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KOVACS, Luciana e CABRAL, Pablo e CHAMMAS, Roger. Mannose receptor 1 expression does not determine the uptake of high-density mannose dendrimers by activated macrophages populations. PLOS ONE, v. 15, n. 10, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0240455. Acesso em: 18 out. 2024.
APA
Kovacs, L., Cabral, P., & Chammas, R. (2020). Mannose receptor 1 expression does not determine the uptake of high-density mannose dendrimers by activated macrophages populations. PLOS ONE, 15( 10). doi:10.1371/journal.pone.0240455
NLM
Kovacs L, Cabral P, Chammas R. Mannose receptor 1 expression does not determine the uptake of high-density mannose dendrimers by activated macrophages populations [Internet]. PLOS ONE. 2020 ; 15( 10):[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0240455
Vancouver
Kovacs L, Cabral P, Chammas R. Mannose receptor 1 expression does not determine the uptake of high-density mannose dendrimers by activated macrophages populations [Internet]. PLOS ONE. 2020 ; 15( 10):[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0240455
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DAIBERT, Raquel Morais de Paiva et al. Lipopolysaccharide triggers different transcriptional signatures in taurine and indicine cattle macrophages: reactive oxygen species and potential outcomes to the development of immune response to infections. PLOS ONE, v. 15, n. 11, p. 1-21, 2020Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0241861. Acesso em: 18 out. 2024.
APA
Daibert, R. M. de P., Biagi Junior, C. A. O. de, Vieira, F. de O., Silva, M. V. G. B. da, Hottz, E. D., Pinheiro, M. B. M., et al. (2020). Lipopolysaccharide triggers different transcriptional signatures in taurine and indicine cattle macrophages: reactive oxygen species and potential outcomes to the development of immune response to infections. PLOS ONE, 15( 11), 1-21. doi:10.1371/journal.pone.0241861
NLM
Daibert RM de P, Biagi Junior CAO de, Vieira F de O, Silva MVGB da, Hottz ED, Pinheiro MBM, Faza DR de LR, Pereira HP, Martins MF, Brandão H de M, Machado MA, Carvalho WA. Lipopolysaccharide triggers different transcriptional signatures in taurine and indicine cattle macrophages: reactive oxygen species and potential outcomes to the development of immune response to infections [Internet]. PLOS ONE. 2020 ; 15( 11): 1-21.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0241861
Vancouver
Daibert RM de P, Biagi Junior CAO de, Vieira F de O, Silva MVGB da, Hottz ED, Pinheiro MBM, Faza DR de LR, Pereira HP, Martins MF, Brandão H de M, Machado MA, Carvalho WA. Lipopolysaccharide triggers different transcriptional signatures in taurine and indicine cattle macrophages: reactive oxygen species and potential outcomes to the development of immune response to infections [Internet]. PLOS ONE. 2020 ; 15( 11): 1-21.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0241861
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BATISTA, Luis F. S. e TOMOKANE, Thaise Y. e LAURENTI, Marcia Dalastra. Canine leishmaniasis: genome-wide analysis and antibody response to Lutzomyia longipalpis saliva. PLOS ONE, v. 13, n. 5, 2018Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0197215. Acesso em: 18 out. 2024.
APA
Batista, L. F. S., Tomokane, T. Y., & Laurenti, M. D. (2018). Canine leishmaniasis: genome-wide analysis and antibody response to Lutzomyia longipalpis saliva. PLOS ONE, 13( 5). doi:10.1371/journal.pone.0197215
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ACUÑA, Stephanie Maia et al. Arginase expression modulates nitric oxide production in Leishmania (Leishmania) amazonensis. PLOS ONE, v. No 2017, n. 11, p. 1-21, 2017Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0187186. Acesso em: 18 out. 2024.
