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Artigo de periodico
Rederivation of a mutant line (prop 1) of zebrafish Danio rerio infected with Pseudoloma neurophilia using in vitro fertilization with eggs from pathogen-free wild-type (AB) females and sperm from prop 1 males (2022)
Fernandes, Bianca Helena Ventura ; Silva, Caroline Caetano da; Bissegato, Debora; Kent, Michael L; Carvalho, Luciani R
Source: Journal of fish diseases. Unidade: FM
Subjects: PEIXES, PARASITOLOGIA, FERTILIZAÇÃO "IN VITRO", MUTAÇÃO GENÉTICA
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ABNT
FERNANDES, Bianca Helena Ventura et al. Rederivation of a mutant line (prop 1) of zebrafish Danio rerio infected with Pseudoloma neurophilia using in vitro fertilization with eggs from pathogen-free wild-type (AB) females and sperm from prop 1 males. Journal of fish diseases, v. 45, n. 1, p. 35-39, 2022Tradução . . Disponível em: https://doi.org/10.1111/jfd.13529. Acesso em: 22 jul. 2024.
APA
Fernandes, B. H. V., Silva, C. C. da, Bissegato, D., Kent, M. L., & Carvalho, L. R. (2022). Rederivation of a mutant line (prop 1) of zebrafish Danio rerio infected with Pseudoloma neurophilia using in vitro fertilization with eggs from pathogen-free wild-type (AB) females and sperm from prop 1 males. Journal of fish diseases, 45( 1), 35-39. doi:10.1111/jfd.13529
NLM
Fernandes BHV, Silva CC da, Bissegato D, Kent ML, Carvalho LR. Rederivation of a mutant line (prop 1) of zebrafish Danio rerio infected with Pseudoloma neurophilia using in vitro fertilization with eggs from pathogen-free wild-type (AB) females and sperm from prop 1 males [Internet]. Journal of fish diseases. 2022 ; 45( 1): 35-39.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1111/jfd.13529
Vancouver
Fernandes BHV, Silva CC da, Bissegato D, Kent ML, Carvalho LR. Rederivation of a mutant line (prop 1) of zebrafish Danio rerio infected with Pseudoloma neurophilia using in vitro fertilization with eggs from pathogen-free wild-type (AB) females and sperm from prop 1 males [Internet]. Journal of fish diseases. 2022 ; 45( 1): 35-39.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1111/jfd.13529
Artigo de periodico
Combined pituitary hormone deficiency caused by PROP1 mutations: update 20 years post-discovery (2019)
Correa, Fernanda A; Nakaguma, Marilena; Madeira, Joao L. O; Nishi, Mirian Y; Abrao, Milena G; Jorge, Alexander Augusto de Lima; Carvalho, Luciani R; Arnhold, Ivo J. P; Mendonça, Berenice Bilharinho de
Source: Archives of endocrinology metabolism. Unidade: FM
Subjects: HORMÔNIOS DA ADENO-HIPÓFISE, MUTAÇÃO GENÉTICA, GENES HOMEOBOX
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ABNT
CORREA, Fernanda A et al. Combined pituitary hormone deficiency caused by PROP1 mutations: update 20 years post-discovery. Archives of endocrinology metabolism, v. 63, n. 2, p. 167-174, 2019Tradução . . Disponível em: https://doi.org/10.20945/2359-3997000000139. Acesso em: 22 jul. 2024.
APA
Correa, F. A., Nakaguma, M., Madeira, J. L. O., Nishi, M. Y., Abrao, M. G., Jorge, A. A. de L., et al. (2019). Combined pituitary hormone deficiency caused by PROP1 mutations: update 20 years post-discovery. Archives of endocrinology metabolism, 63( 2), 167-174. doi:10.20945/2359-3997000000139
NLM
Correa FA, Nakaguma M, Madeira JLO, Nishi MY, Abrao MG, Jorge AA de L, Carvalho LR, Arnhold IJP, Mendonça BB de. Combined pituitary hormone deficiency caused by PROP1 mutations: update 20 years post-discovery [Internet]. Archives of endocrinology metabolism. 2019 ; 63( 2): 167-174.[citado 2024 jul. 22 ] Available from: https://doi.org/10.20945/2359-3997000000139
Vancouver
Correa FA, Nakaguma M, Madeira JLO, Nishi MY, Abrao MG, Jorge AA de L, Carvalho LR, Arnhold IJP, Mendonça BB de. Combined pituitary hormone deficiency caused by PROP1 mutations: update 20 years post-discovery [Internet]. Archives of endocrinology metabolism. 2019 ; 63( 2): 167-174.[citado 2024 jul. 22 ] Available from: https://doi.org/10.20945/2359-3997000000139
Artigo de periodico
Growth hormone deficiency with advanced bone age: phenotypic interaction between GHRH receptor and CYP21A2 mutations diagnosed by sanger and whole exome sequencing (2017)
Correa, Fernanda A.; França, Marcela M; Fang, Qing; Ma, Qianyi; Bachega, Tânia Aparecida Sartori Sanchez; Rodrigues, Andresa; Ozel, Bilge A; Li, Jun Z; Mendonça, Berenice Bilharinho de; Jorge, Alexander Augusto de Lima; Carvalho, Luciani R; Camper, Sally A; Arnhold, Ivo J
Source: Archives of endocrinology metabolism. Unidade: FM
Subjects: HIPERPLASIA SUPRARRENAL CONGÊNITA, HORMÔNIO DO CRESCIMENTO, DOENÇAS DO DESENVOLVIMENTO ÓSSEO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORREA, Fernanda A. et al. Growth hormone deficiency with advanced bone age: phenotypic interaction between GHRH receptor and CYP21A2 mutations diagnosed by sanger and whole exome sequencing. Archives of endocrinology metabolism, v. 61, n. 6, p. 633-636, 2017Tradução . . Disponível em: https://doi.org/10.1590/2359-3997000000311. Acesso em: 22 jul. 2024.
