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Artigo de periodico
DNAJB12 and DNJB14 are non-redundant Hsp40 redox chaperones involved in endoplasmic reticulum protein reflux (2024)
Purificaca, Aline Dias da; Debbas, Victor ; Tanaka, Leonardo Yuji; Gabriel, Gabriele Veronica de Mello; Wosniak Junior, Joao; Bessa, Tiphany Coralie De; Garcia-Rosa, Sheila; Laurindo, Francisco Rafael Martins; Oliveira, Percillia Victoria Santos
Fonte: Biochimica et biophysica acta-general subjects. Unidade: FM
Assuntos: RETÍCULO ENDOPLASMÁTICO, PROTEÍNAS DO CHOQUE TÉRMICO, EXPRESSÃO GÊNICA
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PURIFICACA, Aline Dias da et al. DNAJB12 and DNJB14 are non-redundant Hsp40 redox chaperones involved in endoplasmic reticulum protein reflux. Biochimica et biophysica acta-general subjects, v. 1868, n. 1, 2024Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/57759. Acesso em: 29 jun. 2025.
APA
Purificaca, A. D. da, Debbas, V., Tanaka, L. Y., Gabriel, G. V. de M., Wosniak Junior, J., Bessa, T. C. D., et al. (2024). DNAJB12 and DNJB14 are non-redundant Hsp40 redox chaperones involved in endoplasmic reticulum protein reflux. Biochimica et biophysica acta-general subjects, 1868( 1). doi:10.1016/j.bbagen.2023.130502
NLM
Purificaca AD da, Debbas V, Tanaka LY, Gabriel GV de M, Wosniak Junior J, Bessa TCD, Garcia-Rosa S, Laurindo FRM, Oliveira PVS. DNAJB12 and DNJB14 are non-redundant Hsp40 redox chaperones involved in endoplasmic reticulum protein reflux [Internet]. Biochimica et biophysica acta-general subjects. 2024 ; 1868( 1):[citado 2025 jun. 29 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57759
Vancouver
Purificaca AD da, Debbas V, Tanaka LY, Gabriel GV de M, Wosniak Junior J, Bessa TCD, Garcia-Rosa S, Laurindo FRM, Oliveira PVS. DNAJB12 and DNJB14 are non-redundant Hsp40 redox chaperones involved in endoplasmic reticulum protein reflux [Internet]. Biochimica et biophysica acta-general subjects. 2024 ; 1868( 1):[citado 2025 jun. 29 ] Available from: https://observatorio.fm.usp.br/handle/OPI/57759
Artigo de periodico
Protein disulfide isomerase plasma levels in healthy humans reveal proteomic signatures involved in contrasting endothelial phenotypes (2019)
Oliveira, Percillia Victoria Santos de; Garcia-rosa, Sheila; Sachetto, Ana Teresa Azevedo; Moretti, Ana Iochabel Soares; Debbas, Victor; Bessa, Tiphany Coralie De; Silva, Nathalia Tenguan; Pereira, Alexandre da Costa; Martins-de-souza, Daniel; Santoro, Marcelo Larami
Fonte: Redox biology. Unidade: FM
Assuntos: PROTEÔMICA, CÉLULAS ENDOTELIAIS, ISOENZIMAS, ENZIMAS OXIRREDUTORAS, PROTEÍNAS SANGUÍNEAS
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OLIVEIRA, Percillia Victoria Santos de et al. Protein disulfide isomerase plasma levels in healthy humans reveal proteomic signatures involved in contrasting endothelial phenotypes. Redox biology, v. 22, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2019.101142. Acesso em: 29 jun. 2025.
