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Artigo de periodico
Molecular-based score inspired on metabolic signature improves prognostic stratification for myelodysplastic syndrome (2021)
Silva, Juan Luiz Coelho ; Silveira, Douglas Rafaele Almeida ; Martins, Diego Antonio Pereira ; Rojas, Cesar A. O.; Araujo, Antonio R. Lucena ; Rego, Eduardo Magalhães ; Machado Neto, João Agostinho ; Bendit, Israel ; Rocha, Vanderson Geraldo ; Traina, Fabiola
Source: Scientific Reports. Unidades: FM, ICB, FMRP
Subjects: FARMACOLOGIA, FARMACOGENÉTICA, EXPRESSÃO GÊNICA, CICLO CELULAR, MEDULA ÓSSEA, DOENÇAS SANGUÍNEAS E LINFÁTICAS
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SILVA, Juan Luiz Coelho; SILVEIRA, Douglas Rafaele Almeida; MARTINS, Diego Antonio Pereira; et al. Molecular-based score inspired on metabolic signature improves prognostic stratification for myelodysplastic syndrome. Scientific Reports, London, Nature Publishing Group, v. 11, p. 11 , 2021. Disponível em: < https://doi.org/10.1038/s41598-020-80918-6 > DOI: 10.1038/s41598-020-80918-6.
APA
Silva, J. L. C., Silveira, D. R. A., Martins, D. A. P., Rojas, C. A. O., Araujo, A. R. L., Rego, E. M., et al. (2021). Molecular-based score inspired on metabolic signature improves prognostic stratification for myelodysplastic syndrome. Scientific Reports, 11, 11 . doi:10.1038/s41598-020-80918-6
NLM
Silva JLC, Silveira DRA, Martins DAP, Rojas CAO, Araujo ARL, Rego EM, Machado Neto JA, Bendit I, Rocha VG, Traina F. Molecular-based score inspired on metabolic signature improves prognostic stratification for myelodysplastic syndrome [Internet]. Scientific Reports. 2021 ; 11 11 .Available from: https://doi.org/10.1038/s41598-020-80918-6
Vancouver
Silva JLC, Silveira DRA, Martins DAP, Rojas CAO, Araujo ARL, Rego EM, Machado Neto JA, Bendit I, Rocha VG, Traina F. Molecular-based score inspired on metabolic signature improves prognostic stratification for myelodysplastic syndrome [Internet]. Scientific Reports. 2021 ; 11 11 .Available from: https://doi.org/10.1038/s41598-020-80918-6
Artigo de periodico
Non-canonical roles of connexins (2020)
Campenhout, Raf Van ; Cooreman, Axelle; Leroy, Kaat ; Rusiecka, Olga M; Van Brantegem, Pieter; Annaert, Pieter; Muyldermans, Serge ; Devoogdt, Nick ; Cogliati, Bruno ; Kwak, Brenda R; Vinken, Mathieu
Source: Progressin Biophysics and Molecular Biology. Unidade: FMVZ
Subjects: CONEXINAS, CICLO CELULAR
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CAMPENHOUT, Raf Van; COOREMAN, Axelle; LEROY, Kaat; et al. Non-canonical roles of connexins. Progressin Biophysics and Molecular Biology, Oxford, Elsevier, v. 153, p. 35-41, 2020. Disponível em: < https://dx.doi.org/10.1016/j.pbiomolbio.2020.03.002 > DOI: 10.1016/j.pbiomolbio.2020.03.002.
APA
Campenhout, R. V., Cooreman, A., Leroy, K., Rusiecka, O. M., Van Brantegem, P., Annaert, P., et al. (2020). Non-canonical roles of connexins. Progressin Biophysics and Molecular Biology, 153, 35-41. doi:10.1016/j.pbiomolbio.2020.03.002
NLM
Campenhout RV, Cooreman A, Leroy K, Rusiecka OM, Van Brantegem P, Annaert P, Muyldermans S, Devoogdt N, Cogliati B, Kwak BR, Vinken M. Non-canonical roles of connexins [Internet]. Progressin Biophysics and Molecular Biology. 2020 ; 153 35-41.Available from: https://dx.doi.org/10.1016/j.pbiomolbio.2020.03.002
Vancouver
Campenhout RV, Cooreman A, Leroy K, Rusiecka OM, Van Brantegem P, Annaert P, Muyldermans S, Devoogdt N, Cogliati B, Kwak BR, Vinken M. Non-canonical roles of connexins [Internet]. Progressin Biophysics and Molecular Biology. 2020 ; 153 35-41.Available from: https://dx.doi.org/10.1016/j.pbiomolbio.2020.03.002
Artigo de periodico
Melatonin inhibits human melanoma cells proliferation and invasion via cell cycle arrest and cytoskeleton remodeling (2020)
Moreno, Ana Carolina Ramos ; Saito, Renata de Freitas; Tiago, Manoela; Massaro, Renato Ramos; Pagni, Roberta Liberato; Pegoraro, Rafael; Souza, Patricia da Cruz; Reiter, Russel J. ; Campa, Ana ; Soengas, Maria S; Maria-Engler, Silvya Stuchi
Source: Melatonin Research. Unidades: FCF, ICB
Subjects: MELATONINA, CITOESQUELETO, CICLO CELULAR
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MORENO, Ana Carolina Ramos; SAITO, Renata de Freitas; TIAGO, Manoela; et al. Melatonin inhibits human melanoma cells proliferation and invasion via cell cycle arrest and cytoskeleton remodeling. Melatonin Research, San Antonio, v. 3, n. 2, p. 194-209, 2020. Disponível em: < http://dx.doi.org/10.32794/mr11250057 > DOI: 10.32794/mr11250057.
