Filtros : "Bendtzen, K" Limpar

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  • Source: Endocrinology. Unidade: FMRP

    Assunto: MEDICINA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      SANDLER, S et al. Studies on the mechanisms causing inhibition of inclulin secretion in rat pancreatic islets exposed to human interlenkin 1 b indicate a perturbation in the mitochondrial function. Endocrinology, v. 124, n. 3 , p. 1492-501, 1989Tradução . . Acesso em: 02 ago. 2024.
    • APA

      Sandler, S., Bendtzen, K., Hakan Borg, L. A., Eizirik, D. L., Strandell, E., & Welsh, N. (1989). Studies on the mechanisms causing inhibition of inclulin secretion in rat pancreatic islets exposed to human interlenkin 1 b indicate a perturbation in the mitochondrial function. Endocrinology, 124( 3 ), 1492-501.
    • NLM

      Sandler S, Bendtzen K, Hakan Borg LA, Eizirik DL, Strandell E, Welsh N. Studies on the mechanisms causing inhibition of inclulin secretion in rat pancreatic islets exposed to human interlenkin 1 b indicate a perturbation in the mitochondrial function. Endocrinology. 1989 ;124( 3 ): 1492-501.[citado 2024 ago. 02 ]
    • Vancouver

      Sandler S, Bendtzen K, Hakan Borg LA, Eizirik DL, Strandell E, Welsh N. Studies on the mechanisms causing inhibition of inclulin secretion in rat pancreatic islets exposed to human interlenkin 1 b indicate a perturbation in the mitochondrial function. Endocrinology. 1989 ;124( 3 ): 1492-501.[citado 2024 ago. 02 ]
  • Source: Diabetologia. Unidade: FMRP

    Subjects: INSULINA, INTERLEUCINA 1

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      SANDLER, S et al. Studies on the mechanism causing the impairment of glucose - stimulated insulin release in rat pancreatic islets exposed to human recombinant interleukin 1 beta. Diabetologia, v. 31, p. 538a, 1988Tradução . . Acesso em: 02 ago. 2024.
    • APA

      Sandler, S., Eizirik, D. L., Borg, L. A. H., Strandell, E., Welsh, N., & Bendtzen, K. (1988). Studies on the mechanism causing the impairment of glucose - stimulated insulin release in rat pancreatic islets exposed to human recombinant interleukin 1 beta. Diabetologia, 31, 538a.
    • NLM

      Sandler S, Eizirik DL, Borg LAH, Strandell E, Welsh N, Bendtzen K. Studies on the mechanism causing the impairment of glucose - stimulated insulin release in rat pancreatic islets exposed to human recombinant interleukin 1 beta. Diabetologia. 1988 ;31 538a.[citado 2024 ago. 02 ]
    • Vancouver

      Sandler S, Eizirik DL, Borg LAH, Strandell E, Welsh N, Bendtzen K. Studies on the mechanism causing the impairment of glucose - stimulated insulin release in rat pancreatic islets exposed to human recombinant interleukin 1 beta. Diabetologia. 1988 ;31 538a.[citado 2024 ago. 02 ]
  • Source: Diabetes. Unidade: FMRP

    Assunto: MEDICINA

    How to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      EIZIRIK, D L et al. Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1. Diabetes, v. 37, p. 916-9, 1988Tradução . . Acesso em: 02 ago. 2024.
    • APA

      Eizirik, D. L., Strandell, E., Bendtzen, K., & Sandler, S. (1988). Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1. Diabetes, 37, 916-9.
    • NLM

      Eizirik DL, Strandell E, Bendtzen K, Sandler S. Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1. Diabetes. 1988 ;37 916-9.[citado 2024 ago. 02 ]
    • Vancouver

      Eizirik DL, Strandell E, Bendtzen K, Sandler S. Functional characteristics of rat pancreatic islets maintained in culture following exposure to human intelenkin 1. Diabetes. 1988 ;37 916-9.[citado 2024 ago. 02 ]

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