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Artigo de periodico
Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic‐ and GPR109a‐mediated mechanisms (2019)
Felizardo, Raphael José Ferreira; Almeida, Danilo Candido de ; Pereira, Rafael Luiz; Watanabe, Ingrid Kazue Mizuno; Doimo, Nayara Trevisan dos Santos; Ribeiro, Willian Rodrigues; Cenedeze, Marcos Antonio; Hiyane, Meire Ioshie; Amano, Mariane Tami ; Braga, Tárcio Teodoro ; Ferreira, Caroline Marcantonio ; Parmigiani, Raphael Bessa; Oliveira, Vinicius de Andrade ; Volpini, Rildo Aparecido ; Vinolo, Marco Aurelio Ramirez ; Moreno, Eliana Mariño ; Robert, Remy ; Mackay, Charles Reay ; Câmara, Niels Olsen Saraiva
Fonte: The FASEB Journal. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FELIZARDO, Raphael José Ferreira et al. Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic‐ and GPR109a‐mediated mechanisms. The FASEB Journal, v. 33, n. 11, p. 11894-11908, 2019Tradução . . Disponível em: https://doi.org/10.1096/fj.201901080R. Acesso em: 01 out. 2024.
APA
Felizardo, R. J. F., Almeida, D. C. de, Pereira, R. L., Watanabe, I. K. M., Doimo, N. T. dos S., Ribeiro, W. R., et al. (2019). Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic‐ and GPR109a‐mediated mechanisms. The FASEB Journal, 33( 11), 11894-11908. doi:10.1096/fj.201901080R
NLM
Felizardo RJF, Almeida DC de, Pereira RL, Watanabe IKM, Doimo NT dos S, Ribeiro WR, Cenedeze MA, Hiyane MI, Amano MT, Braga TT, Ferreira CM, Parmigiani RB, Oliveira V de A, Volpini RA, Vinolo MAR, Moreno EM, Robert R, Mackay CR, Câmara NOS. Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic‐ and GPR109a‐mediated mechanisms [Internet]. The FASEB Journal. 2019 ; 33( 11): 11894-11908.[citado 2024 out. 01 ] Available from: https://doi.org/10.1096/fj.201901080R
Vancouver
Felizardo RJF, Almeida DC de, Pereira RL, Watanabe IKM, Doimo NT dos S, Ribeiro WR, Cenedeze MA, Hiyane MI, Amano MT, Braga TT, Ferreira CM, Parmigiani RB, Oliveira V de A, Volpini RA, Vinolo MAR, Moreno EM, Robert R, Mackay CR, Câmara NOS. Gut microbial metabolite butyrate protects against proteinuric kidney disease through epigenetic‐ and GPR109a‐mediated mechanisms [Internet]. The FASEB Journal. 2019 ; 33( 11): 11894-11908.[citado 2024 out. 01 ] Available from: https://doi.org/10.1096/fj.201901080R
Artigo de periodico
CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development (2018)
Braga, Tárcio Teodoro; Costa, Matheus Correa; Silva, Reinaldo Correia; Cruz, Mário Costa; Hiyane, Meire Ioshie; Silva, João Santana da; Perez, Katia Regina; Cuccovia, Iolanda Midea ; Câmara, Niels Olsen Saraiva
Fonte: Inflammopharmacology. Unidades: ICB, FMRP, IQ
Assuntos: MONÓCITOS, QUIMIOCINAS, INFLAMAÇÃO, TÉCNICAS DE DIAGNÓSTICO UROLÓGICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRAGA, Tárcio Teodoro et al. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, v. 26, p. 403-411, 2018Tradução . . Disponível em: https://doi.org/10.1007/s10787-017-0317-4. Acesso em: 01 out. 2024.
