Source: Cellular and Molecular Life Sciences. Unidade: FMRP
Subjects: ADENOCARCINOMA (ETIOLOGIA), TRANSFORMAÇÃO CELULAR NEOPLÁSICA (GENÉTICA), NEOPLASIAS DO COLON, NEURÓGLIA (FISIOLOGIA)
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GARCIA, Sérgio Britto e STOPPER, Helga e KANNEN, Vinicius. The contribution of neuronal–glial–endothelial–epithelial interactions to colon carcinogenesis. Cellular and Molecular Life Sciences, v. 71, n. 17, p. 3191-3197, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00018-014-1642-z. Acesso em: 09 out. 2024.APA
Garcia, S. B., Stopper, H., & Kannen, V. (2014). The contribution of neuronal–glial–endothelial–epithelial interactions to colon carcinogenesis. Cellular and Molecular Life Sciences, 71( 17), 3191-3197. doi:10.1007/s00018-014-1642-zNLM
Garcia SB, Stopper H, Kannen V. The contribution of neuronal–glial–endothelial–epithelial interactions to colon carcinogenesis [Internet]. Cellular and Molecular Life Sciences. 2014 ; 71( 17): 3191-3197.[citado 2024 out. 09 ] Available from: https://doi.org/10.1007/s00018-014-1642-zVancouver
Garcia SB, Stopper H, Kannen V. The contribution of neuronal–glial–endothelial–epithelial interactions to colon carcinogenesis [Internet]. Cellular and Molecular Life Sciences. 2014 ; 71( 17): 3191-3197.[citado 2024 out. 09 ] Available from: https://doi.org/10.1007/s00018-014-1642-z