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Filtros : "Caspi, Rachel R." Removido: "American Association of Immunologists" Limpar

Filtros

Colaboração institucional
Afiliação dos autores externos normalizada
- national institutes of health (nih) (~1)
Afiliação dos autores externos não normalizada
- flinders university, college of medicine and public health, adelaide (1)
- metamorphix inc (1)
- nih, nei, laboratory of immunology, bethesda (1)
- national eye institute (1)
- oregon health and science university, department of pediatrics, portland (1)

Artigo de periodico
T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis (2020)
Napier, Ruth J.; Lee, Ellen J.; Davey, Michael P.; Vance, Emily E.; Furtado, João Marcello Fortes; Snow, Paige E.; Samson, Kimberly A.; Lashley, Sydney J.; Brown, Brieanna R.; Horai, Reiko; Mattapallil, Mary J.; Xu, Biying; Callegan, Michelle C.; Uebelhoer, Luke S.; Lancioni, Christina L.; Vehe, Richard K.; Binstadt, Bryce A.; Smith, Justine R.; Caspi, Rachel R.; Rosenzweig, Holly L.
Fonte: Nature Communications. Unidade: FMRP
Assuntos: LINFÓCITOS T, UVEÍTE, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NAPIER, Ruth J. et al. T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis. Nature Communications, v. 11, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41467-020-18961-0. Acesso em: 02 nov. 2025.
APA
Napier, R. J., Lee, E. J., Davey, M. P., Vance, E. E., Furtado, J. M. F., Snow, P. E., et al. (2020). T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis. Nature Communications, 11( 1). doi:10.1038/s41467-020-18961-0
NLM
Napier RJ, Lee EJ, Davey MP, Vance EE, Furtado JMF, Snow PE, Samson KA, Lashley SJ, Brown BR, Horai R, Mattapallil MJ, Xu B, Callegan MC, Uebelhoer LS, Lancioni CL, Vehe RK, Binstadt BA, Smith JR, Caspi RR, Rosenzweig HL. T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis [Internet]. Nature Communications. 2020 ; 11( 1):[citado 2025 nov. 02 ] Available from: https://doi.org/10.1038/s41467-020-18961-0
Vancouver
Napier RJ, Lee EJ, Davey MP, Vance EE, Furtado JMF, Snow PE, Samson KA, Lashley SJ, Brown BR, Horai R, Mattapallil MJ, Xu B, Callegan MC, Uebelhoer LS, Lancioni CL, Vehe RK, Binstadt BA, Smith JR, Caspi RR, Rosenzweig HL. T cell-intrinsic role for Nod2 in protection against Th17-mediated uveitis [Internet]. Nature Communications. 2020 ; 11( 1):[citado 2025 nov. 02 ] Available from: https://doi.org/10.1038/s41467-020-18961-0
Artigo de periodico
Protective effect of the type IV phosphodiesterase inhibitor rolipram in EUA: Protection is independent of IL-10-inducing activity (1999)
Xu, Hui; Strassmann, Gideon; Chan, Chi-Chao; Rizzo, Luiz Vicente; Silver, Phyllis B.; Wiggert, Barbara; Caspi, Rachel R.
Fonte: Investigative Ophthalmology and Visual Science. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
XU, Hui et al. Protective effect of the type IV phosphodiesterase inhibitor rolipram in EUA: Protection is independent of IL-10-inducing activity. Investigative Ophthalmology and Visual Science, v. 40, p. 942-950, 1999Tradução . . Acesso em: 02 nov. 2025.
APA
Xu, H., Strassmann, G., Chan, C. -C., Rizzo, L. V., Silver, P. B., Wiggert, B., & Caspi, R. R. (1999). Protective effect of the type IV phosphodiesterase inhibitor rolipram in EUA: Protection is independent of IL-10-inducing activity. Investigative Ophthalmology and Visual Science, 40, 942-950.
NLM
Xu H, Strassmann G, Chan C-C, Rizzo LV, Silver PB, Wiggert B, Caspi RR. Protective effect of the type IV phosphodiesterase inhibitor rolipram in EUA: Protection is independent of IL-10-inducing activity. Investigative Ophthalmology and Visual Science. 1999 ; 40 942-950.[citado 2025 nov. 02 ]
Vancouver
Xu H, Strassmann G, Chan C-C, Rizzo LV, Silver PB, Wiggert B, Caspi RR. Protective effect of the type IV phosphodiesterase inhibitor rolipram in EUA: Protection is independent of IL-10-inducing activity. Investigative Ophthalmology and Visual Science. 1999 ; 40 942-950.[citado 2025 nov. 02 ]

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