Fonte: Molecular Endocrinology. Unidade: FM
Assunto: MEDICINA INTERNA
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ABNT
MARTENS, John W. M et al. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology, v. 12, n. 6, p. 775-784, 1998Tradução . . Acesso em: 19 out. 2024.APA
Martens, J. W. M., Post, M. V., Abelin, N. M. A., Ezabella, M., Toledo, S. P. A., Brunner, H. G., & Themmen, A. P. N. (1998). A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology, 12( 6), 775-784.NLM
Martens JWM, Post MV, Abelin NMA, Ezabella M, Toledo SPA, Brunner HG, Themmen APN. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology. 1998 ; 12( 6): 775-784.[citado 2024 out. 19 ]Vancouver
Martens JWM, Post MV, Abelin NMA, Ezabella M, Toledo SPA, Brunner HG, Themmen APN. A homozygous mutation in the luteinizing hormone receptor causes partial leydig cell hypoplasia: correlation between receptor activity and phenotype. Molecular Endocrinology. 1998 ; 12( 6): 775-784.[citado 2024 out. 19 ]