Source: PLOS ONE. Unidades: FM, IB
Subjects: FATORES DE CRESCIMENTO (DEFICIÊNCIA), MUTAÇÃO GENÉTICA, FENÓTIPOS, GENÓTIPOS, BIOLOGIA MOLECULAR, CRANIOSSINOSTOSE (GENÉTICA)
ABNT
YEH, Erika et al. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome. PLOS ONE, v. 8, n. 4, p. 7 , 2013Tradução . . Disponível em: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439. Acesso em: 29 ago. 2024.APA
Yeh, E., Fanganiello, R. D., Sunaga, D. Y., Zhou, X., Holmes, G., Rocha, K. M., et al. (2013). Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome. PLOS ONE, 8( 4), 7 . doi:10.1371/journal.pone.0060439NLM
Yeh E, Fanganiello RD, Sunaga DY, Zhou X, Holmes G, Rocha KM, Alonso N, Matushita H, Wang Y, Jabs EW, Passos-Bueno MR. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome [Internet]. PLOS ONE. 2013 ; 8( 4): 7 .[citado 2024 ago. 29 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439Vancouver
Yeh E, Fanganiello RD, Sunaga DY, Zhou X, Holmes G, Rocha KM, Alonso N, Matushita H, Wang Y, Jabs EW, Passos-Bueno MR. Novel Molecular Pathways Elicited by Mutant FGFR2 May Account for Brain Abnormalities in Apert Syndrome [Internet]. PLOS ONE. 2013 ; 8( 4): 7 .[citado 2024 ago. 29 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0060439