APA
Moreno, A. C. R., Saito, R. de F., Tiago, M., Massaro, R. R., Pagni, R. L., Pegoraro, R., et al. (2020). Melatonin inhibits human melanoma cells proliferation and invasion via cell cycle arrest and cytoskeleton remodeling. Melatonin Research, 3( 2), 194-209. doi:10.32794/mr11250057
NLM
Moreno ACR, Saito R de F, Tiago M, Massaro RR, Pagni RL, Pegoraro R, Souza P da C, Reiter RJ, Campa A, Soengas MS, Maria-Engler SS. Melatonin inhibits human melanoma cells proliferation and invasion via cell cycle arrest and cytoskeleton remodeling [Internet]. Melatonin Research. 2020 ; 3( 2): 194-209.Available from: http://dx.doi.org/10.32794/mr11250057
Vancouver
Moreno ACR, Saito R de F, Tiago M, Massaro RR, Pagni RL, Pegoraro R, Souza P da C, Reiter RJ, Campa A, Soengas MS, Maria-Engler SS. Melatonin inhibits human melanoma cells proliferation and invasion via cell cycle arrest and cytoskeleton remodeling [Internet]. Melatonin Research. 2020 ; 3( 2): 194-209.Available from: http://dx.doi.org/10.32794/mr11250057
Artigo de periodico
CDC7 (cell division cycle 7) (2020)
Vicari, Hugo Passos; Machado Neto, João Agostinho
Source: Atlas of Genetics and Cytogenetics in Oncology and Haematology. Unidade: ICB
Subjects: FARMACOLOGIA, PROTEÍNAS QUINASES, CROMOSSOMOS, DANO AO DNA, CICLO CELULAR, REPLICAÇÃO DO DNA, ANTINEOPLÁSICOS, CÉLULAS CULTIVADAS DE TUMOR
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VICARI, Hugo Passos; MACHADO NETO, João Agostinho. CDC7 (cell division cycle 7). Atlas of Genetics and Cytogenetics in Oncology and Haematology, Poitiers, atlas of genetics and cytogenetics in oncology and haematology, v. 24, n. 10, p. 358-365, 2020. DOI: 10.4267/2042/70816.
APA
Vicari, H. P., & Machado Neto, J. A. (2020). CDC7 (cell division cycle 7). Atlas of Genetics and Cytogenetics in Oncology and Haematology, 24( 10), 358-365. doi:10.4267/2042/70816
NLM
Vicari HP, Machado Neto JA. CDC7 (cell division cycle 7). Atlas of Genetics and Cytogenetics in Oncology and Haematology. 2020 ; 24( 10): 358-365.
Vancouver
Vicari HP, Machado Neto JA. CDC7 (cell division cycle 7). Atlas of Genetics and Cytogenetics in Oncology and Haematology. 2020 ; 24( 10): 358-365.
Artigo de periodico
DNA methyltransferase expression is associated with cell proliferation in salivary mucoepidermoid carcinoma (2020)
Guimarães, Douglas Magno ; Almeida, Fábio de Lucas da Silva; Castilho, Rogerio Moraes; Nör, Jacques Eduardo; Nunes, Fabio Daumas
Source: Journal of Oral Pathology & Medicine. Unidade: FO
Subjects: CICLO CELULAR, CARCINOMA DE CÉLULAS ESCAMOSAS
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GUIMARÃES, Douglas Magno; ALMEIDA, Fábio de Lucas da Silva; CASTILHO, Rogerio Moraes; NÖR, Jacques Eduardo; NUNES, Fabio Daumas. DNA methyltransferase expression is associated with cell proliferation in salivary mucoepidermoid carcinoma. Journal of Oral Pathology & Medicine, Chichester, Wiley-Blackwell Publishing Ltd, v. No 2020, n. 10, p. 1053-60, 2020. DOI: 10.1111/jop.13092.
APA
Guimarães, D. M., Almeida, F. de L. da S., Castilho, R. M., Nör, J. E., & Nunes, F. D. (2020). DNA methyltransferase expression is associated with cell proliferation in salivary mucoepidermoid carcinoma. Journal of Oral Pathology & Medicine, No 2020( 10), 1053-60. doi:10.1111/jop.13092
NLM
Guimarães DM, Almeida F de L da S, Castilho RM, Nör JE, Nunes FD. DNA methyltransferase expression is associated with cell proliferation in salivary mucoepidermoid carcinoma. Journal of Oral Pathology & Medicine. 2020 ; No 2020( 10): 1053-60.
Vancouver
Guimarães DM, Almeida F de L da S, Castilho RM, Nör JE, Nunes FD. DNA methyltransferase expression is associated with cell proliferation in salivary mucoepidermoid carcinoma. Journal of Oral Pathology & Medicine. 2020 ; No 2020( 10): 1053-60.
Artigo de periodico
Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation (2020)
Cavalcante, Isadora Pontes ; Vaczlavik, Anna; Drougat, Ludivine; Lotfi, Claudimara Ferini Pacicco ; Perlemoine, Karine; Ribes, Christopher; Rabin, Marthe Rizk ; Clauser, Eric ; Fragoso, Maria Candida Barisson Villares ; Bertherat, Jérôme ; Ragazzon, Bruno
Source: Endocrine - Related Cancer. Unidades: ICB, FM
Subjects: ANATOMIA, NEOPLASIAS DE TECIDO NERVOSO, MENINGIOMA, CÉLULAS CULTIVADAS DE TUMOR, GENES SUPRESSORES DE TUMOR, REGULAÇÃO NEOPLÁSICA DA EXPRESSÃO GÊNICA, TRANSCRIÇÃO GÊNICA, CICLO CELULAR, MUTAÇÃO GENÉTICA
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ABNT
CAVALCANTE, Isadora Pontes; VACZLAVIK, Anna; DROUGAT, Ludivine; et al. Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation. Endocrine - Related Cancer, Bristol, BioScientifica Ltd, v. 27, n. 4, p. 221-230, 2020. Disponível em: < https://doi.org/10.1530/ERC-19-0502 > DOI: 10.1530/ERC-19-0502.