APA
Oliveira, P. V. S. de, Garcia-rosa, S., Sachetto, A. T. A., Moretti, A. I. S., Debbas, V., Bessa, T. C. D., et al. (2019). Protein disulfide isomerase plasma levels in healthy humans reveal proteomic signatures involved in contrasting endothelial phenotypes. Redox biology, 22. doi:10.1016/j.redox.2019.101142
NLM
Oliveira PVS de, Garcia-rosa S, Sachetto ATA, Moretti AIS, Debbas V, Bessa TCD, Silva NT, Pereira A da C, Martins-de-souza D, Santoro ML. Protein disulfide isomerase plasma levels in healthy humans reveal proteomic signatures involved in contrasting endothelial phenotypes [Internet]. Redox biology. 2019 ; 22[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.redox.2019.101142
Vancouver
Oliveira PVS de, Garcia-rosa S, Sachetto ATA, Moretti AIS, Debbas V, Bessa TCD, Silva NT, Pereira A da C, Martins-de-souza D, Santoro ML. Protein disulfide isomerase plasma levels in healthy humans reveal proteomic signatures involved in contrasting endothelial phenotypes [Internet]. Redox biology. 2019 ; 22[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.redox.2019.101142
Artigo de periodico
Syzygium cumini leaf extract reverts hypertriglyceridemia via downregulation of the hepatic XBP-1s/PDI/MTP axis in monosodium L-glutamate-induced obese rats (2019)
Franca, Lucas Martins; Coelho, Caio Fernando Ferreira; Freitas, Larissa Nara Costa; Souza, Ivana Leticia Santos; Chagas, Vinicyus Teles; Debbas, Victor; Lima, Thais Martins de; Souza, Heraldo Possolo de; Laurindo, Francisco Rafael Martins; Paes, Antonio Marcus de Andrade
Fonte: Oxidative medicine and cellular longevity. Unidade: FM
Assuntos: DOENÇAS METABÓLICAS, EXTRATOS (FORMAS FARMACÊUTICAS), HIPERTRIGLICERIDEMIA, RATOS, GLUTAMATOS
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FRANCA, Lucas Martins et al. Syzygium cumini leaf extract reverts hypertriglyceridemia via downregulation of the hepatic XBP-1s/PDI/MTP axis in monosodium L-glutamate-induced obese rats. Oxidative medicine and cellular longevity, 2019Tradução . . Disponível em: https://doi.org/10.1155/2019/9417498. Acesso em: 29 jun. 2025.
APA
Franca, L. M., Coelho, C. F. F., Freitas, L. N. C., Souza, I. L. S., Chagas, V. T., Debbas, V., et al. (2019). Syzygium cumini leaf extract reverts hypertriglyceridemia via downregulation of the hepatic XBP-1s/PDI/MTP axis in monosodium L-glutamate-induced obese rats. Oxidative medicine and cellular longevity. doi:10.1155/2019/9417498
NLM
Franca LM, Coelho CFF, Freitas LNC, Souza ILS, Chagas VT, Debbas V, Lima TM de, Souza HP de, Laurindo FRM, Paes AM de A. Syzygium cumini leaf extract reverts hypertriglyceridemia via downregulation of the hepatic XBP-1s/PDI/MTP axis in monosodium L-glutamate-induced obese rats [Internet]. Oxidative medicine and cellular longevity. 2019 ;[citado 2025 jun. 29 ] Available from: https://doi.org/10.1155/2019/9417498
Vancouver
Franca LM, Coelho CFF, Freitas LNC, Souza ILS, Chagas VT, Debbas V, Lima TM de, Souza HP de, Laurindo FRM, Paes AM de A. Syzygium cumini leaf extract reverts hypertriglyceridemia via downregulation of the hepatic XBP-1s/PDI/MTP axis in monosodium L-glutamate-induced obese rats [Internet]. Oxidative medicine and cellular longevity. 2019 ;[citado 2025 jun. 29 ] Available from: https://doi.org/10.1155/2019/9417498
Artigo de periodico
Chemotherapy acutely impairs neurovascular and hemodynamic responses in women with breast cancer (2019)
Sales, Allan Robson Kluser; Negrão, Marcelo Vailati; Testa, Laura; Ferreira-Santos, Larissa; Groehs, Raphaela Villar Ramalho; Carvalho, Bruna; Toschi-Dias, Edgar ; Rocha, Natalia Galito; Laurindo, Francisco Rafael Martins; Debbas, Victor; Rondon, Maria Urbana Pinto Brandão ; Mano, Max Sena; Hajjar, Ludhmila Abrahão ; Hoff, Paulo Marcelo Gehm ; Kalil Filho, Roberto ; Negrão, Carlos Eduardo
Fonte: American journal of physiology. Heart and circulatory physiology. Unidades: FM, EEFE
Assuntos: NEOPLASIAS MAMÁRIAS, FLUXO SANGUÍNEO REGIONAL, QUIMIOTERAPIA, SISTEMA NERVOSO SIMPÁTICO
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SALES, Allan Robson Kluser et al. Chemotherapy acutely impairs neurovascular and hemodynamic responses in women with breast cancer. American journal of physiology. Heart and circulatory physiology, v. 317, n. 7, p. H1-H12, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00756.2018. Acesso em: 29 jun. 2025.