APA
Acuña, S. M., Aoki, J. I., Laranjeira-Silva, M. F., Zampieri, R. A., Fernandes, J. C. R., Muxel, S. M., & Floeter-Winter, L. M. (2017). Arginase expression modulates nitric oxide production in Leishmania (Leishmania) amazonensis. PLOS ONE, No 2017( 11), 1-21. doi:10.1371/journal.pone.0187186
NLM
Acuña SM, Aoki JI, Laranjeira-Silva MF, Zampieri RA, Fernandes JCR, Muxel SM, Floeter-Winter LM. Arginase expression modulates nitric oxide production in Leishmania (Leishmania) amazonensis [Internet]. PLOS ONE. 2017 ; No 2017( 11): 1-21.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0187186
Vancouver
Acuña SM, Aoki JI, Laranjeira-Silva MF, Zampieri RA, Fernandes JCR, Muxel SM, Floeter-Winter LM. Arginase expression modulates nitric oxide production in Leishmania (Leishmania) amazonensis [Internet]. PLOS ONE. 2017 ; No 2017( 11): 1-21.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0187186
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NASCIMENTO, Flavia R. F. et al. Interferon regulatory factor (IRF)-1 Is a master regulator of the cross talk between macrophages and L929 fibrosarcoma cells for nitric oxide dependent tumoricidal activity. PLOS ONE, v. 10, n. 2, p. 19 , 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0117782. Acesso em: 18 out. 2024.
APA
Nascimento, F. R. F., Nascimento, E. A. G. de M., Russo, M., & Lepique, A. P. (2015). Interferon regulatory factor (IRF)-1 Is a master regulator of the cross talk between macrophages and L929 fibrosarcoma cells for nitric oxide dependent tumoricidal activity. PLOS ONE, 10( 2), 19 . doi:10.1371/journal.pone.0117782
NLM
Nascimento FRF, Nascimento EAG de M, Russo M, Lepique AP. Interferon regulatory factor (IRF)-1 Is a master regulator of the cross talk between macrophages and L929 fibrosarcoma cells for nitric oxide dependent tumoricidal activity [Internet]. PLOS ONE. 2015 ; 10( 2): 19 .[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0117782
Vancouver
Nascimento FRF, Nascimento EAG de M, Russo M, Lepique AP. Interferon regulatory factor (IRF)-1 Is a master regulator of the cross talk between macrophages and L929 fibrosarcoma cells for nitric oxide dependent tumoricidal activity [Internet]. PLOS ONE. 2015 ; 10( 2): 19 .[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0117782
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SILVA, Bruno Bueno et al. Brazilian red propolis attenuates inflammatory signaling cascade in LPS-activated macrophages. PLOS ONE, v. 10, n. 12 art. e0144954, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0144954. Acesso em: 18 out. 2024.
APA
Silva, B. B., Kawamoto, D., Ando-Suguimoto, E. S., Alencar, S. M. de, Rosalen, P. L., & Mayer, M. P. A. (2015). Brazilian red propolis attenuates inflammatory signaling cascade in LPS-activated macrophages. PLOS ONE, 10( 12 art. e0144954). doi:10.1371/journal.pone.0144954
NLM
Silva BB, Kawamoto D, Ando-Suguimoto ES, Alencar SM de, Rosalen PL, Mayer MPA. Brazilian red propolis attenuates inflammatory signaling cascade in LPS-activated macrophages [Internet]. PLOS ONE. 2015 ; 10( 12 art. e0144954):[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0144954
Vancouver
Silva BB, Kawamoto D, Ando-Suguimoto ES, Alencar SM de, Rosalen PL, Mayer MPA. Brazilian red propolis attenuates inflammatory signaling cascade in LPS-activated macrophages [Internet]. PLOS ONE. 2015 ; 10( 12 art. e0144954):[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0144954
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PARENTE-ROCHA, Juliana Alves et al. Macrophage interaction with Paracoccidioides brasiliensis yeast cells modulates fungal metabolism and generates a response to oxidative stress. PLOS ONE, v. 10, n. 09, p. 1-18, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0137619. Acesso em: 18 out. 2024.