APA
Cavalcante, I. P., Vaczlavik, A., Drougat, L., Lotfi, C. F. P., Perlemoine, K., Ribes, C., et al. (2020). Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation. Endocrine - Related Cancer, 27( 4), 221-230. doi:10.1530/ERC-19-0502
NLM
Cavalcante IP, Vaczlavik A, Drougat L, Lotfi CFP, Perlemoine K, Ribes C, Rabin MR, Clauser E, Fragoso MCBV, Bertherat J, Ragazzon B. Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation [Internet]. Endocrine - Related Cancer. 2020 ; 27( 4): 221-230.Available from: https://doi.org/10.1530/ERC-19-0502
Vancouver
Cavalcante IP, Vaczlavik A, Drougat L, Lotfi CFP, Perlemoine K, Ribes C, Rabin MR, Clauser E, Fragoso MCBV, Bertherat J, Ragazzon B. Cullin 3 targets the tumor suppressor gene ARMC5 for ubiquitination and degradation [Internet]. Endocrine - Related Cancer. 2020 ; 27( 4): 221-230.Available from: https://doi.org/10.1530/ERC-19-0502
Artigo de periodico
Cardioprotection Generated by Aerobic Exercise Training is Not Related to the Proliferation of Cardiomyocytes and Angiotensin-(1-7) Levels in the Hearts of Rats with Supravalvar Aortic Stenosis (2020)
Mota, Gustavo Augusto Ferreira; Souza, Sérgio Luiz Borges de; Silva, Vitor Loureiro da; Gatto, Mariana; Campos, Dijon Henrique Salomé de; Sant’Ana, Paula Grippa; Vileigas, Danielle Fernandes; Padovani, Carlos Roberto; Casarini, Dulce Elena; Oliveira, Edilamar Menezes de ; Bazan, Silméia Garcia Zanati; Fernandes, Tiago ; Sugizaki, Mario Mateus; Cicogna, Antonio Carlos; Gomes, Enéas Ricardo de Morais
Source: Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology. Unidade: EEFE
Subjects: TREINAMENTO FÍSICO, CICLO CELULAR, SISTEMA RENINA-ANGIOTENSINA, INSUFICIÊNCIA CARDÍACA
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MOTA, Gustavo Augusto Ferreira; SOUZA, Sérgio Luiz Borges de; SILVA, Vitor Loureiro da; et al. Cardioprotection Generated by Aerobic Exercise Training is Not Related to the Proliferation of Cardiomyocytes and Angiotensin-(1-7) Levels in the Hearts of Rats with Supravalvar Aortic Stenosis. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, Dusseldorf, v. 54, p. 719-735, 2020. Disponível em: < https://doi.org/10.33594/000000251 > DOI: 10.33594/000000251.
APA
Mota, G. A. F., Souza, S. L. B. de, Silva, V. L. da, Gatto, M., Campos, D. H. S. de, Sant’Ana, P. G., et al. (2020). Cardioprotection Generated by Aerobic Exercise Training is Not Related to the Proliferation of Cardiomyocytes and Angiotensin-(1-7) Levels in the Hearts of Rats with Supravalvar Aortic Stenosis. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 54, 719-735. doi:10.33594/000000251
NLM
Mota GAF, Souza SLB de, Silva VL da, Gatto M, Campos DHS de, Sant’Ana PG, Vileigas DF, Padovani CR, Casarini DE, Oliveira EM de, Bazan SGZ, Fernandes T, Sugizaki MM, Cicogna AC, Gomes ER de M. Cardioprotection Generated by Aerobic Exercise Training is Not Related to the Proliferation of Cardiomyocytes and Angiotensin-(1-7) Levels in the Hearts of Rats with Supravalvar Aortic Stenosis [Internet]. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology. 2020 ; 54 719-735.Available from: https://doi.org/10.33594/000000251
Vancouver
Mota GAF, Souza SLB de, Silva VL da, Gatto M, Campos DHS de, Sant’Ana PG, Vileigas DF, Padovani CR, Casarini DE, Oliveira EM de, Bazan SGZ, Fernandes T, Sugizaki MM, Cicogna AC, Gomes ER de M. Cardioprotection Generated by Aerobic Exercise Training is Not Related to the Proliferation of Cardiomyocytes and Angiotensin-(1-7) Levels in the Hearts of Rats with Supravalvar Aortic Stenosis [Internet]. Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology. 2020 ; 54 719-735.Available from: https://doi.org/10.33594/000000251
Artigo de periodico
RHOAming through the nucleotide excision repair pathway as a mechanism of cellular response against the effects of UV radiation (2020)
Magalhães, Yuli Thamires ; Silva, Gisele E. T; Osaki, Juliana H; Rocha, Clarissa R. R; Forti, Fabio Luis
Source: Frontiers in Cell and Developmental Biology. Unidade: IQ
Subjects: RADIAÇÃO ULTRAVIOLETA, CICLO CELULAR
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ABNT
MAGALHÃES, Yuli Thamires; SILVA, Gisele E. T; OSAKI, Juliana H; ROCHA, Clarissa R. R; FORTI, Fabio Luis. RHOAming through the nucleotide excision repair pathway as a mechanism of cellular response against the effects of UV radiation. Frontiers in Cell and Developmental Biology, Lausanne, v. 8, p. 1-19 art. 816, 2020. Disponível em: < http://dx.doi.org/10.3389/fcell.2020.00816 > DOI: 10.3389/fcell.2020.00816.
