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Artigo de periodico
Toll-like receptor 4 signaling is critical for the adaptive cellular stress response effects induced by intermittent fasting in the mouse brain (2021)
Vasconcelos, Andrea Rodrigues ; Paixão, Amanda Galvão da ; Kinoshita, Paula Fernanda ; Orellana, Ana Maria Marques ; Scavone, Cristoforo ; Kawamoto, Elisa Mitiko
Source: Neuroscience. Unidade: ICB
Subjects: FARMACOLOGIA, MEMORIA, ESTRESSE OXIDATIVO, JEJUNO DE ANIMAL, CÉREBRO, CAMUNDONGOS, PROTEÍNAS QUINASES, TRANSCRIÇÃO GÊNICA, FATORES DE CRESCIMENTO, PROTEÍNAS DO CHOQUE TÉRMICO, HIPOCAMPU DE ANIMAL
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ABNT
VASCONCELOS, Andrea Rodrigues et al. Toll-like receptor 4 signaling is critical for the adaptive cellular stress response effects induced by intermittent fasting in the mouse brain. Neuroscience, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2021.04.022. Acesso em: 05 nov. 2024.
APA
Vasconcelos, A. R., Paixão, A. G. da, Kinoshita, P. F., Orellana, A. M. M., Scavone, C., & Kawamoto, E. M. (2021). Toll-like receptor 4 signaling is critical for the adaptive cellular stress response effects induced by intermittent fasting in the mouse brain. Neuroscience. doi:10.1016/j.neuroscience.2021.04.022
NLM
Vasconcelos AR, Paixão AG da, Kinoshita PF, Orellana AMM, Scavone C, Kawamoto EM. Toll-like receptor 4 signaling is critical for the adaptive cellular stress response effects induced by intermittent fasting in the mouse brain [Internet]. Neuroscience. 2021 ;[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2021.04.022
Vancouver
Vasconcelos AR, Paixão AG da, Kinoshita PF, Orellana AMM, Scavone C, Kawamoto EM. Toll-like receptor 4 signaling is critical for the adaptive cellular stress response effects induced by intermittent fasting in the mouse brain [Internet]. Neuroscience. 2021 ;[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.neuroscience.2021.04.022
Artigo de periodico
21-Benzylidene digoxin decreases proliferation by inhibiting the EGFR/ERK signaling pathway and induces apoptosis in HeLa cells (2019)
Pessôa, Marco Túlio Correa; Valadares, Jéssica Martins de Moura; Rocha, Sayonarah Carvalho ; Silva, Simone Cavalcante ; McDermott, Jeff P. ; Sánchez, Gladis ; Varotti, Fernando de Pilla ; Scavone, Cristoforo ; Ribeiro, Rosy Iara Maciel de Azambuja ; Villar, José A. F. P. ; Blanco, Gustavo; Barbosa, Leandro Augusto De Oliveira
Source: Steroids. Unidade: ICB
Subjects: FARMACOLOGIA, PROLIFERAÇÃO CELULAR, APOPTOSE, ESTEROIDES, TRANSPORTE DE ÍONS, INSULINA, PROTEÍNAS QUINASES, FÁRMACOS (SISTEMA CARDIOVASCULAR), GLICOSÍDEOS
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ABNT
PESSÔA, Marco Túlio Correa et al. 21-Benzylidene digoxin decreases proliferation by inhibiting the EGFR/ERK signaling pathway and induces apoptosis in HeLa cells. Steroids, p. 10 , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.steroids.2019.108551. Acesso em: 05 nov. 2024.
