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Artigo de periodico
The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition (2021)
Queiroz, André L.; Lessard, Sarah J.; Ouchida, Amanda T.; Araujo, Hygor N.; Gonçalves, Dawit A.; Guimarães, Dimitrius Santiago Passos Simões Fróes; Teodoro, Bruno G.; So, Kawai; Espreáfico, Enilza Maria ; Hirshman, Michael F.; Alberici, Luciane Carla ; Kettelhut, Isis do Carmo ; Goodyear, Laurie J.; Silveira, Leonardo R.
Source: Molecular Metabolism. Unidades: FMRP, FCFRP
Subjects: MICRORNAS, MÚSCULO ESQUELÉTICO, MITOCÔNDRIAS, REGULAÇÃO GÊNICA, PROLIFERAÇÃO CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUEIROZ, André L. et al. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, v. 51, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2021.101226. Acesso em: 16 nov. 2024.
APA
Queiroz, A. L., Lessard, S. J., Ouchida, A. T., Araujo, H. N., Gonçalves, D. A., Guimarães, D. S. P. S. F., et al. (2021). The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition. Molecular Metabolism, 51. doi:10.1016/j.molmet.2021.101226
NLM
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Vancouver
Queiroz AL, Lessard SJ, Ouchida AT, Araujo HN, Gonçalves DA, Guimarães DSPSF, Teodoro BG, So K, Espreáfico EM, Hirshman MF, Alberici LC, Kettelhut I do C, Goodyear LJ, Silveira LR. The MicroRNA miR-696 is regulated by SNARK and reduces mitochondrial activity in mouse skeletal muscle through Pgc1α inhibition [Internet]. Molecular Metabolism. 2021 ; 51[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.molmet.2021.101226
Artigo de periodico
Improving cytotoxicity against breast cancer cells by using mixed-ligand ruthenium(II) complexes of 2,2 -bipyridine, amino acid, and nitric oxide derivatives as potential anticancer agents (2021)
Gaspari, Ana Paula Segantin ; Silva, Roberto Santana da; Carneiro, Zumira Aparecida; Carvalho, Marcelo Rodrigues de; Carvalho, Ivone ; Pernomian, Laena ; Ferreira, Lucimara P.; Ramos, Loyanne Carla Barbosa; Souza, Gabriel Aguiar de; Formiga, André Luiz Barboza
Source: Anti-Cancer Agents in Medicinal Chemistry. Unidades: FCFRP, FMRP
Subjects: AMINOÁCIDOS, ANTINEOPLÁSICOS, NEOPLASIAS MAMÁRIAS, LINHAGEM CELULAR, PROLIFERAÇÃO CELULAR, ÓXIDO NÍTRICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GASPARI, Ana Paula Segantin et al. Improving cytotoxicity against breast cancer cells by using mixed-ligand ruthenium(II) complexes of 2,2 -bipyridine, amino acid, and nitric oxide derivatives as potential anticancer agents. Anti-Cancer Agents in Medicinal Chemistry, v. 21, n. 12, p. 1602-1611, 2021Tradução . . Disponível em: https://doi.org/10.2174/0929867327666201020155105. Acesso em: 16 nov. 2024.