APA
Magalhães, Y. T., Silva, G. E. T., Osaki, J. H., Rocha, C. R. R., & Forti, F. L. (2020). RHOAming through the nucleotide excision repair pathway as a mechanism of cellular response against the effects of UV radiation. Frontiers in Cell and Developmental Biology, 8, 1-19 art. 816. doi:10.3389/fcell.2020.00816
NLM
Magalhães YT, Silva GET, Osaki JH, Rocha CRR, Forti FL. RHOAming through the nucleotide excision repair pathway as a mechanism of cellular response against the effects of UV radiation [Internet]. Frontiers in Cell and Developmental Biology. 2020 ; 8 1-19 art. 816.Available from: http://dx.doi.org/10.3389/fcell.2020.00816
Vancouver
Magalhães YT, Silva GET, Osaki JH, Rocha CRR, Forti FL. RHOAming through the nucleotide excision repair pathway as a mechanism of cellular response against the effects of UV radiation [Internet]. Frontiers in Cell and Developmental Biology. 2020 ; 8 1-19 art. 816.Available from: http://dx.doi.org/10.3389/fcell.2020.00816
Artigo de periodico
Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes (2020)
Assis, Leonardo Vinícius Monteiro de ; Mendes, Davi; Silva, Matheus Molina; Kinker, Gabriela Sarti ; Lima, Isabella Pereira; Borges, Maria Nathália de Carvalho Magalhães Moraes Figueira ; Menck, Carlos Frederico Martins ; Castrucci, Ana Maria de Lauro
Source: Biochimica et Biophysica Acta. Molecular Cell Research. Unidades: ICB, IB
Subjects: MICROBIOLOGIA, APOPTOSE, RITMOS BIOLÓGICOS, PIGMENTOS, MELANINAS, MELANOMA, CICLO CELULAR, RADIAÇÃO ULTRAVIOLETA, PELE, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS CUTÂNEAS
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ABNT
ASSIS, Leonardo Vinícius Monteiro de; MENDES, Davi; SILVA, Matheus Molina; et al. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes. Biochimica et Biophysica Acta. Molecular Cell Research, Amsterdam, Elsevier BV, v. 1867, n. 10, p. 10 , 2020. Disponível em: < https://doi.org/10.1016/j.bbamcr.2020.118789 > DOI: 10.1016/j.bbamcr.2020.118789.
APA
Assis, L. V. M. de, Mendes, D., Silva, M. M., Kinker, G. S., Lima, I. P., Borges, M. N. de C. M. M. F., et al. (2020). Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes. Biochimica et Biophysica Acta. Molecular Cell Research, 1867( 10), 10 . doi:10.1016/j.bbamcr.2020.118789
NLM
Assis LVM de, Mendes D, Silva MM, Kinker GS, Lima IP, Borges MN de CMMF, Menck CFM, Castrucci AM de L. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes [Internet]. Biochimica et Biophysica Acta. Molecular Cell Research. 2020 ; 1867( 10): 10 .Available from: https://doi.org/10.1016/j.bbamcr.2020.118789
Vancouver
Assis LVM de, Mendes D, Silva MM, Kinker GS, Lima IP, Borges MN de CMMF, Menck CFM, Castrucci AM de L. Melanopsin mediates UVA-dependent modulation of proliferation, pigmentation, apoptosis, and molecular clock in normal and malignant melanocytes [Internet]. Biochimica et Biophysica Acta. Molecular Cell Research. 2020 ; 1867( 10): 10 .Available from: https://doi.org/10.1016/j.bbamcr.2020.118789
Artigo de periodico
A tetraprenylated benzophenone 7-epiclusianone induces cell cycle arrest at G1/S transition by modulating critical regulators of cell cycle in breast cancer cell lines (2020)
Lamartine-Hanemann, Simone da Silva; Silva, Guilherme Álvaro Ferreira; Horvath, Renato de Oliveira ; Soncini, Roseli ; Caixeta, Ester Siqueira ; Rocha-Sales, Bianca; Niero, Evandro Luís ; Santos, Marcelo Henrique dos; Oliveira, Jaqueline Carvalho de; Miyazawwa, Marta; Ionta, Marisa ; Machado-Santelli, Glaucia Maria
Source: Toxicology in Vitro. Unidade: ICB
Subjects: BIOLOGIA CELULAR, NEOPLASIAS MAMÁRIAS, CÉLULAS CULTIVADAS DE TUMOR, CICLO CELULAR, PROLIFERAÇÃO CELULAR, LINHAGEM CELULAR, APOPTOSE, ANTINEOPLÁSICOS, CICLINA D1
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ABNT
LAMARTINE-HANEMANN, Simone da Silva; SILVA, Guilherme Álvaro Ferreira; HORVATH, Renato de Oliveira; et al. A tetraprenylated benzophenone 7-epiclusianone induces cell cycle arrest at G1/S transition by modulating critical regulators of cell cycle in breast cancer cell lines. Toxicology in Vitro, Oxford, Elsevier Science & Technology, v. 68, p. 14 , 2020. Disponível em: < https://doi.org/10.1016/j.tiv.2020.104927 > DOI: 10.1016/j.tiv.2020.104927.