APA
Sales, A. R. K., Negrão, M. V., Testa, L., Ferreira-Santos, L., Groehs, R. V. R., Carvalho, B., et al. (2019). Chemotherapy acutely impairs neurovascular and hemodynamic responses in women with breast cancer. American journal of physiology. Heart and circulatory physiology, 317( 7), H1-H12. doi:10.1152/ajpheart.00756.2018
NLM
Sales ARK, Negrão MV, Testa L, Ferreira-Santos L, Groehs RVR, Carvalho B, Toschi-Dias E, Rocha NG, Laurindo FRM, Debbas V, Rondon MUPB, Mano MS, Hajjar LA, Hoff PMG, Kalil Filho R, Negrão CE. Chemotherapy acutely impairs neurovascular and hemodynamic responses in women with breast cancer [Internet]. American journal of physiology. Heart and circulatory physiology. 2019 ; 317( 7): H1-H12.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1152/ajpheart.00756.2018
Vancouver
Sales ARK, Negrão MV, Testa L, Ferreira-Santos L, Groehs RVR, Carvalho B, Toschi-Dias E, Rocha NG, Laurindo FRM, Debbas V, Rondon MUPB, Mano MS, Hajjar LA, Hoff PMG, Kalil Filho R, Negrão CE. Chemotherapy acutely impairs neurovascular and hemodynamic responses in women with breast cancer [Internet]. American journal of physiology. Heart and circulatory physiology. 2019 ; 317( 7): H1-H12.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1152/ajpheart.00756.2018
Artigo de periodico
Effect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model (2018)
Guido, Maria C.; Debbas, Victor
Fonte: Oxidative medicine and cellular longevity. Unidade: FM
Assuntos: ANEURISMA AÓRTICO, ANTIOXIDANTES, RATOS
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GUIDO, Maria C. e DEBBAS, Victor. Effect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model. Oxidative medicine and cellular longevity, 2018Tradução . . Disponível em: https://doi.org/10.1155/2018/3967213. Acesso em: 29 jun. 2025.
APA
Guido, M. C., & Debbas, V. (2018). Effect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model. Oxidative medicine and cellular longevity. doi:10.1155/2018/3967213
NLM
Guido MC, Debbas V. Effect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model [Internet]. Oxidative medicine and cellular longevity. 2018 ;[citado 2025 jun. 29 ] Available from: https://doi.org/10.1155/2018/3967213
Vancouver
Guido MC, Debbas V. Effect of the Antioxidant Lipoic Acid in Aortic Phenotype in a Marfan Syndrome Mouse Model [Internet]. Oxidative medicine and cellular longevity. 2018 ;[citado 2025 jun. 29 ] Available from: https://doi.org/10.1155/2018/3967213
Artigo de periodico
Long-term exposure to high-sucrose diet down-regulates hepatic endoplasmic reticulum-stress adaptive pathways and potentiates de novo lipogenesis in weaned male mice (2018)
Flister, Karla Frida Torres; Pinto, Bruno Araujo Serra; Franca, Lucas Martins; Coelho, Caio Fernando Ferreira; Santos, Pamela Costa dos; Vale, Caroline Castro; Kajihara, Daniela; Debbas, Victor; Laurindo, Francisco Rafael Martins; Paes, Antonio Marcus de Andrade
Fonte: Journal of nutritional biochemistry. Unidade: FM
Assuntos: SACAROSE, FÍGADO GORDUROSO, HEPATOPATIAS, DOENÇAS METABÓLICAS, CAMUNDONGOS, RETÍCULO ENDOPLASMÁTICO
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FLISTER, Karla Frida Torres et al. Long-term exposure to high-sucrose diet down-regulates hepatic endoplasmic reticulum-stress adaptive pathways and potentiates de novo lipogenesis in weaned male mice. Journal of nutritional biochemistry, v. 62, p. 155-166, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2018.09.007. Acesso em: 29 jun. 2025.
