Fonte: Neuroimmunomodulation. Unidades: FM, ICB, FMVZ
Assuntos: DOENÇAS DEGENERATIVAS (ETIOLOGIA), IMUNOLOGIA, DOENCA DE CHAGAS (COMPLICAÇÕES), MEDULA ESPINHAL (PARASITOLOGIA), DOENÇAS DO SISTEMA NERVOSO
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BOMBEIRO, André Luis et al. Neurodegeneration and Increased Production of Nitrotyrosine, Nitric Oxide Synthase, IFN-γ and S100β Protein in the Spinal Cord of IL-12p40-Deficient Mice Infected with Trypanosoma cruzi. Neuroimmunomodulation, v. 17, n. 2, p. 67-78, 2010Tradução . . Disponível em: https://doi.org/10.1159/000258689. Acesso em: 19 nov. 2024.APA
Bombeiro, A. L., Lima, M. R. D. 'I., Chadi, G., & Alvarez, J. M. (2010). Neurodegeneration and Increased Production of Nitrotyrosine, Nitric Oxide Synthase, IFN-γ and S100β Protein in the Spinal Cord of IL-12p40-Deficient Mice Infected with Trypanosoma cruzi. Neuroimmunomodulation, 17( 2), 67-78. doi:10.1159/000258689NLM
Bombeiro AL, Lima MRD'I, Chadi G, Alvarez JM. Neurodegeneration and Increased Production of Nitrotyrosine, Nitric Oxide Synthase, IFN-γ and S100β Protein in the Spinal Cord of IL-12p40-Deficient Mice Infected with Trypanosoma cruzi [Internet]. Neuroimmunomodulation. 2010 ; 17( 2): 67-78.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1159/000258689Vancouver
Bombeiro AL, Lima MRD'I, Chadi G, Alvarez JM. Neurodegeneration and Increased Production of Nitrotyrosine, Nitric Oxide Synthase, IFN-γ and S100β Protein in the Spinal Cord of IL-12p40-Deficient Mice Infected with Trypanosoma cruzi [Internet]. Neuroimmunomodulation. 2010 ; 17( 2): 67-78.[citado 2024 nov. 19 ] Available from: https://doi.org/10.1159/000258689