APA
Lamartine-Hanemann, S. da S., Silva, G. Á. F., Horvath, R. de O., Soncini, R., Caixeta, E. S., Rocha-Sales, B., et al. (2020). A tetraprenylated benzophenone 7-epiclusianone induces cell cycle arrest at G1/S transition by modulating critical regulators of cell cycle in breast cancer cell lines. Toxicology in Vitro, 68, 14 . doi:10.1016/j.tiv.2020.104927
NLM
Lamartine-Hanemann S da S, Silva GÁF, Horvath R de O, Soncini R, Caixeta ES, Rocha-Sales B, Niero EL, Santos MH dos, Oliveira JC de, Miyazawwa M, Ionta M, Machado-Santelli GM. A tetraprenylated benzophenone 7-epiclusianone induces cell cycle arrest at G1/S transition by modulating critical regulators of cell cycle in breast cancer cell lines [Internet]. Toxicology in Vitro. 2020 ; 68 14 .Available from: https://doi.org/10.1016/j.tiv.2020.104927
Vancouver
Lamartine-Hanemann S da S, Silva GÁF, Horvath R de O, Soncini R, Caixeta ES, Rocha-Sales B, Niero EL, Santos MH dos, Oliveira JC de, Miyazawwa M, Ionta M, Machado-Santelli GM. A tetraprenylated benzophenone 7-epiclusianone induces cell cycle arrest at G1/S transition by modulating critical regulators of cell cycle in breast cancer cell lines [Internet]. Toxicology in Vitro. 2020 ; 68 14 .Available from: https://doi.org/10.1016/j.tiv.2020.104927
Artigo de periodico
NEK10 interactome and depletion reveal new roles in mitochondria (2020)
Oliveira, Andressa Peres de; Basei, Fernanda Luisa; Slepicka, Priscila Ferreira ; Ferezin, Camila de Castro; Hanchuk, Talita D. Melo ; Souza, Edmarcia Elisa de ; Lima, Tanes I. ; Santos, Valquiria Tiago dos; Mendes, Davi; Silveira, Leonardo Reis; Menck, Carlos Frederico Martins ; Kobarg, Jörg
Source: Proteome Science. Unidade: ICB
Subjects: MICROBIOLOGIA, DANO AO DNA, CICLO CELULAR, PROTEÍNAS QUINASES, MICROSCOPIA CONFOCAL, MITOCÔNDRIAS, METABOLISMO, PROTEÔMICA
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ABNT
OLIVEIRA, Andressa Peres de; BASEI, Fernanda Luisa; SLEPICKA, Priscila Ferreira; et al. NEK10 interactome and depletion reveal new roles in mitochondria. Proteome Science, London, BioMed Central Ltd, v. 18, p. 17 , 2020. Disponível em: < https://doi.org/10.1186/s12953-020-00160-w > DOI: https://doi.org/10.1186/s12953-020-00160-w.
APA
Oliveira, A. P. de, Basei, F. L., Slepicka, P. F., Ferezin, C. de C., Hanchuk, T. D. M., Souza, E. E. de, et al. (2020). NEK10 interactome and depletion reveal new roles in mitochondria. Proteome Science, 18, 17 . doi:https://doi.org/10.1186/s12953-020-00160-w
NLM
Oliveira AP de, Basei FL, Slepicka PF, Ferezin C de C, Hanchuk TDM, Souza EE de, Lima TI, Santos VT dos, Mendes D, Silveira LR, Menck CFM, Kobarg J. NEK10 interactome and depletion reveal new roles in mitochondria [Internet]. Proteome Science. 2020 ; 18 17 .Available from: https://doi.org/10.1186/s12953-020-00160-w
Vancouver
Oliveira AP de, Basei FL, Slepicka PF, Ferezin C de C, Hanchuk TDM, Souza EE de, Lima TI, Santos VT dos, Mendes D, Silveira LR, Menck CFM, Kobarg J. NEK10 interactome and depletion reveal new roles in mitochondria [Internet]. Proteome Science. 2020 ; 18 17 .Available from: https://doi.org/10.1186/s12953-020-00160-w
Tese
BKM-120 reduz a viabilidade celular e secreção de hormônio adrenocorticotrófico em linhagem celular de tumores corticotróficos de camundongo AtT-20/D16v-F2 (2019)
Oliveira, Helen Alves de; Martins, Clarissa Silva (Orientador); Castro, Margaret de (Orientador)
Unidade: FMRP
Subjects: EXPRESSÃO GÊNICA, CICLO CELULAR, FÁRMACOS, HORMÔNIO ADRENOCORTICOTRÓFICO
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ABNT
OLIVEIRA, Helen Alves de; MARTINS, Clarissa Silva; CASTRO, Margaret de. BKM-120 reduz a viabilidade celular e secreção de hormônio adrenocorticotrófico em linhagem celular de tumores corticotróficos de camundongo AtT-20/D16v-F2. 2019.Universidade de São Paulo, Ribeirão Preto, 2019. Disponível em: < http://www.teses.usp.br/teses/disponiveis/17/17138/tde-10032020-135015/ >.
APA
Oliveira, H. A. de, Martins, C. S., & Castro, M. de. (2019). BKM-120 reduz a viabilidade celular e secreção de hormônio adrenocorticotrófico em linhagem celular de tumores corticotróficos de camundongo AtT-20/D16v-F2. Universidade de São Paulo, Ribeirão Preto. Recuperado de http://www.teses.usp.br/teses/disponiveis/17/17138/tde-10032020-135015/
NLM
Oliveira HA de, Martins CS, Castro M de. BKM-120 reduz a viabilidade celular e secreção de hormônio adrenocorticotrófico em linhagem celular de tumores corticotróficos de camundongo AtT-20/D16v-F2 [Internet]. 2019 ;Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-10032020-135015/
Vancouver
Oliveira HA de, Martins CS, Castro M de. BKM-120 reduz a viabilidade celular e secreção de hormônio adrenocorticotrófico em linhagem celular de tumores corticotróficos de camundongo AtT-20/D16v-F2 [Internet]. 2019 ;Available from: http://www.teses.usp.br/teses/disponiveis/17/17138/tde-10032020-135015/
Trabalho de evento-resumo
SCI1 (stigma/style cell-cycle inhibitor 1) and its interaction partner, NtDDX41, are new plant spliceosome-associated factors (2019)
Pinoti, Vitor Favaretto ; Strini, Edward José; Aziani, Rodrigo; Ferreira, Pedro Boscariol ; Lubini, Greice ; Thomé, Vanessa ; Cruz, Joelma de Oliveira ; Quiapim, Andréa Carla; Goldman, Gustavo Henrique ; DePaoli, Henrique Cestari; Goldman, Maria Helena de Souza
Source: Resumos. Conference titles: Brazilian Congress of Genetics. Unidades: FCFRP, FFCLRP, FMRP
Subjects: CICLO CELULAR, RNA, PROLIFERAÇÃO CELULAR, PROTEÍNAS DE PLANTAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINOTI, Vitor Favaretto; STRINI, Edward José; AZIANI, Rodrigo; et al. SCI1 (stigma/style cell-cycle inhibitor 1) and its interaction partner, NtDDX41, are new plant spliceosome-associated factors. Anais.. Águas de Lindóia: Sociedade Brasileira de Genética, 2019.Disponível em: .
