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Artigo de periodico
Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues (2016)
Victório, Jamaira A.; Fontes, Milene T.; Rossoni, Luciana Venturini ; Davel, Ana P.
Fonte: Frontiers in Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
VICTÓRIO, Jamaira A. et al. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, v. 7, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.3389/fphys.2016.00295. Acesso em: 08 jul. 2024.
APA
Victório, J. A., Fontes, M. T., Rossoni, L. V., & Davel, A. P. (2016). Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, 7, 1-10. doi:10.3389/fphys.2016.00295
NLM
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Vancouver
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 jul. 08 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Artigo de periodico
Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway (2015)
Davel, Ana P.; Victorio, Jamaira A.; Delbin, Maria Andréia; Fukuda, Livia Emy; Rossoni, Luciana Venturini
Fonte: Life Sciences. Unidade: ICB
Assuntos: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS
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ABNT
DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 08 jul. 2024.
APA
Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
NLM
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Vancouver
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Artigo de periodico
Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling (2014)
Couto, Gisele Kruger; Davel, Ana P; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Fonte: Experimental physiology. Unidades: ICB, EEFE
Assuntos: FISIOLOGIA, SISTEMA CARDIOVASCULAR
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ABNT
COUTO, Gisele Kruger et al. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, v. 99, n. 10, p. 1427–1438, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2014.079236. Acesso em: 08 jul. 2024.
APA
Couto, G. K., Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, 99( 10), 1427–1438. doi:10.1113/expphysiol.2014.079236
NLM
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Vancouver
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236
Artigo de periodico
Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway (2014)
Davel, Ana P; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Fonte: PLOS ONE. Unidades: EEFE, ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA, DOENÇAS CARDIOVASCULARES
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ABNT
DAVEL, Ana P e BRUM, Patricia Chakur e ROSSONI, Luciana Venturini. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway. PLOS ONE, v. 9, n. 3, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0091877. Acesso em: 08 jul. 2024.
APA
Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway. PLOS ONE, 9( 3). doi:10.1371/journal.pone.0091877
NLM
Davel AP, Brum PC, Rossoni LV. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway [Internet]. PLOS ONE. 2014 ; 9( 3):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.pone.0091877
Vancouver
Davel AP, Brum PC, Rossoni LV. Isoproterenol induces vascular oxidative stress and endothelial dysfunction via a Giα-Coupled β2-adrenoceptor signaling pathway [Internet]. PLOS ONE. 2014 ; 9( 3):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1371/journal.pone.0091877
Artigo de periodico
Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats (2014)
Davel, Ana Paula; Couto, Gisele Kruger; Wenceslau, Camila Ferreira; Peres, Emilia Cristina; Xavier, Fabiano Elias; Rossoni, Luciana Venturini
Fonte: Hormone Molecular Biology and Clinical Investigation. Unidades: ICB, HU
Assuntos: FISIOLOGIA, BIOFÍSICA, ENDOTÉLIO VASCULAR, RATOS, AORTA DE ANIMAL
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ABNT
DAVEL, Ana Paula et al. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, v. 18, n. 2, p. 113-122, 2014Tradução . . Disponível em: https://doi.org/10.1515/hmbci-2013-0058. Acesso em: 08 jul. 2024.
APA
Davel, A. P., Couto, G. K., Wenceslau, C. F., Peres, E. C., Xavier, F. E., & Rossoni, L. V. (2014). Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, 18( 2), 113-122. doi:10.1515/hmbci-2013-0058
NLM
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Vancouver
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Artigo de periodico
Pancreatic islets isolated from Beta2 adrenergic recept or knockout mice show reduced insulin secretion in response to nutrients (2013)
Marçal, Anderson Carlos; Davel, Ana Paula Couto; Carpinelli, Angelo Rafael ; Brum, Patricia Chakur; Rossoni, Luciana Venturini ; Carvalho, Carla Roberta de Oliveira
Fonte: Acta Scientiarum. Biological Sciences. Unidades: ICB, EEFE
Assuntos: ADRENÉRGICOS, FISIOLOGIA, ENDOCRINOLOGIA
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ABNT
MARÇAL, Anderson Carlos et al. Pancreatic islets isolated from Beta2 adrenergic recept or knockout mice show reduced insulin secretion in response to nutrients. Acta Scientiarum. Biological Sciences, v. 35, n. 3, p. 437-443, 2013Tradução . . Disponível em: https://doi.org/10.4025/actascibiolsci.v35i3.15842. Acesso em: 08 jul. 2024.
