ; 299( 6): E1028-E1037.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1152/ajpendo.00395.2010
Trabalho de evento-resumo periodico
Does sympathetic outflow play any role in the Na + /glucose co-transporter SGLT1 in the salivary gland of diabetic hypertensive rats? (2009)
Sabino-Silva, Robinson; Alves-Wagner, Ana Barbara T.; Burgi, Katia; Okamoto, Maristela Mitiko; Alves, Adilson Silva; Freitas, Helayne Soares; Antunes, Vagner Roberto ; Machado, Ubiratan Fabres
Fonte: Journal of Physiological Sciences. Nome do evento: Proceedings of the International Congress of Physiological Sciences (IUPS 2009) - Function of Life: Elements and Integration. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SABINO-SILVA, Robinson et al. Does sympathetic outflow play any role in the Na + /glucose co-transporter SGLT1 in the salivary gland of diabetic hypertensive rats?. Journal of Physiological Sciences. Tokyo: Instituto de Ciências Biomédicas, Universidade de São Paulo. . Acesso em: 23 jul. 2024. , 2009
APA
Sabino-Silva, R., Alves-Wagner, A. B. T., Burgi, K., Okamoto, M. M., Alves, A. S., Freitas, H. S., et al. (2009). Does sympathetic outflow play any role in the Na + /glucose co-transporter SGLT1 in the salivary gland of diabetic hypertensive rats? Journal of Physiological Sciences. Tokyo: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
Sabino-Silva R, Alves-Wagner ABT, Burgi K, Okamoto MM, Alves AS, Freitas HS, Antunes VR, Machado UF. Does sympathetic outflow play any role in the Na + /glucose co-transporter SGLT1 in the salivary gland of diabetic hypertensive rats? Journal of Physiological Sciences. 2009 ; 59[citado 2024 jul. 23 ]
Vancouver
Sabino-Silva R, Alves-Wagner ABT, Burgi K, Okamoto MM, Alves AS, Freitas HS, Antunes VR, Machado UF. Does sympathetic outflow play any role in the Na + /glucose co-transporter SGLT1 in the salivary gland of diabetic hypertensive rats? Journal of Physiological Sciences. 2009 ; 59[citado 2024 jul. 23 ]
Trabalho de evento
Regulação dos genes do GLUT2 pelo PPAR-2 em rim de animais diabéticos e diabétivos tratados (2008)
David-Silva, Aline; Freitas, Helayne Soares; Sabino-Silva, Robinson; Okamoto, Maristela Mitiko; Machado, Ubiratan Fabres
Fonte: Programa. Nome do evento: Reunião Anual da Federação de Sociedades de Biologia Experimental - FeSBE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
DAVID-SILVA, Aline et al. Regulação dos genes do GLUT2 pelo PPAR-2 em rim de animais diabéticos e diabétivos tratados. 2008, Anais.. Águas de Lindóia: Instituto de Ciências Biomédicas, Universidade de São Paulo, 2008. . Acesso em: 23 jul. 2024.
APA
David-Silva, A., Freitas, H. S., Sabino-Silva, R., Okamoto, M. M., & Machado, U. F. (2008). Regulação dos genes do GLUT2 pelo PPAR-2 em rim de animais diabéticos e diabétivos tratados. In Programa. Águas de Lindóia: Instituto de Ciências Biomédicas, Universidade de São Paulo.
NLM
David-Silva A, Freitas HS, Sabino-Silva R, Okamoto MM, Machado UF. Regulação dos genes do GLUT2 pelo PPAR-2 em rim de animais diabéticos e diabétivos tratados. Programa. 2008 ;[citado 2024 jul. 23 ]
Vancouver
David-Silva A, Freitas HS, Sabino-Silva R, Okamoto MM, Machado UF. Regulação dos genes do GLUT2 pelo PPAR-2 em rim de animais diabéticos e diabétivos tratados. Programa. 2008 ;[citado 2024 jul. 23 ]
Artigo de periodico
Insulin but not phlorizin treatment induces a transient increase in GLUT2 gene expression in the kidney of diabetic rats (2007)
Freitas, Helayne Soares; Schaan, Beatriz D'Agord; Silva, Robinson Sabino da; Okamoto, Maristela Mitiko; Oliveira-Souza, Maria; Machado, Ubiratan Fabres
Fonte: Nephron Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FREITAS, Helayne Soares et al. Insulin but not phlorizin treatment induces a transient increase in GLUT2 gene expression in the kidney of diabetic rats. Nephron Physiology, v. 105, n. 3, p. 42-51, 2007Tradução . . Acesso em: 23 jul. 2024.
APA
Freitas, H. S., Schaan, B. D. 'A., Silva, R. S. da, Okamoto, M. M., Oliveira-Souza, M., & Machado, U. F. (2007). Insulin but not phlorizin treatment induces a transient increase in GLUT2 gene expression in the kidney of diabetic rats. Nephron Physiology, 105( 3), 42-51.
NLM
Freitas HS, Schaan BD'A, Silva RS da, Okamoto MM, Oliveira-Souza M, Machado UF. Insulin but not phlorizin treatment induces a transient increase in GLUT2 gene expression in the kidney of diabetic rats. Nephron Physiology. 2007 ; 105( 3): 42-51.[citado 2024 jul. 23 ]
Vancouver
Freitas HS, Schaan BD'A, Silva RS da, Okamoto MM, Oliveira-Souza M, Machado UF. Insulin but not phlorizin treatment induces a transient increase in GLUT2 gene expression in the kidney of diabetic rats. Nephron Physiology. 2007 ; 105( 3): 42-51.[citado 2024 jul. 23 ]
Artigo de periodico
Acute and short-term insulin-induced molecular adaptations of GLUT2 gene expression in the renal cortex of diabetic rats (2005)
Freitas, Helayne Soares; Schaan, Beatriz D'Agord; Seraphim, Patricia Monteiro; Nunes, Maria Tereza ; Machado, Ubiratan Fabres
Fonte: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FREITAS, Helayne Soares et al. Acute and short-term insulin-induced molecular adaptations of GLUT2 gene expression in the renal cortex of diabetic rats. Molecular and Cellular Endocrinology, v. 237, p. 49-57, 2005Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2005.03.005. Acesso em: 23 jul. 2024.
APA
Freitas, H. S., Schaan, B. D. 'A., Seraphim, P. M., Nunes, M. T., & Machado, U. F. (2005). Acute and short-term insulin-induced molecular adaptations of GLUT2 gene expression in the renal cortex of diabetic rats. Molecular and Cellular Endocrinology, 237, 49-57. doi:10.1016/j.mce.2005.03.005
NLM
Freitas HS, Schaan BD'A, Seraphim PM, Nunes MT, Machado UF. Acute and short-term insulin-induced molecular adaptations of GLUT2 gene expression in the renal cortex of diabetic rats [Internet]. Molecular and Cellular Endocrinology. 2005 ; 237 49-57.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.mce.2005.03.005
Vancouver
Freitas HS, Schaan BD'A, Seraphim PM, Nunes MT, Machado UF. Acute and short-term insulin-induced molecular adaptations of GLUT2 gene expression in the renal cortex of diabetic rats [Internet]. Molecular and Cellular Endocrinology. 2005 ; 237 49-57.[citado 2024 jul. 23 ] Available from: https://doi.org/10.1016/j.mce.2005.03.005

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