APA
Flister, K. F. T., Pinto, B. A. S., Franca, L. M., Coelho, C. F. F., Santos, P. C. dos, Vale, C. C., et al. (2018). Long-term exposure to high-sucrose diet down-regulates hepatic endoplasmic reticulum-stress adaptive pathways and potentiates de novo lipogenesis in weaned male mice. Journal of nutritional biochemistry, 62, 155-166. doi:10.1016/j.jnutbio.2018.09.007
NLM
Flister KFT, Pinto BAS, Franca LM, Coelho CFF, Santos PC dos, Vale CC, Kajihara D, Debbas V, Laurindo FRM, Paes AM de A. Long-term exposure to high-sucrose diet down-regulates hepatic endoplasmic reticulum-stress adaptive pathways and potentiates de novo lipogenesis in weaned male mice [Internet]. Journal of nutritional biochemistry. 2018 ; 62 155-166.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.09.007
Vancouver
Flister KFT, Pinto BAS, Franca LM, Coelho CFF, Santos PC dos, Vale CC, Kajihara D, Debbas V, Laurindo FRM, Paes AM de A. Long-term exposure to high-sucrose diet down-regulates hepatic endoplasmic reticulum-stress adaptive pathways and potentiates de novo lipogenesis in weaned male mice [Internet]. Journal of nutritional biochemistry. 2018 ; 62 155-166.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.09.007
Artigo de periodico
CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis (2015)
Luque, Maria Carolina A.; Gutierrez, Paulo S.; Debbas, Victor; Kalil, Jorge; Stolf, Beatriz Simonsen
Fonte: Molecular Immunology. Unidades: FM, ICB
Assuntos: ADERÊNCIA CELULAR, CITOCINAS, INFLAMAÇÃO, ARTERIOSCLEROSE
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LUQUE, Maria Carolina A. et al. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis. Molecular Immunology, v. 67, n. 2, p. 559-567, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.molimm.2015.07.028. Acesso em: 29 jun. 2025.
APA
Luque, M. C. A., Gutierrez, P. S., Debbas, V., Kalil, J., & Stolf, B. S. (2015). CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis. Molecular Immunology, 67( 2), 559-567. doi:10.1016/j.molimm.2015.07.028
NLM
Luque MCA, Gutierrez PS, Debbas V, Kalil J, Stolf BS. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis [Internet]. Molecular Immunology. 2015 ; 67( 2): 559-567.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.molimm.2015.07.028
Vancouver
Luque MCA, Gutierrez PS, Debbas V, Kalil J, Stolf BS. CD100 and plexins B2 and B1 mediate monocyte-endothelial cell adhesion and might take part in atherogenesis [Internet]. Molecular Immunology. 2015 ; 67( 2): 559-567.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.molimm.2015.07.028
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Fonte: Clinical Science. Unidades: FMRP, FM, ICB
Assuntos: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
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CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 29 jun. 2025.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1042/CS20140548
Artigo de periodico
Phage Display Identification of CD100 in Human Atherosclerotic Plaque Macrophages and Foam Cells (2013)
Luque, Maria Carolina Aquino; Gutierrez, Paulo Sampaio; Debbas, Victor; Martins, Waleska Kerllen; Puech-Leao, Pedro; Porto, Georgia; Coelho, Verônica; Boumsell, Laurence; Kalil, Jorge; Stolf, Beatriz Simonsen
Fonte: PLOS ONE. Unidades: FM, ICB
Assuntos: ARTERIOSCLEROSE, BIOMARCADORES, INFARTO DO MIOCÁRDIO (PREVENÇÃO E CONTROLE), AUTÓPSIA, IMUNOHISTOQUÍMICA, IMUNOFLUORESCÊNCIA
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LUQUE, Maria Carolina Aquino et al. Phage Display Identification of CD100 in Human Atherosclerotic Plaque Macrophages and Foam Cells. PLOS ONE, v. 8, n. 9, p. 9 , 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0075772. Acesso em: 29 jun. 2025.
