Fonte: Experimental physiology. Unidades: ICB, EEFE
Assuntos: FISIOLOGIA, SISTEMA CARDIOVASCULAR
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COUTO, Gisele Kruger et al. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, v. 99, n. 10, p. 1427–1438, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2014.079236. Acesso em: 10 set. 2024.APA
Couto, G. K., Davel, A. P., Brum, P. C., & Rossoni, L. V. (2014). Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling. Experimental physiology, 99( 10), 1427–1438. doi:10.1113/expphysiol.2014.079236NLM
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 set. 10 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236Vancouver
Couto GK, Davel AP, Brum PC, Rossoni LV. Double disruption of α2A- and α2C-adrenoceptors induces endothelial dysfunction in mouse small arteries: role of nitric oxide synthase uncoupling [Internet]. Experimental physiology. 2014 ; 99( 10): 1427–1438.[citado 2024 set. 10 ] Available from: https://doi.org/10.1113/expphysiol.2014.079236