APA
Braga, T. T., Costa, M. C., Silva, R. C., Cruz, M. C., Hiyane, M. I., Silva, J. S. da, et al. (2018). CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development. Inflammopharmacology, 26, 403-411. doi:10.1007/s10787-017-0317-4
NLM
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Vancouver
Braga TT, Costa MC, Silva RC, Cruz MC, Hiyane MI, Silva JS da, Perez KR, Cuccovia IM, Câmara NOS. CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development [Internet]. Inflammopharmacology. 2018 ; 26 403-411.[citado 2024 out. 01 ] Available from: https://doi.org/10.1007/s10787-017-0317-4
Artigo de periodico
Macrophage trafficking as key mediator of adenine-induced kidney injury (2014)
Correa-Costa, Matheus; Braga, Tárcio Teodoro; Felizardo, Raphael José Ferreira; Andrade-Oliveira, Vinicius; Perez, Katia Regina; Cuccovia, Iolanda Midea ; Hiyane, Meire Ioshie; Silva, João Santana da; Câmara, Niels Olsen Saraiva
Fonte: Mediators of Inflammation. Unidades: IQ, ICB, FMRP
Assuntos: MACRÓFAGOS, BIOQUÍMICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CORREA-COSTA, Matheus et al. Macrophage trafficking as key mediator of adenine-induced kidney injury. Mediators of Inflammation, p. 1-12 art. 291024, 2014Tradução . . Disponível em: https://doi.org/10.1155/2014/291024. Acesso em: 01 out. 2024.
APA
Correa-Costa, M., Braga, T. T., Felizardo, R. J. F., Andrade-Oliveira, V., Perez, K. R., Cuccovia, I. M., et al. (2014). Macrophage trafficking as key mediator of adenine-induced kidney injury. Mediators of Inflammation, 1-12 art. 291024. doi:10.1155/2014/291024
NLM
Correa-Costa M, Braga TT, Felizardo RJF, Andrade-Oliveira V, Perez KR, Cuccovia IM, Hiyane MI, Silva JS da, Câmara NOS. Macrophage trafficking as key mediator of adenine-induced kidney injury [Internet]. Mediators of Inflammation. 2014 ; 1-12 art. 291024.[citado 2024 out. 01 ] Available from: https://doi.org/10.1155/2014/291024
Vancouver
Correa-Costa M, Braga TT, Felizardo RJF, Andrade-Oliveira V, Perez KR, Cuccovia IM, Hiyane MI, Silva JS da, Câmara NOS. Macrophage trafficking as key mediator of adenine-induced kidney injury [Internet]. Mediators of Inflammation. 2014 ; 1-12 art. 291024.[citado 2024 out. 01 ] Available from: https://doi.org/10.1155/2014/291024
Artigo de periodico
TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis (2012)
Castoldi, Angela; Braga, Tárcio Teodoro; Correa-Costa, Matheus; Aguiar, Cristhiane Fávero; Bassi, Ênio José; Correa-Silva, Reinaldo; Elias, Rosa Maria; Salvador, Fábia; Moraes-Vieira, Pedro Manoel Mendes de; Cenedeze, Marcos Antonio; Reis, Marlene Antônia; Hiyane, Meire Ioshie; Pacheco-Silva, Álvaro; Gonçalves, Giselle Martins; Câmara, Niels Olsen Saraiva
Fonte: PLOS ONE. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CASTOLDI, Angela et al. TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis. PLOS ONE, v. 7, n. 05, p. 1-14, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0037584. Acesso em: 01 out. 2024.
APA
Castoldi, A., Braga, T. T., Correa-Costa, M., Aguiar, C. F., Bassi, Ê. J., Correa-Silva, R., et al. (2012). TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis. PLOS ONE, 7( 05), 1-14. doi:10.1371/journal.pone.0037584
NLM
Castoldi A, Braga TT, Correa-Costa M, Aguiar CF, Bassi ÊJ, Correa-Silva R, Elias RM, Salvador F, Moraes-Vieira PMM de, Cenedeze MA, Reis MA, Hiyane MI, Pacheco-Silva Á, Gonçalves GM, Câmara NOS. TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis [Internet]. PLOS ONE. 2012 ; 7( 05): 1-14.[citado 2024 out. 01 ] Available from: https://doi.org/10.1371/journal.pone.0037584
Vancouver
Castoldi A, Braga TT, Correa-Costa M, Aguiar CF, Bassi ÊJ, Correa-Silva R, Elias RM, Salvador F, Moraes-Vieira PMM de, Cenedeze MA, Reis MA, Hiyane MI, Pacheco-Silva Á, Gonçalves GM, Câmara NOS. TLR2, TLR4 and the MYD88 signaling pathway are crucial for neutrophil migration in acute kidney injury induced by sepsis [Internet]. PLOS ONE. 2012 ; 7( 05): 1-14.[citado 2024 out. 01 ] Available from: https://doi.org/10.1371/journal.pone.0037584

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