APA
Pinoti, V. F., Strini, E. J., Aziani, R., Ferreira, P. B., Lubini, G., Thomé, V., et al. (2019). SCI1 (stigma/style cell-cycle inhibitor 1) and its interaction partner, NtDDX41, are new plant spliceosome-associated factors. In Resumos. Águas de Lindóia: Sociedade Brasileira de Genética. Recuperado de https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
NLM
Pinoti VF, Strini EJ, Aziani R, Ferreira PB, Lubini G, Thomé V, Cruz J de O, Quiapim AC, Goldman GH, DePaoli HC, Goldman MH de S. SCI1 (stigma/style cell-cycle inhibitor 1) and its interaction partner, NtDDX41, are new plant spliceosome-associated factors [Internet]. Resumos. 2019 ;Available from: https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
Vancouver
Pinoti VF, Strini EJ, Aziani R, Ferreira PB, Lubini G, Thomé V, Cruz J de O, Quiapim AC, Goldman GH, DePaoli HC, Goldman MH de S. SCI1 (stigma/style cell-cycle inhibitor 1) and its interaction partner, NtDDX41, are new plant spliceosome-associated factors [Internet]. Resumos. 2019 ;Available from: https://www.sbg.org.br/sites/default/files/e_book_genetica2019_online_0.pdf
Tese
Regulação pré- e pós-transcricional do gene Scratch2 durante a neurogênese no tubo neural posterior (2019)
Goes, Carolina Purcell; Yan, Chao Yun Irene (Orientador)
Unidade: ICB
Subjects: NEURÔNIOS, REGULAÇÃO GÊNICA, DIFERENCIAÇÃO CELULAR, TRANSCRIÇÃO GÊNICA, CICLO CELULAR, EMBRIÃO DE GALINHA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GOES, Carolina Purcell; YAN, Chao Yun Irene. Regulação pré- e pós-transcricional do gene Scratch2 durante a neurogênese no tubo neural posterior. 2019.Universidade de São Paulo, São Paulo, 2019. Disponível em: < https://www.teses.usp.br/teses/disponiveis/42/42134/tde-12022020-153318/ >.
APA
Goes, C. P., & Yan, C. Y. I. (2019). Regulação pré- e pós-transcricional do gene Scratch2 durante a neurogênese no tubo neural posterior. Universidade de São Paulo, São Paulo. Recuperado de https://www.teses.usp.br/teses/disponiveis/42/42134/tde-12022020-153318/
NLM
Goes CP, Yan CYI. Regulação pré- e pós-transcricional do gene Scratch2 durante a neurogênese no tubo neural posterior [Internet]. 2019 ;Available from: https://www.teses.usp.br/teses/disponiveis/42/42134/tde-12022020-153318/
Vancouver
Goes CP, Yan CYI. Regulação pré- e pós-transcricional do gene Scratch2 durante a neurogênese no tubo neural posterior [Internet]. 2019 ;Available from: https://www.teses.usp.br/teses/disponiveis/42/42134/tde-12022020-153318/
Artigo de periodico
Somatic mutations in kinetochore gene KNSTRN are associated with basal proliferating actinic keratoses and cutaneous squamous cell carcinoma (2019)
Schmitz, L; Grinblat, B; Novak, B; Hoeh, A. -K; Haendschke, K; Dobbeler, C. von; Bierhoff, E; Szeimies, R. -M; Gambichler, T; Festa Neto, Cyro
Source: Journal of the european academy of dermatology and venereology. Unidade: FM
Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS, MUTAÇÃO GENÉTICA, CERATOSE, NEOPLASIAS CUTÂNEAS, PROLIFERAÇÃO CELULAR, CICLO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SCHMITZ, L; GRINBLAT, B; NOVAK, B; et al. Somatic mutations in kinetochore gene KNSTRN are associated with basal proliferating actinic keratoses and cutaneous squamous cell carcinoma. Journal of the european academy of dermatology and venereology, Hoboken, v. 33, n. 8, p. 1535-1540, 2019. Disponível em: < http://observatorio.fm.usp.br/handle/OPI/33469 > DOI: 10.1111/jdv.15615.
APA
Schmitz, L., Grinblat, B., Novak, B., Hoeh, A. -K., Haendschke, K., Dobbeler, C. von, et al. (2019). Somatic mutations in kinetochore gene KNSTRN are associated with basal proliferating actinic keratoses and cutaneous squamous cell carcinoma. Journal of the european academy of dermatology and venereology, 33( 8), 1535-1540. doi:10.1111/jdv.15615
NLM
Schmitz L, Grinblat B, Novak B, Hoeh A-K, Haendschke K, Dobbeler C von, Bierhoff E, Szeimies R-M, Gambichler T, Festa Neto C. Somatic mutations in kinetochore gene KNSTRN are associated with basal proliferating actinic keratoses and cutaneous squamous cell carcinoma [Internet]. Journal of the european academy of dermatology and venereology. 2019 ; 33( 8): 1535-1540.Available from: http://observatorio.fm.usp.br/handle/OPI/33469
Vancouver
Schmitz L, Grinblat B, Novak B, Hoeh A-K, Haendschke K, Dobbeler C von, Bierhoff E, Szeimies R-M, Gambichler T, Festa Neto C. Somatic mutations in kinetochore gene KNSTRN are associated with basal proliferating actinic keratoses and cutaneous squamous cell carcinoma [Internet]. Journal of the european academy of dermatology and venereology. 2019 ; 33( 8): 1535-1540.Available from: http://observatorio.fm.usp.br/handle/OPI/33469
Tese
Estresse oxidativo e alterações nos perfis de expressão gênica em idosos com comprometimento cognitivo leve (2019)
Vieira, Johnathan de Andrade; Hojo, Elza Tiemi Sakamoto (Orientador)
Unidade: FMRP
Subjects: IDOSOS, EXPRESSÃO GÊNICA, ESTRESSE OXIDATIVO, COMPROMETIMENTO COGNITIVO LEVE, CICLO CELULAR, ENVELHECIMENTO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VIEIRA, Johnathan de Andrade; HOJO, Elza Tiemi Sakamoto. Estresse oxidativo e alterações nos perfis de expressão gênica em idosos com comprometimento cognitivo leve. 2019.Universidade de São Paulo, Ribeirão Preto, 2019. Disponível em: < http://www.teses.usp.br/teses/disponiveis/17/17135/tde-13022020-105842/ >.
