Source: Hypertension. Unidade: ICB
Assunto: FARMACOLOGIA
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YOGI, Álvaro et al. Renal redox-sensitive signaling, but not blood pressure, is attenuated by NOX1 knockout in angiotensin II-dependent chronic hypertension. Hypertension, v. 51, n. 2, p. 500-506, 2008Tradução . . Acesso em: 31 ago. 2024.APA
Yogi, Á., Mercure, C., Touyz, J., Callera, G. E., Montezano, A. C. I., Aranha, A. B., et al. (2008). Renal redox-sensitive signaling, but not blood pressure, is attenuated by NOX1 knockout in angiotensin II-dependent chronic hypertension. Hypertension, 51( 2), 500-506.NLM
Yogi Á, Mercure C, Touyz J, Callera GE, Montezano ACI, Aranha AB, Passaglia R de CAT, Reudelhuber T, Touyz RM. Renal redox-sensitive signaling, but not blood pressure, is attenuated by NOX1 knockout in angiotensin II-dependent chronic hypertension. Hypertension. 2008 ; 51( 2): 500-506.[citado 2024 ago. 31 ]Vancouver
Yogi Á, Mercure C, Touyz J, Callera GE, Montezano ACI, Aranha AB, Passaglia R de CAT, Reudelhuber T, Touyz RM. Renal redox-sensitive signaling, but not blood pressure, is attenuated by NOX1 knockout in angiotensin II-dependent chronic hypertension. Hypertension. 2008 ; 51( 2): 500-506.[citado 2024 ago. 31 ]