Source: Blood. Conference titles: 65th ASH Annual Meeting Abstracts. Unidades: FMRP, ICB
Subjects: FARMACOLOGIA, HEMATOLOGIA, CÉLULAS MIELOIDES, LEUCEMIA MIELOIDE AGUDA, NEOPLASIAS POR TIPO HISTOLÓGICO, ANTINEOPLÁSICOS
ABNT
MELO, Manuela Albuquerque de et al. Autophagy induction constitutes a targetable vulnerability in FLT3-mutant acute Myeloid Leukemia cells treated with FLT3-inhibitors. Blood, v. 142, n. Supl. 1, p. 5752, 2023Tradução . . Disponível em: https://doi.org/10.1182/blood-2023-190724. Acesso em: 20 ago. 2024.APA
Melo, M. A. de, Macedo, B. G. S., Fonseca, N. P., Scheucher, P. S., Martins, D. A. P., Traina, F., & Machado Neto, J. A. (2023). Autophagy induction constitutes a targetable vulnerability in FLT3-mutant acute Myeloid Leukemia cells treated with FLT3-inhibitors. Blood, 142( Supl. 1), 5752. doi:10.1182/blood-2023-190724NLM
Melo MA de, Macedo BGS, Fonseca NP, Scheucher PS, Martins DAP, Traina F, Machado Neto JA. Autophagy induction constitutes a targetable vulnerability in FLT3-mutant acute Myeloid Leukemia cells treated with FLT3-inhibitors [Internet]. Blood. 2023 ; 142( Supl. 1): 5752.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1182/blood-2023-190724Vancouver
Melo MA de, Macedo BGS, Fonseca NP, Scheucher PS, Martins DAP, Traina F, Machado Neto JA. Autophagy induction constitutes a targetable vulnerability in FLT3-mutant acute Myeloid Leukemia cells treated with FLT3-inhibitors [Internet]. Blood. 2023 ; 142( Supl. 1): 5752.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1182/blood-2023-190724