Source: Abstracts. Conference titles: International Congress of Immunology. Unidade: ICB
Assunto: IMUNOLOGIA
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BORTOLATTO, J. et al. Co-adsorption of allergen with bacterial lipopolysaccharide to Alum suppresses the development of allergic asthma signaling through Toll like receptor 4 and the adaptor molecule MyD88. 2007, Anais.. Rio de Janeiro: Sociedade Brasileira de Imunologia, 2007. . Acesso em: 28 set. 2024.APA
Bortolatto, J., Rodriguez, D., Keller, A., Faquim-Mauro, E., Borducchi, E., Bastos, K., et al. (2007). Co-adsorption of allergen with bacterial lipopolysaccharide to Alum suppresses the development of allergic asthma signaling through Toll like receptor 4 and the adaptor molecule MyD88. In Abstracts. Rio de Janeiro: Sociedade Brasileira de Imunologia.NLM
Bortolatto J, Rodriguez D, Keller A, Faquim-Mauro E, Borducchi E, Bastos K, Candrian S, Schnyder B, Ryffel B, Russo M. Co-adsorption of allergen with bacterial lipopolysaccharide to Alum suppresses the development of allergic asthma signaling through Toll like receptor 4 and the adaptor molecule MyD88. Abstracts. 2007 ;[citado 2024 set. 28 ]Vancouver
Bortolatto J, Rodriguez D, Keller A, Faquim-Mauro E, Borducchi E, Bastos K, Candrian S, Schnyder B, Ryffel B, Russo M. Co-adsorption of allergen with bacterial lipopolysaccharide to Alum suppresses the development of allergic asthma signaling through Toll like receptor 4 and the adaptor molecule MyD88. Abstracts. 2007 ;[citado 2024 set. 28 ]