APA
Pessôa, M. T. C., Valadares, J. M. de M., Rocha, S. C., Silva, S. C., McDermott, J. P., Sánchez, G., et al. (2019). 21-Benzylidene digoxin decreases proliferation by inhibiting the EGFR/ERK signaling pathway and induces apoptosis in HeLa cells. Steroids, 10 . doi:10.1016/j.steroids.2019.108551
NLM
Pessôa MTC, Valadares JM de M, Rocha SC, Silva SC, McDermott JP, Sánchez G, Varotti F de P, Scavone C, Ribeiro RIM de A, Villar JAFP, Blanco G, Barbosa LADO. 21-Benzylidene digoxin decreases proliferation by inhibiting the EGFR/ERK signaling pathway and induces apoptosis in HeLa cells [Internet]. Steroids. 2019 ;10 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.steroids.2019.108551
Vancouver
Pessôa MTC, Valadares JM de M, Rocha SC, Silva SC, McDermott JP, Sánchez G, Varotti F de P, Scavone C, Ribeiro RIM de A, Villar JAFP, Blanco G, Barbosa LADO. 21-Benzylidene digoxin decreases proliferation by inhibiting the EGFR/ERK signaling pathway and induces apoptosis in HeLa cells [Internet]. Steroids. 2019 ;10 .[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.steroids.2019.108551
Artigo de periodico
NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance (2018)
Crunfli, Fernanda ; Mazucanti, Caio Henrique Yokoyama ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Rodrigues, Alice Cristina ; Scavone, Cristoforo ; Torrão, Andréa da Silva
Source: Journal of Alzheimer's Disease. Unidade: ICB
Subjects: INSULINA, DOENÇA DE ALZHEIMER, NEURÔNIOS, PROTEÍNAS QUINASES, METABOLISMO ENERGÉTICO, RECEPTORES DE INSULINA
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ABNT
CRUNFLI, Fernanda et al. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, v. 65, n. 4, p. 1427-1443, 2018Tradução . . Disponível em: https://doi.org/10.3233/JAD-180284. Acesso em: 05 nov. 2024.
APA
Crunfli, F., Mazucanti, C. H. Y., Moraes, R. C. M. de, Costa, A. P., Rodrigues, A. C., Scavone, C., & Torrão, A. da S. (2018). NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, 65( 4), 1427-1443. doi:10.3233/JAD-180284
NLM
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3233/JAD-180284
Vancouver
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 nov. 05 ] Available from: https://doi.org/10.3233/JAD-180284
Artigo de periodico
Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin (2009)
Russo, Lilian Cristina; Goni, Camila Nascimento; Castro, Leandro Mantovani de; Asega, Amanda Francine; Camargo, Antonio Carlos Martins de; Trujillo, Cleber Augusto; Ulrich, Henning ; Glucksman, Marc J.; Scavone, Cristoforo ; Ferro, Emer Suavinho
Source: FEBS Journal. Unidades: ICB, IQ
Subjects: PROTEÍNAS QUINASES, CALMODULINA, OLIGOPEPTÍDEOS
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ABNT
RUSSO, Lilian Cristina et al. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin. FEBS Journal, v. 276, n. 16, p. 4358-4371, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1742-4658.2009.07144.x. Acesso em: 05 nov. 2024.
APA
Russo, L. C., Goni, C. N., Castro, L. M. de, Asega, A. F., Camargo, A. C. M. de, Trujillo, C. A., et al. (2009). Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin. FEBS Journal, 276( 16), 4358-4371. doi:10.1111/j.1742-4658.2009.07144.x
NLM
Russo LC, Goni CN, Castro LM de, Asega AF, Camargo ACM de, Trujillo CA, Ulrich H, Glucksman MJ, Scavone C, Ferro ES. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin [Internet]. FEBS Journal. 2009 ; 276( 16): 4358-4371.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/j.1742-4658.2009.07144.x
Vancouver
Russo LC, Goni CN, Castro LM de, Asega AF, Camargo ACM de, Trujillo CA, Ulrich H, Glucksman MJ, Scavone C, Ferro ES. Inflammatory oedema induced by Lachesis muta muta (Surucucu) venom and LmTX-I in the rat paw and dorsal skin [Internet]. FEBS Journal. 2009 ; 276( 16): 4358-4371.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/j.1742-4658.2009.07144.x

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