APA
Vieira, J. de A., & Hojo, E. T. S. (2019). Estresse oxidativo e alterações nos perfis de expressão gênica em idosos com comprometimento cognitivo leve. Universidade de São Paulo, Ribeirão Preto. Recuperado de http://www.teses.usp.br/teses/disponiveis/17/17135/tde-13022020-105842/
NLM
Vieira J de A, Hojo ETS. Estresse oxidativo e alterações nos perfis de expressão gênica em idosos com comprometimento cognitivo leve [Internet]. 2019 ;Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-13022020-105842/
Vancouver
Vieira J de A, Hojo ETS. Estresse oxidativo e alterações nos perfis de expressão gênica em idosos com comprometimento cognitivo leve [Internet]. 2019 ;Available from: http://www.teses.usp.br/teses/disponiveis/17/17135/tde-13022020-105842/
Artigo de periodico
BIRC5 (baculoviral IAP repeat containing 5) (2019)
Branco, Paola Cristina; Jimenez, Paula Christine; Machado-Neto, João Agostinho ; Lotufo, Leticia Veras Costa
Source: Atlas of Genetics and Cytogenetics in Oncology and Haematology. Unidade: ICB
Subjects: CICLO CELULAR, CÉLULAS CULTIVADAS DE TUMOR, APOPTOSE, NEOPLASIAS, EXPRESSÃO GÊNICA, CARCINOMA, MUTAÇÃO GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BRANCO, Paola Cristina; JIMENEZ, Paula Christine; MACHADO-NETO, João Agostinho; LOTUFO, Leticia Veras Costa. BIRC5 (baculoviral IAP repeat containing 5). Atlas of Genetics and Cytogenetics in Oncology and Haematology, Poitiers, Atlas of Genetics and Cytogenetics in Oncology and Haematology, v. 23, n. 7, p. 168-186, 2019. Disponível em: < https://doi.org/10.4267/2042/70468 > DOI: 10.4267/2042/70468.
APA
Branco, P. C., Jimenez, P. C., Machado-Neto, J. A., & Lotufo, L. V. C. (2019). BIRC5 (baculoviral IAP repeat containing 5). Atlas of Genetics and Cytogenetics in Oncology and Haematology, 23( 7), 168-186. doi:10.4267/2042/70468
NLM
Branco PC, Jimenez PC, Machado-Neto JA, Lotufo LVC. BIRC5 (baculoviral IAP repeat containing 5) [Internet]. Atlas of Genetics and Cytogenetics in Oncology and Haematology. 2019 ; 23( 7): 168-186.Available from: https://doi.org/10.4267/2042/70468
Vancouver
Branco PC, Jimenez PC, Machado-Neto JA, Lotufo LVC. BIRC5 (baculoviral IAP repeat containing 5) [Internet]. Atlas of Genetics and Cytogenetics in Oncology and Haematology. 2019 ; 23( 7): 168-186.Available from: https://doi.org/10.4267/2042/70468
Artigo de periodico
Antiproliferative and apoptotic effects of caffeic acid on SK-Mel-28 human melanoma cancer cells (2019)
Pelinson, Luana Paula; Assmann, Charles Elias; Palma, Tais Vidal; Cruz, Ivana Beatrice Mânica da; Pillat, Micheli Mainardi; Manica, Aline; Stefanello, Naiara; Weis, Grazielle Castagna Cezimbra; Alves, Audrei de Oliveira; Andrade, Cínthia Melazzo de; Ulrich, Henning ; Morsch, Vera Maria Melchiors; Chitolina Schetinger, Maria Rosa; Bagatini, Margarete Dulce
Source: Molecular Biology Reports. Unidade: IQ
Subjects: MELANOMA, EXPRESSÃO GÊNICA, CICLO CELULAR, APOPTOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PELINSON, Luana Paula; ASSMANN, Charles Elias; PALMA, Tais Vidal; et al. Antiproliferative and apoptotic effects of caffeic acid on SK-Mel-28 human melanoma cancer cells. Molecular Biology Reports, Dordrecht, v. 46, n. 2, p. 2085-2092, 2019. Disponível em: < http://dx.doi.org/10.1007/s11033-019-04658-1 > DOI: 10.1007/s11033-019-04658-1.