APA
Marçal, A. C., Davel, A. P. C., Carpinelli, A. R., Brum, P. C., Rossoni, L. V., & Carvalho, C. R. de O. (2013). Pancreatic islets isolated from Beta2 adrenergic recept or knockout mice show reduced insulin secretion in response to nutrients. Acta Scientiarum. Biological Sciences, 35( 3), 437-443. doi:10.4025/actascibiolsci.v35i3.15842
NLM
Marçal AC, Davel APC, Carpinelli AR, Brum PC, Rossoni LV, Carvalho CR de O. Pancreatic islets isolated from Beta2 adrenergic recept or knockout mice show reduced insulin secretion in response to nutrients [Internet]. Acta Scientiarum. Biological Sciences. 2013 ; 35( 3): 437-443.[citado 2024 jul. 08 ] Available from: https://doi.org/10.4025/actascibiolsci.v35i3.15842
Vancouver
Marçal AC, Davel APC, Carpinelli AR, Brum PC, Rossoni LV, Carvalho CR de O. Pancreatic islets isolated from Beta2 adrenergic recept or knockout mice show reduced insulin secretion in response to nutrients [Internet]. Acta Scientiarum. Biological Sciences. 2013 ; 35( 3): 437-443.[citado 2024 jul. 08 ] Available from: https://doi.org/10.4025/actascibiolsci.v35i3.15842
Artigo de periodico
Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay (2012)
Bocalini, Danilo Sales; Dos-Santos, Leonardo; Antonio, Ednei Luiz; Santos, Alexandra Alberta dos; Davel, Ana Paula; Rossoni, Luciana Venturini ; Vassallo, Dalton Valentim; Tucci, Paulo José Ferreira
Fonte: Arquivos Brasileiros de Cardiologia. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BOCALINI, Danilo Sales et al. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay. Arquivos Brasileiros de Cardiologia, v. 98, n. 3, p. 243-251, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0066-782X2012005000016. Acesso em: 08 jul. 2024.
APA
Bocalini, D. S., Dos-Santos, L., Antonio, E. L., Santos, A. A. dos, Davel, A. P., Rossoni, L. V., et al. (2012). Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay. Arquivos Brasileiros de Cardiologia, 98( 3), 243-251. doi:10.1590/S0066-782X2012005000016
NLM
Bocalini DS, Dos-Santos L, Antonio EL, Santos AA dos, Davel AP, Rossoni LV, Vassallo DV, Tucci PJF. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay [Internet]. Arquivos Brasileiros de Cardiologia. 2012 ; 98( 3): 243-251.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
Vancouver
Bocalini DS, Dos-Santos L, Antonio EL, Santos AA dos, Davel AP, Rossoni LV, Vassallo DV, Tucci PJF. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay [Internet]. Arquivos Brasileiros de Cardiologia. 2012 ; 98( 3): 243-251.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
Artigo de periodico
Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update (2011)
Davel, A. P.; Wenceslau, Camilla Ferreira; Akamine, Eliana Hiromi ; Xavier, F. E.; Couto, Gisele Kruger; Oliveira, H. T.; Rossoni, Luciana Venturini
Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
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ABNT
DAVEL, A. P. et al. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 920-932, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500104. Acesso em: 08 jul. 2024.
APA
Davel, A. P., Wenceslau, C. F., Akamine, E. H., Xavier, F. E., Couto, G. K., Oliveira, H. T., & Rossoni, L. V. (2011). Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, 44( 9), 920-932. doi:10.1590/S0100-879X2011007500104
NLM
Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
Vancouver
Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
Artigo de periodico
Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats (2011)
Delbin, Maria Andréia; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Antunes, Edson; Zanesco, Angelina
Fonte: Inflammation. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
DELBIN, Maria Andréia et al. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, v. 34, n. 5, p. 319-325, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10753-010-9237-9. Acesso em: 08 jul. 2024.
APA
Delbin, M. A., Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2011). Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, 34( 5), 319-325. doi:10.1007/s10753-010-9237-9
NLM
Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
Vancouver
Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
Artigo de periodico
The antiapoptotic effect of granulocyte colonystimulating factor reduces infarct size and prevents heart failure development in rats (2011)
Baldo, Marcelo P.; Davel, Ana P. C.; Damas-Souza, Danilo M.; Nicoletti-Carvalho, José Edgar; Bordin, Silvana ; Carvalho, Hernandes F.; Rodrigues, Sérgio L.; Rossoni, Luciana Venturini ; Mill, José G.