APA
Correa, F. A., França, M. M., Fang, Q., Ma, Q., Bachega, T. A. S. S., Rodrigues, A., et al. (2017). Growth hormone deficiency with advanced bone age: phenotypic interaction between GHRH receptor and CYP21A2 mutations diagnosed by sanger and whole exome sequencing. Archives of endocrinology metabolism, 61( 6), 633-636. doi:10.1590/2359-3997000000311
NLM
Correa FA, França MM, Fang Q, Ma Q, Bachega TASS, Rodrigues A, Ozel BA, Li JZ, Mendonça BB de, Jorge AA de L, Carvalho LR, Camper SA, Arnhold IJ. Growth hormone deficiency with advanced bone age: phenotypic interaction between GHRH receptor and CYP21A2 mutations diagnosed by sanger and whole exome sequencing [Internet]. Archives of endocrinology metabolism. 2017 ; 61( 6): 633-636.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1590/2359-3997000000311
Vancouver
Correa FA, França MM, Fang Q, Ma Q, Bachega TASS, Rodrigues A, Ozel BA, Li JZ, Mendonça BB de, Jorge AA de L, Carvalho LR, Camper SA, Arnhold IJ. Growth hormone deficiency with advanced bone age: phenotypic interaction between GHRH receptor and CYP21A2 mutations diagnosed by sanger and whole exome sequencing [Internet]. Archives of endocrinology metabolism. 2017 ; 61( 6): 633-636.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1590/2359-3997000000311
Artigo de periodico
A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) (2016)
Madeira, João L. O.; Jorge, Alexander Augusto de Lima; Martin, Regina M; Montenegro, Luciana Ribeiro; Franca, Marcela M.; Costalong, Everlayny F.; Correa, Fernanda A.; Otto, Aline P.; Arnhold, Ivo J. P; Freitas, Helayne S; Machado, Ubiratan Fabres ; Mendonça, Berenice Bilharinho de; Carvalho, Luciani R
Source: European journal of endocrinology. Unidades: FM, ICB
Subjects: GRAVIDEZ DE GÊMEOS, HORMÔNIO DO CRESCIMENTO, EXPRESSÃO GÊNICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MADEIRA, João L. O. et al. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD). European journal of endocrinology, v. 175, n. 02, p. K7-K15, 2016Tradução . . Disponível em: https://doi.org/10.1530/eje-15-0149. Acesso em: 22 jul. 2024.
APA
Madeira, J. L. O., Jorge, A. A. de L., Martin, R. M., Montenegro, L. R., Franca, M. M., Costalong, E. F., et al. (2016). A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD). European journal of endocrinology, 175( 02), K7-K15. doi:10.1530/eje-15-0149
NLM
Madeira JLO, Jorge AA de L, Martin RM, Montenegro LR, Franca MM, Costalong EF, Correa FA, Otto AP, Arnhold IJP, Freitas HS, Machado UF, Mendonça BB de, Carvalho LR. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) [Internet]. European journal of endocrinology. 2016 ; 175( 02): K7-K15.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1530/eje-15-0149
Vancouver
Madeira JLO, Jorge AA de L, Martin RM, Montenegro LR, Franca MM, Costalong EF, Correa FA, Otto AP, Arnhold IJP, Freitas HS, Machado UF, Mendonça BB de, Carvalho LR. A homozygous point mutation in the GH1 promoter (c.-223C>T) leads to reducd GH1 expression in siblings with isolated GH deficiency (IGHD) [Internet]. European journal of endocrinology. 2016 ; 175( 02): K7-K15.[citado 2024 jul. 22 ] Available from: https://doi.org/10.1530/eje-15-0149

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