APA
Gaspari, A. P. S., Silva, R. S. da, Carneiro, Z. A., Carvalho, M. R. de, Carvalho, I., Pernomian, L., et al. (2021). Improving cytotoxicity against breast cancer cells by using mixed-ligand ruthenium(II) complexes of 2,2 -bipyridine, amino acid, and nitric oxide derivatives as potential anticancer agents. Anti-Cancer Agents in Medicinal Chemistry, 21( 12), 1602-1611. doi:10.2174/0929867327666201020155105
NLM
Gaspari APS, Silva RS da, Carneiro ZA, Carvalho MR de, Carvalho I, Pernomian L, Ferreira LP, Ramos LCB, Souza GA de, Formiga ALB. Improving cytotoxicity against breast cancer cells by using mixed-ligand ruthenium(II) complexes of 2,2 -bipyridine, amino acid, and nitric oxide derivatives as potential anticancer agents [Internet]. Anti-Cancer Agents in Medicinal Chemistry. 2021 ; 21( 12): 1602-1611.[citado 2024 nov. 16 ] Available from: https://doi.org/10.2174/0929867327666201020155105
Vancouver
Gaspari APS, Silva RS da, Carneiro ZA, Carvalho MR de, Carvalho I, Pernomian L, Ferreira LP, Ramos LCB, Souza GA de, Formiga ALB. Improving cytotoxicity against breast cancer cells by using mixed-ligand ruthenium(II) complexes of 2,2 -bipyridine, amino acid, and nitric oxide derivatives as potential anticancer agents [Internet]. Anti-Cancer Agents in Medicinal Chemistry. 2021 ; 21( 12): 1602-1611.[citado 2024 nov. 16 ] Available from: https://doi.org/10.2174/0929867327666201020155105
Artigo de periodico
Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms (2018)
Machado Neto, João Agostinho ; Fenerich, Bruna Alves; Ribeiro, Renata Scopim; Eide, Christopher A; Silva, Juan Luiz Coelho da; Dechandt, Carlos Roberto Porto; Fernandes, Jaqueline Cristina; Alves, Ana Paula Nunes Rodrigues; Scheucher, Priscila Santos; Simões, Belinda Pinto; Alberici, Luciane Carla; Pontes, Lorena Lôbo de Figueiredo; Tognon, Cristina E; Druker, Brian J; Rego, Eduardo Magalhães; Traina, Fabíola
Source: Cell Death & Disease. Unidades: ICB, FMRP, FCFRP, FM
Subjects: METFORMINA, PROLIFERAÇÃO CELULAR, APOPTOSE, MUTAÇÃO GENÉTICA, LEUCEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MACHADO NETO, João Agostinho et al. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms. Cell Death & Disease, v. 9, n. 3, p. 16 , 2018Tradução . . Disponível em: https://doi.org/10.1038/s41419-017-0256-4. Acesso em: 16 nov. 2024.
APA
Machado Neto, J. A., Fenerich, B. A., Ribeiro, R. S., Eide, C. A., Silva, J. L. C. da, Dechandt, C. R. P., et al. (2018). Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms. Cell Death & Disease, 9( 3), 16 . doi:10.1038/s41419-017-0256-4
NLM
Machado Neto JA, Fenerich BA, Ribeiro RS, Eide CA, Silva JLC da, Dechandt CRP, Fernandes JC, Alves APNR, Scheucher PS, Simões BP, Alberici LC, Pontes LL de F, Tognon CE, Druker BJ, Rego EM, Traina F. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms [Internet]. Cell Death & Disease. 2018 ; 9( 3): 16 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41419-017-0256-4
Vancouver
Machado Neto JA, Fenerich BA, Ribeiro RS, Eide CA, Silva JLC da, Dechandt CRP, Fernandes JC, Alves APNR, Scheucher PS, Simões BP, Alberici LC, Pontes LL de F, Tognon CE, Druker BJ, Rego EM, Traina F. Metformin exerts multitarget antileukemia activity in JAK2V617F-positive myeloproliferative neoplasms [Internet]. Cell Death & Disease. 2018 ; 9( 3): 16 .[citado 2024 nov. 16 ] Available from: https://doi.org/10.1038/s41419-017-0256-4
Artigo de periodico
Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation (2014)
Almeida, Luciana O.; Garcia, Cristiana Bernadelli; Silva, Flávia Amoroso Matos e; Curti, Carlos; Leopoldino, Andréia Machado
Source: Biochemical and Biophysical Research Communications. Unidade: FCFRP
Subjects: CARCINOMA DE CÉLULAS ESCAMOSAS, LINHAGEM CELULAR, PROLIFERAÇÃO CELULAR, REGULAÇÃO GÊNICA, NEOPLASIAS DE CABEÇA E PESCOÇO, PROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALMEIDA, Luciana O. et al. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, v. 445, n. 1, p. 196\2013202, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2014.01.175. Acesso em: 16 nov. 2024.
APA
Almeida, L. O., Garcia, C. B., Silva, F. A. M. e, Curti, C., & Leopoldino, A. M. (2014). Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation. Biochemical and Biophysical Research Communications, 445( 1), 196\2013202. doi:10.1016/j.bbrc.2014.01.175
NLM
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175
Vancouver
Almeida LO, Garcia CB, Silva FAM e, Curti C, Leopoldino AM. Accumulated SET protein up-regulates and interacts with hnRNPK, increasing its binding to nucleic acids, the Bcl-xS repression, and cellular proliferation [Internet]. Biochemical and Biophysical Research Communications. 2014 ; 445( 1): 196\2013202.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.bbrc.2014.01.175

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