Fonte: Cellular Physiology and Biochemistry. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BALDO, Marcelo P. et al. The antiapoptotic effect of granulocyte colonystimulating factor reduces infarct size and prevents heart failure development in rats. Cellular Physiology and Biochemistry, v. 28, n. 1, p. 33-40, 2011Tradução . . Disponível em: https://doi.org/10.1159/000331711. Acesso em: 08 jul. 2024.
APA
Baldo, M. P., Davel, A. P. C., Damas-Souza, D. M., Nicoletti-Carvalho, J. E., Bordin, S., Carvalho, H. F., et al. (2011). The antiapoptotic effect of granulocyte colonystimulating factor reduces infarct size and prevents heart failure development in rats. Cellular Physiology and Biochemistry, 28( 1), 33-40. doi:10.1159/000331711
NLM
Baldo MP, Davel APC, Damas-Souza DM, Nicoletti-Carvalho JE, Bordin S, Carvalho HF, Rodrigues SL, Rossoni LV, Mill JG. The antiapoptotic effect of granulocyte colonystimulating factor reduces infarct size and prevents heart failure development in rats [Internet]. Cellular Physiology and Biochemistry. 2011 ; 28( 1): 33-40.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1159/000331711
Vancouver
Baldo MP, Davel APC, Damas-Souza DM, Nicoletti-Carvalho JE, Bordin S, Carvalho HF, Rodrigues SL, Rossoni LV, Mill JG. The antiapoptotic effect of granulocyte colonystimulating factor reduces infarct size and prevents heart failure development in rats [Internet]. Cellular Physiology and Biochemistry. 2011 ; 28( 1): 33-40.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1159/000331711
Artigo de periodico
L-NAME treatment enhances exercise-induced content of myocardial heat shock protein 72 (Hsp72) in rats (2011)
Lunz, Wellington; Capettini, Luciano S. A.; Davel, Ana P. C.; Munhoz, Carolina Demarchi ; Silva, Josiane F. da; Rossoni, Luciana Venturini ; Lemos, Virgínia S.; Baldo, Marcelo P.; Carneiro-Junior, Miguel A.; Natali, Antônio J.; Lacerda, Luiz H. S. de; Mill, José G.
Fonte: Cellular Physiology and Biochemistry. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
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ABNT
LUNZ, Wellington et al. L-NAME treatment enhances exercise-induced content of myocardial heat shock protein 72 (Hsp72) in rats. Cellular Physiology and Biochemistry, v. 27, n. 5, p. 479-486, 2011Tradução . . Disponível em: https://doi.org/10.1159/000329969. Acesso em: 08 jul. 2024.
APA
Lunz, W., Capettini, L. S. A., Davel, A. P. C., Munhoz, C. D., Silva, J. F. da, Rossoni, L. V., et al. (2011). L-NAME treatment enhances exercise-induced content of myocardial heat shock protein 72 (Hsp72) in rats. Cellular Physiology and Biochemistry, 27( 5), 479-486. doi:10.1159/000329969
NLM
Lunz W, Capettini LSA, Davel APC, Munhoz CD, Silva JF da, Rossoni LV, Lemos VS, Baldo MP, Carneiro-Junior MA, Natali AJ, Lacerda LHS de, Mill JG. L-NAME treatment enhances exercise-induced content of myocardial heat shock protein 72 (Hsp72) in rats [Internet]. Cellular Physiology and Biochemistry. 2011 ; 27( 5): 479-486.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1159/000329969
Vancouver
Lunz W, Capettini LSA, Davel APC, Munhoz CD, Silva JF da, Rossoni LV, Lemos VS, Baldo MP, Carneiro-Junior MA, Natali AJ, Lacerda LHS de, Mill JG. L-NAME treatment enhances exercise-induced content of myocardial heat shock protein 72 (Hsp72) in rats [Internet]. Cellular Physiology and Biochemistry. 2011 ; 27( 5): 479-486.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1159/000329969
Artigo de periodico
Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats (2008)
Moraes, Camila de ; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Antunes, Edson; Zanesco, Angelina
Fonte: BMC Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MORAES, Camila de et al. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, v. 8, 2008Tradução . . Disponível em: https://doi.org/10.1186/1472-6793-8-12. Acesso em: 08 jul. 2024.
APA
Moraes, C. de, Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2008). Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, 8. doi:10.1186/1472-6793-8-12
NLM
Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 jul. 08 ] Available from: https://doi.org/10.1186/1472-6793-8-12
Vancouver
Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 jul. 08 ] Available from: https://doi.org/10.1186/1472-6793-8-12

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