APA
Luque, M. C. A., Gutierrez, P. S., Debbas, V., Martins, W. K., Puech-Leao, P., Porto, G., et al. (2013). Phage Display Identification of CD100 in Human Atherosclerotic Plaque Macrophages and Foam Cells. PLOS ONE, 8( 9), 9 . doi:10.1371/journal.pone.0075772
NLM
Luque MCA, Gutierrez PS, Debbas V, Martins WK, Puech-Leao P, Porto G, Coelho V, Boumsell L, Kalil J, Stolf BS. Phage Display Identification of CD100 in Human Atherosclerotic Plaque Macrophages and Foam Cells [Internet]. PLOS ONE. 2013 ; 8( 9): 9 .[citado 2025 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0075772
Vancouver
Luque MCA, Gutierrez PS, Debbas V, Martins WK, Puech-Leao P, Porto G, Coelho V, Boumsell L, Kalil J, Stolf BS. Phage Display Identification of CD100 in Human Atherosclerotic Plaque Macrophages and Foam Cells [Internet]. PLOS ONE. 2013 ; 8( 9): 9 .[citado 2025 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0075772
Artigo de periodico
Effects of High Adherence to Mediterranean or Low-Fat Diets in Medicated Secondary Prevention Patients (2011)
Thomazella, Maria Cristina D.; Góes, Marisa F. S.; Andrade, Cláudia R.; Debbas, Victor; Barbeiro, Denise F.; Correia, Renata L.; Marie, Sueli K. N.; Cardounel, Arturo J.; Luz, Protásio L. da; Laurindo, Francisco R. M.
Fonte: American Journal of Cardiology. Unidade: FM
Assuntos: DIETA REDUTORA (EFEITOS), ADERÊNCIA CELULAR, CORONARIOPATIA (PREVENÇÃO E CONTROLE), ESTRESSE OXIDATIVO (PREVENÇÃO E CONTROLE), QUALIDADE DE VIDA, MODO DE VIDA
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THOMAZELLA, Maria Cristina D. et al. Effects of High Adherence to Mediterranean or Low-Fat Diets in Medicated Secondary Prevention Patients. American Journal of Cardiology, v. 108, n. 11, p. 1523-1529, 2011Tradução . . Disponível em: http://www.sciencedirect.com/science/article/pii/S0002914911022867. Acesso em: 29 jun. 2025.
APA
Thomazella, M. C. D., Góes, M. F. S., Andrade, C. R., Debbas, V., Barbeiro, D. F., Correia, R. L., et al. (2011). Effects of High Adherence to Mediterranean or Low-Fat Diets in Medicated Secondary Prevention Patients. American Journal of Cardiology, 108( 11), 1523-1529. doi:10.1016/j.amjcard.2011.07.008
NLM
Thomazella MCD, Góes MFS, Andrade CR, Debbas V, Barbeiro DF, Correia RL, Marie SKN, Cardounel AJ, Luz PL da, Laurindo FRM. Effects of High Adherence to Mediterranean or Low-Fat Diets in Medicated Secondary Prevention Patients [Internet]. American Journal of Cardiology. 2011 ; 108( 11): 1523-1529.[citado 2025 jun. 29 ] Available from: http://www.sciencedirect.com/science/article/pii/S0002914911022867
Vancouver
Thomazella MCD, Góes MFS, Andrade CR, Debbas V, Barbeiro DF, Correia RL, Marie SKN, Cardounel AJ, Luz PL da, Laurindo FRM. Effects of High Adherence to Mediterranean or Low-Fat Diets in Medicated Secondary Prevention Patients [Internet]. American Journal of Cardiology. 2011 ; 108( 11): 1523-1529.[citado 2025 jun. 29 ] Available from: http://www.sciencedirect.com/science/article/pii/S0002914911022867
Artigo de periodico
Quiescin sulfhydryl oxidase (QSOX) is expressed in the human atheroma core: possible role in apoptosis (2011)
Andrade, Claudia R. de; Stolf, Beatriz Simonsen ; Debbas, Victor; Rosa, Daniela S.; Kalil, Jorge; Coelho, Veronica; Laurindo, Francisco R. M.