APA
Parente-Rocha, J. A., Parente, A. F. A., Baeza, L. C., Bonfim, S. M. R. C., Hernandez, O., McEwen, J. G., et al. (2015). Macrophage interaction with Paracoccidioides brasiliensis yeast cells modulates fungal metabolism and generates a response to oxidative stress. PLOS ONE, 10( 09), 1-18. doi:10.1371/journal.pone.0137619
NLM
Parente-Rocha JA, Parente AFA, Baeza LC, Bonfim SMRC, Hernandez O, McEwen JG, Bailão AM, Taborda CP, Borges CL, Soares CM de A. Macrophage interaction with Paracoccidioides brasiliensis yeast cells modulates fungal metabolism and generates a response to oxidative stress [Internet]. PLOS ONE. 2015 ; 10( 09): 1-18.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0137619
Vancouver
Parente-Rocha JA, Parente AFA, Baeza LC, Bonfim SMRC, Hernandez O, McEwen JG, Bailão AM, Taborda CP, Borges CL, Soares CM de A. Macrophage interaction with Paracoccidioides brasiliensis yeast cells modulates fungal metabolism and generates a response to oxidative stress [Internet]. PLOS ONE. 2015 ; 10( 09): 1-18.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0137619
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ALMEIDA, Fausto Bruno dos Reis et al. Toxoplasma gondii Chitinase induces macrophage activation. PLOS ONE, v. 10, n. 12, p. 1-12, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0144507. Acesso em: 18 out. 2024.
APA
Almeida, F. B. dos R., Sardinha-Silva, A., Silva, T. A. da, Pessoni, A. M., Pinzan, C. F., Alegre-Maller, A. C. P., et al. (2015). Toxoplasma gondii Chitinase induces macrophage activation. PLOS ONE, 10( 12), 1-12. doi:10.1371/journal.pone.0144507
NLM
Almeida FB dos R, Sardinha-Silva A, Silva TA da, Pessoni AM, Pinzan CF, Alegre-Maller ACP, Cecilio NT, Moretti NS, Damásio ARL, Pedersoli WR, Mineo JR, Silva R do N, Roque-Barreira MC. Toxoplasma gondii Chitinase induces macrophage activation [Internet]. PLOS ONE. 2015 ; 10( 12): 1-12.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0144507
Vancouver
Almeida FB dos R, Sardinha-Silva A, Silva TA da, Pessoni AM, Pinzan CF, Alegre-Maller ACP, Cecilio NT, Moretti NS, Damásio ARL, Pedersoli WR, Mineo JR, Silva R do N, Roque-Barreira MC. Toxoplasma gondii Chitinase induces macrophage activation [Internet]. PLOS ONE. 2015 ; 10( 12): 1-12.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0144507
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TOLEDO, Karina Alves et al. Galectin-1 exerts inhibitory effects during DENV-1 infection. PLOS ONE, v. 9, n. 11, p. 1-11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0112474. Acesso em: 18 out. 2024.
APA
Toledo, K. A., Fermino, M. L., Andrade, C. del C., Riul, T. B., Alves, R. T., Muller, V. D. M., et al. (2014). Galectin-1 exerts inhibitory effects during DENV-1 infection. PLOS ONE, 9( 11), 1-11. doi:10.1371/journal.pone.0112474
NLM
Toledo KA, Fermino ML, Andrade C del C, Riul TB, Alves RT, Muller VDM, Russo RR, Stowell SR, Cummings RD, Quintana VHA, Dias-Baruffi M. Galectin-1 exerts inhibitory effects during DENV-1 infection [Internet]. PLOS ONE. 2014 ; 9( 11): 1-11.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0112474
Vancouver
Toledo KA, Fermino ML, Andrade C del C, Riul TB, Alves RT, Muller VDM, Russo RR, Stowell SR, Cummings RD, Quintana VHA, Dias-Baruffi M. Galectin-1 exerts inhibitory effects during DENV-1 infection [Internet]. PLOS ONE. 2014 ; 9( 11): 1-11.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0112474
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FESTUCCIA, William Tadeu Lara et al. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, v. 9, n. 4, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0095432. Acesso em: 18 out. 2024.