APA
Pelinson, L. P., Assmann, C. E., Palma, T. V., Cruz, I. B. M. da, Pillat, M. M., Manica, A., et al. (2019). Antiproliferative and apoptotic effects of caffeic acid on SK-Mel-28 human melanoma cancer cells. Molecular Biology Reports, 46( 2), 2085-2092. doi:10.1007/s11033-019-04658-1
NLM
Pelinson LP, Assmann CE, Palma TV, Cruz IBM da, Pillat MM, Manica A, Stefanello N, Weis GCC, Alves A de O, Andrade CM de, Ulrich H, Morsch VMM, Chitolina Schetinger MR, Bagatini MD. Antiproliferative and apoptotic effects of caffeic acid on SK-Mel-28 human melanoma cancer cells [Internet]. Molecular Biology Reports. 2019 ; 46( 2): 2085-2092.Available from: http://dx.doi.org/10.1007/s11033-019-04658-1
Vancouver
Pelinson LP, Assmann CE, Palma TV, Cruz IBM da, Pillat MM, Manica A, Stefanello N, Weis GCC, Alves A de O, Andrade CM de, Ulrich H, Morsch VMM, Chitolina Schetinger MR, Bagatini MD. Antiproliferative and apoptotic effects of caffeic acid on SK-Mel-28 human melanoma cancer cells [Internet]. Molecular Biology Reports. 2019 ; 46( 2): 2085-2092.Available from: http://dx.doi.org/10.1007/s11033-019-04658-1
Artigo de periodico
Oncogenic signaling pathways in the cancer genome atlas (2018)
Sanchez-Vega, Francisco; Noushmehr, Houtan; Carlotti Júnior, Carlos Gilberto; Santos, José Sebastião dos; Kemp, Rafael; Sankarankutty, Ajith Kumar; Tirapelli, Daniela Pretti da Cunha
Source: Cell. Unidade: FMRP
Subjects: GENÔMICA, NEOPLASIAS, ONCOLOGIA, TERAPÊUTICA, CICLO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANCHEZ-VEGA, Francisco; NOUSHMEHR, Houtan; CARLOTTI JÚNIOR, Carlos Gilberto; et al. Oncogenic signaling pathways in the cancer genome atlas. Cell, Cambridge, v. 173, n. 2, p. 321-337.e1-e4, 2018. Disponível em: < http://dx.doi.org/10.1016/j.cell.2018.03.035 > DOI: 10.1016/j.cell.2018.03.035.
APA
Sanchez-Vega, F., Noushmehr, H., Carlotti Júnior, C. G., Santos, J. S. dos, Kemp, R., Sankarankutty, A. K., & Tirapelli, D. P. da C. (2018). Oncogenic signaling pathways in the cancer genome atlas. Cell, 173( 2), 321-337.e1-e4. doi:10.1016/j.cell.2018.03.035
NLM
Sanchez-Vega F, Noushmehr H, Carlotti Júnior CG, Santos JS dos, Kemp R, Sankarankutty AK, Tirapelli DP da C. Oncogenic signaling pathways in the cancer genome atlas [Internet]. Cell. 2018 ; 173( 2): 321-337.e1-e4.Available from: http://dx.doi.org/10.1016/j.cell.2018.03.035
Vancouver
Sanchez-Vega F, Noushmehr H, Carlotti Júnior CG, Santos JS dos, Kemp R, Sankarankutty AK, Tirapelli DP da C. Oncogenic signaling pathways in the cancer genome atlas [Internet]. Cell. 2018 ; 173( 2): 321-337.e1-e4.Available from: http://dx.doi.org/10.1016/j.cell.2018.03.035
Artigo de periodico
lncRNA epigenetic landscape analysis identifies EPIC1 as an oncogenic lncRNA that interacts with MYC and promotes cell-cycle progression in cancer (2018)
Wang, Zehua; Yang, Bob; Zhang, Min; Guo, Weiwei; Wu, Zhiyuan; Wang, Yue; Jia, Lin; Li, Song; Xie, Wen; Yang, Da; Carlotti Júnior, Carlos Gilberto; Santos, José Sebastião dos; Kemp, Rafael; Sankarankuty, Ajith Kumar; Tirapelli, Daniela Pretti da Cunha
Source: Cancer Cell. Unidade: FMRP
Subjects: NEOPLASIAS MAMÁRIAS, EPIGÊNESE GENÉTICA, METILAÇÃO DE DNA, CICLO CELULAR, FENÓTIPOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WANG, Zehua; YANG, Bob; ZHANG, Min; et al. lncRNA epigenetic landscape analysis identifies EPIC1 as an oncogenic lncRNA that interacts with MYC and promotes cell-cycle progression in cancer. Cancer Cell, Cambridge, v. 33, n. 4, p. 706-720, 2018. Disponível em: < http://dx.doi.org/10.1016/j.ccell.2018.03.006 > DOI: 10.1016/j.ccell.2018.03.006.
APA
Wang, Z., Yang, B., Zhang, M., Guo, W., Wu, Z., Wang, Y., et al. (2018). lncRNA epigenetic landscape analysis identifies EPIC1 as an oncogenic lncRNA that interacts with MYC and promotes cell-cycle progression in cancer. Cancer Cell, 33( 4), 706-720. doi:10.1016/j.ccell.2018.03.006
NLM
Wang Z, Yang B, Zhang M, Guo W, Wu Z, Wang Y, Jia L, Li S, Xie W, Yang D, Carlotti Júnior CG, Santos JS dos, Kemp R, Sankarankuty AK, Tirapelli DP da C. lncRNA epigenetic landscape analysis identifies EPIC1 as an oncogenic lncRNA that interacts with MYC and promotes cell-cycle progression in cancer [Internet]. Cancer Cell. 2018 ; 33( 4): 706-720.Available from: http://dx.doi.org/10.1016/j.ccell.2018.03.006
Vancouver
Wang Z, Yang B, Zhang M, Guo W, Wu Z, Wang Y, Jia L, Li S, Xie W, Yang D, Carlotti Júnior CG, Santos JS dos, Kemp R, Sankarankuty AK, Tirapelli DP da C. lncRNA epigenetic landscape analysis identifies EPIC1 as an oncogenic lncRNA that interacts with MYC and promotes cell-cycle progression in cancer [Internet]. Cancer Cell. 2018 ; 33( 4): 706-720.Available from: http://dx.doi.org/10.1016/j.ccell.2018.03.006

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