Fonte: In vitro cellular and developmental biology - Animal. Unidades: FM, ICB
Assuntos: APOPTOSE, ARTERIOSCLEROSE, LIPÍDEOS (ANÁLISE), NANOPARTÍCULAS, MACRÓFAGOS
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ABNT
ANDRADE, Claudia R. de et al. Quiescin sulfhydryl oxidase (QSOX) is expressed in the human atheroma core: possible role in apoptosis. In vitro cellular and developmental biology - Animal, v. 47, n. 10, p. 716-727, 2011Tradução . . Disponível em: https://doi.org/10.1007/s11626-011-9461-0. Acesso em: 29 jun. 2025.
APA
Andrade, C. R. de, Stolf, B. S., Debbas, V., Rosa, D. S., Kalil, J., Coelho, V., & Laurindo, F. R. M. (2011). Quiescin sulfhydryl oxidase (QSOX) is expressed in the human atheroma core: possible role in apoptosis. In vitro cellular and developmental biology - Animal, 47( 10), 716-727. doi:10.1007/s11626-011-9461-0
NLM
Andrade CR de, Stolf BS, Debbas V, Rosa DS, Kalil J, Coelho V, Laurindo FRM. Quiescin sulfhydryl oxidase (QSOX) is expressed in the human atheroma core: possible role in apoptosis [Internet]. In vitro cellular and developmental biology - Animal. 2011 ; 47( 10): 716-727.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1007/s11626-011-9461-0
Vancouver
Andrade CR de, Stolf BS, Debbas V, Rosa DS, Kalil J, Coelho V, Laurindo FRM. Quiescin sulfhydryl oxidase (QSOX) is expressed in the human atheroma core: possible role in apoptosis [Internet]. In vitro cellular and developmental biology - Animal. 2011 ; 47( 10): 716-727.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1007/s11626-011-9461-0
Artigo de periodico
Moderate exercise training decreases aortic superoxide production in myocardial infarcted rats (2008)
Zanchi, Nelo Eidy; Bechara, Luiz Roberto Grassmann; Tanaka, Leonardo Yuji; Debbas, Victor; Bartholomeu, Teresa; Ramires, P. R.
Fonte: European Journal of Applied Physiology. Unidade: EEFE
Assuntos: FISIOLOGIA DO EXERCÍCIO, BIOQUÍMICA
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ZANCHI, Nelo Eidy et al. Moderate exercise training decreases aortic superoxide production in myocardial infarcted rats. European Journal of Applied Physiology, v. 104, p. 1045-1052, 2008Tradução . . Disponível em: https://doi.org/10.1007/s00421-008-0861-0. Acesso em: 29 jun. 2025.
APA
Zanchi, N. E., Bechara, L. R. G., Tanaka, L. Y., Debbas, V., Bartholomeu, T., & Ramires, P. R. (2008). Moderate exercise training decreases aortic superoxide production in myocardial infarcted rats. European Journal of Applied Physiology, 104, 1045-1052. doi:10.1007/s00421-008-0861-0
NLM
Zanchi NE, Bechara LRG, Tanaka LY, Debbas V, Bartholomeu T, Ramires PR. Moderate exercise training decreases aortic superoxide production in myocardial infarcted rats [Internet]. European Journal of Applied Physiology. 2008 ; 104 1045-1052.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1007/s00421-008-0861-0
Vancouver
Zanchi NE, Bechara LRG, Tanaka LY, Debbas V, Bartholomeu T, Ramires PR. Moderate exercise training decreases aortic superoxide production in myocardial infarcted rats [Internet]. European Journal of Applied Physiology. 2008 ; 104 1045-1052.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1007/s00421-008-0861-0
Artigo de periodico
Thioredoxin-1 promotes survival in cells exposed to S-nitrosoglutathione: correlation with reduction of intracellular levels of nitrosothiols and up-regulation of the ERK1/2 MAP Kinases (2008)
Arai, Roberto J.; Ogata, Fernando Toshio; Batista, Wagner L.; Masutami, Hiroshi; Yodoi, Junji; Debbas, Victor; Augusto, Ohara ; Stern, Arnold; Monteiro, Hugo Pequeno
Fonte: Toxicology and Applied Pharmacology. Unidade: IQ
Assuntos: ÓXIDO NÍTRICO, EXPRESSÃO GÊNICA, TRANSDUÇÃO DE SINAL CELULAR
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ABNT
ARAI, Roberto J. et al. Thioredoxin-1 promotes survival in cells exposed to S-nitrosoglutathione: correlation with reduction of intracellular levels of nitrosothiols and up-regulation of the ERK1/2 MAP Kinases. Toxicology and Applied Pharmacology, v. 233, n. 2, p. 227-237, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2008.07.023. Acesso em: 29 jun. 2025.