APA
Festuccia, W. T. L., Pouliot, P., Bakan, I., Sabatini, D. M., & Laplante, M. (2014). Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide. PLOS ONE, 9( 4), 1-12. doi:10.1371/journal.pone.0095432
NLM
Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0095432
Vancouver
Festuccia WTL, Pouliot P, Bakan I, Sabatini DM, Laplante M. Myeloid-specific rictor deletion induces M1 macrophage polarization and potentiates in vivo pro-inflammatory response to lipopolysaccharide [Internet]. PLOS ONE. 2014 ; 9( 4): 1-12.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0095432
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SECATTO, Adriana et al. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis. PLOS ONE, v. 9, n. 1, p. 1-9, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0085083. Acesso em: 18 out. 2024.
APA
Secatto, A., Soares, E. M., Locachevic, G. A., Assis, P. A. de, Paula-Silva, F. W. G. de, Serezani, C. H. C., et al. (2014). The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis. PLOS ONE, 9( 1), 1-9. doi:10.1371/journal.pone.0085083
NLM
Secatto A, Soares EM, Locachevic GA, Assis PA de, Paula-Silva FWG de, Serezani CHC, Medeiros AI de, Faccioli LH. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis [Internet]. PLOS ONE. 2014 ; 9( 1): 1-9.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0085083
Vancouver
Secatto A, Soares EM, Locachevic GA, Assis PA de, Paula-Silva FWG de, Serezani CHC, Medeiros AI de, Faccioli LH. The leukotriene B4/BLT1 axis is a key determinant in susceptibility and resistance to histoplasmosis [Internet]. PLOS ONE. 2014 ; 9( 1): 1-9.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0085083
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CARNEIRO, Alan Brito et al. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation. PLOS ONE, v. 8, n. 9, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0076233. Acesso em: 18 out. 2024.
APA
Carneiro, A. B., Iaciura, B. M. F., Nohara, L. L., Lopes, C. D., Veas, E. M. C., Mariano, V. S., et al. (2013). Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation. PLOS ONE, 8( 9), 1-9. doi:10.1371/journal.pone.0076233
NLM
Carneiro AB, Iaciura BMF, Nohara LL, Lopes CD, Veas EMC, Mariano VS, Bozza PT, Lopes UG, Atella GC, Almeida IC, Silva-Neto MAC. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation [Internet]. PLOS ONE. 2013 ; 8( 9): 1-9.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0076233
Vancouver
Carneiro AB, Iaciura BMF, Nohara LL, Lopes CD, Veas EMC, Mariano VS, Bozza PT, Lopes UG, Atella GC, Almeida IC, Silva-Neto MAC. Lysophosphatidylcholine triggers TLR2- and TLR4-mediated signaling pathways but counteracts LPS-induced NO synthesis in peritoneal macrophages by inhibiting NF-κB translocation and MAPK/ERK phosphorylation [Internet]. PLOS ONE. 2013 ; 8( 9): 1-9.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0076233
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SORGI, Carlos Arterio et al. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands. PLOS ONE, v. 7, n. 7, p. 1-13, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0040523. Acesso em: 18 out. 2024.
APA
Sorgi, C. A., Rose, S., Court, N., Carlos, D., Paula-Silva, F. W. G. de, Assis, P. A. de, et al. (2012). GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands. PLOS ONE, 7( 7), 1-13. doi:10.1371/journal.pone.0040523
NLM
Sorgi CA, Rose S, Court N, Carlos D, Paula-Silva FWG de, Assis PA de, Frantz FG, Ryffel B, Quesniaux V, Faccioli LH. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands [Internet]. PLOS ONE. 2012 ; 7( 7): 1-13.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0040523
Vancouver
Sorgi CA, Rose S, Court N, Carlos D, Paula-Silva FWG de, Assis PA de, Frantz FG, Ryffel B, Quesniaux V, Faccioli LH. GM-CSF priming drives bone marrow-derived macrophages to a pro-inflammatory pattern and downmodulates PGE2 in response to TLR2 ligands [Internet]. PLOS ONE. 2012 ; 7( 7): 1-13.[citado 2024 out. 18 ] Available from: https://doi.org/10.1371/journal.pone.0040523