APA
Arai, R. J., Ogata, F. T., Batista, W. L., Masutami, H., Yodoi, J., Debbas, V., et al. (2008). Thioredoxin-1 promotes survival in cells exposed to S-nitrosoglutathione: correlation with reduction of intracellular levels of nitrosothiols and up-regulation of the ERK1/2 MAP Kinases. Toxicology and Applied Pharmacology, 233( 2), 227-237. doi:10.1016/j.taap.2008.07.023
NLM
Arai RJ, Ogata FT, Batista WL, Masutami H, Yodoi J, Debbas V, Augusto O, Stern A, Monteiro HP. Thioredoxin-1 promotes survival in cells exposed to S-nitrosoglutathione: correlation with reduction of intracellular levels of nitrosothiols and up-regulation of the ERK1/2 MAP Kinases [Internet]. Toxicology and Applied Pharmacology. 2008 ; 233( 2): 227-237.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.taap.2008.07.023
Vancouver
Arai RJ, Ogata FT, Batista WL, Masutami H, Yodoi J, Debbas V, Augusto O, Stern A, Monteiro HP. Thioredoxin-1 promotes survival in cells exposed to S-nitrosoglutathione: correlation with reduction of intracellular levels of nitrosothiols and up-regulation of the ERK1/2 MAP Kinases [Internet]. Toxicology and Applied Pharmacology. 2008 ; 233( 2): 227-237.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/j.taap.2008.07.023
Artigo de periodico
Nitric oxide and cGMP activate the Ras-Map kinase pathway-stimulating protein tyrosine phosphorylation in rabbit aortic endothelial cells (2003)
Oliveira, Carlos J. R.; Schindler, Fernanda; Ventura, Armando Morais ; Morais, Miriam S.; Arai, Roberto J.; Debbas, Victor; Stern, Arnold; Monteiro, Hugo P.
Fonte: Free Radical Biology & Medicine. Unidade: ICB
Assunto: MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Carlos J. R. et al. Nitric oxide and cGMP activate the Ras-Map kinase pathway-stimulating protein tyrosine phosphorylation in rabbit aortic endothelial cells. Free Radical Biology & Medicine, v. 35, n. 4, p. 381-396, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0891-5849(03)00311-3. Acesso em: 29 jun. 2025.
APA
Oliveira, C. J. R., Schindler, F., Ventura, A. M., Morais, M. S., Arai, R. J., Debbas, V., et al. (2003). Nitric oxide and cGMP activate the Ras-Map kinase pathway-stimulating protein tyrosine phosphorylation in rabbit aortic endothelial cells. Free Radical Biology & Medicine, 35( 4), 381-396. doi:10.1016/s0891-5849(03)00311-3
NLM
Oliveira CJR, Schindler F, Ventura AM, Morais MS, Arai RJ, Debbas V, Stern A, Monteiro HP. Nitric oxide and cGMP activate the Ras-Map kinase pathway-stimulating protein tyrosine phosphorylation in rabbit aortic endothelial cells [Internet]. Free Radical Biology & Medicine. 2003 ; 35( 4): 381-396.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/s0891-5849(03)00311-3
Vancouver
Oliveira CJR, Schindler F, Ventura AM, Morais MS, Arai RJ, Debbas V, Stern A, Monteiro HP. Nitric oxide and cGMP activate the Ras-Map kinase pathway-stimulating protein tyrosine phosphorylation in rabbit aortic endothelial cells [Internet]. Free Radical Biology & Medicine. 2003 ; 35( 4): 381-396.[citado 2025 jun. 29 ] Available from: https://doi.org/10.1016/s0891-5849(03)00311-3

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